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Nature, ISSN 0028-0836, 03/2013, Volume 495, Issue 7440, pp. 251 - 254
Journal Article
Nature Immunology, ISSN 1529-2908, 07/2015, Volume 16, Issue 8, pp. 850 - 858
The success of antitumor immune responses depends on the infiltration of solid tumors by effector T cells, a process guided by chemokines. Here we show that in... 
CD26 EXPRESSION | CELLS | CXCL10 | CHEMOKINE ACTIVITY | IN-VIVO | RECEPTOR | IMMUNOLOGY | CD26/DIPEPTIDYL PEPTIDASE-IV | PROTEINS | CANCER | POSTTRANSLATIONAL MODIFICATION | Dipeptidyl Peptidase 4 - metabolism | Immunotherapy - methods | Male | Adoptive Transfer | Dipeptidyl-Peptidase IV Inhibitors - pharmacology | Cell Movement - immunology | Flow Cytometry | Lymphocytes - immunology | Neoplasms, Experimental - immunology | Chemokine CXCL10 - immunology | Neoplasms, Experimental - genetics | Female | Sitagliptin Phosphate | Dipeptidyl Peptidase 4 - immunology | Chemokines - immunology | Lymphocytes - metabolism | Receptors, CXCR3 - metabolism | Mice, Inbred C57BL | Neoplasms, Experimental - therapy | Dipeptidyl Peptidase 4 - genetics | Mice, Transgenic | Mice, Knockout | Triazoles - pharmacology | Cell Movement - drug effects | Animals | Receptors, CXCR3 - immunology | Cell Line, Tumor | Chemokines - metabolism | Mice, Inbred BALB C | Pyrazines - pharmacology | Chemokine CXCL10 - metabolism | Care and treatment | Usage | Immunotherapy | Development and progression | Inflammation | Health aspects | Chemokines | Tumors | Index Medicus | Neoplasms, Experimental/genetics | Immunotherapy/methods | Cell Movement/drug effects | Neoplasms, Experimental/therapy | Lymphocytes/metabolism | Lymphocytes/immunology | Dipeptidyl Peptidase 4/genetics | Life Sciences | Chemokine CXCL10/immunology | Immunology | Pyrazines/pharmacology | Dipeptidyl Peptidase 4/immunology | Dipeptidyl-Peptidase IV Inhibitors/pharmacology | Chemokines/metabolism | Receptors, CXCR3/immunology | Receptors, CXCR3/metabolism | Neoplasms, Experimental/immunology | Triazoles/pharmacology | Cell Movement/immunology | Chemokines/immunology | Dipeptidyl Peptidase 4/metabolism | Chemokine CXCL10/metabolism
Journal Article
Journal Article
Journal Article
Diabetes, ISSN 0012-1797, 10/2005, Volume 54, Issue 10, pp. 2988 - 2994
Journal Article
Cell Stem Cell, ISSN 1934-5909, 2010, Volume 6, Issue 6, pp. 603 - 615
Recent evidence suggests that a subpopulation of cancer cells, cancer stem cells (CSCs), is responsible for tumor growth in colorectal cancer. However,... 
STEMCELL | CELLCYCLE | BREAST-CANCER | INITIATING CELLS | DIPEPTIDYL-PEPTIDASE-IV | CARCINOMA CELLS | EXTRACELLULAR-MATRIX | ACUTE MYELOID-LEUKEMIA | E-CADHERIN | IDENTIFICATION | TUMOR-GROWTH | HUMAN PANCREATIC-CANCER | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Neoplasm Transplantation | RNA, Small Interfering - genetics | Prognosis | Follow-Up Studies | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Colorectal Neoplasms - genetics | Humans | Apoptosis - genetics | Gene Expression Profiling | Colorectal Neoplasms - diagnosis | Organoplatinum Compounds - pharmacology | Neoplastic Stem Cells - metabolism | Cell Transformation, Neoplastic - genetics | Colorectal Neoplasms - drug therapy | Liver Neoplasms - physiopathology | Neoplastic Stem Cells - pathology | Tumor Burden - genetics | Tumor Cells, Cultured | Liver Neoplasms - secondary | Colorectal Neoplasms - metabolism | Carcinoma - secondary | Carcinoma - drug therapy | Liver Neoplasms - genetics | Dipeptidyl Peptidase 4 - biosynthesis | Liver Neoplasms - drug therapy | Carcinoma - diagnosis | Dipeptidyl Peptidase 4 - genetics | Mice, SCID | Carcinoma - physiopathology | Disease Progression | Colorectal Neoplasms - physiopathology | Drug Resistance, Neoplasm - genetics | Animals | Liver Neoplasms - diagnosis | Tumor Burden - drug effects | Liver Neoplasms - metabolism | Carcinoma - genetics | Biomarkers, Tumor - genetics | Fluorouracil - pharmacology | Mice | Carcinoma - metabolism | Colorectal Neoplasms - pathology | Neoplasm Invasiveness - genetics | Cell Migration Assays | Biomarkers, Tumor - biosynthesis | Index Medicus
Journal Article
Virology, ISSN 0042-6822, 2016, Volume 499, pp. 375 - 382
Abstract Middle East respiratory syndrome (MERS) coronavirus (MERS-CoV) was first identified in 2012, and it continues to threaten human health worldwide. No... 
Infectious Disease | Spike protein | Receptor-binding domain | hDPP4-transgenic mice | Foldon trimerization motif | MERS-CoV | MERS | Protection | Neutralization | ENVELOPE GLYCOPROTEIN TRIMERS | NEUTRALIZING ANTIBODIES | S PROTEIN | BAT CORONAVIRUS | RESPIRATORY SYNDROME CORONAVIRUS | DROMEDARY CAMELS | VIROLOGY | MUCOSAL IMMUNE-RESPONSES | MONOCLONAL-ANTIBODIES | SAUDI-ARABIA | Coronavirus Infections - prevention & control | Spike Glycoprotein, Coronavirus - genetics | Humans | Dipeptidyl Peptidase 4 - genetics | Mice, Transgenic | Receptors, Virus - genetics | Coronavirus Infections - immunology | Middle East Respiratory Syndrome Coronavirus - chemistry | Middle East Respiratory Syndrome Coronavirus - immunology | Antibodies, Neutralizing - immunology | Animals | Coronavirus Infections - virology | Middle East Respiratory Syndrome Coronavirus - genetics | Protein Binding | Protein Domains | Receptors, Virus - immunology | Antibodies, Viral - immunology | Female | Spike Glycoprotein, Coronavirus - immunology | Mice | Mice, Inbred BALB C | Dipeptidyl Peptidase 4 - immunology | Spike Glycoprotein, Coronavirus - chemistry | Coronavirus Infections - enzymology | Viral antibodies | Medical colleges | Biological weapons | Antibodies | Genetic engineering | Blood banks | Biosecurity | Health aspects | Epidemiology | Protein binding | Index Medicus | neutralization | spike protein | receptor-binding domain | foldon trimerization motif | protection
Journal Article
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 08/2017, Volume 102, Issue 8, pp. 2930 - 2940
Context: Dipeptidyl peptidase IV (DPP4) is overexpressed in thyroid cancer and certain malignancies. Furthermore, DPP4 has been identified as a discriminatory... 
CYTOKINES | TGF-BETA | EPITHELIAL-CELLS | PHARMACOLOGY | SITAGLIPTIN | ENDOCRINOLOGY & METABOLISM | STROMAL CELLS | INHIBITOR | SUPPRESSION | EXPRESSION | CANCER | Immunohistochemistry | Dipeptidyl Peptidase 4 - metabolism | Prognosis | Sitagliptin Phosphate - pharmacology | Humans | Middle Aged | Male | Gene Expression Profiling | Molecular Targeted Therapy | Dipeptidyl-Peptidase IV Inhibitors - pharmacology | Gene Knockdown Techniques | Adult | Female | Tumor Stem Cell Assay | Signal Transduction | Dipeptidyl Peptidase 4 - genetics | Blotting, Western | Thyroid Cancer, Papillary | Xenograft Model Antitumor Assays | Cell Movement - drug effects | Thyroid Neoplasms - genetics | Animals | Carcinoma, Papillary | Proto-Oncogene Proteins B-raf - genetics | Cell Line, Tumor | Carcinoma - genetics | Mice | RNA, Small Interfering | Carcinoma - metabolism | Mutation | In Vitro Techniques | Transforming Growth Factor beta - metabolism | Thyroid Neoplasms - metabolism | CD26 antigen | Peptidase | Target recognition | Papillary thyroid cancer | Leukocyte migration | Transforming growth factor | Pharmacology | Gene expression | Thyroid carcinoma | Signal transduction | Signaling | Thyroid cancer | DNA microarrays | Xenografts | Inhibition | Growth factors | Cell migration | Papillary thyroid carcinoma | Tumors | Cancer | Thyroid | Index Medicus | Abridged Index Medicus
Journal Article