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Science (American Association for the Advancement of Science), ISSN 0036-8075, 01/2010, Volume 327, Issue 5961, pp. 88 - 92
Journal Article
Journal Article
Cell death and differentiation, ISSN 1350-9047, 2013, Volume 20, Issue 9, pp. 1194 - 1208
Journal Article
Clinical and Experimental Pharmacology and Physiology, ISSN 1440-1681, 07/2006, Volume 33, Issue 7, pp. 657 - 662
SUMMARY • Duchenne muscular dystrophy (DMD) is a lethal, degenerative muscle disease caused by a genetic mutation that leads to the complete absence of the... 
calcium | mdx mouse | stretch-activated channels | muscle damage | reactive oxygen species | stretch‐activated channels | Reactive oxygen species | Calcium | Muscle damage | Stretch-activated channels | PHYSIOLOGY | MOUSE MUSCLE | TIME-COURSE | PLASMA-MEMBRANE | SKELETAL-MUSCLE | DEPENDENT PROTEOLYSIS | INCREASED SUSCEPTIBILITY | LEAK CHANNELS | PHARMACOLOGY & PHARMACY | DUCHENNE MUSCULAR-DYSTROPHY | ION CHANNELS | ELEVATED FREE CALCIUM | Inflammation - pathology | Muscle, Skeletal - enzymology | Reactive Oxygen Species - metabolism | Calcium - metabolism | Humans | Muscular Dystrophy, Duchenne - enzymology | Cell Membrane Permeability | Muscle, Skeletal - metabolism | Muscular Dystrophy, Duchenne - pathology | Necrosis | Inflammation - metabolism | Animals | Mice, Inbred mdx | TRPC Cation Channels - metabolism | Mice | Muscular Dystrophy, Duchenne - metabolism | Muscle, Skeletal - pathology | Inflammation - enzymology | Ion Channel Gating | Disease Models, Animal | Peptide Hydrolases - metabolism | Calcium, metabolism | Phospholipases A, metabolism | Calpain, pharmacology | Dystrophin, genetics | TRPC Cation Channels, physiology | Muscular Dystrophy, Duchenne, physiopathology | Muscular Dystrophy, Animal, physiopathology | NF-kappa B, metabolism | Muscle Fibers, physiology | Muscular Dystrophy, Animal, genetics | Muscular Dystrophy, Duchenne, genetics | Muscle Spindles, physiology | Muscular Dystrophy, Animal, metabolism | Dystrophin, physiology | Peptide Hydrolases, metabolism | Ion Channels, physiology | Muscle, Skeletal, metabolism | Biomechanics | Ion Channels, metabolism | Muscle, Skeletal, physiology | Calcium Signaling, physiology | Reactive Oxygen Species, metabolism
Journal Article
Nature (London), ISSN 1476-4687, 2017, Volume 547, Issue 7662, pp. 227 - 231
The regenerative capacity of the adult mammalian heart is limited, because of the reduced ability of cardiomyocytes to progress through mitosis(1). Endogenous... 
PRESSURE-OVERLOAD | GENE | ADULT HEART REGENERATION | MULTIDISCIPLINARY SCIENCES | IN-VIVO | MOUSE MODEL | MUSCLE | DUCHENNE MUSCULAR-DYSTROPHY | MICE | Protein-Serine-Threonine Kinases - deficiency | Phosphorylation | Cell Proliferation | Glycoproteins - metabolism | Dystroglycans - metabolism | Male | Phosphoproteins - metabolism | Heart Failure - prevention & control | Multiprotein Complexes - metabolism | Dystrophin - deficiency | Mice, Inbred mdx | Cardiomyopathies | Glycoproteins - deficiency | Multiprotein Complexes - deficiency | Dystrophin - metabolism | Protein-Serine-Threonine Kinases - metabolism | Myocytes, Cardiac - cytology | Mice, Inbred C57BL | Organ Size | Heart Failure - genetics | Pressure | Multiprotein Complexes - chemistry | Animals | Dystrophin - genetics | Myocytes, Cardiac - metabolism | Protein Binding | Mice | Muscular Dystrophy, Duchenne - metabolism | Muscular Dystrophy, Duchenne - genetics | Adaptor Proteins, Signal Transducing - metabolism | Heart | Genes | Homeostasis | Genomes | Kinases | Muscular dystrophy | Cell growth | Actin | Duchenne's muscular dystrophy | Cell cycle | Extracellular matrix | Heart diseases | Dystrophin | Deoxyribonucleic acid--DNA | Heart failure | Dystroglycan | Cardiomyocytes | Glycoproteins | Studies | Yes-associated protein | Regeneration | Point mutation | Cytoskeleton | Mutation | Dystrophy
Journal Article
Nature cell biology, ISSN 1465-7392, 01/2018, Volume 20, Issue 1, pp. 46 - 57
Human pluripotent stem cells (hPSCs) can be directed to differentiate into skeletal muscle progenitor cells (SMPCs). However, the myogenicity of hPSC-SMPCs... 
PLURIPOTENT STEM-CELLS | DIRECTED DIFFERENTIATION | IN-VITRO | MYOGENIC DIFFERENTIATION | RNA-SEQ | FUNCTIONAL-ANALYSIS | DUCHENNE MUSCULAR-DYSTROPHY | SATELLITE CELLS | FETAL | LARGE GENE LISTS | CELL BIOLOGY | Receptor, ErbB-3 - metabolism | Humans | Middle Aged | PAX7 Transcription Factor - genetics | Male | PAX7 Transcription Factor - metabolism | Receptors, Nerve Growth Factor - metabolism | Muscle Fibers, Skeletal - metabolism | Myoblasts - metabolism | Gene Expression Regulation, Developmental | Myoblasts - cytology | Adult | Female | Myosins - metabolism | Cell Differentiation | Induced Pluripotent Stem Cells - cytology | Muscular Dystrophy, Duchenne - therapy | Dystrophin - metabolism | Induced Pluripotent Stem Cells - metabolism | Signal Transduction | Muscular Dystrophy, Duchenne - pathology | Receptor, ErbB-3 - genetics | Myosins - genetics | Nerve Tissue Proteins - genetics | Gene Editing | Receptors, Nerve Growth Factor - genetics | Nerve Tissue Proteins - metabolism | Transforming Growth Factor beta - genetics | Dystrophin - genetics | CRISPR-Cas Systems | Muscle Development - genetics | Muscle Fibers, Skeletal - cytology | Aged | Muscular Dystrophy, Duchenne - metabolism | Muscular Dystrophy, Duchenne - genetics | Transforming Growth Factor beta - metabolism | RNA sequencing | Usage | Growth | Stem cells | Muscle cells | Fetus | Research | Comparative analysis | Transforming growth factors | CRISPR | Satellite RNA | Fetuses | Muscles | Satellite cells | ErbB-3 protein | Ribonucleic acid--RNA | Cell surface | Myogenesis | Muscular dystrophy | Skeletal muscle | Gene sequencing | Musculoskeletal system | Signaling | Receptors | Nerve growth factor receptors | Duchenne's muscular dystrophy | Cells (biology) | Dystrophy | Differentiation | Dystrophin | Pluripotency
Journal Article
Journal Article
Australasian Psychiatry, ISSN 1039-8562, 2008, Volume 42, Issue 8, pp. 662 - 677
During adolescence there is a loss of approximately 30% of the synapses formed in the cortex during childhood. Comprehensive studies of the visual cortex show... 
MuSK | Agrin | Schizophrenia | Neuregulin | Neuroligin | DLPC | DISC1 | Dysbindin | Synapses | CYTOPLASMIC DYNEIN | PSYCHIATRY | neuregulin | schizophrenia | dysbindin | ALPHA-LATROTOXIN RECEPTOR | FASCICULATION-AND-ELONGATION-PROTEIN-ZETA-1 FEZ1 | PREFRONTAL CORTEX | NEUROMUSCULAR-TRANSMISSION | SKELETAL-MUSCLE | synapses | MOOD DISORDERS | VISUAL-CORTEX | agrin | POLYNEURONAL INNERVATION | ORBITOFRONTAL CORTEX | neuroligin | Schizophrenia - metabolism | Age Factors | Humans | Schizophrenia - pathology | Synapses - genetics | Synapses - pathology | Agrin - metabolism | Brain - metabolism | Neuregulin-1 | Cell Adhesion Molecules, Neuronal | Visual Cortex - pathology | Schizophrenia - genetics | Synapses - metabolism | Membrane Proteins - metabolism | Agrin - genetics | Receptors, Cholinergic - metabolism | Membrane Proteins - genetics | Receptor Protein-Tyrosine Kinases - metabolism | Nerve Tissue Proteins - genetics | Nerve Degeneration - pathology | Dystrophin-Associated Proteins | Nerve Tissue Proteins - metabolism | Carrier Proteins - genetics | Magnetic Resonance Imaging | Carrier Proteins - metabolism | Receptor Protein-Tyrosine Kinases - genetics | Brain - pathology | Visual Cortex - metabolism | Receptors, Cholinergic - genetics | Prefrontal Cortex, growth and development | Schizophrenia, physiopathology | Receptors, N-Methyl-D-Aspartate, metabolism | Membrane Proteins, metabolism | Adolescent Development | Receptors, Cholinergic, metabolism | Neuromuscular Junction, metabolism | Receptor Protein-Tyrosine Kinases, metabolism | Acetylcholine, metabolism | Aging | Schizophrenia, metabolism | Dynein ATPase, metabolism | Neuropsychology | Psychopathology | Receptors, Growth Factor, metabolism | Synapses, physiology | Presynaptic Terminals, metabolism | Visual Cortex, physiology | Animals | Prefrontal Cortex, physiology | Models, Biological | Neuronal Plasticity | Neuregulins, metabolism | Signal Transduction, physiology | Adolescent | Nerve Tissue Proteins, metabolism | Dominance, Ocular | Carrier Proteins, metabolism | Visual Cortex, growth and development
Journal Article
Nature (London), ISSN 1476-4687, 2017, Volume 547, Issue 7662, pp. 179 - 184
Journal Article