Articles

Search Articles

Advanced search
Help

Catalogue

Articles

Databases

X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (2919) 2919
Publication (377) 377
Book Review (14) 14
Book Chapter (5) 5
Conference Proceeding (4) 4
Dissertation (1) 1
Newspaper Article (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (2164) 2164
e-selectin (1438) 1438
animals (1184) 1184
index medicus (1086) 1086
e-selectin - genetics (896) 896
male (817) 817
female (768) 768
e-selectin - metabolism (744) 744
mice (731) 731
cells, cultured (682) 682
inflammation (632) 632
expression (568) 568
immunology (475) 475
endothelium (448) 448
endothelium, vascular - metabolism (425) 425
intercellular adhesion molecule-1 - genetics (421) 421
middle aged (414) 414
adult (401) 401
cell adhesion (396) 396
biochemistry & molecular biology (393) 393
atherosclerosis (379) 379
cell biology (379) 379
p-selectin (379) 379
adhesion molecules (351) 351
intercellular adhesion molecule-1 - metabolism (333) 333
endothelium, vascular - cytology (330) 330
vascular cell adhesion molecule-1 - genetics (317) 317
endothelium, vascular - drug effects (304) 304
endothelial cells - metabolism (303) 303
activation (294) 294
endothelial cells (292) 292
tumor necrosis factor-alpha - pharmacology (284) 284
cell adhesion molecules - genetics (279) 279
vascular cell adhesion molecule-1 - metabolism (278) 278
gene expression (277) 277
adhesion (274) 274
e-selectin - biosynthesis (274) 274
mice, inbred c57bl (270) 270
endothelial-cells (268) 268
hematology (258) 258
nf-kappa-b (258) 258
nf-kappa b - metabolism (256) 256
in-vivo (251) 251
rna, messenger - metabolism (244) 244
e-selectin - blood (240) 240
aged (236) 236
cell adhesion - drug effects (232) 232
molecular sequence data (228) 228
ligands (226) 226
research article (226) 226
cytokines (218) 218
icam-1 (217) 217
risk factors (215) 215
tumor-necrosis-factor (212) 212
mice, knockout (211) 211
rna, messenger - genetics (211) 211
research (208) 208
peripheral vascular disease (207) 207
cell line (206) 206
proteins (201) 201
intercellular-adhesion molecule-1 (200) 200
multidisciplinary sciences (200) 200
article (197) 197
cell adhesion molecules - metabolism (197) 197
cell adhesion molecules (194) 194
analysis (193) 193
oncology (192) 192
cell adhesion & migration (191) 191
cells (191) 191
medicine (191) 191
base sequence (185) 185
flow cytometry (183) 183
medicine, research & experimental (182) 182
transfection (182) 182
gene expression regulation (173) 173
gene expression regulation - drug effects (172) 172
endothelial cells - drug effects (171) 171
disease (170) 170
p-selectin - genetics (166) 166
genetic aspects (164) 164
neutrophils (160) 160
metastasis (159) 159
cancer (158) 158
in-vitro (156) 156
vcam-1 (156) 156
intercellular adhesion molecule-1 - biosynthesis (154) 154
immunohistochemistry (152) 152
leukocyte adhesion (152) 152
reverse transcriptase polymerase chain reaction (152) 152
rats (150) 150
disease models, animal (149) 149
p-selectin - metabolism (149) 149
enzyme-linked immunosorbent assay (147) 147
physiological aspects (147) 147
endothelium, vascular - physiology (146) 146
science (145) 145
signal transduction (145) 145
up-regulation (144) 144
biomarkers - blood (140) 140
intercellular adhesion molecule-1 - blood (140) 140
more...
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (2890) 2890
Chinese (20) 20
Russian (4) 4
Japanese (3) 3
French (2) 2
Polish (2) 2
Arabic (1) 1
Swedish (1) 1
Ukrainian (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


1. Full Text NF-κB Directs Dynamic Super Enhancer Formation in Inflammation and Atherogenesis
by Brown, Jonathan D and Lin, Charles Y and Duan, Qiong and Griffin, Gabriel and Federation, Alexander J and Paranal, Ronald M and Bair, Steven and Newton, Gail and Lichtman, Andrew H and Kung, Andrew L and Yang, Tianlun and Wang, Hong and Luscinskas, Francis W and Croce, Kevin J and Bradner, James E and Plutzky, Jorge
Molecular Cell, ISSN 1097-2765, 10/2014, Volume 56, Issue 2, pp. 219 - 231
Proinflammatory stimuli elicit rapid transcriptional responses via transduced signals to master regulatory transcription factors. To explore the role of... 
RNA-POLYMERASE-II | SELECTIVE-INHIBITION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL IDENTITY | DISEASE | GENE-EXPRESSION | ATHEROSCLEROSIS | BROMODOMAIN PROTEIN BRD4 | TRANSCRIPTIONAL ELONGATION | P-TEFB | BET BROMODOMAINS | CELL BIOLOGY | Endothelium, Vascular - cytology | Transcription, Genetic - drug effects | Endothelial Cells | Atherosclerosis - genetics | Humans | Regulatory Sequences, Nucleic Acid | NF-kappa B p50 Subunit - genetics | Transcription Factor RelA - genetics | Cell Movement - genetics | Cell Movement - immunology | NF-kappa B p50 Subunit - immunology | E-Selectin - biosynthesis | Transcription Factor RelA - immunology | Acetylation | Transcription Initiation, Genetic | Transcription Factors - immunology | Macrophages - immunology | Endothelium, Vascular - immunology | Atherosclerosis - immunology | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Transcription Factors - antagonists & inhibitors | Inflammation - immunology | Nuclear Proteins - immunology | Mice, Knockout | Azepines - pharmacology | Cell Adhesion - immunology | Triazoles - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Animals | Enhancer Elements, Genetic | Nuclear Proteins - antagonists & inhibitors | Inflammation - genetics | Protein Binding | RNA Polymerase II - genetics | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Histones - metabolism | SOXF Transcription Factors - genetics | Chromatin - genetics
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
2. Linkage of metabolic defects to activated PIK3CA alleles in endothelial cells derived from lymphatic malformation
by Glaser, Kathryn and Dickie, Peter and Neilson, Derek and Osborn, Alexander and Dickie, Belinda Hsi
Lymphatic Research and Biology, ISSN 1539-6851, 02/2018, Volume 16, Issue 1, pp. 43 - 55
Background: Lymphatic endothelial cells (LECs) derived from lymphatic malformations (LMs) bear activated PIK3CA alleles yet display an inflammatory gene... 
PIK3CA | endothelial cells | lymphatic malformations | MEDICINE, RESEARCH & EXPERIMENTAL | PHYSIOLOGY | PERCUTANEOUS SCLEROTHERAPY | PHOSPHORYLATION | PROTEIN-KINASE | CANCER | FAT STORAGE | PI3K | INSULIN-RESISTANCE | AMPK | MUTATIONS | LIPID DROPLETS | Interleukin-8 - genetics | Heme Oxygenase-1 - metabolism | Class I Phosphatidylinositol 3-Kinases - genetics | Vesicular Transport Proteins - metabolism | Humans | Aminoimidazole Carboxamide - pharmacology | Cyclooxygenase 2 - genetics | E-Selectin - genetics | Lipid Metabolism - genetics | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Repressor Proteins - metabolism | Histone Deacetylases - genetics | Interleukin-6 - genetics | Signal Transduction | Endothelial Cells - metabolism | Membrane Proteins - genetics | Lymphoid Tissue - metabolism | Repressor Proteins - genetics | Lymphatic Abnormalities - pathology | Lymphoid Tissue - pathology | Aminoimidazole Carboxamide - analogs & derivatives | Vascular Endothelial Growth Factor C - metabolism | Angiopoietin-like 4 Protein - metabolism | Mutation | Endothelial Cells - pathology | Autophagy-Related Proteins - metabolism | AMP-Activated Protein Kinases - genetics | Angiopoietin-like 4 Protein - genetics | Lymphatic Abnormalities - genetics | AMP-Activated Protein Kinases - metabolism | Class I Phosphatidylinositol 3-Kinases - metabolism | Ribonucleotides - pharmacology | Heme Oxygenase-1 - genetics | Membrane Proteins - metabolism | Autophagy-Related Proteins - genetics | Cell Line | Vesicular Transport Proteins - genetics | Gene Expression Regulation | Lymphatic Abnormalities - metabolism | E-Selectin - metabolism | Histone Deacetylases - metabolism | Transcription Factors - genetics | Vascular Endothelial Growth Factor C - genetics | Transcription Factors - metabolism | Ankyrins - genetics | Alleles | Ankyrins - metabolism | Cyclooxygenase 2 - metabolism | Primary Cell Culture | Endothelial Cells - drug effects
Journal Article
Details down arrow
3. Full Text Genetic variants in genes of the inflammatory response in association with infective endocarditis
by Weinstock, Melanie and Grimm, Imke and Dreier, Jens and Knabbe, Cornelius and Vollmer, Tanja
PLoS ONE, ISSN 1932-6203, 10/2014, Volume 9, Issue 10, p. e110151
Aims: Inflammation in infective endocarditis (IE) is a complex network including interactions of inflammatory cytokines and other components of host response.... 
MORTALITY | HEART-VALVES | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | SEPTIC SHOCK | PROMOTER POLYMORPHISM | IL-1-BETA | TNF-ALPHA | SEPSIS | SECRETION | EXPRESSION | Inflammation - pathology | Endocarditis - blood | Endocarditis - pathology | Protein Precursors - blood | Humans | Middle Aged | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - genetics | E-Selectin - blood | Male | Interleukin-1beta - genetics | Inflammation - blood | Interleukin-1beta - blood | E-Selectin - genetics | Interleukin-6 - blood | Adult | Female | Intercellular Adhesion Molecule-1 - blood | Leukocyte Count | Endocarditis - genetics | Calcitonin Gene-Related Peptide | Calcitonin - blood | Protein Precursors - genetics | Genetic Association Studies | Interleukin-6 - genetics | Gene Frequency | Genotype | Calcitonin - genetics | Biomarkers - blood | Intercellular Adhesion Molecule-1 - genetics | Adolescent | Enterococcus - pathogenicity | Inflammation - genetics | Polymorphism, Single Nucleotide - genetics | Aged | Heart | Flow cytometry | C-reactive protein | Laboratories | Genes | Interleukin | Procalcitonin | Erythrocytes | Inflammatory response | Cardiovascular disease | Infections | Leukocytes | Blood | Subgroups | Interleukin 6 | Proteins | Cell adhesion | Toll-like receptors | Tumor necrosis factor-TNF | Bacteria | Bioindicators | Genotypes | Deoxyribonucleic acid--DNA | Pathogens | Statistical analysis | Cytokines | Blood & organ donations | Intercellular adhesion molecule 1 | Mortality | Blood cells | Inflammation | Tumor necrosis factor-α | Patients | Blood donors | White blood cells | Genetic variance | Cytometry | Gene frequency | Endocarditis | Interleukin 10 | Biomarkers | Sepsis | Deoxyribonucleic acid | DNA
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
4. Full Text Glaucomatous MYOC mutations activate the IL-1/NF-kappa B inflammatory stress response and the glaucoma marker SELE in trabecular meshwork cells
by Itakura, T and Peters, DM and Fini, ME
MOLECULAR VISION, ISSN 1090-0535, 09/2015, Volume 21, pp. 1071 - 1084
Purpose: Activation of the IL-1/NF-kappa B inflammatory stress pathway and induction of SELE expression in the trabecular meshwork (TBM) is a marker for... 
AQUEOUS OUTFLOW PATHWAY | MATRIX METALLOPROTEINASES | WILD-TYPE | OPEN-ANGLE GLAUCOMA | OXIDATIVE STRESS | PROTEIN | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | OPHTHALMOLOGY | INTRAOCULAR-PRESSURE | EXPRESSION | CALORIE RESTRICTION | Cytoskeletal Proteins - genetics | Glaucoma, Open-Angle - genetics | Transforming Growth Factor beta2 - metabolism | Humans | Actins - metabolism | Glycoproteins - metabolism | Glaucoma, Open-Angle - pathology | NF-kappa B - metabolism | Interleukin-1beta - genetics | Actins - genetics | E-Selectin - genetics | Forkhead Transcription Factors - metabolism | Interleukin-1beta - metabolism | Cytoskeletal Proteins - metabolism | Glaucoma, Open-Angle - metabolism | Lentivirus - genetics | Eye Proteins - genetics | Interleukin-6 - metabolism | Fibroblasts - metabolism | Glycoproteins - genetics | Trabecular Meshwork - pathology | Mutagenesis, Site-Directed | Genetic Vectors - chemistry | Interleukin-6 - genetics | Signal Transduction | Endothelial Cells - metabolism | Gene Expression Regulation | Genetic Vectors - metabolism | E-Selectin - metabolism | Inflammation | Fibroblasts - pathology | Forkhead Transcription Factors - genetics | Eye Proteins - metabolism | NF-kappa B - genetics | Models, Biological | Trabecular Meshwork - metabolism | Forkhead Box Protein O1 | Primary Cell Culture | Transforming Growth Factor beta2 - genetics | Endothelial Cells - pathology | Cell Line, Transformed | Amino Acid Substitution
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
5. Full Text Endothelial cell activation by myeloblasts: Molecular mechanisms of leukostasis and leukemic cell dissemination
by Stucki, Anne and Rivier, Anne-Sophie and Gikic, Milica and Monai, Natacha and Schapira, Marc and Spertini, Olivier
Blood, ISSN 0006-4971, 04/2001, Volume 97, Issue 7, pp. 2121 - 2129
Leukostasis and tissue infiltration by leukemic cells are poorly understood life-threatening complications of acute leukemia. This study has tested the... 
SELECTIN GLYCOPROTEIN LIGAND-1 | INTERCELLULAR-ADHESION MOLECULE-1 | COLONY-STIMULATING FACTOR | IN-VIVO | P-SELECTIN | ACUTE MYELOID-LEUKEMIA | LEUKOCYTE ADHESION | HEMATOLOGY | NF-KAPPA-B | GRANULOCYTE-MACROPHAGE | HUMAN T-CELLS | Neoplastic Stem Cells - cytology | Lymphocyte Function-Associated Antigen-1 - biosynthesis | Coculture Techniques | Humans | Leukemia, Myeloid - genetics | Antigens, CD - genetics | Neoplastic Stem Cells - metabolism | E-Selectin - genetics | E-Selectin - biosynthesis | Leukostasis - genetics | Neoplasm Proteins - genetics | CD18 Antigens - genetics | Gene Expression Regulation, Leukemic | Cell Adhesion | Leukostasis - metabolism | Endothelium, Vascular - metabolism | Lymphocyte Function-Associated Antigen-1 - genetics | Macrophage-1 Antigen - genetics | Intercellular Adhesion Molecule-1 - genetics | Endothelium, Vascular - pathology | Vascular Cell Adhesion Molecule-1 - biosynthesis | Integrin beta1 - genetics | Macrophage-1 Antigen - biosynthesis | Receptors, Lymphocyte Homing - genetics | Tumor Necrosis Factor-alpha - metabolism | Cell Adhesion Molecules - genetics | Leukemic Infiltration - genetics | Lewis X Antigen - genetics | Antigens, CD - biosynthesis | Integrin alphaXbeta2 - biosynthesis | L-Selectin - genetics | Neoplasm Proteins - metabolism | L-Selectin - biosynthesis | Leukemia, Myeloid - pathology | Integrin alphaXbeta2 - genetics | Intercellular Adhesion Molecule-1 - biosynthesis | Vascular Cell Adhesion Molecule-1 - genetics | Tumor Cells, Cultured | Integrin alpha6 | Leukemic Infiltration - metabolism | Interleukin-1 - metabolism | Integrin alpha4 | Integrin alpha5 | Cell Adhesion Molecules - biosynthesis | Neoplasm Proteins - biosynthesis | Cells, Cultured | Integrin beta1 - biosynthesis | Receptors, Lymphocyte Homing - biosynthesis | Leukemia, Myeloid - metabolism | Lewis X Antigen - biosynthesis | CD18 Antigens - biosynthesis
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
6. Full Text MicroRNA-181b regulates NF-κB-mediated vascular inflammation
by Sun, Xinghui and Icli, Basak and Wara, Akm Khyrul and Belkin, Nathan and He, Shaolin and Kobzik, Lester and Hunninghake, Gary M and Vera, Miguel Pinilla and Blackwell, Timothy S and Baron, Rebecca M and Feinberg, Mark W and Dolinay, Tamas and Gazourian, Lee and Fredenburgh, Laura and Massaro, Anthony and Choi, Augustine and MICU Registry
Journal of Clinical Investigation, ISSN 0021-9738, 06/2012, Volume 122, Issue 6, pp. 1973 - 1990
EC activation and dysfunction have been linked to a variety of vascular inflammatory disease states. The function of microRNAs (miRNAs) in vascular EC... 
TRANSCRIPTION FACTORS | MEDICINE, RESEARCH & EXPERIMENTAL | LEUKOCYTE ADHESION MOLECULE-1 | TUMOR-NECROSIS-FACTOR | DEPENDENT INDUCTION | ENDOTHELIAL-CELLS | IN-VIVO | ORGAN DYSFUNCTION | ACUTE MYELOID-LEUKEMIA | PROGNOSTIC-SIGNIFICANCE | EXPRESSION | alpha Karyopherins - metabolism | Endotoxemia - therapy | Leukocytes - pathology | Vasculitis - metabolism | Acute Lung Injury - therapy | Endotoxemia - genetics | Humans | NF-kappa B - immunology | Critical Illness | Acute Lung Injury - genetics | MicroRNAs - metabolism | NF-kappa B - metabolism | Endotoxemia - pathology | Vasculitis - immunology | Leukocytes - immunology | Vasculitis - genetics | alpha Karyopherins - immunology | E-Selectin - genetics | Endotoxemia - immunology | Vascular Cell Adhesion Molecule-1 - genetics | Acute Lung Injury - metabolism | Vascular Cell Adhesion Molecule-1 - immunology | Vasculitis - therapy | Lipopolysaccharides - toxicity | Endothelial Cells - metabolism | E-Selectin - metabolism | E-Selectin - immunology | MicroRNAs - immunology | Acute Lung Injury - pathology | Endothelial Cells - immunology | Animals | NF-kappa B - genetics | alpha Karyopherins - genetics | Acute Lung Injury - immunology | Mice | MicroRNAs - genetics | Endothelial Cells - pathology | Endotoxemia - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Leukocytes - metabolism
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
7. Full Text Association of cell adhesion molecule gene polymorphisms with recurrent aphthous stomatitis
by Alkhateeb, Asem and Karasneh, Jumana and Abbadi, Hebah and Hassan, Ahmad and Thornhill, Martin
Journal of Oral Pathology & Medicine, ISSN 0904-2512, 11/2013, Volume 42, Issue 10, pp. 741 - 746
Background Recurrent aphthous stomatitis (RAS) is a common oral ulcerative condition. At ulcer sites vascular adhesion molecule‐1 (VCAM‐1), E‐selectin and... 
polymorphism | E‐selectin | recurrent aphthous stomatitis | adhesion molecules | Recurrent aphthous stomatitis | E-selectin | Adhesion molecules | Polymorphism | ULCERS | IFN-GAMMA | IL-4 | PATHOLOGY | TUMOR-NECROSIS-FACTOR | DENTISTRY, ORAL SURGERY & MEDICINE | DISEASE | S128R POLYMORPHISM | ORAL ULCERATION | Cell Adhesion Molecules - genetics | Gene Frequency - genetics | Genetic Predisposition to Disease - genetics | Humans | Risk Factors | Thymine | Adenine | Linkage Disequilibrium - genetics | Genotype | Male | L-Selectin - genetics | Nerve Tissue Proteins - genetics | Haplotypes - genetics | Stomatitis, Aphthous - genetics | E-Selectin - genetics | Intercellular Adhesion Molecule-1 - genetics | Alleles | Adult | Female | Polymorphism, Genetic - genetics | Vascular Cell Adhesion Molecule-1 - genetics | Cytosine | Cohort Studies | Genetic research | Stomatitis | Stomatitis, Aphthous | Genetic aspects | Analysis | Genes
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
8. Full Text Factor VII activating protease (FSAP) regulates the expression of inflammatory genes in vascular smooth muscle and endothelial cells
by Byskov, Kristina and Boettger, Thomas and Ruehle, Paul F and Nielsen, Nis Valentin and Etscheid, Michael and Kanse, Sandip M
Atherosclerosis, ISSN 0021-9150, 10/2017, Volume 265, pp. 133 - 139
The factor VII activating protease (FSAP) knockout mice have a bigger neointima after vascular injury and a larger infarct volume after stroke. The Marburg I... 
Atherosclerosis | Protease activated receptors | Inflammation | Gene expression | FSAP | Thrombosis | HABP2 | CARDIAC & CARDIOVASCULAR SYSTEMS | RECEPTOR | RISK | NEOINTIMA FORMATION | PROTEOLYSIS | INDUCTION | IL-6 | INTERLEUKIN-6 EXPRESSION | PERIPHERAL VASCULAR DISEASE | MARBURG-I POLYMORPHISM | PROMOTES | Inflammation - pathology | Interleukin-8 - genetics | Receptor, PAR-1 - metabolism | Humans | Apoptosis - genetics | Gene Expression Profiling | Time Factors | Cyclooxygenase 2 - genetics | E-Selectin - genetics | Inflammation Mediators - metabolism | Serine Endopeptidases - genetics | Amphiregulin - metabolism | Vascular Cell Adhesion Molecule-1 - genetics | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Amphiregulin - genetics | Cell Proliferation - genetics | Interleukin-6 - genetics | Signal Transduction | Myocytes, Smooth Muscle - enzymology | Cells, Cultured | Gene Expression Regulation | E-Selectin - metabolism | Receptor, PAR-1 - genetics | Muscle, Smooth, Vascular - pathology | Human Umbilical Vein Endothelial Cells - enzymology | Cyclooxygenase 2 - metabolism | Inflammation - genetics | Serine Endopeptidases - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Inflammation - enzymology | Muscle, Smooth, Vascular - enzymology | Single nucleotide polymorphisms | Proteases | Genes | Endothelium | Anopheles | Platelet-derived growth factor | Analysis | Genetic research | Smooth muscle | Fibroblast growth factors
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
9. Full Text Relationship between gene expression and lung function in Idiopathic Interstitial Pneumonias
by Steele, Mark P and Luna, Leah G and Coldren, Christopher D and Murphy, Elissa and Hennessy, Corinne E and Heinz, David and Evans, Christopher M and Groshong, Steve and Cool, Carlyne and Cosgrove, Gregory P and Brown, Kevin K and Fingerlin, Tasha E and Schwarz, Marvin I and Schwartz, David A and Yang, Ivana V
BMC Genomics, ISSN 1471-2164, 10/2015, Volume 16, Issue 1, p. 869
Background: Idiopathic interstitial pneumonias (IIPs) are a group of heterogeneous, somewhat unpredictable diseases characterized by progressive scarring of... 
BIOTECHNOLOGY & APPLIED MICROBIOLOGY | E-SELECTIN | DISEASE | GENETICS & HEREDITY | LEVEL | CRYPTOGENIC FIBROSING ALVEOLITIS | HISTOLOGIC PATTERN | PULMONARY-FIBROSIS | BETA | Humans | Middle Aged | Male | Intracellular Signaling Peptides and Proteins - metabolism | Procollagen N-Endopeptidase - metabolism | E-Selectin - genetics | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Idiopathic Interstitial Pneumonias - physiopathology | Adult | Female | Receptor for Advanced Glycation End Products - genetics | Intracellular Signaling Peptides and Proteins - genetics | Serpins - genetics | Serpins - metabolism | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Procollagen N-Endopeptidase - genetics | Receptor for Advanced Glycation End Products - metabolism | Gene Expression Regulation | E-Selectin - metabolism | Lung - physiopathology | ADAMTS4 Protein | Proteins - genetics | ADAM Proteins - metabolism | Serpin E2 - genetics | ADAMTS9 Protein | Idiopathic Interstitial Pneumonias - genetics | Aged | ADAM Proteins - genetics | Serpin E2 - metabolism | Pneumonia | Bacterial pneumonia | Development and progression | Genetic aspects | Forced expiratory volume | Gene expression | Observations | Health aspects
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
10. Full Text Heterogeneous effect of two selectin gene polymorphisms on coronary artery disease risk: A meta-analysis
by Wu, Zhijun and Lou, Yuqing and Lu, Lin and Liu, Yan and Chen, Qiujing and Chen, Xin and Jin, Wei
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, p. e88152
Background: The selectins play important roles in the inflammatory process of coronary artery disease (CAD) and myocardial infarction (MI). Previous studies... 
DIABETES-MELLITUS | HEART-DISEASE | MYOCARDIAL-INFARCTION | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | P-SELECTIN | MECHANISMS | S128R POLYMORPHISM | ASSOCIATION | ATHEROSCLEROSIS RISK | ACC/AHA 2007 GUIDELINES | Myocardial Infarction - genetics | Genetic Predisposition to Disease | E-Selectin - genetics | Humans | Risk Factors | Coronary Artery Disease - genetics | Polymorphism, Genetic - genetics | P-Selectin - genetics | Case-Control Studies | Medical research | Information services | Genes | Medicine, Experimental | Genetic aspects | Heart attack | Risk factors | Information services industry | Genetic polymorphisms | Myocardial infarction | Heart attacks | Bias | Cardiovascular disease | Gene polymorphism | Data bases | Information systems | Cell adhesion & migration | Genotype & phenotype | Epidermal growth factor | Atherosclerosis | Search engines | Cardiology | Heart diseases | Coronary artery | Health risks | Risk reduction | Inflammation | Risk analysis | Angina pectoris | Coronary artery disease | Meta-analysis | Carriers | Scientific papers | Medicine | Studies | E-selectin | Hospitals | Coronary vessels | Alleles | Selectins | Infarction | Acute coronary syndromes | P-selectin | Polymorphism
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
11. Full Text PSGL-1 engagement by E-selectin signals through Src kinase Fgr and ITAM adapters DAP12 and FcRγ to induce slow leukocyte rolling
by Zarbock, Alexander and Abram, Clare L and Hundt, Matthias and Altman, Amnon and Lowell, Clifford A and Ley, Klaus
Journal of Experimental Medicine, ISSN 0022-1007, 09/2008, Volume 205, Issue 10, pp. 2339 - 2347
E-selectin binding to P-selectin glycoprotein ligand-1 (PSGL-1) can activate the beta(2) integrin lymphocyte function-associated antigen-1 by signaling through... 
I-LIKE DOMAIN | MEDICINE, RESEARCH & EXPERIMENTAL | IMMUNE-RESPONSE | ADHESION | ACTIVATION | PROTEIN-TYROSINE KINASES | FAMILY KINASES | P-SELECTIN | DEFICIENT MICE | IMMUNOLOGY | MYELOID CELLS | CUTTING EDGE | GTP-Binding Protein alpha Subunits, Gi-Go - genetics | Neutrophils - cytology | Membrane Glycoproteins - metabolism | Protein-Tyrosine Kinases - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Receptors, IgG - metabolism | Protein-Tyrosine Kinases - genetics | Receptors, IgG - genetics | E-Selectin - genetics | src-Family Kinases - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Proto-Oncogene Proteins - metabolism | Mice, Inbred C57BL | Neutrophils - physiology | Proto-Oncogene Proteins c-hck - metabolism | E-Selectin - metabolism | Proto-Oncogene Proteins - genetics | Membrane Glycoproteins - genetics | Mice, Knockout | Animals | Adaptor Proteins, Signal Transducing - genetics | Leukocyte Rolling - physiology | Proto-Oncogene Proteins c-hck - genetics | Signal Transduction - physiology | Mice | Enzyme Activation | Adaptor Proteins, Signal Transducing - metabolism | src-Family Kinases - genetics | GTP-Binding Protein alpha Subunits, Gi-Go - metabolism
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
12. Full Text Tricin, flavonoid from Njavara reduces inflammatory responses in hPBMCs by modulating the p38MAPK and PI3K/Akt pathways and prevents inflammation associated endothelial dysfunction in HUVECs
by Shalini, V and Pushpan, Chithra K and G., Sindhu and A., Jayalekshmy and A., Helen
Immunobiology, ISSN 0171-2985, 2015, Volume 221, Issue 2, pp. 137 - 144
Highlights • LPS induced hPBMCs and LPS induced HUVECs were used as the model systems. • Tricin acted mainly on p38MAPK and PI3K pathways of LPS induced... 
Allergy and Immunology | Advanced Basic Science | Lipopolysaccharide | Signaling | Inflammation | Tricin | Flavonoid | CELLS | ACTIVATED PROTEIN-KINASE | TRANSCRIPTION | ADHESION MOLECULES | IMMUNOLOGY | INHIBITION | P38 | GENE-EXPRESSION | TNF-ALPHA | NF-KAPPA-B | Chemokine CCL2 - immunology | Humans | Tumor Necrosis Factor-alpha - genetics | MAP Kinase Kinase 4 - immunology | Oryza - chemistry | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Phosphatidylinositol 3-Kinases - immunology | Cyclooxygenase 2 - genetics | E-Selectin - genetics | Human Umbilical Vein Endothelial Cells - cytology | Mitogen-Activated Protein Kinase 1 - genetics | Tumor Necrosis Factor-alpha - immunology | Vascular Cell Adhesion Molecule-1 - immunology | Mitogen-Activated Protein Kinase 1 - immunology | Human Umbilical Vein Endothelial Cells - drug effects | Leukocytes, Mononuclear - drug effects | Mitogen-Activated Protein Kinase 3 - genetics | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | p38 Mitogen-Activated Protein Kinases - immunology | Proto-Oncogene Proteins c-akt - immunology | Intercellular Adhesion Molecule-1 - genetics | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Lipopolysaccharides - pharmacology | MAP Kinase Kinase 4 - genetics | Leukocytes, Mononuclear - cytology | Flavonoids - isolation & purification | Plant Extracts - chemistry | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | NF-kappa B - immunology | Human Umbilical Vein Endothelial Cells - immunology | Anti-Inflammatory Agents, Non-Steroidal - isolation & purification | Proto-Oncogene Proteins c-akt - genetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Leukocytes, Mononuclear - immunology | MAP Kinase Kinase 4 - antagonists & inhibitors | Vascular Cell Adhesion Molecule-1 - genetics | Flavonoids - pharmacology | Cyclooxygenase 2 - immunology | Mitogen-Activated Protein Kinase 3 - immunology | Intercellular Adhesion Molecule-1 - immunology | Gene Expression Regulation | p38 Mitogen-Activated Protein Kinases - genetics | Chemokine CCL2 - genetics | E-Selectin - immunology | Phosphatidylinositol 3-Kinases - genetics | MAP Kinase Signaling System - drug effects | NF-kappa B - genetics | Protein Kinase Inhibitors - pharmacology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Prevention | Analysis | Endothelium | Flavonoids | Flavones | Bioflavonoids | Mitogens
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights