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The Journal of pharmacology and experimental therapeutics, ISSN 0022-3565, 12/2010, Volume 335, Issue 3, pp. 533 - 545
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 10/2013, Volume 110, Issue 43, pp. 17253 - 17258
Phenotypic polarization of macrophages is regulated by a milieu of cues in the local tissue microenvironment. Although much is known about how soluble factors... 
Phenotypes | Cell growth | Cytokines | Stem cells | Cell lines | Actins | Physiological regulation | Macrophages | Endothelial cells | Cells | Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Mannose-Binding Lectins - metabolism | Arginase - metabolism | Lectins, C-Type - immunology | Cell Shape - immunology | Myosin-Light-Chain Kinase - antagonists & inhibitors | Cytochalasin D - pharmacology | Lectins, C-Type - metabolism | Arginase - immunology | Interleukin-4 - pharmacology | Flow Cytometry | Doxorubicin - analogs & derivatives | Inflammation Mediators - metabolism | Female | Immunophenotyping - methods | Cell Polarity - drug effects | Doxorubicin - metabolism | Macrophages - immunology | Cytokines - immunology | Amides - pharmacology | Biomarkers - metabolism | Inflammation Mediators - immunology | Cell Polarity - immunology | Cytokines - metabolism | Myosin-Light-Chain Kinase - metabolism | Mice, Inbred C57BL | Biomarkers - analysis | Cells, Cultured | Receptors, Cell Surface - metabolism | Macrophages - cytology | Interleukin-13 - pharmacology | Receptors, Cell Surface - immunology | Azepines - pharmacology | Cell Shape - drug effects | Macrophages - metabolism | Animals | Mannose-Binding Lectins - immunology | Lipopolysaccharides - pharmacology | Mice | Pyridines - pharmacology | Doxorubicin - immunology | Interferon-gamma - pharmacology | Microscopy, Fluorescence | Physiological aspects | Microbial genetics | Phenotype | Genetic aspects | Research | Index Medicus | Biological Sciences | Physical Sciences
Journal Article
Nature (London), ISSN 1476-4687, 02/2011, Volume 470, Issue 7334, pp. 359 - 365
...) or telomerase RNA component (Terc) genes. Consistent with PGCs as master regulators of mitochondrial physiology and metabolism, telomere dysfunction is associated with impaired mitochondrial... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Cell Proliferation | Reactive Oxygen Species - metabolism | Hematopoietic Stem Cells - pathology | DNA, Mitochondrial - analysis | Adenosine Triphosphate - biosynthesis | Tumor Suppressor Protein p53 - genetics | RNA - genetics | Telomerase - deficiency | Cardiomyopathies - physiopathology | Telomerase - genetics | Myocardium - metabolism | Telomere - metabolism | Gluconeogenesis | Telomere - genetics | Liver - metabolism | Tumor Suppressor Protein p53 - metabolism | Cardiomyopathies - pathology | Transcription Factors - antagonists & inhibitors | Hematopoietic Stem Cells - metabolism | Mitochondria - metabolism | Mitochondria - pathology | Tumor Suppressor Protein p53 - deficiency | Doxorubicin - toxicity | Aging - pathology | Myocardium - cytology | Transcription Factors - metabolism | Telomere - pathology | Animals | Cardiomyopathies - metabolism | Telomere - enzymology | Liver - cytology | Mice | Aging - metabolism | Cardiomyopathies - chemically induced | Telomeres | Mitochondria | Genetic aspects | Chemical properties | Abnormalities | Liver | Cell division | Mitochondrial DNA | Biosynthesis | Biology | DNA repair | Proteins | Studies | Aging | Telomerase | Binding sites | Apoptosis | Index Medicus
Journal Article
Journal of controlled release, ISSN 0168-3659, 09/2016, Volume 238, pp. 139 - 148
Therapeutic nanoparticles (NPs) approved for clinical use in solid tumor therapy provide only modest improvements in patient survival, in part due to... 
Nanoparticles | Intravital microscopy | Multiple particle tracking (MPT) | PEG density | Surface plasmon resonance (SPR) | Tumor tissue penetration | Pharmacology & Pharmacy | Physical Sciences | Chemistry | Life Sciences & Biomedicine | Chemistry, Multidisciplinary | Science & Technology | Neoplasms - metabolism | Nanoparticles - analysis | Humans | Polyethylene Glycols - analysis | Drug Carriers - analysis | Albumin-Bound Paclitaxel - administration & dosage | Antineoplastic Agents - administration & dosage | Polyglycolic Acid - analysis | Breast - metabolism | Breast Neoplasms - metabolism | Antineoplastic Agents - metabolism | Nanoparticles - metabolism | Surface Properties | Doxorubicin - analogs & derivatives | Female | Doxorubicin - metabolism | Polyglycolic Acid - metabolism | Diffusion | Lactic Acid - analysis | Doxorubicin - administration & dosage | Albumin-Bound Paclitaxel - metabolism | Polyethylene Glycols - metabolism | Drug Carriers - metabolism | Lactic Acid - metabolism | Proteoglycans - metabolism | Breast Neoplasms - drug therapy | Polyethylene Glycols - administration & dosage | Neoplasms - drug therapy | Particle Size | Collagen - metabolism | Animals | Breast - drug effects | Mice, Nude | Cell Line, Tumor | Mice | Laminin - metabolism | Drug Combinations | Drugs | Ethylene glycol | Drug delivery systems | Drug therapy | Drug approval | Tumors | Vehicles | Medical colleges | Cancer | Index Medicus | multiple particle tracking (MPT) | tumor tissue penetration | surface plasmon resonance (SPR) | nanoparticles | intravital microscopy
Journal Article
Cardiovascular Research, ISSN 0008-6363, 3/2010, Volume 85, Issue 4, pp. 773 - 784
Journal Article
Cardiovascular research, ISSN 1755-3245, 12/2012, Volume 96, Issue 3, pp. 456 - 465
Active autophagy has recently been reported in doxorubicin-induced cardiotoxicity; here we investigated its pathophysiological role. Acute cardiotoxicity was... 
Heart failure | Autophagy | Doxorubicin | Cardiac & Cardiovascular Systems | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | AMP-Activated Protein Kinases - metabolism | Microtubule-Associated Proteins - genetics | Ventricular Function, Left | Microtubule-Associated Proteins - metabolism | Heart Failure - physiopathology | Green Fluorescent Proteins - genetics | Ventricular Dysfunction, Left - chemically induced | Autophagy - drug effects | Autophagy-Related Protein-1 Homolog | Starvation - metabolism | Heart Failure - prevention & control | Hypertrophy, Left Ventricular - chemically induced | Time Factors | Hypertrophy, Left Ventricular - pathology | Adenosine Triphosphate - metabolism | Ventricular Dysfunction, Left - pathology | Protein-Serine-Threonine Kinases - metabolism | Green Fluorescent Proteins - metabolism | Hypertrophy, Left Ventricular - metabolism | Hypertrophy, Left Ventricular - prevention & control | Cells, Cultured | Rats | Mice, Transgenic | Heart Failure - metabolism | Antibiotics, Antineoplastic | Heart Failure - pathology | Cathepsin D - metabolism | Ventricular Dysfunction, Left - physiopathology | Ventricular Dysfunction, Left - prevention & control | Stroke Volume | Ventricular Dysfunction, Left - metabolism | Myocytes, Cardiac - pathology | Animals | Energy Metabolism | Myocytes, Cardiac - metabolism | Mice | Ventricular Pressure | Hypertrophy, Left Ventricular - physiopathology | Heart Failure - chemically induced | Starvation - complications | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 05/2009, Volume 296, Issue 5, pp. 1466 - 1483
Doxorubicin (DOX) is a potent available antitumor agent; however, its clinical use is limited because of its cardiotoxicity. Cell death is a key component in... 
Heart failure | Hemodynamics | Inducible nitric oxide synthase | Apoptosis | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Free Radical Scavengers - pharmacology | Antibiotics, Antineoplastic - toxicity | Mitochondria, Heart - metabolism | Heart Diseases - metabolism | Apoptosis - drug effects | Mitochondria, Heart - pathology | Antioxidants - metabolism | Myocardial Contraction - drug effects | Heart Diseases - prevention & control | Male | Mitochondria, Heart - drug effects | Tyrosine - analogs & derivatives | Necrosis | Dose-Response Relationship, Drug | Matrix Metalloproteinase 9 - metabolism | Nitric Oxide Synthase Type II - antagonists & inhibitors | Superoxides - metabolism | Nitric Oxide Donors - pharmacology | Heart Diseases - chemically induced | Cell Line | Heart Diseases - physiopathology | Matrix Metalloproteinase 2 - metabolism | Mice, Inbred C57BL | Nitric Oxide Synthase Type II - deficiency | Ventricular Function, Left - drug effects | Enzyme Inhibitors - pharmacology | Ventricular Pressure - drug effects | Doxorubicin - toxicity | Mice, Knockout | Myocytes, Cardiac - pathology | Poly(ADP-ribose) Polymerases - metabolism | Tyrosine - metabolism | Animals | Myocytes, Cardiac - drug effects | Nitric Oxide Synthase Type II - genetics | Peroxynitrous Acid - metabolism | Myocytes, Cardiac - metabolism | Mice | Nitric Oxide - metabolism | Heart Diseases - pathology | Index Medicus | heart failure | apoptosis | hemodynamics | inducible nitric oxide synthase
Journal Article
Cell (Cambridge), ISSN 0092-8674, 09/2007, Volume 130, Issue 6, pp. 1120 - 1133
Coordination of cell proliferation and cell death is essential to attain proper organ size during development and for maintaining tissue homeostasis throughout... 
DEVBIO | SIGNALING | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Protein Kinases - metabolism | Phosphorylation | Cell Proliferation | Large Neutral Amino Acid-Transporter 1 - metabolism | Humans | Liver Neoplasms, Experimental - chemically induced | Homeostasis | Cytoplasm - metabolism | Drosophila Proteins - metabolism | Doxorubicin | Drosophila - enzymology | Liver Neoplasms, Experimental - metabolism | Cell Nucleus - metabolism | Transfection | Trans-Activators - genetics | Active Transport, Cell Nucleus | Liver Neoplasms, Experimental - pathology | Nuclear Proteins - genetics | Protein-Serine-Threonine Kinases - metabolism | Drosophila - genetics | Cell Line | Mammals - metabolism | Mammals - growth & development | Signal Transduction | Mice, Inbred C57BL | Cell Cycle Proteins - metabolism | Protein-Serine-Threonine Kinases - genetics | Intracellular Signaling Peptides and Proteins | Organ Size | Drosophila - cytology | Mice, Transgenic | Nuclear Proteins - metabolism | Transcription Factors - genetics | Serine - metabolism | Transcription Factors - metabolism | Animals | Liver Neoplasms, Experimental - enzymology | Trans-Activators - metabolism | Drosophila - growth & development | Mice | Drosophila - metabolism | Drosophila Proteins - genetics | Mutation | Adaptor Proteins, Signal Transducing - metabolism | Apoptosis | Drosophila | Knowledge-based systems | Index Medicus
Journal Article