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Nature Immunology, ISSN 1529-2908, 07/2015, Volume 16, Issue 8, pp. 850 - 858
The success of antitumor immune responses depends on the infiltration of solid tumors by effector T cells, a process guided by chemokines. Here we show that in... 
CD26 EXPRESSION | CELLS | CXCL10 | CHEMOKINE ACTIVITY | IN-VIVO | RECEPTOR | IMMUNOLOGY | CD26/DIPEPTIDYL PEPTIDASE-IV | PROTEINS | CANCER | POSTTRANSLATIONAL MODIFICATION | Dipeptidyl Peptidase 4 - metabolism | Immunotherapy - methods | Male | Adoptive Transfer | Dipeptidyl-Peptidase IV Inhibitors - pharmacology | Cell Movement - immunology | Flow Cytometry | Lymphocytes - immunology | Neoplasms, Experimental - immunology | Chemokine CXCL10 - immunology | Neoplasms, Experimental - genetics | Female | Sitagliptin Phosphate | Dipeptidyl Peptidase 4 - immunology | Chemokines - immunology | Lymphocytes - metabolism | Receptors, CXCR3 - metabolism | Mice, Inbred C57BL | Neoplasms, Experimental - therapy | Dipeptidyl Peptidase 4 - genetics | Mice, Transgenic | Mice, Knockout | Triazoles - pharmacology | Cell Movement - drug effects | Animals | Receptors, CXCR3 - immunology | Cell Line, Tumor | Chemokines - metabolism | Mice, Inbred BALB C | Pyrazines - pharmacology | Chemokine CXCL10 - metabolism | Care and treatment | Usage | Immunotherapy | Development and progression | Inflammation | Health aspects | Chemokines | Tumors | Neoplasms, Experimental/genetics | Immunotherapy/methods | Cell Movement/drug effects | Neoplasms, Experimental/therapy | Lymphocytes/metabolism | Lymphocytes/immunology | Dipeptidyl Peptidase 4/genetics | Life Sciences | Chemokine CXCL10/immunology | Immunology | Pyrazines/pharmacology | Dipeptidyl Peptidase 4/immunology | Dipeptidyl-Peptidase IV Inhibitors/pharmacology | Chemokines/metabolism | Receptors, CXCR3/immunology | Receptors, CXCR3/metabolism | Neoplasms, Experimental/immunology | Triazoles/pharmacology | Cell Movement/immunology | Chemokines/immunology | Dipeptidyl Peptidase 4/metabolism | Chemokine CXCL10/metabolism
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 07/2016, Volume 185, Issue 1, pp. 1 - 21
Journal Article
Clinical & Experimental Immunology, ISSN 0009-9104, 06/2016, Volume 184, Issue 3, pp. 265 - 283
Summary Dipeptidyl peptidase (DPP) 4 (CD26, DPP4) is a multi‐functional protein involved in T cell activation by co‐stimulation via its association with... 
DPP4 activity and/or structure homologue proteins (DASH) | CD26 | antigen presentation/processing | co‐stimulation | signal transduction | Signal transduction | Antigen presentation/processing | Co-stimulation | IV ACTIVITY | MOLECULAR CHARACTERIZATION | RHEUMATOID-ARTHRITIS | INSULIN-SECRETION | SUBCELLULAR-LOCALIZATION | ENTEROINSULAR AXIS | IMMUNOLOGY | co-stimulation | IMPROVED GLUCOSE-TOLERANCE | NEUROPEPTIDE-Y NPY | SUBSTRATE-SPECIFICITY | FIBROBLAST-ACTIVATION-PROTEIN | Humans | Neoplasms - diagnosis | Caveolin 1 - immunology | Isoenzymes - immunology | Antigen Presentation | Neoplasms - genetics | Serine Endopeptidases - genetics | Serine Endopeptidases - immunology | Cell Differentiation | T-Lymphocytes - pathology | Dipeptidyl Peptidase 4 - immunology | Chemokines - immunology | Gelatinases - immunology | Signal Transduction | Adenosine Deaminase - genetics | Isoenzymes - genetics | Membrane Proteins - genetics | Caveolin 1 - genetics | Dipeptidyl Peptidase 4 - genetics | Gene Expression Regulation, Neoplastic - immunology | Membrane Proteins - immunology | Inflammation | Chemokines - genetics | Gelatinases - genetics | Neoplasms - immunology | T-Lymphocytes - immunology | Biomarkers, Tumor - genetics | Structural Homology, Protein | Neoplasms - pathology | Biomarkers, Tumor - immunology | Adenosine Deaminase - immunology | Proteins | Rodents | antigen presentation | DPP4 activity and | processing | Review | or structure homologue proteins (DASH)
Journal Article
Nature Immunology, ISSN 1529-2908, 03/2019, Volume 20, Issue 3, pp. 257 - 264
Post-translational modification of chemokines mediated by the dipeptidyl peptidase DPP4 (CD26) has been shown to negatively regulate lymphocyte trafficking,... 
EPITHELIAL-CELLS | ROLES | CHEMOTAXIS | INFLAMMATION | IMMUNOLOGY | AMINO-TERMINAL TRUNCATION | IL-33 | EOTAXIN | EXPRESSION | CD8(+) T | Dipeptidyl Peptidase 4 - metabolism | Eosinophils - metabolism | Sitagliptin Phosphate - pharmacology | Eosinophils - drug effects | Chemokine CCL11 - metabolism | Humans | Mice, Inbred C57BL | Neoplasms, Experimental - prevention & control | Dipeptidyl-Peptidase IV Inhibitors - pharmacology | Eosinophils - immunology | Interleukin-33 - immunology | Mice, Knockout | Cell Movement - immunology | Cell Movement - drug effects | Animals | Neoplasms, Experimental - immunology | Interleukin-33 - metabolism | Cell Line, Tumor | Chemokine CCL11 - immunology | Mice, Inbred BALB C | Neoplasms, Experimental - metabolism | Dipeptidyl Peptidase 4 - immunology | Disease Models, Animal | Post-translational modification | Sitagliptin | T cells | Cancer | Care and treatment | Usage | Enzyme inhibitors | Interleukins | Development and progression | Health aspects | Eosinophils | Tumors | Animal models | CD26 antigen | Leukocyte migration | Trafficking | Hepatocellular carcinoma | Lymphocytes T | Immunity | Cell adhesion & migration | Lymphocytes | Post-translation | Inhibition | Peptidase | Immunoregulation | Leukocytes (eosinophilic) | Breast cancer | Ablation | Depletion | Inhibitors | Immune checkpoint | CXCL10 protein | Degranulation | Solid tumors | Chemokines | Cell migration | Life Sciences | Immunology
Journal Article
Virology, ISSN 0042-6822, 2016, Volume 499, pp. 375 - 382
Abstract Middle East respiratory syndrome (MERS) coronavirus (MERS-CoV) was first identified in 2012, and it continues to threaten human health worldwide. No... 
Infectious Disease | Spike protein | Receptor-binding domain | hDPP4-transgenic mice | Foldon trimerization motif | MERS-CoV | MERS | Protection | Neutralization | ENVELOPE GLYCOPROTEIN TRIMERS | NEUTRALIZING ANTIBODIES | S PROTEIN | BAT CORONAVIRUS | RESPIRATORY SYNDROME CORONAVIRUS | DROMEDARY CAMELS | VIROLOGY | MUCOSAL IMMUNE-RESPONSES | MONOCLONAL-ANTIBODIES | SAUDI-ARABIA | Coronavirus Infections - prevention & control | Spike Glycoprotein, Coronavirus - genetics | Humans | Dipeptidyl Peptidase 4 - genetics | Mice, Transgenic | Receptors, Virus - genetics | Coronavirus Infections - immunology | Middle East Respiratory Syndrome Coronavirus - chemistry | Middle East Respiratory Syndrome Coronavirus - immunology | Antibodies, Neutralizing - immunology | Animals | Coronavirus Infections - virology | Middle East Respiratory Syndrome Coronavirus - genetics | Protein Binding | Protein Domains | Receptors, Virus - immunology | Antibodies, Viral - immunology | Female | Spike Glycoprotein, Coronavirus - immunology | Mice | Mice, Inbred BALB C | Dipeptidyl Peptidase 4 - immunology | Spike Glycoprotein, Coronavirus - chemistry | Coronavirus Infections - enzymology | Viral antibodies | Medical colleges | Biological weapons | Antibodies | Genetic engineering | Blood banks | Biosecurity | Health aspects | Epidemiology | Protein binding | neutralization | spike protein | receptor-binding domain | foldon trimerization motif | protection
Journal Article
Hepatology, ISSN 0270-9139, 08/2014, Volume 60, Issue 2, pp. 487 - 496
The pathogenesis of hepatitis C virus (HCV) infection is strongly influenced by the nature of the host's antiviral immunity. Counterintuitively, elevated serum... 
NATURAL-KILLER-CELLS | INJECTION-DRUG USERS | INTERFERON | VIRUS-INFECTION | NK CELLS | APPARENT RESISTANCE | PROTEIN-10 | REACTIVITY | RIBAVIRIN | GASTROENTEROLOGY & HEPATOLOGY | HCV INFECTION | Dipeptidyl Peptidase 4 - metabolism | Prospective Studies | Follow-Up Studies | Humans | Middle Aged | Hepatitis C, Chronic - virology | Male | Interferon-gamma - metabolism | T-Lymphocytes - virology | Young Adult | T-Lymphocytes - metabolism | Chemokine CXCL10 - immunology | Interleukin-10 - metabolism | Killer Cells, Natural - immunology | Adult | Female | Adaptive Immunity - immunology | Dipeptidyl Peptidase 4 - immunology | Chemokine CXCL10 - blood | Killer Cells, Natural - virology | Immunity, Innate - immunology | Interferon-gamma - immunology | Hepatitis C, Chronic - immunology | T-Lymphocytes - immunology | Killer Cells, Natural - metabolism | Interleukin-10 - immunology | Longitudinal Studies | Hepatitis | Infections | Drug therapy | Hepatology | Chemokines | Immune system | Killer Cells, Natural/immunology | Interferon-gamma/immunology | Chemokine CXCL10/blood | Life Sciences | Chemokine CXCL10/immunology | Immunology | Hepatitis C, Chronic/immunology | Interleukin-10/immunology | T-Lymphocytes/metabolism | Dipeptidyl Peptidase 4/immunology | Hepatitis C, Chronic/virology | Immunity, Innate/immunology | Adaptive Immunity/immunology | T-Lymphocytes/immunology | Interferon-gamma/metabolism | T-Lymphocytes/virology | Killer Cells, Natural/metabolism | Killer Cells, Natural/virology | Dipeptidyl Peptidase 4/metabolism | Interleukin-10/metabolism
Journal Article
Trends in Immunology, ISSN 1471-4906, 2008, Volume 29, Issue 6, pp. 295 - 301
Journal Article