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PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, p. e77986
Diabetic neuropathy is a severe complication of long-standing diabetes and one of the major etiologies of neuropathic pain. Diabetes is associated with an... 
OXIDATIVE STRESS | ION-CHANNEL TRPA1 | SERUM-ALBUMIN | MULTIDISCIPLINARY SCIENCES | RENAL TUBULAR CELLS | COVALENT MODIFICATION | PERIPHERAL NEUROPATHY | CALCIUM LEVELS | DIABETIC-NEUROPATHY | ACTIVATE TRPA1 | GLYOXALASE-I | Acute Pain - metabolism | Transient Receptor Potential Channels - antagonists & inhibitors | Pyruvaldehyde - adverse effects | Lactoylglutathione Lyase - genetics | Acute Pain - chemically induced | Diabetic Neuropathies - genetics | Insulin-Secreting Cells - metabolism | Madin Darby Canine Kidney Cells | Sensory Receptor Cells - metabolism | Transient Receptor Potential Channels - metabolism | Acetanilides - pharmacology | Pyruvaldehyde - pharmacology | Sensory Receptor Cells - pathology | Lactoylglutathione Lyase - metabolism | Purines - pharmacology | Glucose - genetics | Mice, Knockout | Acute Pain - genetics | Animals | Calcium Signaling - drug effects | Transient Receptor Potential Channels - genetics | TRPA1 Cation Channel | Diabetic Neuropathies - pathology | Dogs | Glucose - metabolism | Mice | Insulin-Secreting Cells - pathology | Calcium Signaling - genetics | Diabetic Neuropathies - metabolism | Acute Pain - pathology | Type 2 diabetes | Pancreatic beta cells | Diabetic neuropathies | Blood sugar monitoring | Neurons | Glucose | Glucose tolerance tests | Dextrose | Reactive oxygen species | Calcium | Diabetic neuropathy | Neuropathy | Peripheral neuropathy | Hyperglycemia | Pain | Metabolites | Etiology | Rodents | Penicillin | Sensory neurons | Physiology | Pancreas | Calcium (intracellular) | Cold | Hypersensitivity | Diabetes mellitus | Detoxification | Pharmacology | Glucose tolerance | In vivo methods and tests | Diabetes | Intracellular | Pyruvaldehyde | Apoptosis
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 06/2008, Volume 28, Issue 26, pp. 6652 - 6658
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 6/2010, Volume 107, Issue 22, pp. 10190 - 10195
An insertion polymorphism of the angiotensin-I converting enzyme gene (ACE) is common in humans and the higher expressing allele is associated with an... 
Diabetic neuropathies | Receptors | Diabetes complications | Diabetic nephropathies | Diabetes | Type 2 diabetes mellitus | Genetic mutation | Bradykinin receptors | Type 1 diabetes mellitus | Blood plasma | Kinins | Diabetes mellitus complications | ACE GENE | MYOCARDIAL-INFARCTION | MULTIDISCIPLINARY SCIENCES | diabetes mellitus complications | DELETION POLYMORPHISM | GENE POLYMORPHISM | KININ RECEPTORS | kinins | ANGIOTENSIN-CONVERTING-ENZYME | TISSUE GROWTH-FACTOR | C-OXIDASE ACTIVITY | NITRIC-OXIDE | PERIPHERAL NEUROPATHY | Receptor, Bradykinin B2 - genetics | Diabetic Neuropathies - etiology | Diabetic Nephropathies - etiology | Humans | Diabetes Mellitus, Experimental - genetics | Male | Osteoporosis - etiology | Diabetic Neuropathies - genetics | Receptor, Bradykinin B1 - genetics | DNA, Mitochondrial - genetics | Osteoporosis - metabolism | Mice, Mutant Strains | Osteoporosis - genetics | Diabetes Mellitus, Experimental - complications | Diabetes Mellitus, Experimental - metabolism | Diabetic Neuropathies - physiopathology | Diabetic Nephropathies - pathology | Bone Density | Diabetic Nephropathies - metabolism | Mice, Inbred C57BL | Diabetic Nephropathies - genetics | Mice, Knockout | Phenotype | Animals | Receptor, Bradykinin B1 - deficiency | Receptor, Bradykinin B2 - deficiency | Mice | Diabetic Neuropathies - metabolism | Complications and side effects | Bradykinin | Angiotensin | Mitochondrial DNA | Properties | Observations | Genetic polymorphisms | Biological Sciences
Journal Article
Experimental Neurology, ISSN 0014-4886, 03/2012, Volume 234, Issue 1, pp. 62 - 69
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 02/2017, Volume 127, Issue 2, pp. 608 - 622
Sensory neurons have the capacity to produce, release, and respond to acetylcholine (ACh), but the functional role of cholinergic systems in adult mammalian... 
MEDICINE, RESEARCH & EXPERIMENTAL | AXONAL DEGENERATION | OXIDATIVE STRESS | ACTIVATED PROTEIN-KINASE | NEURITE OUTGROWTH | ADULT SENSORY NEURONS | DORSAL-ROOT GANGLIA | MITOCHONDRIAL DYSFUNCTION | CHOLINE-ACETYLTRANSFERASE | DIABETIC-NEUROPATHY | ACETYLCHOLINE-RECEPTOR | Diabetes Mellitus, Experimental - drug therapy | Diabetes Mellitus, Experimental - genetics | Male | Receptor, Muscarinic M1 - antagonists & inhibitors | Diabetic Neuropathies - genetics | Mice, Mutant Strains | Muscarinic Antagonists - pharmacology | Sensory Receptor Cells - metabolism | Diabetes Mellitus, Experimental - metabolism | Sensory Receptor Cells - pathology | Hyperalgesia - metabolism | Rats | Diabetic Neuropathies - drug therapy | Mitochondria - metabolism | Neurites - metabolism | Mitochondria - pathology | Neurites - pathology | Receptor, Muscarinic M1 - genetics | Animals | Hyperalgesia - drug therapy | Hyperalgesia - genetics | Diabetic Neuropathies - pathology | Diabetes Mellitus, Experimental - pathology | Mice | Diabetic Neuropathies - metabolism | Care and treatment | Cholinergic mechanisms | Polyneuropathies | Sensory receptors | Development and progression | Genetic aspects | Muscarinic receptors | Health aspects | Motility | Neurons | Colleges & universities | Committees | Diabetic neuropathy | Metabolism | Mammals | Peripheral neuropathy | Mitochondria | Microscopy | Plasmids | Rodents | Diabetes
Journal Article
American Journal of Physiology - Endocrinology and Metabolism, ISSN 0193-1849, 11/2011, Volume 301, Issue 5, pp. E844 - E852
Yamakawa I, Kojima H, Terashima T, Katagi M, Oi J, Urabe H, Sanada M, Kawai H, Chan L, Yasuda H, Maegawa H, Kimura H. Inactivation of TNF-alpha ameliorates... 
Diabetic neuropathy | Infliximab | Dorsal root ganglion | Tumor necrosis factor | Nerve conduction velocity | MECHANICAL ALLODYNIA | PROTEIN-KINASE-C | EXPRESSING CELLS | PHYSIOLOGY | ENDOTHELIAL DYSFUNCTION | diabetic neuropathy | infliximab | NITRIC-OXIDE SYNTHASE | nerve conduction velocity | SENSORY NEURONS | tumor necrosis factor-alpha | TUMOR-NECROSIS-FACTOR | dorsal root ganglion | ENDOCRINOLOGY & METABOLISM | PERIPHERAL NEUROPATHY | KAPPA-B PATHWAY | ALDOSE REDUCTASE | Tumor Necrosis Factor-alpha - metabolism | Streptozocin | Tumor Necrosis Factor-alpha - genetics | Diabetes Mellitus, Experimental - genetics | Antibodies, Monoclonal - therapeutic use | Molecular Targeted Therapy | Diabetic Neuropathies - genetics | Anti-Inflammatory Agents - therapeutic use | Diabetes Mellitus, Experimental - chemically induced | Diabetes Mellitus, Experimental - complications | Drug Evaluation, Preclinical | Anti-Inflammatory Agents - pharmacology | Antibodies, Monoclonal - pharmacology | Mice, Inbred C57BL | Diabetic Neuropathies - drug therapy | Mice, Knockout | Gene Expression Regulation - drug effects | Animals | Transcription Factor RelA - metabolism | Diabetic Neuropathies - pathology | Ganglia, Spinal - pathology | Tumor Necrosis Factor-alpha - physiology | Diabetes Mellitus, Experimental - pathology | Mice | Diabetic Neuropathies - metabolism | Gene Silencing - physiology | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Ganglia, Spinal - metabolism | tumor necrosis factor-α
Journal Article
Diabetologia, ISSN 0012-186X, 6/2016, Volume 59, Issue 6, pp. 1297 - 1306
Journal Article