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endocytosis (253) 253
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mice (183) 183
biochemistry & molecular biology (177) 177
rats (174) 174
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phosphorylation (131) 131
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mitochondria (97) 97
endocytosis - physiology (96) 96
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cells (81) 81
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research (79) 79
fission (74) 74
mitochondria - metabolism (73) 73
multidisciplinary sciences (72) 72
internalization (70) 70
clathrin - metabolism (69) 69
drp1 (69) 69
enzyme inhibitors - pharmacology (67) 67
rats, sprague-dawley (66) 66
research article (66) 66
clathrin (65) 65
hela cells (65) 65
physiological aspects (65) 65
mutation (63) 63
neurons (61) 61
transfection (60) 60
cell line, tumor (58) 58
activation (57) 57
cell membrane - metabolism (57) 57
dynamins (57) 57
mediated endocytosis (57) 57
oxidative stress (56) 56
mitochondrial proteins - metabolism (54) 54
fusion (52) 52
gene expression (52) 52
medicine (52) 52
female (51) 51
kinases (50) 50
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analysis (49) 49
biochemistry (49) 49
biology (49) 49
dynamin ii - metabolism (49) 49
dynamin-related protein-1 (49) 49
gtp phosphohydrolases - genetics (49) 49
mitochondrial fission (49) 49
neurons - metabolism (49) 49
inhibition (48) 48
mitochondria - drug effects (48) 48
plasma-membrane (48) 48
protein (48) 48
science (48) 48
clathrin-mediated endocytosis (46) 46
pharmacology & pharmacy (46) 46
trafficking (45) 45
mitochondrial dna (44) 44
protein binding (44) 44
actin (43) 43
apoptosis - drug effects (42) 42
gtp phosphohydrolases - antagonists & inhibitors (42) 42
protein transport (42) 42
calcium (41) 41
calcium - metabolism (41) 41
rodents (41) 41
reactive oxygen species - metabolism (40) 40
endocrine system (39) 39
physiology (39) 39
rna interference (39) 39
dynamin i - metabolism (38) 38
brain (36) 36
hek293 cells (36) 36
neurons - drug effects (36) 36
time factors (36) 36
dynamin ii - antagonists & inhibitors (35) 35
mice, inbred c57bl (35) 35
mitochondrial dynamics - drug effects (35) 35
blotting, western (34) 34
microtubule-associated proteins - metabolism (34) 34
recombinant fusion proteins - metabolism (34) 34
synaptic vesicle endocytosis (34) 34
membrane proteins - metabolism (33) 33
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Free Radical Biology and Medicine, ISSN 0891-5849, 11/2017, Volume 112, pp. 336 - 349
Aberrant modulation of mitochondrial dynamic network, which shifts the balance of fusion and fission towards fission, is involved in brain damage of various... 
Mdivi-1 | Inflammation | Early brain injury | Blood-brain barrier disruption | Subarachnoid hemorrhage | Apoptosis | OXYGEN SPECIES PRODUCTION | OXIDATIVE STRESS | BIOCHEMISTRY & MOLECULAR BIOLOGY | COMPLEX I INHIBITOR | HIPPOCAMPAL-NEURONS | RAT MODEL | MITOCHONDRIAL FISSION | CELL-DEATH | SIGNALING PATHWAY | MOUSE MODEL | ENDOCRINOLOGY & METABOLISM | NF-KAPPA-B | Interleukin-6 - antagonists & inhibitors | Reactive Oxygen Species - metabolism | Apoptosis - drug effects | Proto-Oncogene Proteins c-bcl-2 - agonists | Tumor Necrosis Factor-alpha - genetics | Male | NF-kappa B - metabolism | Interleukin-1beta - genetics | Subarachnoid Space - drug effects | Occludin - metabolism | Matrix Metalloproteinase 9 - metabolism | Interleukin-1beta - metabolism | Matrix Metalloproteinase 9 - genetics | Subarachnoid Hemorrhage - mortality | Zonula Occludens-1 Protein - metabolism | Interleukin-6 - metabolism | Subarachnoid Hemorrhage - drug therapy | Interleukin-1beta - antagonists & inhibitors | Zonula Occludens-1 Protein - genetics | NF-kappa B - antagonists & inhibitors | Endoplasmic Reticulum Stress - drug effects | Interleukin-6 - genetics | bcl-2-Associated X Protein - metabolism | Dynamins - genetics | Rats | Subarachnoid Hemorrhage - genetics | Rats, Sprague-Dawley | Blood-Brain Barrier - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Survival Analysis | Subarachnoid Space - metabolism | Tumor Necrosis Factor-alpha - metabolism | Dynamins - metabolism | Mitochondrial Dynamics - genetics | Claudin-5 - metabolism | Occludin - genetics | Proto-Oncogene Proteins c-bcl-2 - metabolism | Mitochondria - genetics | Quinazolinones - pharmacology | bcl-2-Associated X Protein - genetics | Dynamins - antagonists & inhibitors | Claudin-5 - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Subarachnoid Hemorrhage - pathology | bcl-2-Associated X Protein - antagonists & inhibitors | Blood-Brain Barrier - drug effects | Animals | Mitochondrial Dynamics - drug effects | NF-kappa B - genetics | Subarachnoid Space - pathology | Proto-Oncogene Proteins c-bcl-2 - genetics | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Stroke (Disease) | Proteins | Medical research | Brain | Neurons | Analysis | Medicine, Experimental | Brain damage | Injuries | Index Medicus
Journal Article
Developmental Cell, ISSN 1534-5807, 03/2017, Volume 40, Issue 6, pp. 583 - 594.e6
Mitochondrial fission mediated by the GTPase dynamin-related protein 1 (Drp1) is an attractive drug target in numerous maladies that range from heart disease... 
succinate | bioenergetics | mitochondria | fission | neuron | reverse electron transfer | respiration | superoxide | brain | fragmentation | ISCHEMIA-REPERFUSION INJURY | BRAIN MITOCHONDRIA | LIFE-SPAN | CELLS | ROS | DYNAMIN-RELATED PROTEIN-1 | DEVELOPMENTAL BIOLOGY | RAT-BRAIN | FISSION | DIVISION | DAMAGE | CELL BIOLOGY | Dynamins - metabolism | Reactive Oxygen Species - metabolism | Microtubule-Associated Proteins - metabolism | Humans | Cercopithecus aethiops | Saccharomyces cerevisiae - drug effects | GTP Phosphohydrolases - antagonists & inhibitors | Electron Transport Complex I - metabolism | Fibroblasts - ultrastructure | Saccharomyces cerevisiae - metabolism | Mitochondrial Proteins - metabolism | Cell Respiration - drug effects | Oxidation-Reduction - drug effects | Neurons - metabolism | NAD - metabolism | Quinazolinones - pharmacology | Fibroblasts - metabolism | Dynamins - antagonists & inhibitors | Electron Transport Complex I - antagonists & inhibitors | Mitochondrial Proteins - antagonists & inhibitors | Mitochondria - metabolism | Microtubule-Associated Proteins - antagonists & inhibitors | Mitochondria - drug effects | Rats, Sprague-Dawley | Mice, Knockout | Animals | GTP Phosphohydrolases - metabolism | Oxygen Consumption - drug effects | Saccharomyces cerevisiae Proteins - metabolism | Mice | COS Cells | Brain | Medical colleges | Nervous system diseases | Heart diseases | Injuries | Biomedical engineering | Neurons | Superoxide | Index Medicus
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 04/2014, Volume 171, Issue 8, pp. 1988 - 1999
Background and PurposeDynamin-related protein 1 (Drp1) mediates mitochondrial fission and is thought to promote Bax/Bak-induced cytochrome c release during... 
mitochondria | fission | Bim | primed for death | Bax | apoptosis | Bcl | Bak | oxygen | caspases | Bcl-xL | PROAPOPTOTIC BAX | CRISTAE | DEATH | MECHANISMS | BH3 MIMETIC ABT-737 | MITOCHONDRIAL FISSION | BRAIN MITOCHONDRIA | INHIBITION | NEURAL CELL | PHARMACOLOGY & PHARMACY | PROTEINS | Up-Regulation | Proto-Oncogene Proteins c-bcl-2 - physiology | Microtubule-Associated Proteins - genetics | Humans | Mitochondrial Proteins - genetics | GTP Phosphohydrolases - antagonists & inhibitors | bcl-2 Homologous Antagonist-Killer Protein - metabolism | Biphenyl Compounds - pharmacology | Dynamins - physiology | Nitrophenols - pharmacology | Cell Death - drug effects | Quinazolinones - pharmacology | Fibroblasts - metabolism | Cytochromes c - antagonists & inhibitors | Dynamins - antagonists & inhibitors | GTP Phosphohydrolases - physiology | Cytochromes c - metabolism | Mitochondrial Proteins - antagonists & inhibitors | Cells, Cultured | bcl-2-Associated X Protein - metabolism | Dynamins - genetics | Mitochondrial Proteins - physiology | Microtubule-Associated Proteins - antagonists & inhibitors | Sulfonamides - pharmacology | Piperazines - pharmacology | Mice, Knockout | Microtubule-Associated Proteins - physiology | Animals | Cell Death - physiology | GTP Phosphohydrolases - genetics | Oxygen Consumption - drug effects | Fibroblasts - drug effects | Proto-Oncogene Proteins c-bcl-2 - antagonists & inhibitors | Mice | Proto-Oncogene Proteins c-bcl-2 - genetics | Cytochrome c | Proteins | Mitochondrial DNA | Apoptosis | Rodents | Index Medicus
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 11/2010, Volume 17, Issue 11, pp. 1785 - 1794
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 12/2013, Volume 123, Issue 12, pp. 5371 - 5388
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2014, Volume 289, Issue 32, pp. 22258 - 22267
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2016, Volume 11, Issue 4, pp. e0154280 - e0154280
Endoplasmic reticulum (ER)-mitochondrial contact sites play a pivotal role in exchange of lipids and ions between the two organelles. How size and function of... 
Mitochondrial Dynamics | RNA, Small Interfering - genetics | Dynamins - metabolism | Saccharomyces cerevisiae - genetics | Vesicular Transport Proteins - metabolism | Humans | Caenorhabditis elegans Proteins - metabolism | Endoplasmic Reticulum - metabolism | Saccharomyces cerevisiae - ultrastructure | GTP Phosphohydrolases - antagonists & inhibitors | Mitochondria - ultrastructure | Endoplasmic Reticulum - ultrastructure | Saccharomyces cerevisiae - metabolism | Endoplasmic Reticulum - chemistry | Vesicular Transport Proteins - antagonists & inhibitors | Conserved Sequence | Ion Transport | Mitochondria - chemistry | Caenorhabditis elegans - metabolism | Signal Transduction | Caenorhabditis elegans - genetics | Saccharomyces cerevisiae Proteins - antagonists & inhibitors | Vesicular Transport Proteins - genetics | Gene Expression Regulation | Dynamins - genetics | Lipid Metabolism | Mitochondria - metabolism | Monomeric GTP-Binding Proteins - genetics | Saccharomyces cerevisiae Proteins - genetics | Intracellular Membranes - chemistry | Monomeric GTP-Binding Proteins - antagonists & inhibitors | Animals | GTP Phosphohydrolases - metabolism | Caenorhabditis elegans Proteins - antagonists & inhibitors | Monomeric GTP-Binding Proteins - metabolism | GTP Phosphohydrolases - genetics | Saccharomyces cerevisiae Proteins - metabolism | HeLa Cells | Mutation | Caenorhabditis elegans Proteins - genetics | Intracellular Membranes - metabolism | Amino Acid Substitution | RNA, Small Interfering - metabolism | Physiological aspects | Mitochondria | Genetic aspects | Research | Endoplasmic reticulum | Temperature | Neuropathology | Glycerol | Tubules | Lipids | Biosynthesis | Biology | Gene expression | Organelles | Proteins | Clonal deletion | Morphology | Dynamin | Nematodes | Deletion | Cleavage | Curvature | Guanosinetriphosphatase | Index Medicus
Journal Article
Journal of Cell Science, ISSN 0021-9533, 02/2013, Volume 126, Issue 3, pp. 789 - 802
Journal Article
Blood Cells, Molecules and Diseases, ISSN 1079-9796, 03/2016, Volume 57, pp. 85 - 90
Granulocyte-macrophage colony stimulating factor (GM-CSF) induces procoagulant activity of macrophages. Tissue factor (TF) is a membrane-bound glycoprotein and... 
Neurokinin 1 receptor | Granulocyte-macrophage colony-stimulating factor | Substance P | Tissue factor | ACTIVATION | ADIPOCYTES | MECHANISM | MONOCYTES | NEUROKININ-1 RECEPTOR | INDUCTION | MICROPARTICLES | ENDOTHELIAL-CELLS | INHIBITOR | HEMATOLOGY | PROCOAGULANT ACTIVITY | Free Radical Scavengers - pharmacology | RNA, Small Interfering - genetics | Adipocytes - secretion | Dynamins - metabolism | Humans | NADPH Oxidases - metabolism | Adipocytes - cytology | Arrestins - genetics | Substance P - pharmacology | Phosphorylcholine - analogs & derivatives | Arrestins - metabolism | rho-Associated Kinases - antagonists & inhibitors | Pyrrolidines - pharmacology | Macrophages - secretion | Neurokinin-1 Receptor Antagonists - pharmacology | Thiocarbamates - pharmacology | rho-Associated Kinases - metabolism | Substance P - biosynthesis | Phosphorylcholine - pharmacology | Acetophenones - pharmacology | NADPH Oxidases - genetics | Benzopyrans - pharmacology | Thromboplastin - biosynthesis | Amides - pharmacology | Dynamins - antagonists & inhibitors | Signal Transduction | NADPH Oxidases - antagonists & inhibitors | Gene Expression Regulation | Dynamins - genetics | Granulocyte-Macrophage Colony-Stimulating Factor - pharmacology | Morpholines - pharmacology | rho-Associated Kinases - genetics | Macrophages - cytology | Arrestins - antagonists & inhibitors | beta-Arrestin 2 | beta-Arrestins | Macrophages - drug effects | Protein Kinase Inhibitors - pharmacology | Pyridines - pharmacology | Primary Cell Culture | Macrophage Activation - drug effects | RNA, Small Interfering - metabolism | Thromboplastin - secretion | Index Medicus
Journal Article
Diabetes, ISSN 0012-1797, 05/2017, Volume 66, Issue 5, pp. 1247 - 1257
In pancreatic -cells, mitochondrial bioenergetics control glucose-stimulated insulin secretion. Mitochondrial dynamics are generally associated with quality... 
FUSION | BETA-CELL APOPTOSIS | DISEASE | ENDOCRINOLOGY & METABOLISM | DYNAMIN-RELATED PROTEIN-1 | DYSFUNCTION | STRESS | GTPASE | DIVISION | DRP1 | PROTECTS | Dynamins - antagonists & inhibitors | Microtubule-Associated Proteins - genetics | Mitochondrial Dynamics - genetics | Humans | Mitochondrial Proteins - antagonists & inhibitors | Mitochondria - metabolism | Mitochondrial Proteins - genetics | GTP Phosphohydrolases - antagonists & inhibitors | Microtubule-Associated Proteins - antagonists & inhibitors | Mitochondria - pathology | Gene Knockdown Techniques | Microscopy, Confocal | Insulin - metabolism | Insulin-Secreting Cells - metabolism | Animals | Insulin-Secreting Cells - drug effects | GTP Phosphohydrolases - genetics | Islets of Langerhans - metabolism | Adenosine Triphosphate - metabolism | Glucose - metabolism | Mice | Energy Metabolism - genetics | Insulin Secretion | Pyruvic Acid - pharmacology | Energy metabolism | Mitochondrial biogenesis | Research | Bioenergetics | Mitochondria | Phosphorylation | Oxidative phosphorylation | Secretion | Quality control | Pyruvic acid | Glucose | Islets of Langerhans | Pancreas | Insulin | Cells | Index Medicus | Abridged Index Medicus | Clinical Medicine | Medical and Health Sciences | Endokrinologi och diabetes | Klinisk medicin | Medicin och hälsovetenskap | Endocrinology and Diabetes
Journal Article