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Journal Article
Annals of Neurology, ISSN 0364-5134, 03/2014, Volume 75, Issue 3, pp. 351 - 362
Journal Article
Cell Stem Cell, ISSN 1934-5909, 2011, Volume 8, Issue 3, pp. 267 - 280
Studies of Parkinson's disease (PD) have been hindered by lack of access to affected human dopaminergic (DA) neurons. Here, we report generation of induced... 
PLURIPOTENT STEM-CELLS | KINASE-ACTIVITY | ALZHEIMERS-DISEASE | ANIMAL-MODELS | HUMAN FIBROBLASTS | ALPHA-SYNUCLEIN | LEWY-BODY-DISEASE | SUBSTANTIA-NIGRA | MIDBRAIN DOPAMINERGIC-NEURONS | PARKINSONS-DISEASE | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Neurons - pathology | Humans | Middle Aged | rho-Associated Kinases - antagonists & inhibitors | Amino Acid Substitution - drug effects | Oxidopamine - pharmacology | rho-Associated Kinases - metabolism | Female | Cell Death - drug effects | Neurons - drug effects | Dopamine - metabolism | Protein-Serine-Threonine Kinases - metabolism | Induced Pluripotent Stem Cells - metabolism | Mesencephalon - pathology | Amides - pharmacology | Induced Pluripotent Stem Cells - pathology | Parkinson Disease - pathology | Induced Pluripotent Stem Cells - drug effects | Hydrogen Peroxide - pharmacology | Protein-Serine-Threonine Kinases - genetics | Parkinson Disease - genetics | Mutation - genetics | Leucine-Rich Repeat Serine-Threonine Protein Kinase-2 | Leupeptins - pharmacology | Phenotype | Animals | Cell Differentiation - drug effects | Parkinson Disease - enzymology | Mice | Pyridines - pharmacology | Oxidative Stress - drug effects | Index Medicus | α-synuclein(SNCA) | 6-hydroxydopamine | p.G2019S | hydrogen peroxide | Leucine Rich Repeat Kinase 2 (LRRK2) | human embryonic stem cells (hESCs) | induced pluripotent stem cells (iPSCs) | midbrain dopaminergic neurons (mDA) | Parkinson’s disease | MG-132
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2012, Volume 109, Issue 9, pp. 3213 - 3219
We used in vivo amperometry to monitor changes in synaptic dopamine (DA) release in the striatum induced by overexpression of human wild-type α-synuclein in... 
Electrodes | Axons | Nervous system diseases | Neurons | Innervation | Substantia nigra | Reuptake | Parkinson disease | Mathematical vectors | T tests | Neurodegeneration | Synaptic transmission | PATHOGENESIS | RAT SUBSTANTIA-NIGRA | TRANSPORTER | MULTIDISCIPLINARY SCIENCES | neurodegeneration | synaptic transmission | RELEASE | INHIBITORS | NIGROSTRIATAL SYSTEM | MICE LACKING | MODULATION | PARKINSONS-DISEASE | Dependovirus - genetics | Up-Regulation | Synaptic Transmission - physiology | Putamen - pathology | Dopaminergic Neurons - pathology | Humans | Caudate Nucleus - pathology | Substantia Nigra - cytology | Dose-Response Relationship, Drug | Dopamine - secretion | alpha-Synuclein - biosynthesis | Axons - ultrastructure | Female | alpha-Synuclein - genetics | Transgenes | Dopaminergic Neurons - secretion | Nomifensine - pharmacology | Dopamine Uptake Inhibitors - pharmacology | Microinjections | alpha-Synuclein - toxicity | Rats | Recombinant Fusion Proteins - toxicity | Electrodes, Implanted | Potassium - pharmacology | Rats, Sprague-Dawley | Genetic Vectors - genetics | Animals | Genetic Vectors - toxicity | Dopamine | Parkinson's disease | Neurodegenerative diseases | potassium chloride | Synuclein | amperometry | Neostriatum | Dopamine transporter | Degeneration | Movement disorders | Neurotransmission | Index Medicus | Biological Sciences | Basic Medicine | Neurosciences | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Neurovetenskaper
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 01/2009, Volume 119, Issue 1, pp. 182 - 192
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2006, Volume 103, Issue 28, pp. 10793 - 10798
Journal Article
Journal of Neuroscience, ISSN 0270-6474, 07/2012, Volume 32, Issue 27, pp. 9248 - 9264
Journal Article
Nature, ISSN 0028-0836, 06/2007, Volume 447, Issue 7148, pp. 1081 - 1086
Why dopamine-containing neurons of the brain's substantia nigra pars compacta die in Parkinson's disease has been an enduring mystery. Our studies suggest that... 
PATHOGENESIS | SUBSTANTIA-NIGRA NEURONS | STRIATAL LEVEL | CHOLINERGIC INTERNEURONS | MULTIDISCIPLINARY SCIENCES | CA2+ CHANNELS | CALCIUM-ANTAGONISTS | MECHANISMS | MITOCHONDRIAL-DNA DELETIONS | MEDIUM SPINY NEURONS | MIDBRAIN DOPAMINERGIC-NEURONS | Neurons - pathology | Antiparkinson Agents - pharmacology | Substantia Nigra - pathology | Calcium - metabolism | Male | Neurons - cytology | Electric Conductivity | Parkinson Disease - drug therapy | Substantia Nigra - metabolism | Substantia Nigra - cytology | Gene Deletion | Rotenone - pharmacology | Aging | Neurons - metabolism | Parkinson Disease - metabolism | Neurons - drug effects | Dopamine - metabolism | Disease Models, Animal | 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine - pharmacology | Dendrites - metabolism | Parkinson Disease - pathology | Calcium - pharmacology | Mice, Inbred C57BL | Mitochondria - drug effects | Disease Progression | Parkinson Disease - prevention & control | Animals | Calcium Channels, L-Type - genetics | Calcium Channels, L-Type - deficiency | Mice | Calcium Channels, L-Type - metabolism | Models, Neurological | Parkinson's disease | Brain stem | Rejuvenation | Physiological aspects | Development and progression | Research | Dopaminergic mechanisms | Medical research | Models | Brain research | Neurons | Rodents | Parkinsons disease | Biomedical materials | Progressions | In vivo testing | In vivo tests | Parkinson disease | Strategy | Channels | Index Medicus
Journal Article