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Epilepsia, ISSN 0013-9580, 05/2007, Volume 48, Issue 5, pp. 1015 - 1018
Journal Article
Epilepsy Research, ISSN 0920-1211, 2011, Volume 97, Issue 1, pp. 190 - 197
Summary Background Although the US FDA recommends screening for HLA-B*1502 allele in most of Asian ancestry before initiating carbamazepine therapy, the HLA... 
Neurology | Hypersensitivity syndrome (HSS) | Human leukocyte antigen (HLA) | Toxic epidermal necrolysis (TEN) | Carbamazepine | Stevens–Johnson syndrome (SJS) | Stevens-Johnson syndrome (SJS) | B LOCUS | TOXIC EPIDERMAL NECROLYSIS | CLINICAL NEUROLOGY | RISK-FACTOR | CLASS-I | HYPERSENSITIVITY REACTIONS | DIVERSITY | HLA-B-ASTERISK-1502 ALLELE | JAPANESE PATIENTS | STEVENS-JOHNSON-SYNDROME | ASSOCIATION | Predictive Value of Tests | Humans | Middle Aged | Asian Continental Ancestry Group - genetics | Male | Drug Eruptions - ethnology | Young Adult | Stevens-Johnson Syndrome - chemically induced | Anticonvulsants - adverse effects | Epilepsy - ethnology | Stevens-Johnson Syndrome - ethnology | Adult | Epilepsy - genetics | Female | Drug Eruptions - genetics | Republic of Korea | Pharmacogenetics - statistics & numerical data | Genetic Predisposition to Disease - genetics | Histocompatibility Testing | Gene Frequency | Genotype | Histocompatibility Antigens Class I - genetics | Stevens-Johnson Syndrome - genetics | Carbamazepine - adverse effects | Epilepsy - drug therapy | Genetic Predisposition to Disease - ethnology | Aged | Asian Continental Ancestry Group - statistics & numerical data | Antigen-antibody reactions | Complications and side effects | Allergy | Medical colleges | Histocompatibility antigens | HLA histocompatibility antigens | Genetic aspects | Koreans | Allergic reaction
Journal Article
European Journal of Immunology, ISSN 0014-2980, 10/2015, Volume 45, Issue 10, pp. 2847 - 2857
The pathogenesis of inflammatory skin diseases such as psoriasis involves the release of numerous proinflammatory cytokines, including members of the IL‐1... 
IL‐1R | Skin inflammation | NLRP3 inflammasome | Imiquimod | MyD88 | IL-1R | INTERLEUKIN-1 | RECEPTOR | KERATINOCYTES | CYTOKINE | IMMUNOLOGY | IL-22 | DISEASE | DEFICIENT | ABSENCE | EXPRESSION | PSORIASIS | Adjuvants, Immunologic - pharmacology | Receptors, Interleukin-1 Type I - immunology | Drug Eruptions - pathology | NLR Family, Pyrin Domain-Containing 3 Protein | Adjuvants, Immunologic - adverse effects | Signal Transduction - immunology | Toll-Like Receptor 7 - immunology | Myeloid Differentiation Factor 88 - immunology | Membrane Glycoproteins - immunology | Aminoquinolines - pharmacology | Cytokines - genetics | Drug Eruptions - genetics | Carrier Proteins - immunology | Drug Eruptions - immunology | Skin - pathology | Cytokines - immunology | Skin - immunology | Toll-Like Receptor 7 - genetics | Myeloid Differentiation Factor 88 - genetics | Signal Transduction - genetics | Inflammasomes - genetics | Membrane Glycoproteins - genetics | Mice, Knockout | Carrier Proteins - genetics | Animals | Signal Transduction - drug effects | Inflammasomes - immunology | Receptors, Interleukin-1 Type I - genetics | Mice | Aminoquinolines - adverse effects | Skin | Inflammation | Psoriasis | RNA | Dermatitis | Rodents | Life Sciences | Human health and pathology | Immunology | Dermatology | Biochemistry, Molecular Biology | Innate immunity | Biomolecules | Adaptive immunology
Journal Article
Molecules, ISSN 1420-3049, 2018, Volume 23, Issue 6, p. 1477
Journal Article
Journal of Immunology, ISSN 0022-1767, 10/2014, Volume 193, Issue 7, pp. 3693 - 3703
IFN-gamma mediates chemically induced skin inflammation; however, the mechanism by which IFN-gamma-producing cells are recruited to the sites of inflammation... 
RESPONSES | ACTIVATION | DERMATITIS | DISEASE | IMMUNOPATHOGENESIS | KILLER T-CELLS | FACTOR MIF | MICE | PSORIATIC SKIN | IMMUNOLOGY | CHEMOKINE RECEPTORS | Inflammation - chemically induced | Inflammation - pathology | Intramolecular Oxidoreductases - immunology | Tetradecanoylphorbol Acetate - adverse effects | Tetradecanoylphorbol Acetate - pharmacology | Drug Eruptions - pathology | Humans | Macrophage Migration-Inhibitory Factors - immunology | Neovascularization, Pathologic - pathology | Carcinogens - pharmacology | Macrophage Migration-Inhibitory Factors - genetics | Natural Killer T-Cells - pathology | Antigens, Differentiation, B-Lymphocyte - immunology | Neovascularization, Pathologic - immunology | Interferon-gamma - genetics | Drug Eruptions - genetics | Drug Eruptions - immunology | Disease Models, Animal | Antigens, Differentiation, B-Lymphocyte - genetics | Cell Line | Drug Eruptions - etiology | Acute Disease | Epidermis - pathology | Inflammation - immunology | Receptors, Interleukin-8B - immunology | Neovascularization, Pathologic - chemically induced | Keratinocytes - immunology | Keratinocytes - pathology | Animals | Histocompatibility Antigens Class II - immunology | Epidermis - immunology | Intramolecular Oxidoreductases - genetics | Interferon-gamma - immunology | Receptors, Interleukin-8B - genetics | Inflammation - genetics | Neovascularization, Pathologic - genetics | Mice | Histocompatibility Antigens Class II - genetics | Chronic Disease | Natural Killer T-Cells - immunology
Journal Article
British Journal of Dermatology, ISSN 0007-0963, 07/2012, Volume 167, Issue 1, pp. 44 - 49
Journal Article
JAMA Dermatology, ISSN 2168-6068, 04/2018, Volume 154, Issue 4, pp. 441 - 446
IMPORTANCE: Dapsone-induced hypersensitivity syndrome (DHS) is a life-threatening adverse drug reaction. Based on available epidemiologic studies, HLA... 
ALLELE | RISK | LEPROSY PATIENTS | HYPERSENSITIVITY SYNDROME | HLA | DERMATOLOGY | Dapsone - adverse effects | Anti-Infective Agents - adverse effects | Humans | Drug Hypersensitivity Syndrome - genetics | Genotype | Drug Eruptions - epidemiology | Drug Eruptions - genetics | HLA-B13 Antigen - genetics | Online First | Original Investigation | Research
Journal Article
Clinical Pharmacology & Therapeutics, ISSN 0009-9236, 12/2012, Volume 92, Issue 6, pp. 757 - 765