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Neuron, ISSN 0896-6273, 2005, Volume 47, Issue 6, pp. 817 - 831
The molecular mechanisms controlling the differentiation of neural progenitors into distinct subtypes of neurons during neocortical development are unknown.... 
AREA IDENTITY | NEOCORTICAL NEURONS | CELL FATE SPECIFICATION | CEREBRAL CORTICAL DEVELOPMENT | LAYER NEURONS | CENTRAL-NERVOUS-SYSTEM | SUBCORTICAL TARGETS | ZINC-FINGER GENE | SUBVENTRICULAR ZONE | NEUROSCIENCES | ZEBRAFISH FOREBRAIN | Membrane Glycoproteins - metabolism | Embryo, Mammalian | Homeodomain Proteins - metabolism | Nerve Tissue Proteins - deficiency | S100 Proteins - genetics | POU Domain Factors - genetics | Forkhead Transcription Factors - metabolism | In Situ Hybridization - methods | Repressor Proteins - metabolism | Gene Expression Regulation, Developmental - physiology | POU Domain Factors - metabolism | Animals, Newborn | DNA-Binding Proteins - physiology | Motor Neurons - physiology | Tumor Suppressor Proteins - metabolism | Membrane Proteins - genetics | Cell Cycle Proteins - metabolism | S100 Proteins - metabolism | In Situ Nick-End Labeling - methods | Apoptosis Regulatory Proteins - metabolism | Mice, Knockout | Immunohistochemistry - methods | Annexin A2 - genetics | Cell Death - physiology | Green Fluorescent Proteins - biosynthesis | Mice | Annexin A2 - metabolism | Age Factors | Phosphopyruvate Hydratase - metabolism | Cell Movement - physiology | DNA-Binding Proteins - deficiency | DNA-Binding Proteins - metabolism | Amino Acids - metabolism | Motor Neurons - cytology | Tumor Suppressor Proteins - genetics | Cell Cycle Proteins - genetics | Membrane Proteins - metabolism | Bromodeoxyuridine - metabolism | Cell Differentiation - physiology | Pyramidal Tracts - physiology | Nerve Tissue Proteins - physiology | Neocortex - growth & development | Nuclear Proteins - metabolism | DNA-Binding Proteins - genetics | Dopamine and cAMP-Regulated Phosphoprotein 32 - metabolism | Nerve Tissue Proteins - genetics | Homeodomain Proteins - genetics | Membrane Glycoproteins - genetics | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Cell Count - methods | Neocortex - cytology | Receptors, Antigen, T-Cell, alpha-beta - metabolism | Neurosciences | Neurons | Developmental biology | Stem cells | Studies | Brain | Ultrasonic imaging | Transcription factors | Hybridization | Neurogenesis | Index Medicus
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 07/2015, Volume 6, Issue 1, pp. 7735 - 7735
Journal Article
Glia, ISSN 0894-1491, 11/2016, Volume 64, Issue 11, pp. 1841 - 1856
Journal Article
GLIA, ISSN 0894-1491, 04/2010, Volume 58, Issue 5, pp. 588 - 598
Neural injury leads to inflammation and activation of microglia that in turn may participate in progression of neurodegeneration. The mechanisms involved in... 
Neurodegenerative diseases | Innate immune system | Heme oxygenase-1 | OXIDATIVE STRESS | ACTIVATION | LEWY BODY DISEASE | heme oxygenase-1 | NEUROSCIENCES | innate immune system | CELL-DEATH | neurodegenerative diseases | TRANSCRIPTION FACTOR NRF2 | BRAIN INFLAMMATION | MOUSE MODEL | THERAPEUTIC TARGET | ENHANCES SUSCEPTIBILITY | Dopamine Plasma Membrane Transport Proteins - metabolism | Tyrosine 3-Monooxygenase - metabolism | Flow Cytometry - methods | Leukocyte Common Antigens - metabolism | Reactive Oxygen Species - metabolism | Culture Media, Conditioned - pharmacology | Male | Glial Fibrillary Acidic Protein - metabolism | Brain - metabolism | MPTP Poisoning - complications | Inflammation - metabolism | Microglia - physiology | MPTP Poisoning - pathology | Antigens, Differentiation - metabolism | Neurons - metabolism | Neurons - drug effects | Dopamine - metabolism | Gliosis - etiology | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Cytokines - metabolism | Neurons - chemistry | Mice, Inbred C57BL | Gene Expression Regulation - physiology | NF-E2-Related Factor 2 - deficiency | Inflammation - etiology | Mice, Knockout | Gene Expression Regulation - drug effects | Animals | Analysis of Variance | Microfilament Proteins | NF-E2-Related Factor 2 - metabolism | Cyclooxygenase 2 - metabolism | Brain - pathology | Mice | CD11b Antigen - metabolism | Cell Line, Transformed | Nitric Oxide Synthase Type II - metabolism | Index Medicus
Journal Article
Molecular Pharmacology, ISSN 0026-895X, 2010, Volume 78, Issue 3, pp. 466 - 477
Parkinson's disease (PD) is the second most common neurodegenerative disease characterized by a progressive loss of dopamine (DA) neurons in the substantia... 
RESPONSES | IN-VITRO | MICROGLIAL ACTIVATION | PHOSPHORYLATION | GENE-EXPRESSION | NITRIC-OXIDE | PHARMACOLOGY & PHARMACY | NECROSIS-FACTOR-ALPHA | NADPH OXIDASE ACTIVITY | PARKINSONS-DISEASE | DEGENERATION | Microglia - metabolism | Mesencephalon - cytology | Reactive Oxygen Species - metabolism | Embryo, Mammalian | Rats, Inbred F344 | NADPH Oxidases - metabolism | Lipopolysaccharides - antagonists & inhibitors | Anti-Inflammatory Agents - metabolism | Nerve Degeneration - metabolism | Neuroprotective Agents - metabolism | Microglia - physiology | Neuroprotective Agents - pharmacology | Stilbenes | Dopamine - physiology | Anti-Inflammatory Agents - therapeutic use | Polyphenols | Neurons - metabolism | Dopamine - metabolism | Phenols - pharmacology | Anti-Inflammatory Agents - pharmacology | NADPH Oxidases - antagonists & inhibitors | Rats | Reactive Oxygen Species - therapeutic use | Lipopolysaccharides - pharmacology | Mitogen-Activated Protein Kinases - pharmacology | Neurotoxicity Syndromes - metabolism | Mice | NADPH Oxidases - physiology | Neuroprotective Agents - therapeutic use | Mesencephalon - metabolism | Reactive Oxygen Species - pharmacology | Lipopolysaccharides - metabolism | Parkinson Disease - drug therapy | Substantia Nigra - metabolism | Flavonoids - therapeutic use | Neuroglia - drug effects | Nerve Degeneration - prevention & control | Neurons - physiology | Female | Flavonoids - pharmacology | Parkinson Disease - metabolism | Neurons - drug effects | Microglia - drug effects | Mice, Inbred C57BL | Dopamine - pharmacology | Phenols - metabolism | Pregnancy | Animals | Flavonoids - metabolism | Phenols - therapeutic use | Neuroglia - metabolism | Mitogen-Activated Protein Kinases - metabolism | Index Medicus
Journal Article
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 09/2003, Volume 112, Issue 6, pp. 892 - 901
Parkinson disease (PD) is a neurodegenerative disorder characterized by a loss of the nigrostriatal dopaminergic neurons accompanied by a deficit in... 
ELECTRON-TRANSPORT CHAIN | RAT-LIVER | MEDICINE, RESEARCH & EXPERIMENTAL | COMPLEX-I | 1-METHYL-4-PHENYL-1,2,3,6-TETRAHYDROPYRIDINE MOUSE MODEL | NITRIC-OXIDE | NEURODEGENERATION | DIETARY RESTRICTION | MPTP | DOPAMINERGIC NEUROTOXICITY | BRAIN
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 12/2016, Volume 311, Issue 6, pp. H1360 - H1366
Anesthesia can affect respiratory, circulatory, and endocrine systems but is necessary for certain experimental procedures such as echocardiography and blood... 
Ketamine-xylazine | Pentobarbital sodium | Isoflurane | pentobarbital sodium | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ketamine-xylazine | MECHANISMS | BODY-TEMPERATURE | COMBINATION | BLOOD-PRESSURE | INSULIN | IMPACT | PERIPHERAL VASCULAR DISEASE | MICE | isoflurane | STRESS | HYPERTENSIVE-RATS | Renin - metabolism | Anesthetics, Dissociative - pharmacology | Isoflurane - pharmacology | Pentobarbital - pharmacology | Male | Fatty Acids, Nonesterified - metabolism | Heart Rate - drug effects | Aldosterone - metabolism | Xylazine - pharmacology | Renin - drug effects | Blood Pressure - drug effects | Dopamine - metabolism | Anesthetics, Inhalation - pharmacology | Angiotensin II - drug effects | Echocardiography | Angiotensin II - metabolism | Cholesterol, HDL - drug effects | Cholesterol, HDL - metabolism | Aorta - drug effects | Insulin Resistance | Rats | Cholesterol, LDL - drug effects | Cholesterol - metabolism | Rats, Sprague-Dawley | Triglycerides - metabolism | Blood Glucose - drug effects | Insulin - metabolism | Animals | Norepinephrine - metabolism | Lipid Metabolism - drug effects | Cholesterol, LDL - metabolism | Glucose - metabolism | Renin-Angiotensin System - drug effects | Hemodynamics - drug effects | Hypnotics and Sedatives - pharmacology | Blood Glucose - metabolism | Ketamine - pharmacology | Epinephrine - metabolism | Lipids | Anesthesia | Ultrasonic imaging | Glucose | Metabolism | Rodents | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2015, Volume 10, Issue 11, pp. e0143852 - e0143852
Understanding the mechanisms in the generation of neural stem cells from pluripotent stem cells is a fundamental step towards successful management of... 
HUMAN ES | RETINOIC ACID | NUCLEAR-FACTOR | MULTIDISCIPLINARY SCIENCES | SWITCH CONTROLS | TRANSCRIPTION | GROWTH | NEURONS | RECEPTOR | PROLIFERATION | DIFFERENTIATION | Tyrosine 3-Monooxygenase - metabolism | Retinoid X Receptor alpha - metabolism | TOR Serine-Threonine Kinases - metabolism | Homeodomain Proteins - metabolism | Humans | tau Proteins - metabolism | Stem Cells - cytology | Stem Cells - metabolism | Dopaminergic Neurons - cytology | Morphogenesis | Cell Differentiation - genetics | Multiprotein Complexes - metabolism | Parkinson Disease - metabolism | Trophoblasts - cytology | Proto-Oncogene Proteins c-akt - metabolism | Calcium Signaling | Calcium-Calmodulin-Dependent Protein Kinase Type 2 - metabolism | Disease Models, Animal | Cell Line | Nuclear Pore Complex Proteins - metabolism | Gene Expression Regulation | Axons - metabolism | Rats | Receptors, Retinoic Acid - metabolism | Parkinson Disease - genetics | Glycogen Synthase Kinase 3 - metabolism | beta Catenin - metabolism | Protein Transport | Transcription Factors - metabolism | Cyclic AMP Response Element-Binding Protein - genetics | Tyrosine 3-Monooxygenase - genetics | Animals | Receptor, trkA - metabolism | Signal Transduction - drug effects | Cell Differentiation - drug effects | Models, Biological | Cyclic AMP Response Element-Binding Protein - metabolism | Protein Binding | Stem Cells - drug effects | GTP-Binding Proteins - metabolism | Stem cell research | Analysis | Influence | Cellular signal transduction | G proteins | Research | Gene expression | Flow cytometry | Parkinson's disease | Substantia nigra | Genes | Stem cell transplantation | Kinases | Tyrosine 3-monooxygenase | Proteins | β-catenin | Signal transduction | Pathways | Rodents | TrkA protein | TrkA receptors | Movement disorders | Dopamine receptors | Tyrosine | Dopamine | Neurodegenerative diseases | Neurons | Axonogenesis | Hydroxylase | Implantation | Progenitor cells | Neurological diseases | Regeneration | Signaling | Brain-derived neurotrophic factor | Microscopy | Stem cells | Regulatory mechanisms (biology) | Cells (biology) | Epigenetics | Neural stem cells | Protein interaction | Differentiation | Retinoic acid | Pluripotency | Index Medicus
Journal Article
Journal Article