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Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 2/2001, Volume 98, Issue 4, pp. 1519 - 1524
The E2F transcription factors play a key role in the regulation of cellular proliferation and terminal differentiation. E2F6 is the most recently identified... 
Proteins | Yeasts | Biological Sciences | Immunoprecipitation | Plasmids | DNA | Genes | Antibodies | Amino acids | Repression | Western blotting | GROUP GENES | FACTOR YY1 | APOPTOSIS | retinoblastoma protein pathway | S-PHASE ENTRY | INTERACTS | MULTIDISCIPLINARY SCIENCES | MYC TRANSGENIC MICE | CELL-PROLIFERATION | FAMILY PROTEINS | REPRESSOR | DROSOPHILA | Cell proliferation | Research | Retinoblastoma
Journal Article
The Journal of Pathology, ISSN 0022-3417, 12/2004, Volume 204, Issue 5, pp. 528 - 537
The Polycomb group (PcG) of proteins comprises a family of repressors of homeobox genes that play key roles in body formation, haematopoiesis and cell cycle... 
Polycomb | Hodgkin | NFκB | tissue microarray | E2F6 | lymphoma | STEM-CELLS | BMI-1 | PROLIFERATION | PATHOLOGY | TARGET GENES | DISEASE TUMOR-CELLS | ONCOLOGY | POLYCOMB-GROUP GENES | INTERACTS | MYC TRANSGENIC MICE | GROUP-COMPLEXES | NF kappa B | STERNBERG CELLS | Tissue microarray | Lymphoma
Journal Article
Oncogene, ISSN 0950-9232, 01/2006, Volume 25, Issue 2, pp. 230 - 239
ARHI is a maternally imprinted tumor suppressor gene whose expression is markedly downregulated in breast cancer. Reactivation of ARHI expression in breast... 
E2F | Breast cancer | ARHI | Transcriptional regulation | HDAC | Tumor suppressor gene | TRANSCRIPTION FACTORS | METHYLATION | transcriptional regulation | BIOCHEMISTRY & MOLECULAR BIOLOGY | breast cancer | CELL-CYCLE PROGRESSION | CELL BIOLOGY | CARCINOGENESIS | REPLICATION | REPRESSION | ONCOLOGY | HISTONE H3 | IN-VIVO | GENETICS & HEREDITY | EXPRESSION | tumor suppressor gene | E2F2 Transcription Factor - genetics | Epithelial Cells - metabolism | Luciferases - metabolism | Humans | Mammary Glands, Human - metabolism | Bone Neoplasms - pathology | E2F2 Transcription Factor - metabolism | Promoter Regions, Genetic - genetics | Bone Neoplasms - metabolism | Breast Neoplasms - metabolism | E2F2 Transcription Factor - antagonists & inhibitors | Cell Nucleus - metabolism | Chromatin Immunoprecipitation | E2F6 Transcription Factor - antagonists & inhibitors | Female | Bone Neoplasms - genetics | E2F1 Transcription Factor - antagonists & inhibitors | Electrophoretic Mobility Shift Assay | Acetylation | Epithelial Cells - cytology | Binding Sites | E2F4 Transcription Factor - metabolism | Genes, Tumor Suppressor | Hydroxamic Acids - pharmacology | Osteosarcoma - metabolism | Mammary Glands, Human - cytology | E2F6 Transcription Factor - metabolism | Response Elements | Gene Expression Regulation - genetics | rho GTP-Binding Proteins - genetics | RNA, Small Interfering - pharmacology | Retinoblastoma Protein - metabolism | Cells, Cultured | E2F6 Transcription Factor - genetics | Enzyme Inhibitors - pharmacology | Histone Deacetylases - metabolism | E2F1 Transcription Factor - metabolism | E2F4 Transcription Factor - genetics | Breast Neoplasms - genetics | Breast Neoplasms - pathology | rho GTP-Binding Proteins - metabolism | Histone Deacetylase Inhibitors | Histones - metabolism | Osteosarcoma - genetics | E2F1 Transcription Factor - genetics | E2F4 Transcription Factor - antagonists & inhibitors | Osteosarcoma - pathology
Journal Article
Biochemical Journal, ISSN 0264-6021, 11/2004, Volume 383, Issue 3, pp. 529 - 536
E2F6 is widely expressed in human tissues and cell lines. Recent studies have demonstrated its involvement in developmental patterning and in the regulation of... 
Chromatin immunoprecipitation (ChIP) | E2F gene | NRF-1/α-PAL (nuclear respiratory factor-1/α-palindrome-binding protein) | Promoter analysis | Gene regulation | DNA-BINDING | NUCLEAR RESPIRATORY FACTOR-1 | EUKARYOTIC INITIATION FACTOR-2-ALPHA | BIOCHEMISTRY & MOLECULAR BIOLOGY | FACTOR-I | NRF-1/alpha-PAL (nuclear respiratory factor-1/alpha-palindrome-binding protein) | IDENTIFICATION | chromatin immunoprecipitation (ChIP) | CYTOCHROME-C | GENE | gene regulation | promoter analysis | CELL-CYCLE | REPRESSOR | EXPRESSION | Conserved Sequence - genetics | Protein Binding - genetics | Transcriptional Activation - genetics | Conserved Sequence - physiology | DNA, Neoplasm - metabolism | Humans | Molecular Sequence Data | Bone Neoplasms - pathology | DNA Footprinting - methods | Nuclear Respiratory Factors | Trans-Activators - physiology | Base Sequence | Binding Sites - physiology | Female | E2F6 Transcription Factor | Nuclear Respiratory Factor 1 | DNA-Binding Proteins - physiology | Transcription Factors - physiology | Rats | Binding Sites - genetics | Transcription Factors - genetics | Deoxyribonuclease I - metabolism | Sequence Alignment - methods | Animals | Promoter Regions, Genetic - physiology | Breast Neoplasms - pathology | Cell Line, Tumor | NF-E2-Related Factor 1 | Mice | Osteosarcoma - pathology | Protein Binding - physiology | α-palindrome-binding protein | α-PAL, nuclear respiratory factor-1 | eIF, eukaryotic translation-initiation factor | PcG, polycomb group proteins | ICAM-1, intercellular cell-adhesion molecule 1 | FMR1, fragile X mental retardation 1 | DBD-Gal4, DNA-binding domain of Gal4 | ChIP, chromatin immunoprecipitation | EMSA, electrophoretic mobility-shift assay | α-PAL (nuclear respiratory factor-1 | IvT, in vitro translated | NRF-1 | DN-NRF-1, dominant-negative NRF-1
Journal Article
Journal Article
Journal Article