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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 11/2013, Volume 305, Issue 9, pp. H1344 - H1353
Ca+ mishandling due to impaired activity of cardiac sarco(endo) plasmic reticulum Ca2+ ATPase (SERCA2a) has been associated with the development of left... 
Heart | Oxidative stress | Calcium | Metabolic syndrome | Sarco(endo)plasmic reticulum Ca | ATPase 2a | SUCROSE-FED RATS | CARDIAC CONTRACTILE DYSFUNCTION | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | calcium | HEART-FAILURE | HYPERTRIGLYCERIDEMIC RATS | ANTIOXIDANT ENZYMES | heart | metabolic syndrome | VENTRICULAR DIASTOLIC DYSFUNCTION | SARCOPLASMIC-RETICULUM CA2 | sarco(endo) plasmic reticulum Ca2+ ATPase 2a | INSULIN-RESISTANCE | RYANODINE RECEPTORS | PERIPHERAL VASCULAR DISEASE | TYPE-2 DIABETES-MELLITUS | oxidative stress | Phosphorylation | Reactive Oxygen Species - metabolism | Rats, Wistar | Calcium - metabolism | Male | Metabolic Syndrome - chemically induced | Myocytes, Cardiac - enzymology | Metabolic Syndrome - blood | Hyperinsulinism - enzymology | Ventricular Dysfunction, Left - enzymology | Dietary Sucrose | Hyperinsulinism - blood | Calcium Signaling | Disease Models, Animal | Calcium-Binding Proteins - metabolism | Metabolic Syndrome - enzymology | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Oxidation-Reduction | Down-Regulation | Insulin Resistance | Rats | Ventricular Dysfunction, Left - etiology | Antioxidants - pharmacology | Obesity, Abdominal - enzymology | Hypertriglyceridemia - blood | Hypertriglyceridemia - enzymology | Obesity, Abdominal - blood | Animals | Myocytes, Cardiac - drug effects | Oxidative Stress - drug effects | Heart cells | Physiological aspects | Insulin resistance | Metabolic diseases | Research | Health aspects | Pathology | Cardiovascular disease | Insulin
Journal Article
Journal Article
Pharmacology and Therapeutics, ISSN 0163-7258, 07/2015, Volume 151, pp. 87 - 98
Journal Article
Circulation, ISSN 0009-7322, 07/2016, Volume 134, Issue 1, pp. 61 - 72
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 7/2008, Volume 105, Issue 27, pp. 9415 - 9420
Journal Article
Physiology & Behavior, ISSN 0031-9384, 2017, Volume 177, pp. 230 - 241
Abstract Down syndrome (DS) is caused by three copies of human chromosome 21 (Hsa21) and results in phenotypes including intellectual disability and skeletal... 
Psychiatry | Trisomy 21 | Mouse model | Cognition | Down syndrome | Bone | EGCG | YOUNG-ADULTS | DEFECTS | DYRK1A | COGNITIVE IMPAIRMENT | PROTEIN-KINASES | OVEREXPRESSION | CONCENTRIC SQUARE FIELD | BEHAVIORAL SCIENCES | MICE | PSYCHOLOGY, BIOLOGICAL | BRAIN | Motor Activity - physiology | Protein-Tyrosine Kinases - metabolism | Brain - enzymology | Motor Activity - drug effects | Male | Femur - diagnostic imaging | Recognition (Psychology) - physiology | Protease Inhibitors - pharmacology | Protein-Tyrosine Kinases - genetics | Femur - enzymology | Treatment Failure | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Cognition - physiology | Catechin - pharmacology | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Down Syndrome - pathology | Maze Learning - physiology | Administration, Oral | Protein-Serine-Threonine Kinases - genetics | Femur - drug effects | Mice, Transgenic | Down Syndrome - enzymology | Random Allocation | Cognition - drug effects | Maze Learning - drug effects | Mice, Inbred C3H | Brain - drug effects | Phenotype | Animals | Recognition (Psychology) - drug effects | Down Syndrome - drug therapy | Catechin - analogs & derivatives | Down Syndrome - psychology | Protein-Tyrosine Kinases - antagonists & inhibitors | Catechin | Genetic aspects | Green tea | Analysis | bone | mouse model | cognition
Journal Article
Journal Article
PLoS genetics, ISSN 1553-7390, 2010, Volume 6, Issue 12, pp. e1001240 - 17
Fragile X Tremor Ataxia Syndrome (FXTAS) is a common inherited neurodegenerative disorder caused by expansion of a CGG trinucleotide repeat in the 5'UTR of the... 
PREMUTATION CARRIERS | MEDIATED NEURODEGENERATION | FULL MUTATION | DROSOPHILA MODEL | MOUSE MODEL | PROTEIN EXPRESSION | GENETICS & HEREDITY | PERIPHERAL-BLOOD | INTRANUCLEAR INCLUSIONS | FMR1 MESSENGER-RNA | CEREBELLAR-TREMOR/ATAXIA-SYNDROME | Trinucleotide Repeats | Eye - pathology | Humans | Middle Aged | Eye - enzymology | Fragile X Syndrome - pathology | Male | Fragile X Mental Retardation Protein - metabolism | Drosophila Proteins - metabolism | Drosophila melanogaster - genetics | Fragile X Syndrome - drug therapy | Fragile X Syndrome - enzymology | Aged, 80 and over | Eye - innervation | Adult | Acetylation | Disease Models, Animal | Fragile X Syndrome - genetics | Histone Deacetylases - genetics | Histone Deacetylase 6 | Down-Regulation | Enzyme Inhibitors - pharmacology | Gene Silencing | Histone Deacetylases - metabolism | Animals | Histone Acetyltransferases - antagonists & inhibitors | Drosophila melanogaster - enzymology | Fragile X Mental Retardation Protein - genetics | Drosophila Proteins - genetics | Histones - metabolism | Messenger RNA | Gene mutations | Physiological aspects | Genetic aspects | Research | Fragile X syndrome | Health aspects | Risk factors | Research & development | R&D | Chromatin | Insects | Neurodegeneration | Pathogenesis | Ataxia | Mutation | Experiments | Drug dosages | Patients | Molecular weight
Journal Article
Journal Article
BBA - Molecular Basis of Disease, ISSN 0925-4439, 07/2014, Volume 1842, Issue 7, pp. 1144 - 1153
Journal Article