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Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 09/2011, Volume 108, Issue 37, pp. 15196 - 15200
Journal Article
PloS one, ISSN 1932-6203, 11/2014, Volume 9, Issue 11, p. e113330
Mitochondria provide energy in form of ATP in eukaryotic cells. However, it is not known when, during embryonic cardiac development, mitochondria become able... 
PERMEABILITY TRANSITION PORE | CYTOCHROME-C-OXIDASE | OXIDATIVE-PHOSPHORYLATION | RESPIRATORY-CHAIN | FUNCTIONAL ASSAYS | PROTEIN | METABOLISM | MULTIDISCIPLINARY SCIENCES | IN-VIVO | DIFFERENTIATION | CELL-DEATH | Electron Transport Complex III - genetics | Electron Transport Complex III - metabolism | Electron Transport Complex II - genetics | Mitochondria, Heart - metabolism | Age Factors | Embryo, Mammalian | Electron Transport - genetics | Mice, Inbred C57BL | Protein Multimerization | Oxidative Phosphorylation | Electron Transport Complex IV - genetics | Electron Transport Complex I - metabolism | Electron Transport Complex IV - metabolism | Mitochondrial Membranes - metabolism | Animals | Electron Transport Complex I - genetics | Electron Transport Complex II - metabolism | Gene Expression Regulation, Developmental | Myocardium - metabolism | Mice | Mitochondria, Heart - genetics | Heart | Embryonic development | Mitochondrial DNA | Electron transport | Cytochrome | Tricarboxylic acid cycle | Pediatrics | Phosphorylation | Immunoprecipitation | Sucrose | Activation | Krebs cycle | Proteins | Electron transport chain | Mitochondria | Embryogenesis | Ubiquinone | Bioenergetics | Cardiology | Heart diseases | Medical research | Oxygen | Oxygen consumption | Permeability | Embryos | Cytochrome c | Oxidative phosphorylation | Respiration | Laboratory animals | Electrons
Journal Article
The EMBO Journal, ISSN 0261-4189, 11/2011, Volume 30, Issue 22, pp. 4652 - 4664
Journal Article
American journal of human genetics, ISSN 0002-9297, 07/2010, Volume 87, Issue 1, pp. 52 - 59
Journal Article
PLoS genetics, ISSN 1553-7404, 2012, Volume 8, Issue 1, p. e1002456
Pink1 is a mitochondrial kinase involved in Parkinson's disease, and loss of Pink1 function affects mitochondrial morphology via a pathway involving Parkin and... 
LIFE-SPAN | OXIDATIVE STRESS | DROSOPHILA-PARKIN MUTANTS | ALTERNATIVE OXIDASE | QUINONE OXIDOREDUCTASE | NDI1 GENE | HUMAN-CELLS | INCREASED SENSITIVITY | GENETICS & HEREDITY | MITOCHONDRIAL-DYSFUNCTION | RESERVE POOL | Electron Transport Complex III - genetics | Electron Transport Complex III - metabolism | Cytoskeletal Proteins - genetics | Saccharomyces cerevisiae - genetics | Humans | Male | Drosophila Proteins - metabolism | Ciona intestinalis - genetics | Drosophila melanogaster - genetics | Electron Transport Complex I - metabolism | GTP-Binding Proteins - genetics | Electron Transport Complex IV - metabolism | Drosophila melanogaster - metabolism | Mitochondria - genetics | Electron Transport Complex I - genetics | Mitochondrial Proteins - metabolism | Cytoskeletal Proteins - metabolism | Membrane Proteins - metabolism | Plant Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Oxidoreductases - metabolism | Membrane Proteins - genetics | Gene Expression Regulation | Protein-Serine-Threonine Kinases - genetics | Ubiquitin-Protein Ligases - metabolism | Mitochondria - metabolism | Electron Transport Complex IV - genetics | Parkinson Disease - genetics | Saccharomyces cerevisiae Proteins - genetics | Animals, Genetically Modified - metabolism | Animals | Animals, Genetically Modified - genetics | Saccharomyces cerevisiae Proteins - metabolism | Drosophila Proteins - genetics | Mutation | Ubiquitin-Protein Ligases - genetics | GTP-Binding Proteins - metabolism | Parkinson's disease | Physiological aspects | NADH dehydrogenase | Genetic aspects | Mitochondrial DNA | Research | Electron transport | Risk factors | Enzymes | Mitochondria | Yeast | Insects | Parkinsons disease | Genetic engineering | Grants | Experiments | Evacuations & rescues | Deoxyribonucleic acid--DNA | Defects | Deoxyribonucleic acid | DNA
Journal Article
Current genetics, ISSN 1432-0983, 2019, Volume 65, Issue 6, pp. 1347 - 1353
The respiratory chain has been proposed as an attractive target for the development of new therapies to tackle human fungal pathogens. This arises from the... 
Biochemistry, general | Yeast | Microbiology | Respirometry | Metabolism | Cell Biology | Life Sciences | Mitochondria | Microbial Genetics and Genomics | Candida | Proteomics | Respiration | Plant Sciences | COMPLEX I | OXIDATIVE STRESS | ALTERNATIVE OXIDASE | SUSCEPTIBILITY | CANDIDA-ALBICANS | GENETICS & HEREDITY | RESISTANCE | OXIDE-RELEASING NANOPARTICLES | FLUCONAZOLE | Antifungal Agents - pharmacology | Candida albicans - pathogenicity | Virulence - drug effects | Electron Transport Complex III - metabolism | Fungi - drug effects | Oxidoreductases - metabolism | Oxidoreductases - drug effects | Humans | Electron Transport Complex III - drug effects | Mitochondria - metabolism | Mitochondria - drug effects | Candida albicans - growth & development | Fungi - metabolism | Electron Transport Complex I - drug effects | Electron Transport Complex I - metabolism | Antifungal Agents - therapeutic use | Electron Transport Complex IV - metabolism | Candida albicans - drug effects | Electron Transport Complex II - metabolism | Electron Transport Complex IV - drug effects | Mycoses - drug therapy | Fungi - pathogenicity | Drug Therapy, Combination | Electron Transport Complex II - drug effects | Fungi, Pathogenic | Usage | Control | Drug targeting | Virulence (Microbiology) | Health aspects | Methods | Fungi | Electron transport chain | Pathogens | Inhibitors | Fungicides | Cell walls | Pathogenesis | Virulence | Electron transport | Mini-Review
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