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Molecular Cell, ISSN 1097-2765, 10/2014, Volume 56, Issue 2, pp. 219 - 231
Proinflammatory stimuli elicit rapid transcriptional responses via transduced signals to master regulatory transcription factors. To explore the role of... 
RNA-POLYMERASE-II | SELECTIVE-INHIBITION | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL IDENTITY | DISEASE | GENE-EXPRESSION | ATHEROSCLEROSIS | BROMODOMAIN PROTEIN BRD4 | TRANSCRIPTIONAL ELONGATION | P-TEFB | BET BROMODOMAINS | CELL BIOLOGY | Endothelium, Vascular - cytology | Transcription, Genetic - drug effects | Endothelial Cells | Atherosclerosis - genetics | Humans | Regulatory Sequences, Nucleic Acid | NF-kappa B p50 Subunit - genetics | Transcription Factor RelA - genetics | Cell Movement - genetics | Cell Movement - immunology | NF-kappa B p50 Subunit - immunology | E-Selectin - biosynthesis | Transcription Factor RelA - immunology | Acetylation | Transcription Initiation, Genetic | Transcription Factors - immunology | Macrophages - immunology | Endothelium, Vascular - immunology | Atherosclerosis - immunology | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Transcription Factors - antagonists & inhibitors | Inflammation - immunology | Nuclear Proteins - immunology | Mice, Knockout | Azepines - pharmacology | Cell Adhesion - immunology | Triazoles - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Animals | Enhancer Elements, Genetic | Nuclear Proteins - antagonists & inhibitors | Inflammation - genetics | Protein Binding | RNA Polymerase II - genetics | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Histones - metabolism | SOXF Transcription Factors - genetics | Chromatin - genetics
Journal Article
Lymphatic Research and Biology, ISSN 1539-6851, 02/2018, Volume 16, Issue 1, pp. 43 - 55
Background: Lymphatic endothelial cells (LECs) derived from lymphatic malformations (LMs) bear activated PIK3CA alleles yet display an inflammatory gene... 
PIK3CA | endothelial cells | lymphatic malformations | MEDICINE, RESEARCH & EXPERIMENTAL | PHYSIOLOGY | PERCUTANEOUS SCLEROTHERAPY | PHOSPHORYLATION | PROTEIN-KINASE | CANCER | FAT STORAGE | PI3K | INSULIN-RESISTANCE | AMPK | MUTATIONS | LIPID DROPLETS | Interleukin-8 - genetics | Heme Oxygenase-1 - metabolism | Class I Phosphatidylinositol 3-Kinases - genetics | Vesicular Transport Proteins - metabolism | Humans | Aminoimidazole Carboxamide - pharmacology | Cyclooxygenase 2 - genetics | E-Selectin - genetics | Lipid Metabolism - genetics | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Repressor Proteins - metabolism | Histone Deacetylases - genetics | Interleukin-6 - genetics | Signal Transduction | Endothelial Cells - metabolism | Membrane Proteins - genetics | Lymphoid Tissue - metabolism | Repressor Proteins - genetics | Lymphatic Abnormalities - pathology | Lymphoid Tissue - pathology | Aminoimidazole Carboxamide - analogs & derivatives | Vascular Endothelial Growth Factor C - metabolism | Angiopoietin-like 4 Protein - metabolism | Mutation | Endothelial Cells - pathology | Autophagy-Related Proteins - metabolism | AMP-Activated Protein Kinases - genetics | Angiopoietin-like 4 Protein - genetics | Lymphatic Abnormalities - genetics | AMP-Activated Protein Kinases - metabolism | Class I Phosphatidylinositol 3-Kinases - metabolism | Ribonucleotides - pharmacology | Heme Oxygenase-1 - genetics | Membrane Proteins - metabolism | Autophagy-Related Proteins - genetics | Cell Line | Vesicular Transport Proteins - genetics | Gene Expression Regulation | Lymphatic Abnormalities - metabolism | E-Selectin - metabolism | Histone Deacetylases - metabolism | Transcription Factors - genetics | Vascular Endothelial Growth Factor C - genetics | Transcription Factors - metabolism | Ankyrins - genetics | Alleles | Ankyrins - metabolism | Cyclooxygenase 2 - metabolism | Primary Cell Culture | Endothelial Cells - drug effects
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2014, Volume 9, Issue 10, p. e110151
Aims: Inflammation in infective endocarditis (IE) is a complex network including interactions of inflammatory cytokines and other components of host response.... 
MORTALITY | HEART-VALVES | MULTIDISCIPLINARY SCIENCES | SUSCEPTIBILITY | SEPTIC SHOCK | PROMOTER POLYMORPHISM | IL-1-BETA | TNF-ALPHA | SEPSIS | SECRETION | EXPRESSION | Inflammation - pathology | Endocarditis - blood | Endocarditis - pathology | Protein Precursors - blood | Humans | Middle Aged | Tumor Necrosis Factor-alpha - blood | Tumor Necrosis Factor-alpha - genetics | E-Selectin - blood | Male | Interleukin-1beta - genetics | Inflammation - blood | Interleukin-1beta - blood | E-Selectin - genetics | Interleukin-6 - blood | Adult | Female | Intercellular Adhesion Molecule-1 - blood | Leukocyte Count | Endocarditis - genetics | Calcitonin Gene-Related Peptide | Calcitonin - blood | Protein Precursors - genetics | Genetic Association Studies | Interleukin-6 - genetics | Gene Frequency | Genotype | Calcitonin - genetics | Biomarkers - blood | Intercellular Adhesion Molecule-1 - genetics | Adolescent | Enterococcus - pathogenicity | Inflammation - genetics | Polymorphism, Single Nucleotide - genetics | Aged | Heart | Flow cytometry | C-reactive protein | Laboratories | Genes | Interleukin | Procalcitonin | Erythrocytes | Inflammatory response | Cardiovascular disease | Infections | Leukocytes | Blood | Subgroups | Interleukin 6 | Proteins | Cell adhesion | Toll-like receptors | Tumor necrosis factor-TNF | Bacteria | Bioindicators | Genotypes | Deoxyribonucleic acid--DNA | Pathogens | Statistical analysis | Cytokines | Blood & organ donations | Intercellular adhesion molecule 1 | Mortality | Blood cells | Inflammation | Tumor necrosis factor-α | Patients | Blood donors | White blood cells | Genetic variance | Cytometry | Gene frequency | Endocarditis | Interleukin 10 | Biomarkers | Sepsis | Deoxyribonucleic acid | DNA
Journal Article
MOLECULAR VISION, ISSN 1090-0535, 09/2015, Volume 21, pp. 1071 - 1084
Purpose: Activation of the IL-1/NF-kappa B inflammatory stress pathway and induction of SELE expression in the trabecular meshwork (TBM) is a marker for... 
AQUEOUS OUTFLOW PATHWAY | MATRIX METALLOPROTEINASES | WILD-TYPE | OPEN-ANGLE GLAUCOMA | OXIDATIVE STRESS | PROTEIN | GENE | BIOCHEMISTRY & MOLECULAR BIOLOGY | OPHTHALMOLOGY | INTRAOCULAR-PRESSURE | EXPRESSION | CALORIE RESTRICTION | Cytoskeletal Proteins - genetics | Glaucoma, Open-Angle - genetics | Transforming Growth Factor beta2 - metabolism | Humans | Actins - metabolism | Glycoproteins - metabolism | Glaucoma, Open-Angle - pathology | NF-kappa B - metabolism | Interleukin-1beta - genetics | Actins - genetics | E-Selectin - genetics | Forkhead Transcription Factors - metabolism | Interleukin-1beta - metabolism | Cytoskeletal Proteins - metabolism | Glaucoma, Open-Angle - metabolism | Lentivirus - genetics | Eye Proteins - genetics | Interleukin-6 - metabolism | Fibroblasts - metabolism | Glycoproteins - genetics | Trabecular Meshwork - pathology | Mutagenesis, Site-Directed | Genetic Vectors - chemistry | Interleukin-6 - genetics | Signal Transduction | Endothelial Cells - metabolism | Gene Expression Regulation | Genetic Vectors - metabolism | E-Selectin - metabolism | Inflammation | Fibroblasts - pathology | Forkhead Transcription Factors - genetics | Eye Proteins - metabolism | NF-kappa B - genetics | Models, Biological | Trabecular Meshwork - metabolism | Forkhead Box Protein O1 | Primary Cell Culture | Transforming Growth Factor beta2 - genetics | Endothelial Cells - pathology | Cell Line, Transformed | Amino Acid Substitution
Journal Article
Blood, ISSN 0006-4971, 04/2001, Volume 97, Issue 7, pp. 2121 - 2129
Leukostasis and tissue infiltration by leukemic cells are poorly understood life-threatening complications of acute leukemia. This study has tested the... 
SELECTIN GLYCOPROTEIN LIGAND-1 | INTERCELLULAR-ADHESION MOLECULE-1 | COLONY-STIMULATING FACTOR | IN-VIVO | P-SELECTIN | ACUTE MYELOID-LEUKEMIA | LEUKOCYTE ADHESION | HEMATOLOGY | NF-KAPPA-B | GRANULOCYTE-MACROPHAGE | HUMAN T-CELLS | Neoplastic Stem Cells - cytology | Lymphocyte Function-Associated Antigen-1 - biosynthesis | Coculture Techniques | Humans | Leukemia, Myeloid - genetics | Antigens, CD - genetics | Neoplastic Stem Cells - metabolism | E-Selectin - genetics | E-Selectin - biosynthesis | Leukostasis - genetics | Neoplasm Proteins - genetics | CD18 Antigens - genetics | Gene Expression Regulation, Leukemic | Cell Adhesion | Leukostasis - metabolism | Endothelium, Vascular - metabolism | Lymphocyte Function-Associated Antigen-1 - genetics | Macrophage-1 Antigen - genetics | Intercellular Adhesion Molecule-1 - genetics | Endothelium, Vascular - pathology | Vascular Cell Adhesion Molecule-1 - biosynthesis | Integrin beta1 - genetics | Macrophage-1 Antigen - biosynthesis | Receptors, Lymphocyte Homing - genetics | Tumor Necrosis Factor-alpha - metabolism | Cell Adhesion Molecules - genetics | Leukemic Infiltration - genetics | Lewis X Antigen - genetics | Antigens, CD - biosynthesis | Integrin alphaXbeta2 - biosynthesis | L-Selectin - genetics | Neoplasm Proteins - metabolism | L-Selectin - biosynthesis | Leukemia, Myeloid - pathology | Integrin alphaXbeta2 - genetics | Intercellular Adhesion Molecule-1 - biosynthesis | Vascular Cell Adhesion Molecule-1 - genetics | Tumor Cells, Cultured | Integrin alpha6 | Leukemic Infiltration - metabolism | Interleukin-1 - metabolism | Integrin alpha4 | Integrin alpha5 | Cell Adhesion Molecules - biosynthesis | Neoplasm Proteins - biosynthesis | Cells, Cultured | Integrin beta1 - biosynthesis | Receptors, Lymphocyte Homing - biosynthesis | Leukemia, Myeloid - metabolism | Lewis X Antigen - biosynthesis | CD18 Antigens - biosynthesis
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 06/2012, Volume 122, Issue 6, pp. 1973 - 1990
EC activation and dysfunction have been linked to a variety of vascular inflammatory disease states. The function of microRNAs (miRNAs) in vascular EC... 
TRANSCRIPTION FACTORS | MEDICINE, RESEARCH & EXPERIMENTAL | LEUKOCYTE ADHESION MOLECULE-1 | TUMOR-NECROSIS-FACTOR | DEPENDENT INDUCTION | ENDOTHELIAL-CELLS | IN-VIVO | ORGAN DYSFUNCTION | ACUTE MYELOID-LEUKEMIA | PROGNOSTIC-SIGNIFICANCE | EXPRESSION | alpha Karyopherins - metabolism | Endotoxemia - therapy | Leukocytes - pathology | Vasculitis - metabolism | Acute Lung Injury - therapy | Endotoxemia - genetics | Humans | NF-kappa B - immunology | Critical Illness | Acute Lung Injury - genetics | MicroRNAs - metabolism | NF-kappa B - metabolism | Endotoxemia - pathology | Vasculitis - immunology | Leukocytes - immunology | Vasculitis - genetics | alpha Karyopherins - immunology | E-Selectin - genetics | Endotoxemia - immunology | Vascular Cell Adhesion Molecule-1 - genetics | Acute Lung Injury - metabolism | Vascular Cell Adhesion Molecule-1 - immunology | Vasculitis - therapy | Lipopolysaccharides - toxicity | Endothelial Cells - metabolism | E-Selectin - metabolism | E-Selectin - immunology | MicroRNAs - immunology | Acute Lung Injury - pathology | Endothelial Cells - immunology | Animals | NF-kappa B - genetics | alpha Karyopherins - genetics | Acute Lung Injury - immunology | Mice | MicroRNAs - genetics | Endothelial Cells - pathology | Endotoxemia - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Leukocytes - metabolism
Journal Article
Atherosclerosis, ISSN 0021-9150, 10/2017, Volume 265, pp. 133 - 139
The factor VII activating protease (FSAP) knockout mice have a bigger neointima after vascular injury and a larger infarct volume after stroke. The Marburg I... 
Atherosclerosis | Protease activated receptors | Inflammation | Gene expression | FSAP | Thrombosis | HABP2 | CARDIAC & CARDIOVASCULAR SYSTEMS | RECEPTOR | RISK | NEOINTIMA FORMATION | PROTEOLYSIS | INDUCTION | IL-6 | INTERLEUKIN-6 EXPRESSION | PERIPHERAL VASCULAR DISEASE | MARBURG-I POLYMORPHISM | PROMOTES | Inflammation - pathology | Interleukin-8 - genetics | Receptor, PAR-1 - metabolism | Humans | Apoptosis - genetics | Gene Expression Profiling | Time Factors | Cyclooxygenase 2 - genetics | E-Selectin - genetics | Inflammation Mediators - metabolism | Serine Endopeptidases - genetics | Amphiregulin - metabolism | Vascular Cell Adhesion Molecule-1 - genetics | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Amphiregulin - genetics | Cell Proliferation - genetics | Interleukin-6 - genetics | Signal Transduction | Myocytes, Smooth Muscle - enzymology | Cells, Cultured | Gene Expression Regulation | E-Selectin - metabolism | Receptor, PAR-1 - genetics | Muscle, Smooth, Vascular - pathology | Human Umbilical Vein Endothelial Cells - enzymology | Cyclooxygenase 2 - metabolism | Inflammation - genetics | Serine Endopeptidases - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Inflammation - enzymology | Muscle, Smooth, Vascular - enzymology | Single nucleotide polymorphisms | Proteases | Genes | Endothelium | Anopheles | Platelet-derived growth factor | Analysis | Genetic research | Smooth muscle | Fibroblast growth factors
Journal Article
BMC Genomics, ISSN 1471-2164, 10/2015, Volume 16, Issue 1, p. 869
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2014, Volume 9, Issue 2, p. e88152
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 09/2008, Volume 205, Issue 10, pp. 2339 - 2347
Journal Article
Immunobiology, ISSN 0171-2985, 2015, Volume 221, Issue 2, pp. 137 - 144
Highlights • LPS induced hPBMCs and LPS induced HUVECs were used as the model systems. • Tricin acted mainly on p38MAPK and PI3K pathways of LPS induced... 
Allergy and Immunology | Advanced Basic Science | Lipopolysaccharide | Signaling | Inflammation | Tricin | Flavonoid | CELLS | ACTIVATED PROTEIN-KINASE | TRANSCRIPTION | ADHESION MOLECULES | IMMUNOLOGY | INHIBITION | P38 | GENE-EXPRESSION | TNF-ALPHA | NF-KAPPA-B | Chemokine CCL2 - immunology | Humans | Tumor Necrosis Factor-alpha - genetics | MAP Kinase Kinase 4 - immunology | Oryza - chemistry | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Phosphatidylinositol 3-Kinases - immunology | Cyclooxygenase 2 - genetics | E-Selectin - genetics | Human Umbilical Vein Endothelial Cells - cytology | Mitogen-Activated Protein Kinase 1 - genetics | Tumor Necrosis Factor-alpha - immunology | Vascular Cell Adhesion Molecule-1 - immunology | Mitogen-Activated Protein Kinase 1 - immunology | Human Umbilical Vein Endothelial Cells - drug effects | Leukocytes, Mononuclear - drug effects | Mitogen-Activated Protein Kinase 3 - genetics | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | p38 Mitogen-Activated Protein Kinases - immunology | Proto-Oncogene Proteins c-akt - immunology | Intercellular Adhesion Molecule-1 - genetics | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Lipopolysaccharides - pharmacology | MAP Kinase Kinase 4 - genetics | Leukocytes, Mononuclear - cytology | Flavonoids - isolation & purification | Plant Extracts - chemistry | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | NF-kappa B - immunology | Human Umbilical Vein Endothelial Cells - immunology | Anti-Inflammatory Agents, Non-Steroidal - isolation & purification | Proto-Oncogene Proteins c-akt - genetics | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Leukocytes, Mononuclear - immunology | MAP Kinase Kinase 4 - antagonists & inhibitors | Vascular Cell Adhesion Molecule-1 - genetics | Flavonoids - pharmacology | Cyclooxygenase 2 - immunology | Mitogen-Activated Protein Kinase 3 - immunology | Intercellular Adhesion Molecule-1 - immunology | Gene Expression Regulation | p38 Mitogen-Activated Protein Kinases - genetics | Chemokine CCL2 - genetics | E-Selectin - immunology | Phosphatidylinositol 3-Kinases - genetics | MAP Kinase Signaling System - drug effects | NF-kappa B - genetics | Protein Kinase Inhibitors - pharmacology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Prevention | Analysis | Endothelium | Flavonoids | Flavones | Bioflavonoids | Mitogens
Journal Article