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PloS one, ISSN 1932-6203, 02/2013, Volume 8, Issue 2, pp. e54442 - e54442
... Pollock 1 , Amanda Mather 1 Introduction Diabetic nephropathy is the leading cause of end stage kidney disease and its escalating incidence is a challenge to health... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Diabetes Mellitus - pathology | Epithelial Cells - metabolism | Gene Expression - drug effects | Diabetes Mellitus - genetics | Transcription Factor AP-1 - genetics | Epithelial Cells - drug effects | Humans | Diabetic Nephropathies - drug therapy | NF-kappa B - metabolism | Smad3 Protein - metabolism | Transcription Factor AP-1 - metabolism | Sodium-Glucose Transporter 1 - genetics | Smad3 Protein - genetics | Sodium-Glucose Transporter 1 - metabolism | Phosphorylation - drug effects | Interleukin-6 - metabolism | Transforming Growth Factor beta1 - pharmacology | Sodium-Glucose Transporter 2 - genetics | Collagen Type IV - metabolism | Sodium-Glucose Transporter 1 - antagonists & inhibitors | Promoter Regions, Genetic | Diabetic Nephropathies - pathology | Glucosides - pharmacology | Kidney Tubules, Proximal - pathology | Interleukin-6 - genetics | Sodium-Glucose Transporter 2 - metabolism | Diabetic Nephropathies - metabolism | Diabetes Mellitus - drug therapy | Diabetes Mellitus - metabolism | Toll-Like Receptor 4 - genetics | Diabetic Nephropathies - genetics | Epithelial Cells - pathology | Glucose - pharmacology | Toll-Like Receptor 4 - metabolism | Sodium-Glucose Transporter 2 - antagonists & inhibitors | Hypoglycemic Agents - pharmacology | NF-kappa B - genetics | Kidney Tubules, Proximal - metabolism | Collagen Type IV - genetics | Protein Binding | Benzhydryl Compounds - pharmacology | Kidney Tubules, Proximal - drug effects | Physiological aspects | Care and treatment | Genetic aspects | Research | Transforming growth factors | Diabetic nephropathies | Chromatin | Immunoprecipitation | Transforming growth factor-b | Interleukin | Clinical trials | Systematic review | Smad3 protein | Glucose | Kinases | Interleukin 6 | Proteins | Hyperglycemia | Rodents | Toll-like receptors | Collagen (type IV) | Hypoglycemic agents | Inhibition | Growth factors | Deoxyribonucleic acid--DNA | Immune system | Binding | Glucose transporter | Medical research | NF-κB protein | Diabetes mellitus | Markers | Activator protein 1 | Blocking | Reabsorption | Inflammation | Gene expression | Glucose transport | Medicine | High mobility group proteins | Hospitals | Inhibitors | Nephropathy | Sodium | Fibrosis | Kidney diseases | Diabetes | Transporter | Adenosine triphosphatase | Kidney transplantation | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal Article
Nature reviews. Nephrology, ISSN 1759-5061, 2014, Volume 10, Issue 9, pp. 517 - 530
Journal Article
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 11/2017, Volume 21, Issue 11, pp. 2732 - 2747
...)‐induced diabetic nephropathy, and dynamically regulated in cultured mouse podocytes stimulated with high glucose, which showed a trend from rise to decline... 
MALAT1 | β‐catenin | high glucose | diabetic nephropathy | SRSF1 | podocyte | β-catenin | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Cell Biology | Research & Experimental Medicine | RNA, Small Interfering - genetics | Diabetes Mellitus, Experimental - genetics | Male | Serine-Arginine Splicing Factors - metabolism | Serine-Arginine Splicing Factors - genetics | Glucose - toxicity | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - metabolism | Wnt Signaling Pathway | Promoter Regions, Genetic | Diabetic Nephropathies - pathology | Podocytes - metabolism | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Diabetic Nephropathies - chemically induced | Gene Expression Regulation | Diabetic Nephropathies - genetics | RNA, Long Noncoding - genetics | Podocytes - pathology | beta Catenin - metabolism | beta Catenin - genetics | Protein Transport | Serine-Arginine Splicing Factors - antagonists & inhibitors | Feedback, Physiological | Animals | Podocytes - drug effects | beta Catenin - antagonists & inhibitors | Diabetes Mellitus, Experimental - pathology | Protein Binding | Mice | Streptozocin - toxicity | RNA, Long Noncoding - antagonists & inhibitors | RNA, Long Noncoding - metabolism | Cell Line, Transformed | RNA, Small Interfering - metabolism | Index Medicus | Original
Journal Article