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Journal Article
Diabetes, ISSN 0012-1797, 09/2018, Volume 67, Issue 9, pp. 1867 - 1879
Intermittent fasting (IF) protects against the development of metabolic diseases and cancer, but whether it can prevent diabetic microvascular complications is... 
DIABETIC-RETINOPATHY | BILE-ACID | CELLS | ACTIVATION | BACTERIA | OBESITY | METABOLISM | FARNESOID X RECEPTOR | C57BL/6 MICE | ENDOCRINOLOGY & METABOLISM | TAUROURSODEOXYCHOLIC ACID | Retina - drug effects | Leukocytes - pathology | Receptors, G-Protein-Coupled - metabolism | Colon - drug effects | Microvessels - metabolism | Microvessels - pathology | Colon - immunology | Male | Ganglia, Sensory - metabolism | Retina - immunology | Intestinal Mucosa - drug effects | Diabetic Retinopathy - complications | Retinal Vessels - pathology | Feces - microbiology | Intestinal Mucosa - immunology | Bacteroidetes - isolation & purification | Firmicutes - growth & development | Diabetic Retinopathy - immunology | Firmicutes - isolation & purification | Retinal Vessels - metabolism | Colon - pathology | Retinal Vessels - drug effects | Dysbiosis - therapy | Goblet Cells - metabolism | Colon - metabolism | Ganglia, Sensory - pathology | Diabetic Retinopathy - prevention & control | Gastrointestinal Microbiome - drug effects | Bile Acids and Salts - therapeutic use | Survival Analysis | Leukocytes - drug effects | Diabetes Mellitus, Type 2 - pathology | Dysbiosis - pathology | Retina - pathology | Intestinal Mucosa - pathology | Intestinal Mucosa - metabolism | Retina - metabolism | Diabetes Mellitus, Type 2 - microbiology | Ganglia, Sensory - drug effects | Goblet Cells - drug effects | Diabetic Retinopathy - pathology | Receptors, G-Protein-Coupled - agonists | Dysbiosis - microbiology | Microvessels - immunology | Ganglia, Sensory - immunology | Goblet Cells - immunology | Leukocytes - immunology | Retinal Vessels - immunology | Mice, Mutant Strains | Diabetes Mellitus, Type 2 - therapy | Bacteroidetes - immunology | Mice, Inbred DBA | Verrucomicrobia - growth & development | Verrucomicrobia - immunology | Diabetes Mellitus, Type 2 - complications | Fasting | Microvessels - drug effects | Firmicutes - immunology | Verrucomicrobia - isolation & purification | Dysbiosis - complications | Animals | Bacteroidetes - growth & development | Gastrointestinal Microbiome - immunology | Goblet Cells - pathology | Prevention | Diabetic retinopathy | Usage | Diet therapy | Low-calorie diet | Rattus | Rats | Health aspects | Neuroprotection | Retinopathy | Diabetes mellitus | Peptidoglycans | Microbiomes | Retina | mRNA | Tumor necrosis factor-α | Feeding | Microbiota | Mucin | Metabolites | Cell number | Rodents | Hemoglobin | Microvasculature | Diabetes | Capillaries | Ganglion cells | Bile | Cancer | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 2009, Volume 4, Issue 12, pp. e8158 - e8158
Inflammation affects the formation and the progression of various vitreoretinal diseases. We performed a comprehensive analysis of inflammatory immune... 
C-C CHEMOKINES | INTRAVITREAL TRIAMCINOLONE | PROLIFERATIVE DIABETIC-RETINOPATHY | VITREOUS FLUID | MACULAR EDEMA | MULTIDISCIPLINARY SCIENCES | MULTIPLEX BEAD ANALYSIS | RETINAL VEIN OCCLUSION | MONOCYTE CHEMOTACTIC PROTEIN-1 | NF-KAPPA-B | ENDOTHELIAL GROWTH-FACTOR | Vitreoretinopathy, Proliferative - blood | Retinal Diseases - immunology | Humans | Middle Aged | Retinal Diseases - blood | Male | Diabetic Retinopathy - pathology | Retinal Vein Occlusion - pathology | Inflammation Mediators - metabolism | Retinal Diseases - pathology | Female | Cytokines - blood | Diabetic Retinopathy - immunology | Retinal Vein Occlusion - immunology | Vitreoretinopathy, Proliferative - pathology | Inflammation Mediators - blood | Solubility | Vascular Endothelial Growth Factor A - blood | Vitreoretinopathy, Proliferative - immunology | Retinal Vein Occlusion - blood | Diabetic Retinopathy - blood | Models, Biological | Vitreous Body - immunology | Vitreous Body - pathology | Aged | Diabetic retinopathy | Development and progression | Inflammation | Interleukins | Vascular endothelial growth factor | Analysis | Occlusion | Multiplexing | Membranes | Retinopathy | Pathogenesis | Interleukin | Retina | Interleukin 6 | Proteins | Angiogenesis | Ischemia | Granulocytes | Tumor necrosis factor-TNF | Growth factors | Interleukin 8 | Edema | Factor analysis | Cytokines | Diabetes mellitus | Patients | Diseases | Detachment | Correlation analysis | Monocyte chemoattractant protein | Medical prognosis | Photoreceptors | Hypoxia | Diabetes | Chemokines | Monocyte chemoattractant protein 1 | Apoptosis | Index Medicus
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 05/2017, Volume 41, Issue 3, pp. 960 - 972
Background/Aims: Interleukin (IL)-17A, a proinflammatory cytokine, has been implicated in several autoimmune diseases. However, it is unclear whether IL-17A is... 
Retinal Müller cells | Diabetic retinopathy | Interleukin-17A | Interleukin-17 receptor A | ACTIVATION | PHYSIOLOGY | NEURODEGENERATION | DEGENERATION | CELL BIOLOGY | INTRAVITREAL INJECTION | RETINAL GANGLION-CELLS | INTERLEUKIN-17 | INFLAMMATION | Retinal Muller cells | MOUSE MODEL | EXPRESSION | T-CELLS | Retina - drug effects | Glial Fibrillary Acidic Protein - genetics | Intravitreal Injections | Streptozocin | Receptors, Interleukin-17 - immunology | Interleukin-17 - immunology | Male | Receptors, Interleukin-17 - genetics | Retina - immunology | Diabetic Retinopathy - pathology | Glial Fibrillary Acidic Protein - immunology | Excitatory Amino Acid Transporter 1 - genetics | Vascular Endothelial Growth Factor A - genetics | Diabetic Retinopathy - chemically induced | Diabetes Mellitus, Experimental - chemically induced | Ependymoglial Cells - pathology | Diabetic Retinopathy - immunology | Signal Transduction | Antibodies, Monoclonal - pharmacology | Mice, Inbred C57BL | Gene Expression Regulation | Interleukin-17 - genetics | Vascular Endothelial Growth Factor A - immunology | Receptors, Interleukin-17 - antagonists & inhibitors | Diabetic Retinopathy - therapy | Ependymoglial Cells - drug effects | Ependymoglial Cells - immunology | Diabetes Mellitus, Experimental - immunology | Glutamate-Ammonia Ligase - immunology | Animals | Interleukin-17 - antagonists & inhibitors | Glutamate-Ammonia Ligase - genetics | Excitatory Amino Acid Transporter 1 - immunology | Diabetes Mellitus, Experimental - pathology | Immunization, Passive | Mice | Retina - pathology | Immunoglobulins | Cytokines | Research & development--R&D | Retina | Permeability | Gene expression | Kinases | Hyperglycemia | Rodents | Diabetes | Vascular endothelial growth factor | Apoptosis | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2014, Volume 9, Issue 10, pp. e108508 - e108508
Inflammation in the diabetic retina is mediated by leukocyte adhesion to the retinal vasculature and alteration of the blood-retinal barrier (BRB). We... 
ADHESION MOLECULE-1 | CELLS | PROTEIN-1 | MACULAR EDEMA | MICROGLIA | MULTIDISCIPLINARY SCIENCES | OCCLUDIN | INJURY | NEOVASCULARIZATION | EXPRESSION | ENDOTHELIAL PERMEABILITY | Blood-Retinal Barrier - cytology | Monocytes - cytology | Cell Count | Humans | Capillary Permeability - drug effects | Male | Blood-Retinal Barrier - drug effects | Dose-Response Relationship, Drug | Inflammation - metabolism | Chemokine CCL2 - metabolism | Cell Line | Blood-Retinal Barrier - immunology | Diabetic Retinopathy - immunology | Endothelial Cells - metabolism | Rats | Chemokine CCL2 - genetics | Glucose - pharmacology | Macrophages - cytology | Gene Knockout Techniques | Chemokine CCL2 - deficiency | Rats, Sprague-Dawley | Diabetic Retinopathy - metabolism | Diabetic Retinopathy - genetics | Up-Regulation - drug effects | Cell Movement - drug effects | Animals | Macrophages - drug effects | Mice | Endothelial Cells - drug effects | Diabetic retinopathy | Inflammation | Glucose | Gene expression | Analysis | Dextrose | Cell culture | Flow cytometry | Retinopathy | Streptozocin | Trafficking | Retina | CX3CR1 protein | Kinases | Macrophages | Blood | Reduction | Rodents | Animal tissues | Conditioning | Vascular endothelial growth factor | Recombinant | Edema | CD11b antigen | Therapeutic applications | Diabetes mellitus | Injection | Endothelial cells | Microglia | White blood cells | Cytometry | Monocytes | Infiltration | Chemokines | Monocyte chemoattractant protein 1 | Index Medicus
Journal Article
The FASEB journal : official publication of the Federation of American Societies for Experimental Biology, ISSN 0892-6638, 2002, Volume 16, Issue 3, pp. 438 - 440
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 08/2017, Volume 138, pp. 130 - 139
Diabetic retinopathy (DR) is the most frequent complication of diabetes and one of leading causes of blindness worldwide. Early phases of DR are characterized... 
Pericytes | Diabetic retinopathy | Inflammation | IL-1β | P2X7 receptor | Molecular modeling | MOLECULAR-DYNAMICS | ACTIVATION | MECHANISM | IL-1 beta | RELEASE | ASTROCYTES | ATP-BINDING | NETWORK ANALYSIS | IN-VITRO | PHARMACOLOGY & PHARMACY | BINDING-SITES | P2X-INDUCED PORE FORMATION | Niacinamide - analogs & derivatives | Diabetic Retinopathy - drug therapy | Pericytes - drug effects | Purinergic P2X Receptor Antagonists - pharmacokinetics | Apoptosis - drug effects | Expert Systems | Humans | Molecular Conformation | Anti-Inflammatory Agents, Non-Steroidal - chemistry | Piperazines - metabolism | Purinergic P2X Receptor Antagonists - pharmacology | Piperazines - chemistry | Diabetic Retinopathy - pathology | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Niacinamide - chemistry | Pericytes - pathology | Databases, Protein | Niacinamide - pharmacokinetics | Anti-Inflammatory Agents, Non-Steroidal - metabolism | Piperazines - pharmacokinetics | Biomarkers - metabolism | Diabetic Retinopathy - immunology | Niacinamide - metabolism | Allosteric Site - drug effects | Pericytes - metabolism | Cells, Cultured | Computational Biology | Models, Molecular | Piperazines - pharmacology | Diabetic Retinopathy - metabolism | Pericytes - immunology | Animals | Databases, Chemical | Purinergic P2X Receptor Antagonists - chemistry | Anti-Inflammatory Agents, Non-Steroidal - pharmacokinetics | Purinergic P2X Receptor Antagonists - metabolism | Receptors, Purinergic P2X7 - metabolism | Ligands | ROC Curve | Molecular Docking Simulation | Structural Homology, Protein | Niacinamide - pharmacology | Receptors, Purinergic P2X7 - chemistry | Medical colleges | Analysis | Blindness | Physiological aspects | Glucose | Dextrose | Index Medicus
Journal Article
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 11/2017, Volume 493, Issue 2, pp. 1136 - 1142
Diabetic retinopathy (DR) is a major microvascular complication of diabetes, resulting in neuronal dysfunction, retinal vascular leakage, and apoptosis within... 
Diabetic retinopathy | NF-κB | TLR7 | Inflammation | MAPKs | OXIDATIVE STRESS | NF-kappa B | BIOPHYSICS | BIOCHEMISTRY & MOLECULAR BIOLOGY | NF-KAPPA-B | Inflammation - pathology | Retinal Pigment Epithelium - metabolism | Retina - metabolism | Cytokines - analysis | Diabetes Mellitus, Type 2 - genetics | Male | Retina - immunology | Diabetic Retinopathy - pathology | Retinal Pigment Epithelium - pathology | Diabetes Mellitus, Type 2 - immunology | Gene Deletion | Toll-Like Receptor 7 - immunology | Diabetic Retinopathy - etiology | Membrane Glycoproteins - immunology | Diabetes Mellitus, Type 2 - complications | Cytokines - immunology | Toll-Like Receptor 7 - genetics | Cell Line | Diabetic Retinopathy - immunology | Mice, Inbred C57BL | Inflammation - immunology | Inflammation - etiology | Diabetic Retinopathy - genetics | Membrane Glycoproteins - genetics | Mice, Knockout | Animals | Retinal Pigment Epithelium - immunology | Inflammation - genetics | Diabetes Mellitus, Type 2 - pathology | Retina - pathology | Type 2 diabetes | Development and progression | Interleukins | Vascular endothelial growth factor | Fructose | Endothelial growth factors | Analysis | Mice | Ophthalmology | Mitogens | Protein kinases | Index Medicus | APOPTOSIS | IMMUNITY | NEOPLASMS | PHOSPHORYLATION | 60 APPLIED LIFE SCIENCES | PATHOGENESIS | STREPTOZOCIN | FRUCTOSE | INHIBITION | NECROSIS | LYMPHOKINES | ANOXIA | IN VITRO | INFLAMMATION | RHODOPSIN | KNOCK-OUT REACTIONS | PHOSPHOTRANSFERASES | MICE | RETINA | RECEPTORS | THICKNESS
Journal Article
PLoS ONE, ISSN 1932-6203, 09/2015, Volume 10, Issue 9, pp. e0137358 - e0137358
Macrophages are involved in low-grade inflammation in diabetes, and play pathogenic roles in proliferative diabetic retinopathy (PDR) by producing... 
VEGF | MULTIDISCIPLINARY SCIENCES | IL-17 | INTERLEUKIN-31 | EXPRESSION | T-CELLS | ANGIOGENIC FACTORS | IL-31 | Tumor Necrosis Factor-alpha - metabolism | Eye - pathology | Inflammation - pathology | Humans | Middle Aged | Male | Diabetic Retinopathy - pathology | Inflammation - metabolism | Retinal Perforations - metabolism | Th17 Cells - metabolism | Vitreous Body - metabolism | Sarcoidosis - immunology | Tumor Necrosis Factor-alpha - immunology | Aged, 80 and over | Adult | Female | Vitrectomy | Macrophages - immunology | Th2 Cells - pathology | Cytokines - blood | Cytokines - immunology | Uveitis - metabolism | Eye - metabolism | Diabetic Retinopathy - immunology | Cytokines - metabolism | Macrophages - pathology | Inflammation - immunology | Diabetic Retinopathy - surgery | Immunity, Innate | Th2 Cells - metabolism | Diabetic Retinopathy - blood | Macrophages - metabolism | Th17 Cells - immunology | Vitreous Body - immunology | Vitreous Body - pathology | Aged | Retinal Perforations - pathology | Inflammation - surgery | Sarcoidosis - metabolism | Uveitis - pathology | Sarcoidosis - pathology | Diabetic retinopathy | Cytokines | Physiological aspects | Development and progression | Genetic aspects | Research | T cells | Retinopathy | Pathogenesis | Helper cells | Homeostasis | Interleukin 22 | Retina | Interleukin 23 | Lymphocytes T | Macrophages | Dermatitis | Hemorrhage | Inflammatory diseases | Interleukin 6 | Interleukin 4 | Lymphocytes | Quality | Surgery | Uveitis | Interleukin 21 | Immune response | Sarcoidosis | Review boards | Diabetes mellitus | Inflammation | Tumor necrosis factor-α | Patients | Allergies | γ-Interferon | Interleukin 10 | Ligands | Infiltration | Interferon | Diabetes | Index Medicus
Journal Article