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Journal of the American Society of Nephrology, ISSN 1533-3450, 10/2005, Volume 16, Issue 10, pp. 2941 - 2952
.... For determining the causal relationship between podocyte depletion and glomerulosclerosis, a transgenic rat strain in which the human diphtheria toxin receptor is specifically expressed in podocytes was developed... 
Life Sciences & Biomedicine | Urology & Nephrology | Science & Technology | Diphtheria Toxin - pharmacology | Cell Count | Humans | Intercellular Signaling Peptides and Proteins | Rats, Inbred F344 | Rats | Heparin-binding EGF-like Growth Factor | Animals | Podocytes - drug effects | Podocytes - physiology | Glomerulosclerosis, Focal Segmental - etiology | Receptors, Cell Surface - biosynthesis | Transgenes | Receptors, Cell Surface - genetics | Index Medicus
Journal Article
ChemMedChem, ISSN 1860-7179, 04/2018, Volume 13, Issue 7, pp. 754 - 761
Cytotoxic necrotizing factor 1 (CNF1) is a toxin produced by pathogenic strains of Escherichia coli responsible for extra... 
inhibitors | CNF1 | screening | Rac1 | drug discovery | Pharmacology & Pharmacy | Life Sciences & Biomedicine | Chemistry, Medicinal | Science & Technology | Human Umbilical Vein Endothelial Cells | Immunoassay | Small Molecule Libraries - pharmacology | Humans | Monensin - pharmacology | Escherichia coli Proteins - metabolism | Drug Repositioning | Escherichia coli - chemistry | Lasalocid - pharmacology | Bacterial Toxins - adverse effects | Escherichia coli Proteins - adverse effects | Bacterial Toxins - antagonists & inhibitors | rac1 GTP-Binding Protein - immunology | Amodiaquine - pharmacology | Bacterial Toxins - metabolism | Diphtheria Toxin - adverse effects | Escherichia coli Proteins - antagonists & inhibitors | HeLa Cells | rac1 GTP-Binding Protein - chemistry | rac1 GTP-Binding Protein - metabolism | Bepridil - pharmacology | Drugs | Bacterial infections | Drug discovery | Drug approval | Gastrointestinal diseases | Escherichia coli | Biodegradation | Lasalocid | Monensin | Cytotoxic necrotizing factor | Fluorescence | Cytotoxicity | Rac1 protein | Inflammation | Infections | Diphtheria | Degradation | Inhibitors | E coli | Intestine | Cytotoxic necrotizing factor 1 | Proteasomes | Necrotizing factor 1 | Amodiaquine | Intoxication | Diphtheria toxin | Index Medicus | Bacterial Toxins/adverse effects | rac1 GTP-Binding Protein/immunology | Diphtheria Toxin/adverse effects | Lasalocid/pharmacology | Cellular Biology | Bacterial Toxins/antagonists & inhibitors | Life Sciences | Bepridil/pharmacology | Monensin/pharmacology | Small Molecule Libraries/pharmacology | Bacterial Toxins/metabolism | Subcellular Processes | rac1 GTP-Binding Protein/chemistry | Cell Behavior | Amodiaquine/pharmacology | Escherichia coli Proteins/antagonists & inhibitors | Bacteriology | Microbiology and Parasitology | Escherichia coli Proteins/adverse effects | Escherichia coli Proteins/metabolism | Escherichia coli/chemistry | rac1 GTP-Binding Protein/metabolism
Journal Article
Trends in pharmacological sciences (Regular ed.), ISSN 0165-6147, 08/2020, Volume 41, Issue 8, pp. 511 - 513
AB toxins with built-in cell targeting and endosomal escape mechanisms are attractive intracellular delivery vehicles... 
siRNA delivery | diphtheria toxin | glioblastoma | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Nucleic acids | Therapeutics | Homeopathy | Materia medica and therapeutics | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, pp. e0166477 - e0166477
Journal Article
Journal of immunological methods, ISSN 0022-1759, 09/2017, Volume 448, pp. 51 - 58
Ontak® is a FDA-approved diphtheria toxin-based recombinant fusion toxin for treatment of human CD25... 
Ontak | CD25 | Pichia Pastoris | Diphtheria toxin | CTCL | IL-2 fusion toxin | Biochemistry & Molecular Biology | Immunology | Life Sciences & Biomedicine | Biochemical Research Methods | Science & Technology | Immunoconjugates - toxicity | Diphtheria Toxin - pharmacology | Skin Neoplasms - drug therapy | Recombinant Fusion Proteins - pharmacology | Lymphoma, T-Cell, Cutaneous - immunology | Humans | Diphtheria Toxin - toxicity | Lymphoma, T-Cell, Cutaneous - drug therapy | Antineoplastic Agents - toxicity | Antineoplastic Agents - metabolism | Dose-Response Relationship, Drug | Immunoconjugates - pharmacology | Pichia - metabolism | Time Factors | Antineoplastic Agents - pharmacology | Interleukin-2 Receptor alpha Subunit - immunology | Skin Neoplasms - pathology | Skin Neoplasms - immunology | Immunoconjugates - metabolism | Recombinant Fusion Proteins - toxicity | Mice, SCID | Interleukin-2 - toxicity | Safety-Based Drug Withdrawals | Mice, Knockout | Lymphoma, T-Cell, Cutaneous - pathology | Xenograft Model Antitumor Assays | Interleukin-2 - genetics | Animals | Interleukin-2 Receptor alpha Subunit - metabolism | Tumor Burden - drug effects | Immunoconjugates - genetics | Pichia - genetics | Cell Line, Tumor | Interleukin-2 - pharmacology | Mice, Inbred NOD | Interleukin-2 - biosynthesis | Non-Hodgkin's lymphomas | T cells | Drug approval | Analysis | Index Medicus | diphtheria toxin
Journal Article
Journal Article