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humans (253) 253
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Oncogene, ISSN 0950-9232, 01/2008, Volume 27, Issue 3, pp. 366 - 377
The activating protein-1 (AP-1) transcription factor transduces growth signals through signal transduction pathways to the nucleus, leading to the expression... 
Gene expression regulation | Proliferation arrest | Cyclin D1 | AP-1 factor | Breast cancer cells | E2F factors | TRANSFORMATION | proliferation arrest | breast cancer cells | KINASE-ACTIVITY | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | FOS | CELL BIOLOGY | gene expression regulation | OVEREXPRESSION | GENE | ONCOLOGY | GENETICS & HEREDITY | BINDING-SITE | cyclin D1 | DIFFERENTIATION | C-JUN | EXPRESSION | Cyclin D1 - metabolism | E2F2 Transcription Factor - genetics | Cell Proliferation | Transcription Factor DP1 - metabolism | Humans | Cyclins - genetics | Gene Expression Regulation, Neoplastic | RNA, Messenger - metabolism | E2F2 Transcription Factor - metabolism | Transcription Factor AP-1 - metabolism | E2F Transcription Factors - metabolism | Cyclin D1 - antagonists & inhibitors | Chromatin Immunoprecipitation | Cyclins - metabolism | RNA, Messenger - antagonists & inhibitors | Female | E2F4 Transcription Factor - metabolism | Promoter Regions, Genetic | Peptide Fragments - metabolism | Down-Regulation | E2F1 Transcription Factor - metabolism | E2F4 Transcription Factor - genetics | Transcription Factor AP-1 - antagonists & inhibitors | Breast Neoplasms - genetics | Cyclin D1 - genetics | Breast Neoplasms - pathology | Proto-Oncogene Proteins c-jun - metabolism | Cell Line, Tumor | E2F1 Transcription Factor - genetics | E2F Transcription Factors - genetics | Care and treatment | Cellular signal transduction | Genetic aspects | Breast cancer | Research | DNA binding proteins | Health aspects | Proteins | Signal transduction | Cell growth | Cyclin-dependent kinases | Oncology | Gene expression | Cells
Journal Article
Genes and Development, ISSN 0890-9369, 04/2011, Volume 25, Issue 8, pp. 801 - 813
In the absence of growth signals, cells exit the cell cycle and enter into G0 or quiescence. Alternatively, cells enter senescence in response to inappropriate... 
Phosphorylation | Down syndrome | Growth arrest | Retinoblastoma-like protein p130 | Cell cycle | Cellular senescence | PRB | cell cycle | DEVELOPMENTAL BIOLOGY | CELL-CYCLE CONTROL | IDENTIFICATION | DROSOPHILA | CELL BIOLOGY | cellular senescence | CAENORHABDITIS-ELEGANS | IN-VIVO | DOWN-SYNDROME | GENETICS & HEREDITY | growth arrest | E2F | TUMOR-SUPPRESSOR | B-MYB | phosphorylation | retinoblastoma-like protein p130 | Cell Cycle - genetics | NIH 3T3 Cells | Cell Proliferation | Transcription Factor DP1 - metabolism | Protein-Tyrosine Kinases - metabolism | Humans | Retinoblastoma-Binding Protein 4 - genetics | Transcription Factor DP1 - genetics | Protein-Tyrosine Kinases - genetics | Retinoblastoma-Like Protein p130 - metabolism | Tumor Suppressor Proteins - genetics | Trans-Activators - genetics | Nuclear Proteins - genetics | E2F4 Transcription Factor - metabolism | Protein-Serine-Threonine Kinases - metabolism | Retinoblastoma-Binding Protein 4 - metabolism | Cell Line | Cellular Senescence - genetics | Tumor Suppressor Proteins - metabolism | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Cellular Senescence - physiology | Nuclear Proteins - metabolism | E2F4 Transcription Factor - genetics | Animals | Cell Cycle - physiology | Cell Line, Tumor | Trans-Activators - metabolism | Mice | Retinoblastoma-Like Protein p130 - genetics | Ras genes | Analysis | Aging | Physiological aspects | Genetic aspects | Research | Research Paper
Journal Article
Journal Article
Developmental Cell, ISSN 1534-5807, 05/2017, Volume 41, Issue 4, pp. 382 - 391.e5
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 01/2010, Volume 30, Issue 2, pp. 524 - 536
Journal Article
Journal Article
Journal Article
Cell Cycle, ISSN 1538-4101, 12/2016, Volume 15, Issue 23, pp. 3183 - 3190
The E2F family of transcription factors is a key determinant of cell proliferation in response to extra- and intra-cellular signals. Within this family, E2F4... 
RB | stem cells | development | differentiation | cell cycle | regeneration | E2F | cancer | E2F4 | GENOTOXIC STRESS | EMBRYONIC STEM | CELL-CYCLE PROGRESSION | CELL BIOLOGY | REPRESSOR COMPLEX | TARGET GENES | DISTINCT MECHANISMS | PROSTATE CARCINOMA | IN-VIVO | GENE-EXPRESSION | TUMOR-SUPPRESSOR | Animals | Cell Cycle | Disease | Models, Biological | Humans | Models, Animal | Embryonic Development | E2F4 Transcription Factor - metabolism
Journal Article
Cell Cycle, ISSN 1538-4101, 10/2014, Volume 13, Issue 19, pp. 3037 - 3058
The predominant function of the tumor suppressor p53 is transcriptional regulation. It is generally accepted that p53-dependent transcriptional activation... 
E2F/RB complex | CDKN1A | DREAM complex | genome-wide meta-analysis | p53 | Genome-wide meta-analysis | E2F/FB complex | WILD-TYPE P53 | NUCLEAR ANTIGEN PROMOTER | CDE | DREAM | CHR | ChIP | TUMOR-SUPPRESSOR P53 | chromatin immunoprecipitation | HPV | CELL BIOLOGY | AUTOREGULATORY FEEDBACK LOOP | cell cycle genes homology region | MANGANESE SUPEROXIDE-DISMUTASE | E2F | cell cycle-dependent element | E2F4 | and MuvB complex | Nuclear factor Y | RB-like | NF-Y | STRESS-INDUCED APOPTOSIS | cdk | human papilloma virus | ALPHA-FETOPROTEIN GENE | BREAST-CANCER CELLS | RB complex | P53-DEPENDENT DOWN-REGULATION | cyclin-dependent kinase | MAMMARY EPITHELIAL-CELLS | Promoter Regions, Genetic | Meta-Analysis as Topic | RNA, Untranslated - metabolism | Signal Transduction | HCT116 Cells | Humans | Retinoblastoma Protein - metabolism | Transcriptional Activation | Tumor Suppressor Protein p53 - metabolism | Repressor Proteins - genetics | RNA, Messenger - metabolism | E2F Transcription Factors - metabolism | Tumor Suppressor Protein p53 - genetics | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Chromatin Immunoprecipitation | Retinoblastoma Protein - genetics | E2F Transcription Factors - chemistry | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Protein Binding | Tumor Suppressor Protein p53 - chemistry | Binding Sites | E2F Transcription Factors - genetics | Real-Time Polymerase Chain Reaction | Repressor Proteins - metabolism
Journal Article