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BBA - Molecular and Cell Biology of Lipids, ISSN 1388-1981, 06/2016, Volume 1861, Issue 6, pp. 491 - 500
Journal Article
The EMBO Journal, ISSN 1460-2075, 2007, Volume 26, Issue 24, pp. 5020 - 5032
The signaling lipid molecule 15‐deoxy‐delta 12,14‐prostaglandin J2 (15d‐PGJ2) has multiple cellular functions, including anti‐inflammatory and antineoplastic... 
15d‐PGJ2 | translation | proliferation | inflammation | eIF4A | Translation | Inflammation | Proliferation | 15d-PGJ2 | TRANSFORMATION SUPPRESSOR PDCD4 | KAPPA-B ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | 15-DEOXY-DELTA(12,14)-PROSTAGLANDIN J | BINDING-PROTEIN | INITIATION CODON | CELL BIOLOGY | HEPATITIS-C VIRUS | PATHWAY | STRESS GRANULE FORMATION | PRODUCT PATEAMINE-A | RNA HELICASE EIF4A | Tumor Necrosis Factor-alpha - metabolism | Arachidonic Acid - metabolism | Protein Biosynthesis | Sodium Compounds - metabolism | Hypoglycemic Agents - metabolism | Humans | TNF Receptor-Associated Factor 2 - genetics | Cytoplasmic Granules - chemistry | PPAR gamma - metabolism | Anti-Inflammatory Agents - metabolism | Chromans - metabolism | Poly(A)-Binding Proteins - metabolism | Prostaglandin D2 - metabolism | Cytoplasmic Granules - metabolism | Eukaryotic Initiation Factor-2 - metabolism | Prostaglandin D2 - analogs & derivatives | T-Cell Intracellular Antigen-1 | Eukaryotic Initiation Factor-4A - genetics | Emetine - metabolism | Enzyme Inhibitors - metabolism | Poly(A)-Binding Proteins - genetics | Gene Expression Regulation | TNF Receptor-Associated Factor 2 - metabolism | Protein Synthesis Inhibitors - metabolism | Thiazolidinediones - metabolism | Arsenites - metabolism | Dinoprostone - metabolism | Eukaryotic Initiation Factor-2 - genetics | Inflammation - genetics | Cyclopentanes - metabolism | Signal Transduction - physiology | Prostaglandins A - metabolism | HeLa Cells | Eukaryotic Initiation Factor-4A - metabolism | Lipids | Cellular biology | Molecular biology | Binding sites | Inflammatory diseases
Journal Article
Circulation, ISSN 0009-7322, 07/2003, Volume 108, Issue 2, pp. 198 - 204
Background-Intravitreal neovascular diseases, as in ischemic retinopathies, are a major cause of blindness. Because inflammatory mechanisms influence vitreal... 
Prostaglandins | Ischemia | Diabetes mellitus | Vasculature | vasculature | APOPTOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | ANGIOGENESIS | OXYGEN-INDUCED RETINOPATHY | diabetes mellitus | OCULAR NEOVASCULARIZATION | ischemia | RETINAL NEOVASCULARIZATION | INHIBITION | IN-VIVO | prostaglandins | PERIPHERAL VASCULAR DISEASE | PLATELET-ACTIVATING-FACTOR | GROWTH-FACTOR | PROSTAGLANDIN E-2 | Retina - drug effects | Endothelial Growth Factors - metabolism | Vascular Endothelial Growth Factors | Humans | Middle Aged | Astrocytes - pathology | Male | Astrocytes - enzymology | Receptors, Lipoprotein - metabolism | Diabetic Retinopathy - complications | Receptors, Prostaglandin E, EP4 Subtype | Retinal Vessels - pathology | Lymphokines - metabolism | Ischemia - pathology | Disease Models, Animal | Enzyme Inhibitors - pharmacology | Rats | Retinal Vessels - drug effects | Rats, Sprague-Dawley | Receptors, Prostaglandin E, EP3 Subtype | Cell Division - drug effects | Dinoprostone - metabolism | Membrane Proteins | Receptors, Scavenger | Neovascularization, Pathologic - drug therapy | Receptors, Immunologic | Mice | Thrombospondin 1 - metabolism | Vitreoretinopathy, Proliferative - enzymology | Retina - pathology | Diabetic Retinopathy - drug therapy | Vascular Endothelial Growth Factor A | Vitreoretinopathy, Proliferative - complications | Cyclooxygenase 2 | Ischemia - enzymology | Diabetic Retinopathy - pathology | Neovascularization, Pathologic - pathology | Intercellular Signaling Peptides and Proteins - metabolism | CD36 Antigens - metabolism | Ischemia - complications | Isoenzymes - metabolism | Retina - enzymology | Adult | Female | Astrocytes - drug effects | Vitreoretinopathy, Proliferative - drug therapy | Mice, Inbred C57BL | Vitreoretinopathy, Proliferative - pathology | Cells, Cultured | Receptors, Prostaglandin E - drug effects | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Animals | Diabetic Retinopathy - enzymology | Prostaglandin-Endoperoxide Synthases - metabolism | Aged | Isoenzymes - antagonists & inhibitors | Receptors, Prostaglandin E - metabolism | Enzyme Inhibitors | Astrocytes | Antigens, CD36 | Neurons and Cognition | Endothelial Growth Factors | Dinoprostone | Intercellular Signaling Peptides | Life Sciences | Cell Division | Diabetic Retinopathy
Journal Article
Nature (London), ISSN 1476-4687, 2016, Volume 531, Issue 7595, pp. 528 - 532
Journal Article
Gut, ISSN 0017-5749, 08/2014, Volume 63, Issue 8, pp. 1300 - 1312
Objective Enteric glia activation has been reported to amplify intestinal inflammation via the enteroglial-specific S100B protein. This neurotrophin promotes... 
NITRIC-OXIDE PRODUCTION | INHIBITION | CROHNS-DISEASE | MACROPHAGES | ULCERATIVE-COLITIS | FATTY-ACID AMIDE | S100B PROTEIN | N-PALMITOYL-ETHANOLAMINE | GLIAL-CELLS | GASTROENTEROLOGY & HEPATOLOGY | BOWEL-DISEASE | Tumor Necrosis Factor-alpha - metabolism | Dextran Sulfate | Ethanolamines - pharmacology | Endocannabinoids - pharmacology | Rectum - pathology | Humans | Middle Aged | Palmitic Acids - therapeutic use | Male | NF-kappa B - metabolism | Glial Fibrillary Acidic Protein - metabolism | Nitric Oxide - analysis | Colon, Sigmoid - chemistry | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Neuroglia - drug effects | Colitis - chemically induced | Female | Indoles - pharmacology | Colitis - drug therapy | S100 Calcium Binding Protein beta Subunit - metabolism | Colitis, Ulcerative - drug therapy | Palmitic Acids - pharmacology | Colon, Sigmoid - pathology | Endocannabinoids - therapeutic use | Neutrophil Infiltration - drug effects | Severity of Illness Index | PPAR alpha - antagonists & inhibitors | Signal Transduction | Colitis, Ulcerative - metabolism | Cells, Cultured | Colitis, Ulcerative - pathology | Toll-Like Receptor 4 - metabolism | Dinoprostone - metabolism | Nerve Tissue Proteins - metabolism | Animals | PPAR gamma - antagonists & inhibitors | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Anilides - pharmacology | Colitis - metabolism | Cyclooxygenase 2 - metabolism | Rectum - chemistry | Ethanolamines - therapeutic use | Neuroglia - metabolism | Mice | PPAR alpha - metabolism | Nitric Oxide - metabolism | Nitric Oxide Synthase Type II - metabolism | Influence | Amides | Inflammation | Neuroglia | Analysis | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
Reviews in the Neurosciences, ISSN 0334-1763, 04/2012, Volume 23, Issue 2, pp. 163 - 177
Journal Article
International Journal of Urology, ISSN 0919-8172, 01/2018, Volume 25, Issue 1, pp. 7 - 17
There is growing evidence of the presence of cancer stem cells in urothelial carcinoma. Cancer stem cells have the ability to self‐renew and to differentiate... 
signal pathways | epithelial–mesenchymal transition | urothelial carcinoma | cancer stem cell | bladder cancer | marker | INITIATING CELLS | BLADDER-CANCER | SIDE POPULATION | NOTCH PATHWAY | CD133 EXPRESSION | MYRTUCOMMULONE-A | RIBONUCLEASE INHIBITOR | EMERGING ROLE | SIGNALING PATHWAY | UROLOGY & NEPHROLOGY | TUMOR-SUPPRESSOR | epithelial-mesenchymal transition | Dinoprostone - biosynthesis | Polycomb Repressive Complex 1 - metabolism | Receptors, Notch - metabolism | Humans | Receptors, Androgen - metabolism | Retinal Dehydrogenase - metabolism | Hedgehog Proteins - metabolism | MicroRNAs - metabolism | Carcinoma, Transitional Cell - therapy | Autophagy | Neoplastic Stem Cells - metabolism | Isoenzymes - metabolism | Immunotherapy | Epithelial-Mesenchymal Transition | Hyaluronan Receptors - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | STAT3 Transcription Factor - metabolism | Wnt Signaling Pathway | Biomarkers - metabolism | Carcinoma, Transitional Cell - metabolism | Carcinoma, Transitional Cell - pathology | AC133 Antigen - metabolism | Cyclooxygenase 2 - metabolism | SOX Transcription Factors - metabolism | Polycomb Repressive Complex 2 - metabolism | Transforming Growth Factor beta - metabolism | Transplantation | Carcinoma | Health aspects | Stem cells | Cancer | Cell proliferation | Urothelial cancer | Wnt protein | Mesenchyme | Stat3 protein | Clinical trials | Prostaglandin E2 | AKT protein | Drug development | Cancer therapies | Cell adhesion & migration | 1-Phosphatidylinositol 3-kinase | Reviews | CD44 antigen | Hedgehog protein | PD-L1 protein | Xenografts | miRNA | Cyclooxygenase-2 | Urothelial carcinoma | Phagocytosis | Cell migration
Journal Article
Journal Article
Molecular neurobiology, ISSN 1559-1182, 2015, Volume 53, Issue 9, pp. 6426 - 6443
Artemether, a lipid-soluble derivative of artemisinin has been reported to possess anti-inflammatory properties. In this study, we have investigated the... 
Neurology | NF-κB | Neurosciences | Biomedicine | BV2 microglia | HT22 hippocampal neurons | Nrf2 | Neurobiology | Artemether | Neuroinflammation | Cell Biology | ALZHEIMERS-DISEASE | INFLAMMATORY REACTIONS | NEUROSCIENCES | BV-2 CELLS | PROTECTS | STAT3 ACTIVATION | NF-kappa B | CULTURED ASTROCYTES | HEME OXYGENASE-1 | NF-KAPPA-B | ARTEMISININ | Neuroprotective Agents - therapeutic use | Inflammation - pathology | Microglia - metabolism | Heme Oxygenase-1 - metabolism | Transcriptional Activation - genetics | Antioxidants - metabolism | Transcriptional Activation - drug effects | NF-kappa B - metabolism | Lipopolysaccharides | Artemisinins - pharmacology | Cell Nucleus - metabolism | Neuroprotective Agents - pharmacology | Inflammation - drug therapy | Amyloid beta-Peptides - metabolism | Microglia - pathology | NF-E2-Related Factor 2 - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Artemisinins - chemistry | Cell Line | Cell Survival - drug effects | Microglia - drug effects | Prostaglandin-E Synthases - metabolism | Neurotoxins - toxicity | Antimalarials - therapeutic use | Antimalarials - pharmacology | Dinoprostone - metabolism | Amyloid Precursor Protein Secretases - metabolism | Animals | Artemisinins - therapeutic use | MAP Kinase Signaling System - drug effects | Aspartic Acid Endopeptidases - metabolism | NF-E2-Related Factor 2 - metabolism | Antimalarials - chemistry | Cyclooxygenase 2 - metabolism | Interleukin-6 - biosynthesis | NAD(P)H Dehydrogenase (Quinone) - metabolism | Mice | Cell Nucleus - drug effects | Nitric Oxide - metabolism | Tumor Necrosis Factor-alpha - biosynthesis | Nitric Oxide Synthase Type II - metabolism | Nervous system diseases | Neurons | Nitric oxide | Heme | Amyloid beta-protein | Synthetic prostaglandins E | Mitogens | Protein kinases | Proteins | Enzymes | Inflammation | Kinases | Index Medicus
Journal Article