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Cell reports (Cambridge), ISSN 2211-1247, 06/2012, Volume 1, Issue 6, pp. 703 - 714
To model human neural-cell-fate specification and to provide cells for regenerative therapies, we have developed a method to generate human neural progenitors and neurons from human embryonic stem... 
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Body Patterning - drug effects | Organ Specificity - drug effects | Embryonic Stem Cells - metabolism | Embryonic Stem Cells - cytology | Humans | Cell Survival - genetics | Motor Activity - drug effects | Neurons - cytology | Neural Stem Cells - cytology | Cell Culture Techniques - methods | Cell Lineage - drug effects | Neural Tube - drug effects | Cell Differentiation - genetics | Organ Specificity - genetics | Dopamine - secretion | Dopaminergic Neurons - metabolism | Telencephalon - drug effects | Dopaminergic Neurons - drug effects | Neural Stem Cells - transplantation | Neurons - metabolism | Neurons - drug effects | Cell Lineage - genetics | Cell Survival - drug effects | Telencephalon - metabolism | Telencephalon - cytology | Glycogen Synthase Kinase 3 - antagonists & inhibitors | Cells, Cultured | Neural Stem Cells - drug effects | Rats | Glycogen Synthase Kinase 3 - metabolism | Aging - pathology | Gene Expression Regulation - drug effects | Phenotype | Animals | Wnt Signaling Pathway - drug effects | Embryonic Stem Cells - drug effects | Wnt Signaling Pathway - genetics | Cell Differentiation - drug effects | Cell Proliferation - drug effects | Protein Kinase Inhibitors - pharmacology | Neural Tube - embryology | Body Patterning - genetics | Neural Stem Cells - metabolism | Electrophysiological Phenomena - drug effects | Index Medicus | Biological Sciences | Biologi | Naturvetenskap | Cellbiologi | Natural Sciences
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 03/2012, Volume 73, Issue 6, pp. 1173 - 1183
Salient but aversive stimuli inhibit the majority of dopamine (DA) neurons in the ventral tegmental area (VTA... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Tyrosine 3-Monooxygenase - metabolism | Action Potentials - genetics | Electroshock - adverse effects | Analgesics, Opioid - pharmacology | Channelrhodopsins | Conditioning, Operant - physiology | GABAergic Neurons - physiology | Ventral Tegmental Area - cytology | Apomorphine - pharmacology | Conditioning, Operant - drug effects | Time Factors | G Protein-Coupled Inwardly-Rectifying Potassium Channels - deficiency | Dopaminergic Neurons - drug effects | Dopaminergic Neurons - physiology | Escape Reaction - drug effects | Optics and Photonics | Haloperidol - pharmacology | Action Potentials - drug effects | Dopamine Agonists - pharmacology | Ventral Tegmental Area - drug effects | Morphine - pharmacology | Mice, Inbred C57BL | Bacterial Proteins - genetics | GABAergic Neurons - drug effects | Glutamate Decarboxylase - genetics | Mice, Transgenic | G Protein-Coupled Inwardly-Rectifying Potassium Channels - genetics | Escape Reaction - physiology | Tyrosine 3-Monooxygenase - genetics | Animals | Analysis of Variance | Dopamine Antagonists - pharmacology | Bacterial Proteins - metabolism | Luminescent Proteins - genetics | Mice | In Vitro Techniques | Luminescent Proteins - metabolism | GABA | Neurons | Lasers | Rodents | Light | Addictive behaviors | Behavior | Charitable foundations | Experiments | Footshock | Ventral tegmentum | Dopamine | gamma -Aminobutyric acid A receptors | Aversion | Information processing | Data processing | Genetics | Optics | Neurotransmission | Index Medicus
Journal Article
Nature neuroscience, ISSN 1097-6256, 03/2013, Volume 16, Issue 3, pp. 300 - 308
Journal Article
The Journal of biological chemistry, ISSN 0021-9258, 12/2014, Volume 289, Issue 50, pp. 34743 - 34767
Journal Article
Cell stem cell, ISSN 1934-5909, 03/2011, Volume 8, Issue 3, pp. 267 - 280
Journal Article
The Journal of neuroscience, ISSN 0270-6474, 07/2016, Volume 36, Issue 29, pp. 7693 - 7706
Journal Article
Journal Article
Brain (London, England : 1878), ISSN 0006-8950, 10/2012, Volume 135, Issue 11, pp. 3355 - 3370
... and improve survival of lesioned central nervous system neurons. In this study, we examined the neuroprotective potential of pharmacological rho kinase inhibition mediated... 
axonal degeneration | Parkinson's disease | rho kinase | 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine | dopaminergic cell death | Neurosciences | Clinical Neurology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Neuroprotective Agents - therapeutic use | Rats, Wistar | Dopaminergic Neurons - pathology | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Male | Corpus Striatum - metabolism | Nerve Degeneration - chemically induced | Neuroprotective Agents - metabolism | rho-Associated Kinases - antagonists & inhibitors | Parkinson Disease, Secondary - pathology | Neuroprotective Agents - pharmacology | Parkinson Disease, Secondary - drug therapy | Dopaminergic Neurons - physiology | Behavior, Animal - drug effects | Parkinson Disease, Secondary - chemically induced | Proto-Oncogene Proteins c-akt - metabolism | Cell Survival - physiology | Dopamine - metabolism | Disease Models, Animal | Substantia Nigra - enzymology | Cell Survival - drug effects | rho-Associated Kinases - physiology | Nerve Degeneration - enzymology | Mice, Inbred C57BL | Cells, Cultured | Axons - drug effects | Rats | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - therapeutic use | Behavior, Animal - physiology | MPTP Poisoning - drug therapy | MPTP Poisoning - enzymology | Parkinson Disease, Secondary - enzymology | Neurites - pathology | Dopaminergic Neurons - enzymology | Substantia Nigra - drug effects | Animals | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Axons - pathology | 1-Methyl-4-phenylpyridinium - toxicity | Mice | Nerve Degeneration - drug therapy | Proto-Oncogene Proteins c-akt - drug effects | Index Medicus | Abridged Index Medicus | Cell culture | Neuroprotection | Animal models | Cell survival | Dopamine | Neurodegenerative diseases | Substantia nigra | Central nervous system | AKT protein | Motor task performance | Rho-associated kinase | Axons | Regeneration | Metabolites | Neurodegeneration | Neostriatum | Axon guidance | Movement disorders | Original | Parkinson’s disease
Journal Article