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Nature Communications, ISSN 2041-1723, 2014, Volume 5, Issue 1, pp. 5811 - 5811
Epidermal growth factor receptor (EGFR) overexpression plays an important oncogenic role in cancer. Regular EGFR protein levels are increased in cancer cells... 
AUTOCRINE LOOP | RECEPTOR TYROSINE KINASES | MULTIDISCIPLINARY SCIENCES | EPIDERMAL-GROWTH-FACTOR | GENE-EXPRESSION | HUMAN GLIOMAS | CANCER-THERAPY | TRANSGENIC MICE | INNATE IMMUNITY | BRAIN-TUMORS | FACTOR-ALPHA | Receptor, Epidermal Growth Factor - genetics | Shc Signaling Adaptor Proteins - metabolism | Apoptosis - drug effects | Humans | Brain Neoplasms - pathology | Gene Expression Regulation, Neoplastic | Extracellular Signal-Regulated MAP Kinases - metabolism | Extracellular Signal-Regulated MAP Kinases - genetics | Interferon Regulatory Factor-3 - genetics | Brain Neoplasms - metabolism | Proto-Oncogene Proteins c-akt - genetics | Receptor, Epidermal Growth Factor - metabolism | Glioblastoma - genetics | Glioblastoma - metabolism | Antineoplastic Agents - pharmacology | Membrane Proteins - metabolism | Phosphorylation - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Protein-Serine-Threonine Kinases - metabolism | Membrane Proteins - genetics | Protein-Serine-Threonine Kinases - genetics | Brain Neoplasms - genetics | Signal Transduction - genetics | TNF-Related Apoptosis-Inducing Ligand - metabolism | Carrier Proteins - genetics | Carrier Proteins - metabolism | Shc Signaling Adaptor Proteins - genetics | Glioblastoma - pathology | Cell Line, Tumor | Interferon Regulatory Factor-3 - metabolism | Epidermal Growth Factor - pharmacology | Primary Cell Culture | TNF-Related Apoptosis-Inducing Ligand - genetics | Index Medicus
Journal Article
Nature Cell Biology, ISSN 1465-7392, 01/2016, Volume 18, Issue 2, pp. 213 - 224
Although long non-coding RNAs (lncRNAs) predominately reside in the nucleus and exert their functions in many biological processes, their potential involvement... 
METASTASIS | LONG NONCODING RNAS | TYROSINE KINASE | EPIDERMAL-GROWTH-FACTOR | BINDING PROTEINS | DISEASE | EPIGENETIC REGULATION | HYPOXIA | TUMOR-GROWTH | PROMOTES | CELL BIOLOGY | Heparin-binding EGF-like Growth Factor - pharmacology | Phosphorylation | Prognosis | Membrane Glycoproteins - metabolism | Protein-Tyrosine Kinases - metabolism | Humans | Multiprotein Complexes | Serine | Gene Expression Regulation, Neoplastic | Proline | Cytoplasm - metabolism | E1A-Associated p300 Protein - metabolism | Neoplasm Proteins - metabolism | Receptor, Epidermal Growth Factor - metabolism | Transfection | RNA Interference | Time Factors | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Triple Negative Breast Neoplasms - pathology | Female | Transcription, Genetic | Protein Stability | Protein-Serine-Threonine Kinases - metabolism | Tyrosine | Hydroxylation | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | RNA, Long Noncoding - genetics | Leucine-Rich Repeat Serine-Threonine Protein Kinase-2 | Animals | Triple Negative Breast Neoplasms - genetics | Signal Transduction - drug effects | Mice, Nude | Cell Line, Tumor | Glycolysis | RNA, Long Noncoding - metabolism | Index Medicus | glycoprotein non-metastatic b (GPNMB) | Epidermal Growth Factor Receptor (EGFR) | Triple-negative Breast Cancer (TNBC) | Long Noncoding RNA | Signaling Transduction | Glycolysis Reprogramming | HIF1α
Journal Article
Clinical Cancer Research, ISSN 1078-0432, 11/2017, Volume 23, Issue 22, pp. 7084 - 7096
Journal Article
Cancer Science, ISSN 1347-9032, 12/2018, Volume 109, Issue 12, pp. 3874 - 3882
Autocrine and paracrine factors, including glutamate and epidermal growth factor ( EGF ), are potent inducers of brain tumor cell invasion, a pathological... 
xCT | glioma | epidermal growth factor receptor | GluN2B | glutamate | MIGRATION | CELLS | TYROSINE PHOSPHORYLATION | NMDA RECEPTORS | EGFR | INVASION | ONCOLOGY | BIOLOGY | DEFICIENT | EXPRESSION | MODULATION | Neoplasm Transplantation | Phosphorylation | Humans | Receptors, N-Methyl-D-Aspartate - metabolism | Sulfasalazine - pharmacology | Brain Neoplasms - metabolism | Glioma - metabolism | Amino Acid Transport System y+ - metabolism | Receptors, N-Methyl-D-Aspartate - chemistry | Dizocilpine Maleate - administration & dosage | Protein Domains | Cell Survival - drug effects | ErbB Receptors - metabolism | Brain Neoplasms - drug therapy | Disease Progression | Drug Synergism | Sulfasalazine - administration & dosage | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Glutamic Acid - metabolism | Mice | Epidermal Growth Factor - pharmacology | Dizocilpine Maleate - pharmacology | Glioma - drug therapy | Methyl aspartate | Epidermal growth factor | Gliomas | Brain tumors | Development and progression | Aspartate | Glutamate | Immunoglobulins | Cell survival | Epidermal growth factor receptors | Paracrine signalling | N-Methyl-D-aspartic acid receptors | Glutamic acid receptors | Kinases | Cell adhesion & migration | Glioma cells | Conflicts of interest | Sulfasalazine | Autocrine signalling | Cell migration | Tumors | Index Medicus | Original
Journal Article
Cancer, ISSN 0008-543X, 10/2014, Volume 120, Issue 19, pp. 2980 - 2985
Simultaneous targeting of the IGF‐1R and EGFR signaling pathways for more effective downstream inhibition of proliferation and survival did not improve... 
pancreatic cancer | erlotinib signaling | randomized phase II | IGF‐1R | cixutumumab | EGFR | targeted treatment | Erlotinib signaling | Targeted treatment | Randomized phase II | Pancreatic cancer | Cixutumumab | IGF-1R | CARCINOMA-CELLS | DUCTAL ADENOCARCINOMA | MONOCLONAL-ANTIBODY | SINGLE-AGENT CETUXIMAB | BREAST-CANCER | INHIBITION | K-RAS | ONCOLOGY | THERAPEUTIC TARGET | C-MET | RESISTANCE | Erlotinib Hydrochloride | Pancreatic Neoplasms - metabolism | Humans | Middle Aged | Antibodies, Monoclonal - adverse effects | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Receptor, Epidermal Growth Factor - drug effects | Male | Insulin-Like Growth Factor I - drug effects | Pancreatic Neoplasms - drug therapy | Receptor, Epidermal Growth Factor - metabolism | Adenocarcinoma - metabolism | Adult | Deoxycytidine - adverse effects | Female | Quinazolines - administration & dosage | Drug Administration Schedule | Deoxycytidine - administration & dosage | Pancreatic Neoplasms - pathology | Kaplan-Meier Estimate | Treatment Outcome | Adenocarcinoma - drug therapy | Adenocarcinoma - secondary | Disease-Free Survival | Signal Transduction - drug effects | Antibodies, Monoclonal - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Quinazolines - adverse effects | Aged | Deoxycytidine - analogs & derivatives | Insulin-Like Growth Factor I - metabolism | Index Medicus | Abridged Index Medicus
Journal Article
Journal Article
Journal of Investigative Dermatology, ISSN 0022-202X, 02/2019, Volume 139, Issue 2, pp. 369 - 379
Kindler syndrome is an autosomal recessive genodermatosis that results from mutations in the gene encoding t kindlin-1. Kindlin-1 localizes to focal adhesion... 
KINDLER-SYNDROME | LOCALIZATION | DOMAIN | FOCAL ADHESION | PROTEIN | GENE | MIGFILIN | INTEGRIN | MUTATIONS | CELL | DERMATOLOGY | MST, microscale thermophoresis | FRET, fluorescence resonance energy transfer | KS, Kindler syndrome | PBS, phosphate buffered saline | WT, wild type | EGFR, epidermal growth factor receptor | IP, immunoprecipitation
Journal Article
Cancer Research, ISSN 0008-5472, 06/2015, Volume 75, Issue 12, pp. 2489 - 2500
Journal Article
Oncogene, ISSN 0950-9232, 02/2016, Volume 35, Issue 6, pp. 738 - 747
Journal Article
Cancer Discovery, ISSN 2159-8274, 2012, Volume 2, Issue 10, pp. 934 - 947
Journal Article