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Nature, ISSN 0028-0836, 01/2016, Volume 529, Issue 7587, pp. 541 - 545
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2012, Volume 7, Issue 11, p. e49589
Journal Article
Journal Article
Scientific Reports, ISSN 2045-2322, 2015, Volume 5, Issue 1, p. 9382
Chronic infection by high risk human papillomavirus (HPV) strains may lead to cancer. Expression of the two viral oncoproteins E6 and E7 is largely responsible... 
ONCOPROTEIN | CERVICAL-CARCINOMA CELLS | MECHANISM | PDZ DOMAINS | DIMERIC INHIBITOR | MULTIDISCIPLINARY SCIENCES | HIGH-AFFINITY | PHAGE DISPLAY | ASSOCIATION | BINDING-PROTEINS | P53 | Adaptor Proteins, Signal Transducing - chemistry | Immunoprecipitation | Oncogene Proteins, Viral - chemistry | Humans | Molecular Sequence Data | Peptide Library | DNA-Binding Proteins - metabolism | Escherichia coli - metabolism | Oncogene Proteins, Viral - genetics | Membrane Proteins - metabolism | Human papillomavirus 16 - chemistry | Binding Sites | Repressor Proteins - metabolism | Oncogene Proteins, Viral - metabolism | Recombinant Proteins - metabolism | Amino Acid Sequence | Gene Expression | Repressor Proteins - chemistry | Protein Structure, Secondary | Membrane Proteins - genetics | Models, Molecular | Recombinant Proteins - chemistry | Repressor Proteins - genetics | Recombinant Proteins - genetics | Human papillomavirus 18 - chemistry | DNA-Binding Proteins - genetics | DNA-Binding Proteins - chemistry | PDZ Domains - genetics | Membrane Proteins - chemistry | Escherichia coli - genetics | Adaptor Proteins, Signal Transducing - genetics | Protein Binding | HeLa Cells | Adaptor Proteins, Signal Transducing - metabolism | Ubiquitin | E6 protein | Risk groups | Chronic infection | C-Terminus | Proteins | Phage display | Human papillomavirus | Immortalization | N-Terminus | Affinity | Ubiquitin-protein ligase | Cancer | Medical Biotechnology | Medical and Health Sciences | Medicin och hälsovetenskap | Medical Biotechnology (with a focus on Cell Biology (including Stem Cell Biology), Molecular Biology, Microbiology, Biochemistry or Biopharmacy) | Medicinsk bioteknologi (med inriktning mot cellbiologi (inklusive stamcellsbiologi), molekylärbiologi, mikrobiologi, biokemi eller biofarmaci) | Medicinsk bioteknologi
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2000, Volume 97, Issue 23, pp. 12513 - 12518
Most cervical carcinomas express high-risk human papillomaviruses (HPVs) E6 and E7 proteins, which neutralize cellular tumor suppressor function. To determine... 
Biological Sciences | Carcinoma | Messenger RNA | RNA | DNA | Cell lines | Cell cycle | HeLa cells | Infections | Repression | Cyclins | HPV18 REGULATORY REGION | GROWTH ARREST | MULTIDISCIPLINARY SCIENCES | RETINOBLASTOMA GENE-PRODUCT | CYCLE PROGRESSION | FAMILY PROTEINS | BINDING-SITES | E2 PROTEIN | P53 | Uterine Cervical Neoplasms | Tumor Suppressor Protein p53 - biosynthesis | Humans | E2F Transcription Factors | Papillomavirus E7 Proteins | Retinoblastoma-Like Protein p107 | Proto-Oncogene Proteins - biosynthesis | Viral Proteins - metabolism | Papillomaviridae - genetics | Bovine papillomavirus 1 - genetics | DNA-Binding Proteins - metabolism | Proteins | Cyclins - biosynthesis | Cattle | DNA - biosynthesis | Female | Oncogene Proteins, Viral - genetics | Bovine papillomavirus 1 - metabolism | Nuclear Proteins - genetics | Genes, Tumor Suppressor | Oncogenes | Repressor Proteins - metabolism | Carrier Proteins | Signal Transduction | Cyclin-Dependent Kinase Inhibitor p21 | Repressor Proteins - genetics | Viral Proteins - genetics | Phosphoproteins - genetics | DNA-Binding Proteins - genetics | Transcription Factor DP1 | Transcription Factors - metabolism | Proto-Oncogene Proteins c-mdm2 | Animals | Gene Expression Regulation, Viral | Retinoblastoma Protein - genetics | Retinoblastoma-Like Protein p130 | HeLa Cells | Retinoblastoma-Binding Protein 1 | Cell Cycle Proteins | Papillomaviruses | Tumor suppressor genes | Genetic aspects | Analysis | Cervical cancer
Journal Article
Virology, ISSN 0042-6822, 2016, Volume 492, pp. 145 - 154
Abstract Asian-American (AA) HPV-16 variants are associated with higher risk of cancer. Abnormal activation of intracellular signaling play a critical role in... 
Infectious Disease | HPV-16 | Transformation | Molecular variants | Intracellular signaling | Migration | PROTEIN | 16 E6 VARIANTS | KINASE | RISK | CERVICAL-CANCER | SUFFICIENT | VIROLOGY | GENE | UP-REGULATION | IMMORTALIZATION | RNA, Small Interfering - genetics | Cell Proliferation | Keratinocytes - virology | Humans | Glycogen Synthase Kinase 3 beta | Male | Phosphatidylinositol 3-Kinases - metabolism | Proto-Oncogene Proteins c-akt - genetics | Papillomavirus E7 Proteins - genetics | MAP Kinase Kinase 1 - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Oncogene Proteins, Viral - genetics | p38 Mitogen-Activated Protein Kinases - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Repressor Proteins - metabolism | Oncogene Proteins, Viral - metabolism | MAP Kinase Kinase 1 - antagonists & inhibitors | Transduction, Genetic | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Gene Expression Regulation | Repressor Proteins - genetics | p38 Mitogen-Activated Protein Kinases - genetics | Genotype | MAP Kinase Kinase 1 - metabolism | Glycogen Synthase Kinase 3 - metabolism | Human papillomavirus 16 - metabolism | Host-Pathogen Interactions | Phosphatidylinositol 3-Kinases - genetics | Keratinocytes - pathology | Papillomavirus E7 Proteins - metabolism | Glycogen Synthase Kinase 3 - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Keratinocytes - metabolism | Primary Cell Culture | Human papillomavirus 16 - genetics | Cell Movement | Mitogen-Activated Protein Kinase 1 - metabolism | RNA, Small Interfering - metabolism | Proteins | Medical colleges | Development and progression | Papillomavirus infections | Analysis | Stem cells | Tyrosine | Evaluation | Glycogen | Heat shock proteins | Papillomaviruses | Synthesis | Exhibitions | Mitogens | Health aspects | Protein kinases | Protein binding | Index Medicus
Journal Article
Molecular Cell, ISSN 1097-2765, 2006, Volume 22, Issue 6, pp. 783 - 794
Journal Article
PLOS PATHOGENS, ISSN 1553-7366, 12/2017, Volume 13, Issue 12, p. e1006781
Papillomavirus E6 proteins bind to LXXLL peptide motifs displayed on targeted cellular proteins. Alpha genus HPV E6 proteins associate with the cellular... 
CANINE PAPILLOMAVIRUS | EPIDERMODYSPLASIA-VERRUCIFORMIS | CETACEAN PAPILLOMAVIRUSES | FAMILY PAPILLOMAVIRIDAE | MICROBIOLOGY | MULTIPLE SEQUENCE ALIGNMENT | COMPLETE GENOME SEQUENCE | VIROLOGY | CUTANEOUS BETA | SQUAMOUS-CELL CARCINOMA | MOLECULAR-CLONING | NUCLEOTIDE-SEQUENCE | PARASITOLOGY | Amino Acid Sequence | Cell Line | Papillomaviridae - metabolism | Repressor Proteins - chemistry | Oncogene Proteins, Viral - chemistry | Species Specificity | Humans | Ubiquitin-Protein Ligases - metabolism | Models, Molecular | Repressor Proteins - genetics | Phylogeny | Papillomaviridae - genetics | DNA-Binding Proteins - metabolism | Host-Pathogen Interactions | Papillomaviridae - pathogenicity | Transcription Factors - metabolism | Animals | HEK293 Cells | Protein Binding | Oncogene Proteins, Viral - genetics | Protein Interaction Domains and Motifs | Repressor Proteins - metabolism | Evolution, Molecular | Oncogene Proteins, Viral - metabolism | Ubiquitin | Papillomaviruses | Cellular proteins | Peptides | Ligases | Amino acids | Papillomavirus infections | Porpoises | Protein binding | Physiological aspects | Genetic aspects | Research | E6 protein | Divergence | Transcription | Genes | Viruses | Marine mammals | Degradation | Proteins | Human papillomavirus | Phylogenetics | Evolution | Bioinformatics | Ubiquitin-protein ligase | Binding | Pigs | Pathology | Signaling | Acids | Notch protein | Mutation | Dolphins | Structure-function relationships
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2016, Volume 11, Issue 2, p. e0149845
Journal Article