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1. Full Text E2f1-3 switch from activators in progenitor cells to repressors in differentiating cells
by Chong, Jean-Leon and Wang, Shu-Huei and Ouseph, Madhu and Leone, Gustavo and Pipas, James M and Robinson, Michael L and Wenzel, Pamela L and Cantalupo, Paul G and Sharma, Nidhi and Culp, Brian and Sáenz-Robles, M. Teresa and Nair, Vivek and Mezache, Louise and Winton, Douglas J and Hagan, John P and Sansom, Owen J and Chen, Danian and Ferrey, Antoney and Chen, Hui-Zi and Bremner, Rod and Trikha, Prashant and Gomez, Yorman M
Nature (London), ISSN 0028-0836, 12/2009, Volume 462, Issue 7275, pp. 930 - 934
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Fundamental and applied biological sciences. Psychology | Intestine. Mesentery | Vertebrates: digestive system | Biological and medical sciences | Medical sciences | Tumor cell | Tumors | General aspects (metabolism, cell proliferation, established cell line...) | E2F Transcription Factors - deficiency | Cell Cycle - genetics | Embryonic Stem Cells - metabolism | E2F2 Transcription Factor - genetics | Cell Proliferation | Embryonic Stem Cells - cytology | E2F2 Transcription Factor - deficiency | E2F2 Transcription Factor - metabolism | E2F Transcription Factors - metabolism | Embryo, Mammalian - metabolism | E2F1 Transcription Factor - deficiency | Repressor Proteins - deficiency | Female | Cell Differentiation | Repressor Proteins - metabolism | Retinoblastoma Protein - metabolism | Gene Expression Regulation | Repressor Proteins - genetics | E2F3 Transcription Factor - genetics | Mice, Transgenic | Retinoblastoma Protein - deficiency | E2F1 Transcription Factor - metabolism | E2F3 Transcription Factor - deficiency | Animals | Intestine, Small - cytology | Embryo, Mammalian - cytology | Alleles | E2F3 Transcription Factor - metabolism | Cell Cycle - physiology | Mice | Intestine, Small - metabolism | E2F1 Transcription Factor - genetics | E2F Transcription Factors - genetics | Apoptosis | Physiological aspects | Transcription factors | Genetic aspects | Research | Embryonic stem cells | Cell cycle | Rodents | Stem cells | Cell division | Gene expression | Independent sample | Index Medicus | E2F | tumor suppressor | cell cycle | retinoblastoma | Small intestine
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2. Full Text Canonical and atypical E2Fs regulate the mammalian endocycle
by Chen, Hui-Zi and Ouseph, Madhu M and Li, Jing and Pécot, Thierry and Chokshi, Veda and Kent, Lindsey and Bae, Sooin and Byrne, Morgan and Duran, Camille and Comstock, Grant and Trikha, Prashant and Mair, Markus and Senapati, Shantibhusan and Martin, Chelsea K and Gandhi, Sagar and Wilson, Nicholas and Liu, Bin and Huang, Yi-Wen and Thompson, John C and Raman, Sundaresan and Singh, Shantanu and Leone, Marcelo and Machiraju, Raghu and Huang, Kun and Mo, Xiaokui and Fernandez, Soledad and Kalaszczynska, Ilona and Wolgemuth, Debra J and Sicinski, Piotr and Huang, Tim and Jin, Victor and Leone, Gustavo
Nature cell biology, ISSN 1465-7392, 11/2012, Volume 14, Issue 11, pp. 1192 - 1202
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Immunohistochemistry | Cell Cycle - genetics | E2F2 Transcription Factor - genetics | Hepatocytes - metabolism | E2F7 Transcription Factor - genetics | E2F7 Transcription Factor - metabolism | E2F2 Transcription Factor - metabolism | E2F Transcription Factors - metabolism | Hepatocytes - cytology | Flow Cytometry | Chromatin Immunoprecipitation | Female | Repressor Proteins - metabolism | Microscopy, Electron, Transmission | Trophoblasts - metabolism | Giant Cells - metabolism | Repressor Proteins - genetics | E2F3 Transcription Factor - genetics | Giant Cells - cytology | E2F1 Transcription Factor - metabolism | Pregnancy | Microscopy, Confocal | Animals | E2F3 Transcription Factor - metabolism | Cell Cycle - physiology | Mice | E2F1 Transcription Factor - genetics | E2F Transcription Factors - genetics | Microscopy, Fluorescence | Physiological aspects | DNA synthesis | Transcription factors | Research | Cell cycle | Index Medicus
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3. Full Text Division and apoptosis of E2f-deficient retinal progenitors
by Chen, Danian and Pacal, Marek and Wenzel, Pamela and Knoepfler, Paul S and Leone, Gustavo and Bremner, Rod
Nature (London), ISSN 0028-0836, 12/2009, Volume 462, Issue 7275, pp. 925 - 929
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Fundamental and applied biological sciences. Psychology | Vertebrates: nervous system and sense organs | Eye and associated structures. Visual pathways and centers. Vision | Biological and medical sciences | Medical sciences | Tumor cell | Tumors | General aspects (metabolism, cell proliferation, established cell line...) | E2F Transcription Factors - deficiency | Sirtuin 1 - metabolism | E2F2 Transcription Factor - genetics | Retina - metabolism | E2F2 Transcription Factor - deficiency | Stem Cells - cytology | Stem Cells - metabolism | E2F2 Transcription Factor - metabolism | E2F Transcription Factors - metabolism | E2F1 Transcription Factor - deficiency | Neuroglia - cytology | Retina - cytology | Cell Division | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Fibroblasts | Acetylation | Cell Survival | Tumor Suppressor Protein p53 - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - antagonists & inhibitors | E2F3 Transcription Factor - genetics | E2F1 Transcription Factor - metabolism | E2F3 Transcription Factor - deficiency | Mice, Knockout | Animals | E2F3 Transcription Factor - metabolism | Neuroglia - metabolism | Mice | E2F1 Transcription Factor - genetics | E2F Transcription Factors - genetics | Apoptosis | Physiological aspects | Retina | Genetic aspects | DNA binding proteins | Research | Proteins | Studies | Rodents | Cyclin-dependent kinases | Kinases | Index Medicus
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4. Full Text Transcription factor C/EBP-β induces tumor-suppressor phosphatase PHLPP2 through repression of the miR-17-92 cluster in differentiating AML cells
by Yan, Y and Hanse, E.A and Stedman, K and Benson, J.M and Lowman, X.H and Subramanian, S and Kelekar, A
Cell death and differentiation, ISSN 1350-9047, 07/2016, Volume 23, Issue 7, pp. 1232 - 1242
Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | MicroRNAs - antagonists & inhibitors | Humans | Leukemia, Myeloid, Acute - metabolism | Phosphoprotein Phosphatases - metabolism | Transcriptional Activation - drug effects | MicroRNAs - metabolism | E2F Transcription Factors - metabolism | Base Sequence | Antineoplastic Agents - pharmacology | 3' Untranslated Regions | Tretinoin - pharmacology | Promoter Regions, Genetic | Leukemia, Myeloid, Acute - pathology | CCAAT-Enhancer-Binding Protein-beta - genetics | Proto-Oncogene Proteins c-myc - metabolism | CCAAT-Enhancer-Binding Protein-beta - metabolism | Up-Regulation - drug effects | Bcl-2-Like Protein 11 - metabolism | Phosphoprotein Phosphatases - genetics | Mutagenesis | Cell Differentiation - drug effects | Cell Line, Tumor | HL-60 Cells | MicroRNAs - genetics | Antagomirs - metabolism | Index Medicus | Original Paper
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5. Full Text Redeployment of Myc and E2f1-3 drives Rb-deficient cell cycles
by Liu, Huayang and Tang, Xing and Srivastava, Arunima and Pécot, Thierry and Daniel, Piotr and Hemmelgarn, Benjamin and Reyes, Stephan and Fackler, Nicholas and Bajwa, Amneet and Kladney, Raleigh and Koivisto, Christopher and Chen, Zhong and Wang, Qianben and Huang, Kun and Machiraju, Raghu and Sáenz-Robles, Maria Teresa and Cantalupo, Paul and Pipas, James M and Leone, Gustavo
Nature cell biology, ISSN 1465-7392, 08/2015, Volume 17, Issue 8, pp. 1036 - 1048
Life Sciences & Biomedicine | Science & Technology | Cell Biology | E2F Transcription Factors - deficiency | E2F2 Transcription Factor - genetics | Cell Proliferation | Epithelial Cells - metabolism | Intestine, Small - pathology | Male | Mice, 129 Strain | E2F2 Transcription Factor - metabolism | E2F Transcription Factors - metabolism | S Phase Cell Cycle Checkpoints | Time Factors | G1 Phase Cell Cycle Checkpoints | Female | Transcription, Genetic | Binding Sites | Promoter Regions, Genetic | Signal Transduction | Mice, Inbred C57BL | Gene Expression Regulation | Chromatin Assembly and Disassembly | E2F3 Transcription Factor - genetics | Epithelial Cells - pathology | Genotype | Retinoblastoma Protein - deficiency | E2F1 Transcription Factor - metabolism | Proto-Oncogene Proteins c-myc - metabolism | G2 Phase Cell Cycle Checkpoints | Mice, Knockout | Cell Cycle Checkpoints | Phenotype | Animals | Retinoblastoma Protein - genetics | Proto-Oncogene Proteins c-myc - deficiency | E2F3 Transcription Factor - metabolism | Proto-Oncogene Proteins c-myc - genetics | Intestine, Small - metabolism | E2F1 Transcription Factor - genetics | E2F Transcription Factors - genetics | Cell proliferation | Transcription factors | Genetic aspects | Genetic transcription | Properties | Health aspects | Index Medicus | Life Sciences | Animal genetics | Genetics | Molecular biology | Biochemistry, Molecular Biology | Cancer
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6. Full Text E2F transcription factor 1 regulates cellular and organismal senescence by inhibiting forkhead box O transcription factors
by Xie, Qi and Peng, Shengyi and Tao, Li and Ruan, Haihe and Yang, Yanglu and Li, Tie-Mei and Adams, Ursula and Meng, Songshu and Bi, Xiaolin and Dong, Meng-Qiu and Yuan, Zengqiang
The Journal of biological chemistry, ISSN 0021-9258, 12/2014, Volume 289, Issue 49, pp. 34205 - 34213
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | RNA, Small Interfering - genetics | Superoxide Dismutase - genetics | Embryo, Mammalian | Humans | Caenorhabditis elegans Proteins - metabolism | E2F Transcription Factors - metabolism | Aging - genetics | Forkhead Transcription Factors - metabolism | Gene Expression Regulation, Developmental | HEK293 Cells | E2F1 Transcription Factor - antagonists & inhibitors | Protein Interaction Domains and Motifs | Binding Sites | Superoxide Dismutase - metabolism | Fibroblasts - metabolism | Cell Line | Cellular Senescence - genetics | Caenorhabditis elegans - metabolism | Signal Transduction | Caenorhabditis elegans - genetics | Catalase - genetics | E2F1 Transcription Factor - metabolism | Longevity - genetics | Transcription Factors - genetics | Forkhead Transcription Factors - genetics | Catalase - metabolism | Transcription Factors - metabolism | Animals | Protein Binding | Fibroblasts - cytology | Mice | E2F1 Transcription Factor - genetics | Forkhead Box Protein O3 | Caenorhabditis elegans Proteins - genetics | E2F Transcription Factors - genetics | Aging - metabolism | RNA, Small Interfering - metabolism | Index Medicus | FOXO | Transcription Regulation | Senescence | Oxidative Stress | E2F Transcription Factor
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7. Full Text The AP-1 transcription factor regulates breast cancer cell growth via cyclins and E2F factors
by Shen, Q and Uray, I.P and Li, Y and Krisko, T.I and Strecker, T.E and Kim, H.-T and Brown, P.H
Oncogene, ISSN 0950-9232, 01/2008, Volume 27, Issue 3, pp. 366 - 377
Gene expression regulation | Proliferation arrest | Cyclin D1 | AP-1 factor | Breast cancer cells | E2F factors | Biochemistry & Molecular Biology | Oncology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Fundamental and applied biological sciences. Psychology | Gynecology. Andrology. Obstetrics | Mammary gland diseases | Biological and medical sciences | Cell physiology | Molecular and cellular biology | Medical sciences | Cell transformation and carcinogenesis. Action of oncogenes and antioncogenes | Tumors | Cyclin D1 - metabolism | E2F2 Transcription Factor - genetics | Cell Proliferation | Transcription Factor DP1 - metabolism | Humans | Cyclins - genetics | Gene Expression Regulation, Neoplastic | RNA, Messenger - metabolism | E2F2 Transcription Factor - metabolism | Transcription Factor AP-1 - metabolism | E2F Transcription Factors - metabolism | Cyclin D1 - antagonists & inhibitors | Chromatin Immunoprecipitation | Cyclins - metabolism | RNA, Messenger - antagonists & inhibitors | Female | E2F4 Transcription Factor - metabolism | Promoter Regions, Genetic | Peptide Fragments - metabolism | Down-Regulation | E2F1 Transcription Factor - metabolism | E2F4 Transcription Factor - genetics | Transcription Factor AP-1 - antagonists & inhibitors | Breast Neoplasms - genetics | Cyclin D1 - genetics | Breast Neoplasms - pathology | Proto-Oncogene Proteins c-jun - metabolism | Cell Line, Tumor | E2F1 Transcription Factor - genetics | E2F Transcription Factors - genetics | Care and treatment | Cellular signal transduction | Genetic aspects | Breast cancer | Research | DNA binding proteins | Health aspects | Proteins | Signal transduction | Cell growth | Cyclin-dependent kinases | Gene expression | Cells | Index Medicus
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8. Full Text The transcription factor p53: Not a repressor, solely an activator
by Fischer, Martin and Steiner, Lydia and Engeland, Kurt
Cell cycle (Georgetown, Tex.), ISSN 1538-4101, 10/2014, Volume 13, Issue 19, pp. 3037 - 3058
E2F/RB complex | CDKN1A | DREAM complex | genome-wide meta-analysis | Genome-wide meta-analysis | E2F/FB complex | p53 | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Promoter Regions, Genetic | Meta-Analysis as Topic | RNA, Untranslated - metabolism | Signal Transduction | HCT116 Cells | Humans | Retinoblastoma Protein - metabolism | Transcriptional Activation | Tumor Suppressor Protein p53 - metabolism | Repressor Proteins - genetics | RNA, Messenger - metabolism | E2F Transcription Factors - metabolism | Tumor Suppressor Protein p53 - genetics | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Chromatin Immunoprecipitation | Retinoblastoma Protein - genetics | E2F Transcription Factors - chemistry | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Protein Binding | Tumor Suppressor Protein p53 - chemistry | Binding Sites | E2F Transcription Factors - genetics | Real-Time Polymerase Chain Reaction | Repressor Proteins - metabolism | Index Medicus
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