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PloS one, ISSN 1932-6203, 08/2015, Volume 10, Issue 8, pp. e0134984 - e0134984
Protein tyrosine phosphatases dephosphorylate tyrosine residues of proteins, whereas, dual specificity phosphatases (DUSPs... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Dual Specificity Phosphatase 1 - genetics | Signal Transduction | Dual-Specificity Phosphatases - metabolism | Humans | Phosphoprotein Phosphatases - metabolism | Substrate Specificity | Dual Specificity Phosphatase 3 - genetics | cdc25 Phosphatases - genetics | Phylogeny | Recombinant Proteins | Mitogen-Activated Protein Kinase Phosphatases - genetics | Amino Acid Motifs | Phosphotyrosine - metabolism | Phosphoprotein Phosphatases - genetics | Dual-Specificity Phosphatases - genetics | Protein Array Analysis | Dual Specificity Phosphatase 1 - metabolism | cdc25 Phosphatases - metabolism | Dual Specificity Phosphatase 3 - metabolism | Mitogen-Activated Protein Kinase Phosphatases - metabolism | Physiological aspects | Phosphorylation | Genetic aspects | Phosphatases | Research | Cluster analysis | Residues | Peptides | Laboratories | Phosphotyrosine | Biochemistry | Catalytic activity | Dephosphorylation | Structure-activity relationships | Kinases | Phosphatase | Crystallography | Subgroups | Proteins | Signal transduction | Cell growth | Pathways | Substrate specificity | Armed forces | Catalysis | Localization | Pharmaceutical sciences | Tyrosine | Smallpox | Medical research | Enzymes | Cell division | Clustering | Substrates | Signaling | Chemistry | Infectious diseases | Proteomes | Alzheimers disease | Recognition | Cancer | Index Medicus
Journal Article
Cancer research (Chicago, Ill.), ISSN 1538-7445, 09/2017, Volume 77, Issue 17, pp. 4602 - 4612
.... Mechanistic investigations revealed that TP53INP1 downregulation in early-stage HCC cells promoted metastasis via DUSP10 phosphatase-mediated activation of the ERK pathway... 
Life Sciences & Biomedicine | Oncology | Science & Technology | Prognosis | Humans | Lung Neoplasms - metabolism | Tumor Protein p73 - genetics | Gene Expression Profiling | Tumor Protein p73 - metabolism | Heat-Shock Proteins - genetics | Neoplasm Metastasis | Lung Neoplasms - secondary | Carcinoma, Hepatocellular - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Dual-Specificity Phosphatases - genetics | Liver Neoplasms - pathology | Tumor Cells, Cultured | Lung Neoplasms - genetics | Liver Neoplasms - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Dual-Specificity Phosphatases - metabolism | Heat-Shock Proteins - metabolism | Mitogen-Activated Protein Kinase Phosphatases - genetics | Xenograft Model Antitumor Assays | Carrier Proteins - genetics | Animals | Carrier Proteins - metabolism | Mitogen-Activated Protein Kinase 3 - metabolism | Mice, Nude | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Protein Array Analysis | Mice | Mice, Inbred BALB C | Carcinoma, Hepatocellular - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Mitogen-Activated Protein Kinase Phosphatases - metabolism | Liver | Extracellular signal-regulated kinase | Hepatocellular carcinoma | Metastasis | Metastases | Liver cancer | Signal transduction | Signaling | Stress response | Binding sites | Cellular stress response | Tumors | Apoptosis | Cancer | Index Medicus | Liver Neoplasms | Lung Neoplasms | Life Sciences | Tumor Protein p73 | Heat-Shock Proteins | Mitogen-Activated Protein Kinase Phosphatases | Carcinoma, Hepatocellular | Dual-Specificity Phosphatases | Mitogen-Activated Protein Kinase 1 | Mitogen-Activated Protein Kinase 3 | Carrier Proteins
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 1522-1539, 02/2012, Volume 302, Issue 3, pp. H801 - H810
Journal Article
Journal Article
American journal of physiology. Heart and circulatory physiology, ISSN 1522-1539, 10/2008, Volume 295, Issue 4, pp. H1580 - H1586
.... IPCD reduced infarct size by 58% in WT mice (33 ± 1% vs. 14 ± 3% for control and IPCD, respectively, P < 0.05... 
Obesity | Infarction | Phosphatase | Kinase | Cardiac & Cardiovascular Systems | Physiology | Peripheral Vascular Disease | Life Sciences & Biomedicine | Cardiovascular System & Cardiology | Science & Technology | Myocardial Reperfusion Injury - etiology | Myocardial Ischemia - metabolism | AMP-Activated Protein Kinases - metabolism | Phosphorylation | Phosphoprotein Phosphatases - metabolism | Male | Myocardial Reperfusion Injury - pathology | Myocardial Infarction - pathology | Myocardium - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Myocardial Infarction - etiology | Disease Models, Animal | Ribosomal Protein S6 Kinases, 70-kDa - metabolism | Signal Transduction | Obesity - complications | Mice, Inbred C57BL | Protein Phosphatase 2C | PTEN Phosphohydrolase - metabolism | Myocardium - pathology | Myocardial Infarction - metabolism | Myocardial Ischemia - pathology | Obesity - metabolism | Obesity - pathology | Dual Specificity Phosphatase 6 - metabolism | Myocardial Reperfusion Injury - metabolism | Myocardium - enzymology | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Myocardial Ischemia - complications | Myocardial Ischemia - therapy | Mice, Obese | Mice | Myocardial Infarction - prevention & control | Myocardial Reperfusion Injury - prevention & control | Mitogen-Activated Protein Kinase 1 - metabolism | Heart | Heart attacks | Kinases | Rodents | Index Medicus | Life Sciences | Biochemistry, Molecular Biology | postconditioning | pathology | Myocardial Ischemia | AMP-Activated Protein Kinases | Dual Specificity Phosphatase 6 | PTEN Phosphohydrolase | Ribosomal Protein S6 Kinases, 70-kDa | Phosphoprotein Phosphatases | complications | therapy | prevention & control | enzymology | etiology | Proto-Oncogene Proteins c-akt | Myocardial Infarction | kinase | Myocardium | Myocardial Reperfusion Injury | metabolism | phosphatase | Mitogen-Activated Protein Kinase 1 | Mitogen-Activated Protein Kinase 3
Journal Article