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Cancer Research, ISSN 0008-5472, 05/2010, Volume 70, Issue 9, pp. 3606 - 3617
Curcumin induces cancer cell growth arrest and apoptosis in vitro, but its poor bioavailability in vivo limits its antitumor efficacy. We have previously... 
GROWTH-FACTOR RECEPTOR | APOPTOSIS | TO-MESENCHYMAL TRANSITION | INHIBITION | ONCOLOGY | ADENOCARCINOMA | DOWN-REGULATION | ORTHOTOPIC MODEL | FACTOR-KAPPA-B | MICRORNA-21 | LINES | Pancreatic Neoplasms - metabolism | Triterpenes - pharmacology | Apoptosis - drug effects | DNA, Neoplasm - metabolism | Dinoprostone - biosynthesis | Humans | DNA, Complementary - genetics | Apoptosis - genetics | Deoxycytidine - pharmacology | NF-kappa B - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Curcumin - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Vascular Endothelial Growth Factor A - secretion | Pancreatic Neoplasms - drug therapy | Transfection | Gene Expression Regulation, Neoplastic - genetics | PTEN Phosphohydrolase - genetics | NF-kappa B - antagonists & inhibitors | Deoxycytidine - administration & dosage | Pancreatic Neoplasms - pathology | Curcumin - pharmacology | Gene Silencing | MicroRNAs - biosynthesis | Pancreatic Neoplasms - genetics | Drug Synergism | Oligonucleotides, Antisense - genetics | NF-kappa B - genetics | Cell Line, Tumor | MicroRNAs - genetics | DNA, Neoplasm - genetics | Dinoprostone - antagonists & inhibitors | Triterpenes - administration & dosage | Deoxycytidine - analogs & derivatives
Journal Article
Journal Article
Journal Article
Journal Article
Bioorganic & Medicinal Chemistry Letters, ISSN 0960-894X, 05/2016, Volume 26, Issue 10, pp. 2531 - 2538
Journal Article
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 11/2003, Volume 140, Issue 6, pp. 1077 - 1087
Although capsaicin analogs might be a potential strategy to manipulate inflammation, the mechanism is still unclear. In this study, the effects and action... 
macrophages | Capsaicin | JNK | capsazepine | COX‐2 | IKK | TRPV1 | ERK | COX-2 | Macrophages | Capsazepine | KAPPA-B ACTIVATION | ROOT GANGLION NEURONS | FATTY-ACIDS | PROSTAGLANDIN ENDOPEROXIDE SYNTHASE-2 | INTERFERON-GAMMA | MESSENGER-RNA | capsaicin | PHARMACOLOGY & PHARMACY | MOLECULAR-CLONING | RAT PERITONEAL-MACROPHAGES | TRANSCRIPTION FACTOR-KAPPA | VANILLOID RECEPTOR ANTAGONIST | Prostaglandin-Endoperoxide Synthases - biosynthesis | Tetradecanoylphorbol Acetate - pharmacology | Capsaicin - pharmacology | Dinoprostone - biosynthesis | Receptors, Drug - genetics | Cyclooxygenase 2 | Nitric Oxide Synthase - antagonists & inhibitors | Male | NF-kappa B - metabolism | Nitric Oxide Synthase - genetics | RNA, Messenger - metabolism | Nitric Oxide Synthase Type II | Transcription Factor AP-1 - metabolism | DNA-Binding Proteins - metabolism | Dose-Response Relationship, Drug | Enzyme Induction - drug effects | JNK Mitogen-Activated Protein Kinases | Protein-Serine-Threonine Kinases - metabolism | RNA, Messenger - drug effects | Diterpenes - pharmacology | Prostaglandin-Endoperoxide Synthases - genetics | Cell Line | Signal Transduction | Isoenzymes - genetics | RNA, Messenger - genetics | Rats | Capsaicin - analogs & derivatives | Enzyme Activation - drug effects | I-kappa B Kinase | Rats, Sprague-Dawley | Receptors, Drug - metabolism | Macrophages - enzymology | Neurotoxins - pharmacology | Macrophages - metabolism | Animals | Lipopolysaccharides - pharmacology | STAT1 Transcription Factor | Macrophages - drug effects | Trans-Activators - metabolism | Nitric Oxide Synthase - metabolism | Ganglia, Spinal - drug effects | Isoenzymes - biosynthesis | Interferon-gamma - pharmacology | Ganglia, Spinal - metabolism | Mitogen-Activated Protein Kinases - metabolism | Papers
Journal Article
Inflammation, ISSN 0360-3997, 4/2014, Volume 37, Issue 2, pp. 542 - 554
Plumbagin has been reported to modulate cellular redox status and suppress NF-κB. In the present study, we investigated the effect of plumbagin on... 
Pathology | Medicine & Public Health | MAPKinases | Rheumatology | endotoxemia | reactive oxygen species | Internal Medicine | cytokines | Pharmacology/Toxicology | GSH | ACTIVATED PROTEIN-KINASE | DOWN-REGULATION | CYTOKINE | IMMUNOLOGY | MURINE | CELL BIOLOGY | THIOREDOXIN | INDUCED INHIBITION | TRANSCRIPTION FACTOR | T-CELLS | SEVERE SEPSIS | LYMPHOCYTES | Tumor Necrosis Factor-alpha - metabolism | Spleen - immunology | Male | NF-kappa B - metabolism | Shock, Septic - prevention & control | Spleen - drug effects | Extracellular Signal-Regulated MAP Kinases - metabolism | Shock, Septic - immunology | Lipopolysaccharides | Dose-Response Relationship, Drug | Liver - immunology | Liver - drug effects | Time Factors | Shock, Septic - blood | Inflammation Mediators - metabolism | Lung - metabolism | Interleukin-6 - metabolism | Macrophages - immunology | Disease Models, Animal | Cell Line | Shock, Septic - chemically induced | Anti-Inflammatory Agents - pharmacology | Down-Regulation | Liver - metabolism | Naphthoquinones - pharmacology | Antioxidants - pharmacology | Dinoprostone - metabolism | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Spleen - metabolism | Lung - drug effects | Macrophages - drug effects | Mice | Enzyme Activation | Oxidative Stress - drug effects | Nitric Oxide - metabolism | Lung - immunology
Journal Article
Immunology, ISSN 0019-2805, 03/2010, Volume 129, Issue 3, pp. 375 - 385
Summary The synthetic phospho‐ceramide analogue‐1 (PCERA‐1) down‐regulates production of the pro‐inflammatory cytokine tumour necrosis factor‐α (TNF‐α) and... 
macrophages | cyclic adenosine monophosphate response element binding protein | lipopolysaccharide | inflammation | interleukin‐10 | tumour necrosis factor‐α | Cyclic adenosine monophosphate response element binding protein | Lipopolysaccharide | Inflammation | Tumour necrosis factor-α | Interleukin-10 | Macrophages | PROTEIN-KINASE-C | TRANSCRIPTIONAL ACTIVATION | VASOACTIVE-INTESTINAL-PEPTIDE | BETA-ADRENOCEPTOR | IMMUNOLOGY | interleukin-10 | CYTOKINE EXPRESSION | THYROID FRTL-5 CELLS | LIPOPOLYSACCHARIDE INDUCTION | CERAMIDE 1-PHOSPHATE | HIGHLY POTENT INHIBITIONS | TNF-ALPHA | tumour necrosis factor-alpha | Tumor Necrosis Factor-alpha - metabolism | Guanosine Triphosphate - metabolism | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Ribosomal Protein S6 Kinases, 90-kDa - metabolism | Isoquinolines - pharmacology | Transfection | Cyclic AMP - analogs & derivatives | Ribosomal Protein S6 Kinases, 90-kDa - antagonists & inhibitors | Interleukin-10 - metabolism | Indoles - pharmacology | Cell Membrane - metabolism | Response Elements - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Phosphorylation - drug effects | Rolipram - pharmacology | Cyclic AMP - metabolism | Cell Membrane - drug effects | Cyclic AMP-Dependent Protein Kinases - metabolism | Cell Line | Dinoprostone - pharmacology | Enzyme Inhibitors - pharmacology | Genes, Reporter - genetics | Imidazoles - pharmacology | Adenylyl Cyclases - metabolism | Sulfonamides - pharmacology | Ceramides - pharmacology | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Calcium Signaling - drug effects | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cyclic AMP Response Element-Binding Protein - metabolism | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Signal Transduction - physiology | Bucladesine - pharmacology | Mice | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | tumour necrosis factor-α | Original
Journal Article
Pharmacological Research, ISSN 1043-6618, 11/2016, Volume 113, Issue Pt A, pp. 500 - 514
Journal Article