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1. Full Text Uterine relaxant effects of Curcuma aeruginosa Roxb. rhizome extracts
by Thaina, Peerarat and Tungcharoen, Pattreeya and Wongnawa, Malinee and Reanmongkol, Wantana and Subhadhirasakul, Sanan
Journal of Ethnopharmacology, ISSN 0378-8741, 2009, Volume 121, Issue 3, pp. 433 - 443
The effects and plausible mechanism of action of Roxb. (Zingiberaceae) rhizome chloroform and methanol extracts on the uterine contraction were investigated... 
Relaxation | Extract | Rat | Uterus | Rhizome | Curcuma aeruginosa | PROSTAGLANDIN-F2-ALPHA | CHEMISTRY, MEDICINAL | CONTRACTILITY | ESSENTIAL OIL | MECHANISMS | ANTAGONISM | MYOMETRIUM | PLANT SCIENCES | SMOOTH-MUSCLE | INTEGRATIVE & COMPLEMENTARY MEDICINE | CALCIUM | DRUGS | OXYTOCIN | PHARMACOLOGY & PHARMACY | Oxytocics - pharmacology | Diclofenac - pharmacology | Oxytocin - pharmacology | Vasodilator Agents - pharmacology | Rats, Wistar | Acetylcholine - pharmacology | Plant Extracts - pharmacology | Uterus - drug effects | Rats | Atropine - pharmacology | Calcium Channel Blockers - pharmacology | Parasympatholytics - pharmacology | Verapamil - pharmacology | Uterine Contraction - drug effects | Propranolol - pharmacology | Animals | Isoproterenol - pharmacology | Female | Curcuma | Dinoprost - pharmacology
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2. Full Text Hypoxia enhances the relaxing influence of perivascular adipose tissue in isolated mice aorta
by Maenhaut, Nele and Boydens, Charlotte and Van de Voorde, Johan
European Journal of Pharmacology, ISSN 0014-2999, 2010, Volume 641, Issue 2, pp. 207 - 212
Adipose tissue releases an “adipocyte-derived relaxing factor” (ADRF) lowering tone of isolated arteries. The potential influence of hypoxia on the... 
Relaxation | Hypoxia | Adipose tissue | Arteries | ADIPOCYTES | ADENOSINE | HYDROGEN-SULFIDE | INFLAMMATION | DYSREGULATION | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | VASODILATION | ARTERIAL TONE | EXPRESSION | Norepinephrine - pharmacology | Tetraethylammonium - pharmacology | Theophylline - analogs & derivatives | Cell Hypoxia - physiology | Prostaglandins - pharmacology | Male | Soluble Guanylyl Cyclase | Arteries - drug effects | Adenosine - pharmacology | Receptors, Cytoplasmic and Nuclear - pharmacology | Potassium - pharmacology | Adipose Tissue - metabolism | Animals | Guanylate Cyclase - pharmacology | Glyburide - pharmacology | Aorta, Thoracic - physiology | 15-Hydroxy-11 alpha,9 alpha-(epoxymethano)prosta-5,13-dienoic Acid - pharmacology | Theophylline - pharmacology | Mice | Aorta, Thoracic - drug effects | Vasodilation - physiology | Dinoprost - pharmacology | Sulfides - pharmacology | Adipose tissues | Lactates | Glibenclamide | Theophylline
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3. Full Text Endothelium-derived hyperpolarizing factor mediated relaxations in pig coronary arteries do not involve Gi/o proteins
by Ng, KFJ and Leung, SWS and Man, RYK and Vanhoutte, PM
Acta Pharmacologica Sinica, ISSN 1671-4083, 12/2008, Volume 29, Issue 12, pp. 1419 - 1424
Aim: Endothelium-dependent relaxations to certain neurohumoral substances are mediated by pertussis toxin-sensitive Gi/o protein. Our experiments were designed... 
CELLS | DEPENDENT RELAXATION | ANANDAMIDE | MESENTERIC-ARTERY | NATRIURETIC PEPTIDE | CHEMISTRY, MULTIDISCIPLINARY | endothelium-derived hyperpolarizing factor | Gi/o protein | pig coronary artery | L-ARGININE | pertussis toxin | PHARMACOLOGY & PHARMACY | RECEPTORS | EXPRESSION | EDHF | Endothelium-Dependent Relaxing Factors - pharmacology | Hemostatics - pharmacology | Peptide Fragments - pharmacology | Substance P - pharmacology | Apamin - pharmacology | Serotonin - pharmacology | Dose-Response Relationship, Drug | Thrombin - pharmacology | Charybdotoxin - pharmacology | Neurotransmitter Agents - pharmacology | Dinoprost - pharmacology | Bradykinin - pharmacology | Ionophores - pharmacology | Pertussis Toxin - pharmacology | NG-Nitroarginine Methyl Ester - pharmacology | Ketanserin - pharmacology | Coronary Vessels - drug effects | Vasodilator Agents - pharmacology | Coronary Vessels - physiology | Enzyme Inhibitors - pharmacology | Serotonin Antagonists - pharmacology | Calcimycin - pharmacology | Neurotoxins - pharmacology | Animals | Muscle Relaxation - drug effects | GTP-Binding Protein alpha Subunits, Gi-Go - metabolism | Sus scrofa | Biological Factors - pharmacology | Tocolytic agents | Swine | Tachykinins | Bradykinin | Thrombin | Indomethacin | Carbonates | Endothelium-derived relaxing factors | Peptide hormones | Arteries | Endothelium | Index Medicus
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4. Full Text Reciprocal Relationship Between Reactive Oxygen Species and Cyclooxygenase-2 and Vascular Dysfunction in Hypertension
by Martínez-Revelles, Sonia and Avendaño, María S and García-Redondo, Ana B and Álvarez, Yolanda and Aguado, Andrea and Pérez-Girón, Jose V and García-Redondo, Laura and Esteban, Vanesa and Redondo, Juan M and Alonso, María J and Briones, Ana M and Salaices, Mercedes
Antioxidants & Redox Signaling, ISSN 1523-0864, 01/2013, Volume 18, Issue 1, pp. 51 - 65
Aims: This study evaluates a possible relationship between reactive oxygen species (ROS) and cyclooxygenase (COX)-2-derived products in conductance and... 
Original Research Communications | II-INFUSED MICE | RESPONSES | CELLS | OXIDATIVE STRESS | IMPAIRS ENDOTHELIAL FUNCTION | RESISTANCE ARTERIES | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOCRINOLOGY & METABOLISM | RATS | ANGIOTENSIN-II | HYDROGEN-PEROXIDE | NADPH OXIDASES | Reactive Oxygen Species - metabolism | Oxidative Stress | Rats, Inbred WKY | Endothelium, Vascular - drug effects | Male | Dinoprost - metabolism | Endothelium, Vascular - enzymology | Vasodilation | Phenylephrine - pharmacology | Cyclooxygenase 2 - genetics | Acetophenones - pharmacology | Aorta - physiopathology | Hypertension - enzymology | Aorta - enzymology | Pyrazoles - pharmacology | Rats, Inbred SHR | Vasoconstrictor Agents - pharmacology | Vasodilator Agents - pharmacology | Cyclooxygenase 2 Inhibitors - pharmacology | Mice, Inbred C57BL | Endothelium, Vascular - physiopathology | Rats | Cyclooxygenase 2 - physiology | Nitric Oxide - physiology | Antioxidants - pharmacology | Celecoxib | Sulfonamides - pharmacology | Hypertension - physiopathology | Animals | Cyclooxygenase 2 - metabolism | Mice | Cyclooxygenase 1 - metabolism | In Vitro Techniques | Nitric Oxide - metabolism | Hypertension | Cyclooxygenases | Development and progression | Reactive oxygen species | Research
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5. Full Text Prostaglandin ethanolamides attenuate damage in a human explant colitis model
by Nicotra, Lauren L and Vu, Megan and Harvey, Benjamin S and Smid, Scott D
Prostaglandins and Other Lipid Mediators, ISSN 1098-8823, 01/2013, Volume 100-101, Issue 1, pp. 22 - 29
► Cytokines evoke colitis-like mucosal damage in human colonic explant tissue. ► Both exogenous and endogenous prostamides inhibit mucosal damage. ► The... 
Prostamides | Colitis | Anandamide | Bimatoprost | COX-2 METABOLITE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ENDOGENOUS CANNABINOID SYSTEM | RECEPTOR | CELL BIOLOGY | MATRIX METALLOPROTEINASES | INFLAMMATORY-BOWEL-DISEASE | UP-REGULATION | EXPRESSION | T-CELLS | Immunohistochemistry | Interleukin-1beta - pharmacology | Endocannabinoids - pharmacology | Humans | Middle Aged | Male | Receptors, Prostaglandin - metabolism | Colon, Sigmoid - metabolism | Young Adult | Cloprostenol - pharmacology | Adult | Female | Dinoprost - analogs & derivatives | Colon, Sigmoid - pathology | Dinoprost - pharmacology | Receptors, Prostaglandin - antagonists & inhibitors | Amides - pharmacology | Dinoprostone - pharmacology | Tissue Culture Techniques | Colon, Sigmoid - drug effects | Sulfonamides - pharmacology | Arachidonic Acids - pharmacology | Cloprostenol - analogs & derivatives | Cyclooxygenase Inhibitors - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Colitis - metabolism | Cyclooxygenase 2 - metabolism | Polyunsaturated Alkamides - pharmacology | Dinoprostone - analogs & derivatives | Oxazoles - pharmacology | Colitis - prevention & control | Metabolites | Prostaglandins
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6. Full Text Antihypertensive effects of the flavonoid quercetin in spontaneously hypertensive rats
by Duarte, Juan and Pérez‐Palencia, Raquel and Vargas, Felix and Angeles Ocete, Maria and Pérez‐Vizcaino, Francisco and Zarzuelo, Antonio and Tamargo, Juan
British Journal of Pharmacology, ISSN 0007-1188, 05/2001, Volume 133, Issue 1, pp. 117 - 124
The effects of an oral daily dose (10 mg kg−1) of the flavonoid quercetin for 5 weeks in spontaneously hypertensive (SHR) and normotensive Wistar Kyoto rats... 
antioxidant | flavonoid | SHR | hypertension | Quercetin | Hypertension | Antioxidant | Flavonoid | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | RISK | CORONARY HEART-DISEASE | VASCULAR SMOOTH-MUSCLE | BLOOD-PRESSURE | PLASMA | ANTIOXIDANT STATUS | IN-VIVO | NITRIC-OXIDE | CARDIOVASCULAR-DISEASE | quercetin | PHARMACOLOGY & PHARMACY | Norepinephrine - pharmacology | Kidney - pathology | Body Weight - drug effects | Rats, Inbred WKY | Endothelium, Vascular - drug effects | Hypertension - drug therapy | Male | Nitroprusside - pharmacology | Endothelium, Vascular - physiology | Heart Rate - drug effects | Malondialdehyde - blood | Potassium Chloride - pharmacology | Quercetin - administration & dosage | Blood Pressure - drug effects | Dinoprost - analogs & derivatives | Oxidants - blood | Oxidants - urine | Heart Ventricles - pathology | Rats, Inbred SHR | Vasoconstrictor Agents - pharmacology | Kidney - drug effects | Vasodilator Agents - pharmacology | Dinoprost - urine | Acetylcholine - pharmacology | Quercetin - therapeutic use | Aorta - drug effects | Rats | Antioxidants - pharmacology | Antioxidants - therapeutic use | Organ Size - drug effects | Animals | Quercetin - pharmacology | Antioxidants - administration & dosage | Aorta - cytology | Vasodilation - drug effects | In Vitro Techniques | Aorta - physiology | Heart Ventricles - drug effects | Papers
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7. Full Text Suppression of endoplasmic reticulum stress improves endothelium-dependent contractile responses in aorta of the spontaneously hypertensive rat
by Spitler, Kathryn M and Matsumoto, Takayuki and Webb, R. Clinton
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 08/2013, Volume 305, Issue 3, pp. H344 - H353
A contributing factor to increased peripheral resistance seen during hypertension is an increased production of endothelium-derived contractile factors... 
Cyclooxygenase | EDCF | ERK1/2 | cPLA | Endoplasmic reticulum stress | CYTOSOLIC PHOSPHOLIPASE A | OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | MESENTERIC-ARTERIES | ER STRESS | OLETF RATS | cyclooxygenase | endoplasmic reticulum stress | PROSTANOID RECEPTORS | UNFOLDED PROTEIN RESPONSE | GENE-EXPRESSION | RENAL-ARTERIES | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | Antihypertensive Agents - pharmacology | Epoprostenol - metabolism | Phosphorylation | Muscle, Smooth, Vascular - metabolism | Rats, Inbred WKY | Endothelium, Vascular - drug effects | Hypertension - drug therapy | Male | Aorta - metabolism | Dinoprost - metabolism | Muscle, Smooth, Vascular - physiopathology | Dose-Response Relationship, Drug | Taurochenodeoxycholic Acid - pharmacology | Phenylbutyrates - pharmacology | tert-Butylhydroperoxide - pharmacology | Blood Pressure - drug effects | Membrane Proteins - metabolism | Aorta - physiopathology | Myocytes, Smooth Muscle - drug effects | Myocytes, Smooth Muscle - metabolism | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Rats, Inbred SHR | Vasoconstrictor Agents - pharmacology | Vasodilator Agents - pharmacology | Endoplasmic Reticulum Stress - drug effects | Acetylcholine - pharmacology | Aorta - drug effects | Cells, Cultured | Endothelium, Vascular - physiopathology | bcl-2-Associated X Protein - metabolism | Rats | Vasoconstriction - drug effects | Hypertension - physiopathology | Phospholipases A2, Cytosolic - metabolism | Hydrogen Peroxide - metabolism | Hypertension - metabolism | Thromboxane A2 - metabolism | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Endothelium, Vascular - metabolism | Arachidonic Acid - pharmacology | Cyclooxygenase 1 - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Hypertension | Physiological aspects | Stress (Physiology) | Endoplasmic reticulum | Health aspects | Muscle contraction | cPLA2 | Vascular Biology and Microcirculation | ERK1
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8. Full Text Interaction of human and rat organic anion transporter 2 with various cephalosporin antibiotics
by Khamdang, Suparat and Takeda, Michio and Babu, Ellappan and Noshiro, Rie and Onozato, Maristela Lika and Tojo, Akihiro and Enomoto, Atsushi and Huang, Xiu-Lin and Narikawa, Shinichi and Anzai, Naohiko and Piyachaturawat, Pawinee and Endou, Hitoshi
European Journal of Pharmacology, ISSN 0014-2999, 2003, Volume 465, Issue 1, pp. 1 - 7
Cephalosporin antibiotics are thought to be excreted into the urine via organic anion transporters (OATs) and OAT can mediate nephrotoxicity by cephalosporins,... 
Proximal tubule | Cell line | Transport | Cephalosporin antibiotic | Organic anion transporter | cephalosporin antibiotic | organic anion transporter | transport | MEDIATE | IDENTIFICATION | CATION TRANSPORTERS | BETA-LACTAM ANTIBIOTICS | PHARMACOLOGY & PHARMACY | MOLECULAR-CLONING | INHIBITORS | proximal tubule | KIDNEY | cell line | EXPRESSION | CEPHALORIDINE | NEPHROTOXICITY | Cefoperazone - pharmacology | Organic Anion Transporters, Sodium-Independent - genetics | Humans | Ceftriaxone - pharmacology | Dose-Response Relationship, Drug | Cefamandole - pharmacology | Transfection | Organic Anion Transporters, Sodium-Independent - metabolism | Cefadroxil - pharmacology | Biological Transport - drug effects | Cephalosporins - pharmacology | Cell Line | Cell Survival - drug effects | Cefazolin - pharmacology | Dinoprost - pharmacokinetics | Cephalothin - pharmacology | Probenecid - pharmacology | Rats | Mice, Transgenic | Cephaloridine - pharmacology | Animals | Uricosuric Agents - pharmacology | Cefotaxime - pharmacology | Anti-Bacterial Agents - pharmacology | Mice | Kinetics
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9. Full Text Endocannabinoid-mediated modulation of Gq/11 protein-coupled receptor signaling-induced vasoconstriction and hypertension
by Szekeres, Mária and Nádasy, György L and Turu, Gábor and Soltész-Katona, Eszter and Benyó, Zoltán and Offermanns, Stefan and Ruisanchez, Éva and Szabó, Eszter and Takáts, Zoltán and Bátkai, Sándor and Tóth, Zsuzsanna E and Hunyady, László
Molecular and Cellular Endocrinology, ISSN 0303-7207, 03/2015, Volume 403, pp. 46 - 56
Activation of G protein-coupled receptors (GPCRs) can induce vasoconstriction via calcium signal-mediated and Rho-dependent pathways. Earlier reports have... 
G protein-coupled receptor | Calcium signaling | Blood pressure | Endocannabinoid | Angiotensin II | Vasoconstriction | Lipoprotein Lipase - antagonists & inhibitors | Endocannabinoids - pharmacology | Muscle, Smooth, Vascular - metabolism | Rats, Wistar | Calcium - metabolism | Monoacylglycerol Lipases - antagonists & inhibitors | GTP-Binding Protein alpha Subunits, Gq-G11 - deficiency | Hypertension - drug therapy | Male | Lactones - pharmacology | Monoacylglycerol Lipases - genetics | Monoacylglycerol Lipases - metabolism | Phenylephrine - pharmacology | Piperidines - pharmacology | Hypertension - genetics | Myocytes, Smooth Muscle - drug effects | Dinoprost - pharmacology | Lipoprotein Lipase - metabolism | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Calcium Signaling | Muscle, Smooth, Vascular - drug effects | GTP-Binding Protein alpha Subunits, Gq-G11 - genetics | Angiotensin II - pharmacology | Lipoprotein Lipase - genetics | Tissue Culture Techniques | Aorta - drug effects | Gene Expression Regulation | Rats | Muscle, Smooth, Vascular - cytology | Vasoconstriction - drug effects | Arachidonic Acids - pharmacology | Hypertension - physiopathology | Hypertension - metabolism | Mice, Knockout | Receptor, Cannabinoid, CB1 - metabolism | Glycerides - pharmacology | Animals | Receptor, Cannabinoid, CB1 - genetics | Mice | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Benzodioxoles - pharmacology
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10. Full Text Linalool elicits vasorelaxation of mouse aortae through activation of guanylyl cyclase and K+ channels
by Kang, Purum and Seol, Geun Hee
Journal of Pharmacy and Pharmacology, ISSN 0022-3573, 05/2015, Volume 67, Issue 5, pp. 714 - 719
Objectives The aim of this study was to investigate the cardiovascular relaxing properties of monoterpene alcohol (‐)‐linalool (LIN), a principal component of... 
K+ channel | (‐)‐linalool | soluble guanylyl cyclase | vasorelaxation | channel | (-)-linalool | Vasorelaxation | Soluble guanylyl cyclase | K + channel | SMOOTH-MUSCLE | POTASSIUM CHANNELS | ESSENTIAL OIL | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | STRESS | HYPERTENSIVE-RATS | BLOOD-PRESSURE | NG-Nitroarginine Methyl Ester - pharmacology | Tetraethylammonium - pharmacology | Vasodilator Agents - pharmacology | Calcium - metabolism | Aorta - drug effects | Endoplasmic Reticulum - metabolism | Guanylate Cyclase - metabolism | Male | Aorta - metabolism | Enzyme Activation - drug effects | Dose-Response Relationship, Drug | Potassium Channels - metabolism | Quinoxalines - pharmacology | Calcium Chloride - pharmacology | Monoterpenes - pharmacology | Animals | Oxadiazoles - pharmacology | Calcium Chloride - antagonists & inhibitors | Dinoprost - antagonists & inhibitors | Mice | Monoterpenes - antagonists & inhibitors | Vasodilation - drug effects | Dinoprost - pharmacology | Blood vessels | Dilatation | Nitric oxide
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11. Full Text Urocortin 1 expression and secretion by human umbilical vein endothelial cells: In vitro effects of interleukin 8, interferon γ, lipopolysaccharide, endothelin 1, prostaglandin F-2α, estradiol, progesterone and dexamethasone
by Borges, Lavínia E and Bloise, Enrrico and Dela Cruz, Cynthia and Galleri, Letizia and Apa, Rosanna and Petraglia, Felice and Reis, Fernando M
Peptides, ISSN 0196-9781, 12/2015, Volume 74, pp. 64 - 69
Urocortin 1 (Ucn1) is a 40-amino-acid peptide that has vasodilatory activity and displays immunomodulatory and antioxidant properties. Maternal and cord plasma... 
Urocortin | Dexamethasone | Interferon-γ | Endothelin-1 | HUVEC | Interleukin-8 | LPS | Interferon-gamma | ENDOMETRIUM | BIOCHEMISTRY & MOLECULAR BIOLOGY | INNERVATION | HUMAN PLACENTAL CELLS | BLOOD-FLOW | PEPTIDE | ACTIVIN-A | CORTICOTROPIN-RELEASING-FACTOR | HORMONE | MESSENGER-RNA | PHARMACOLOGY & PHARMACY | INFLAMMATORY CYTOKINES | Gene Expression | Human Umbilical Vein Endothelial Cells - drug effects | Human Umbilical Vein Endothelial Cells - metabolism | Endothelin-1 - pharmacology | Humans | Cells, Cultured | Urocortins - genetics | Pregnancy | Urocortins - metabolism | Dexamethasone - pharmacology | Lipopolysaccharides - pharmacology | Female | Interleukin-8 - pharmacology | Dinoprost - pharmacology | Progesterone - pharmacology | Estradiol - pharmacology | Interferon-gamma - pharmacology
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12. Full Text Comparison of the mechanisms underlying the relaxation induced by two nitric oxide donors: Sodium nitroprusside and a new ruthenium complex
by Bonaventura, Daniella and de Lima, Renata Galvão and Vercesi, Juliana A and da Silva, Roberto Santana and Bendhack, Lusiane M
Vascular Pharmacology, ISSN 1537-1891, 2006, Volume 46, Issue 3, pp. 215 - 222
Abstract We studied the mechanisms involved in the relaxation induced by nitric oxide (NO) donors, ruthenium complex ([Ru(terpy)(bdq)NO+ ]3+ -TERPY) and sodium... 
Cardiovascular | Nitric oxide species | Nitric oxide donor | K + channels and vasorelaxation | Guanylyl cyclase | channels and vasorelaxation | ACTIVATION | nitric oxide species | ENDOTHELIAL DYSFUNCTION | POTASSIUM CHANNELS | MESENTERIC-ARTERY | PULMONARY-ARTERY | SOLUBLE GUANYLATE-CYCLASE | GLYCERYL TRINITRATE | VASCULAR SMOOTH-MUSCLE | guanylyl cyclase | K+ channels and vasorelaxation | PHARMACOLOGY & PHARMACY | nitric oxide donor | RAT AORTIC RINGS | VISIBLE-LIGHT IRRADIATION | Free Radical Scavengers - pharmacology | Norepinephrine - pharmacology | Oxyhemoglobins - metabolism | Vasodilator Agents - pharmacology | Rats, Wistar | Enzyme Inhibitors - pharmacology | Rats | Endothelium, Vascular - drug effects | Male | Guanylate Cyclase - antagonists & inhibitors | Nitroprusside - pharmacology | Animals | Phenylephrine - pharmacology | Cyclic GMP - metabolism | Cysteine - pharmacology | Sarcoplasmic Reticulum Calcium-Transporting ATPases - antagonists & inhibitors | Aorta, Thoracic - drug effects | Nitric Oxide Donors - pharmacology | Vasodilation - drug effects | In Vitro Techniques | Dinoprost - pharmacology | Potassium Channel Blockers - pharmacology | Ruthenium Compounds - pharmacology | Sodium nitroferricyanide | Ruthenium | Comparative analysis | Nitric oxide
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13. Full Text Mechanisms involved in increased sensitivity to adenosine A(2A) receptor activation and hypoxia-induced vasodilatation in porcine coronary arteries
by Hedegaard, ER and Nielsen, BD and Mogensen, S and Rembold, CM and Frobert, O and Simonsen, U and Region Örebro län
EUROPEAN JOURNAL OF PHARMACOLOGY, ISSN 0014-2999, 01/2014, Volume 723, pp. 216 - 226
Hypoxia-induced coronary vasorelaxation is a compensatory mechanism increasing blood flow. We hypothesized that hypoxia shares pathways with adenosine and... 
Force suppression | MLC | PROTEIN-KINASE | SWINE | MYOCARDIAL-ISCHEMIA | RELAXATION | Vasorelaxation | DILATION | VASCULAR SMOOTH-MUSCLE | HSP20 PHOSPHORYLATION | INHIBITION | NITRIC-OXIDE | Hypoxia | Coronary | PHARMACOLOGY & PHARMACY | HSP20 | CAMP | K+ CHANNELS | Coronary Vessels - drug effects | Triazines - pharmacology | Phenethylamines - pharmacology | Oxygen - physiology | Coronary Vessels - physiology | Adenosine-5'-(N-ethylcarboxamide) - pharmacology | Purinergic P1 Receptor Agonists - pharmacology | Receptors, Purinergic P1 - physiology | Adenosine - pharmacology | HSP20 Heat-Shock Proteins - metabolism | Purinergic P1 Receptor Antagonists - pharmacology | Triazoles - pharmacology | Animals | Swine | Adenosine - analogs & derivatives | Hypoxia - physiopathology | Vasodilation - drug effects | Vasodilation - physiology | In Vitro Techniques | Cyclic AMP - metabolism | Dinoprost - pharmacology | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Farmakologi och toxikologi | Pharmacology and Toxicology
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