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1. Full Text Bone vascular niche E-selectin induces mesenchymal-epithelial transition and Wnt activation in cancer cells to promote bone metastasis
by Esposito, Mark and Mondal, Nandini and Greco, Todd M and Wei, Yong and Spadazzi, Chiara and Lin, Song-Chang and Zheng, Hanqiu and Cheung, Corey and Magnani, John L and Lin, Sue-Hwa and Cristea, Ileana M and Sackstein, Robert and Kang, Yibin
Nature cell biology, ISSN 1465-7392, 05/2019, Volume 21, Issue 5, pp. 627 - 639
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Fucosyltransferases - genetics | Sialoglycoproteins - genetics | Cell Proliferation - genetics | Transcriptional Activation - genetics | Humans | Bone Neoplasms - secondary | Signal Transduction - genetics | Epithelial-Mesenchymal Transition - genetics | Bone Neoplasms - pathology | Cell Movement - genetics | Xenograft Model Antitumor Assays | Neoplasm Metastasis - genetics | Animals | Neoplasms - genetics | Wnt Signaling Pathway - genetics | E-Selectin - genetics | Neoplasm Metastasis - pathology | Receptors, Fibroblast Growth Factor - genetics | Cell Line, Tumor | Neoplastic Stem Cells - pathology | Bone Neoplasms - genetics | Mice | Neoplasms - pathology | Stem Cell Niche - genetics | Ethylenediaminetetraacetic acid | Metastasis | Cancer cells | Stem cells | Cancer | Medical examination | Growth | Development and progression | Bones | Health aspects | Risk factors | Cell adhesion molecules | Binding | Cell survival | Wnt protein | Mesenchyme | Organs | Metastases | E-selectin | Bone growth | Bone cancer | Colonization | Niches | Tumors | Index Medicus | Wnt signaling | α1-3 Fucosyltransferase | bone metastasis | mesenchymal-epithelial transition | Golgi glycoprotein 1 | tumor-stromal interaction | vascular niche | breast cancer | HCELL
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2. Full Text NF-κB Directs Dynamic Super Enhancer Formation in Inflammation and Atherogenesis
by Brown, Jonathan D and Lin, Charles Y and Duan, Qiong and Griffin, Gabriel and Federation, Alexander J and Paranal, Ronald M and Bair, Steven and Newton, Gail and Lichtman, Andrew H and Kung, Andrew L and Yang, Tianlun and Wang, Hong and Luscinskas, Francis W and Croce, Kevin J and Bradner, James E and Plutzky, Jorge
Molecular cell, ISSN 1097-2765, 10/2014, Volume 56, Issue 2, pp. 219 - 231
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Endothelium, Vascular - cytology | Transcription, Genetic - drug effects | Endothelial Cells | Atherosclerosis - genetics | Humans | Regulatory Sequences, Nucleic Acid | NF-kappa B p50 Subunit - genetics | Transcription Factor RelA - genetics | Cell Movement - genetics | Cell Movement - immunology | NF-kappa B p50 Subunit - immunology | E-Selectin - biosynthesis | Transcription Factor RelA - immunology | Acetylation | Transcription Initiation, Genetic | Transcription Factors - immunology | Macrophages - immunology | Endothelium, Vascular - immunology | Atherosclerosis - immunology | Signal Transduction | Mice, Inbred C57BL | Cells, Cultured | Transcription Factors - antagonists & inhibitors | Inflammation - immunology | Nuclear Proteins - immunology | Mice, Knockout | Azepines - pharmacology | Cell Adhesion - immunology | Triazoles - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Animals | Enhancer Elements, Genetic | Nuclear Proteins - antagonists & inhibitors | Inflammation - genetics | Protein Binding | RNA Polymerase II - genetics | Vascular Cell Adhesion Molecule-1 - biosynthesis | Mice | Histones - metabolism | SOXF Transcription Factors - genetics | Chromatin - genetics | Index Medicus
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3. Linkage of metabolic defects to activated PIK3CA alleles in endothelial cells derived from lymphatic malformation
by Glaser, Kathryn and Dickie, Peter and Neilson, Derek and Osborn, Alexander and Dickie, Belinda Hsi
Lymphatic research and biology, ISSN 1539-6851, 02/2018, Volume 16, Issue 1, pp. 43 - 55
PIK3CA | endothelial cells | lymphatic malformations | Physiology | Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Interleukin-8 - genetics | Heme Oxygenase-1 - metabolism | Class I Phosphatidylinositol 3-Kinases - genetics | Vesicular Transport Proteins - metabolism | Humans | Aminoimidazole Carboxamide - pharmacology | Cyclooxygenase 2 - genetics | E-Selectin - genetics | Lipid Metabolism - genetics | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Repressor Proteins - metabolism | Histone Deacetylases - genetics | Interleukin-6 - genetics | Signal Transduction | Endothelial Cells - metabolism | Membrane Proteins - genetics | Lymphoid Tissue - metabolism | Repressor Proteins - genetics | Lymphatic Abnormalities - pathology | Lymphoid Tissue - pathology | Aminoimidazole Carboxamide - analogs & derivatives | Vascular Endothelial Growth Factor C - metabolism | Angiopoietin-like 4 Protein - metabolism | Mutation | Endothelial Cells - pathology | Autophagy-Related Proteins - metabolism | AMP-Activated Protein Kinases - genetics | Angiopoietin-like 4 Protein - genetics | Lymphatic Abnormalities - genetics | AMP-Activated Protein Kinases - metabolism | Class I Phosphatidylinositol 3-Kinases - metabolism | Ribonucleotides - pharmacology | Heme Oxygenase-1 - genetics | Membrane Proteins - metabolism | Autophagy-Related Proteins - genetics | Cell Line | Vesicular Transport Proteins - genetics | Gene Expression Regulation | Lymphatic Abnormalities - metabolism | E-Selectin - metabolism | Histone Deacetylases - metabolism | Transcription Factors - genetics | Vascular Endothelial Growth Factor C - genetics | Transcription Factors - metabolism | Ankyrins - genetics | Alleles | Ankyrins - metabolism | Cyclooxygenase 2 - metabolism | Primary Cell Culture | Endothelial Cells - drug effects
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4. Full Text Multiple Genes for Essential-Hypertension Susceptibility on Chromosome 1q
by Chang, Yen-Pei Christy and Liu, Xin and Kim, James Dae Ok and Ikeda, Morna A and Layton, Marnie R and Weder, Alan B and Cooper, Richard S and Kardia, Sharon L.R and Rao, D.C and Hunt, Steve C and Luke, Amy and Boerwinkle, Eric and Chakravarti, Aravinda
American journal of human genetics, ISSN 0002-9297, 2007, Volume 80, Issue 2, pp. 253 - 264
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Fundamental and applied biological sciences. Psychology | Classical genetics, quantitative genetics, hybrids | Human | Cardiology. Vascular system | General aspects. Genetic counseling | Arterial hypertension. Arterial hypotension | Medical genetics | Biological and medical sciences | Molecular and cellular biology | Genetics of eukaryotes. Biological and molecular evolution | Medical sciences | Blood and lymphatic vessels | RGS Proteins - genetics | Genetic Predisposition to Disease | Sodium-Potassium-Exchanging ATPase - genetics | Humans | Middle Aged | Male | Phenotype | E-Selectin - genetics | Adolescent | Alleles | Adult | Female | Polymorphism, Single Nucleotide | Hypertension - genetics | Chromosomes, Human, Pair 1 - genetics | Genome, Human | Genetic Linkage | Hypertension | Genetic aspects | Heart diseases | Risk factors | Index Medicus
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5. Full Text Glaucomatous MYOC mutations activate the IL-1/NF-kappa B inflammatory stress response and the glaucoma marker SELE in trabecular meshwork cells
by Itakura, Tatsuo and Peters, Donna M and Fini, M. Elizabeth
Molecular vision, ISSN 1090-0535, 09/2015, Volume 21, pp. 1071 - 1084
Biochemistry & Molecular Biology | Ophthalmology | Life Sciences & Biomedicine | Science & Technology | Cytoskeletal Proteins - genetics | Glaucoma, Open-Angle - genetics | Transforming Growth Factor beta2 - metabolism | Humans | Actins - metabolism | Glycoproteins - metabolism | Glaucoma, Open-Angle - pathology | NF-kappa B - metabolism | Interleukin-1beta - genetics | Actins - genetics | E-Selectin - genetics | Forkhead Transcription Factors - metabolism | Interleukin-1beta - metabolism | Cytoskeletal Proteins - metabolism | Glaucoma, Open-Angle - metabolism | Lentivirus - genetics | Eye Proteins - genetics | Interleukin-6 - metabolism | Fibroblasts - metabolism | Glycoproteins - genetics | Trabecular Meshwork - pathology | Mutagenesis, Site-Directed | Genetic Vectors - chemistry | Interleukin-6 - genetics | Signal Transduction | Endothelial Cells - metabolism | Gene Expression Regulation | Genetic Vectors - metabolism | E-Selectin - metabolism | Inflammation | Fibroblasts - pathology | Forkhead Transcription Factors - genetics | Eye Proteins - metabolism | NF-kappa B - genetics | Models, Biological | Trabecular Meshwork - metabolism | Forkhead Box Protein O1 | Primary Cell Culture | Transforming Growth Factor beta2 - genetics | Endothelial Cells - pathology | Cell Line, Transformed | Amino Acid Substitution | Index Medicus
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6. Full Text MicroRNA-181b regulates NF-κB-mediated vascular inflammation
by Sun, Xinghui and Icli, Basak and Wara, Akm Khyrul and Belkin, Nathan and He, Shaolin and Kobzik, Lester and Hunninghake, Gary M and Vera, Miguel Pinilla and Blackwell, Timothy S and Baron, Rebecca M and Feinberg, Mark W and Dolinay, Tamas and Gazourian, Lee and Fredenburgh, Laura and Massaro, Anthony and Choi, Augustine and MICU Registry
The Journal of clinical investigation, ISSN 0021-9738, 06/2012, Volume 122, Issue 6, pp. 1973 - 1990
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | alpha Karyopherins - metabolism | Endotoxemia - therapy | Leukocytes - pathology | Vasculitis - metabolism | Acute Lung Injury - therapy | Endotoxemia - genetics | Humans | NF-kappa B - immunology | Critical Illness | Acute Lung Injury - genetics | MicroRNAs - metabolism | NF-kappa B - metabolism | Endotoxemia - pathology | Vasculitis - immunology | Leukocytes - immunology | Vasculitis - genetics | alpha Karyopherins - immunology | E-Selectin - genetics | Endotoxemia - immunology | Vascular Cell Adhesion Molecule-1 - genetics | Acute Lung Injury - metabolism | Vascular Cell Adhesion Molecule-1 - immunology | Vasculitis - therapy | Lipopolysaccharides - toxicity | Endothelial Cells - metabolism | E-Selectin - metabolism | E-Selectin - immunology | MicroRNAs - immunology | Acute Lung Injury - pathology | Endothelial Cells - immunology | Animals | NF-kappa B - genetics | alpha Karyopherins - genetics | Acute Lung Injury - immunology | Mice | MicroRNAs - genetics | Endothelial Cells - pathology | Endotoxemia - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Leukocytes - metabolism | Index Medicus | Abridged Index Medicus
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7. Full Text Critical role of C5a in sickle cell disease
by Vercellotti, Gregory M and Dalmasso, Agustin P and Schaid, Terry R and Nguyen, Julia and Chen, Chunsheng and Ericson, Marna E and Abdulla, Fuad and Killeen, Trevor and Lindorfer, Margaret A and Taylor, Ronald P and Belcher, John D
American journal of hematology, ISSN 0361-8609, 03/2019, Volume 94, Issue 3, pp. 327 - 337
Life Sciences & Biomedicine | Hematology | Science & Technology | Receptor, Anaphylatoxin C5a - genetics | Liver - pathology | Kidney - blood supply | Kidney - pathology | Humans | Male | Complement Membrane Attack Complex - immunology | Liver - immunology | Liver - drug effects | E-Selectin - genetics | Cerebrovascular Disorders - immunology | Anemia, Sickle Cell - immunology | Complement C3 - genetics | Disease Models, Animal | Vascular Cell Adhesion Molecule-1 - immunology | Kidney - drug effects | Signal Transduction | Toll-Like Receptor 4 - genetics | Mice, Transgenic | Toll-Like Receptor 4 - immunology | P-Selectin - genetics | Complement C3 - immunology | Intercellular Adhesion Molecule-1 - genetics | Lung - drug effects | Complement C5a - genetics | Cerebrovascular Disorders - genetics | Mice | Receptor, Anaphylatoxin C5a - antagonists & inhibitors | Anemia, Sickle Cell - genetics | Anemia, Sickle Cell - pathology | Cerebrovascular Disorders - drug therapy | Kidney - immunology | NF-kappa B - immunology | Anemia, Sickle Cell - drug therapy | Complement Membrane Attack Complex - genetics | Receptor, Anaphylatoxin C5a - immunology | Vascular Cell Adhesion Molecule-1 - genetics | P-Selectin - immunology | P-Selectin - antagonists & inhibitors | Lung - pathology | Intercellular Adhesion Molecule-1 - immunology | Gene Expression Regulation | Antibodies, Neutralizing - pharmacology | E-Selectin - immunology | Immunity, Innate | Complement C5a - antagonists & inhibitors | Liver - blood supply | Animals | NF-kappa B - genetics | Complement C5a - immunology | Lung - blood supply | Cerebrovascular Disorders - pathology | Lung - immunology | Analysis | Sickle cell anemia | Occlusion | Kidneys | Complement component C5a | Immunoglobulin G | Liver | Blocking antibodies | Inflammation | TLR4 protein | Leukocytes | Alternative pathway | Endothelial cells | Endothelium | Complement activation | Reperfusion | Ischemia | Rodents | Complement component C3 | Von Willebrand factor | Toll-like receptors | Hypoxia | Microvasculature | Skin | Sickle cell disease | Umbilical vein | Index Medicus
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8. Full Text Endothelial cell activation by myeloblasts: Molecular mechanisms of leukostasis and leukemic cell dissemination
by Stucki, Anne and Rivier, Anne-Sophie and Gikic, Milica and Monai, Natacha and Schapira, Marc and Spertini, Olivier
Blood, ISSN 0006-4971, 04/2001, Volume 97, Issue 7, pp. 2121 - 2129
Life Sciences & Biomedicine | Hematology | Science & Technology | Hematologic and hematopoietic diseases | Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis | Biological and medical sciences | Medical sciences | Neoplastic Stem Cells - cytology | Lymphocyte Function-Associated Antigen-1 - biosynthesis | Coculture Techniques | Humans | Leukemia, Myeloid - genetics | Antigens, CD - genetics | Neoplastic Stem Cells - metabolism | E-Selectin - genetics | E-Selectin - biosynthesis | Leukostasis - genetics | Neoplasm Proteins - genetics | CD18 Antigens - genetics | Gene Expression Regulation, Leukemic | Cell Adhesion | Leukostasis - metabolism | Endothelium, Vascular - metabolism | Lymphocyte Function-Associated Antigen-1 - genetics | Macrophage-1 Antigen - genetics | Intercellular Adhesion Molecule-1 - genetics | Endothelium, Vascular - pathology | Vascular Cell Adhesion Molecule-1 - biosynthesis | Integrin beta1 - genetics | Macrophage-1 Antigen - biosynthesis | Receptors, Lymphocyte Homing - genetics | Tumor Necrosis Factor-alpha - metabolism | Cell Adhesion Molecules - genetics | Leukemic Infiltration - genetics | Lewis X Antigen - genetics | Antigens, CD - biosynthesis | Integrin alphaXbeta2 - biosynthesis | L-Selectin - genetics | Neoplasm Proteins - metabolism | L-Selectin - biosynthesis | Leukemia, Myeloid - pathology | Integrin alphaXbeta2 - genetics | Intercellular Adhesion Molecule-1 - biosynthesis | Vascular Cell Adhesion Molecule-1 - genetics | Tumor Cells, Cultured | Integrin alpha6 | Leukemic Infiltration - metabolism | Interleukin-1 - metabolism | Integrin alpha4 | Integrin alpha5 | Cell Adhesion Molecules - biosynthesis | Neoplasm Proteins - biosynthesis | Cells, Cultured | Integrin beta1 - biosynthesis | Receptors, Lymphocyte Homing - biosynthesis | Leukemia, Myeloid - metabolism | Lewis X Antigen - biosynthesis | CD18 Antigens - biosynthesis | Index Medicus | Abridged Index Medicus
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