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Journal Article
Journal of Cellular Physiology, ISSN 0021-9541, 03/2019, Volume 234, Issue 3, pp. 2916 - 2928
Vascular endothelial cell (VEC) dysfunction plays an important role in the ischemia–reperfusion injury (IRI)‐related diseases, and microRNAs (miRNAs) are key... 
microRNA‐26a | PFKFB3 | vascular endothelial cell injury | ischemia–reperfusion injury | AMPK pathway | microRNA-26a | MIGRATION | APOPTOSIS | PHYSIOLOGY | VON-WILLEBRAND-FACTOR | DAMAGE | CELL BIOLOGY | ischemia-reperfusion injury | BREAST-CANCER | IN-VITRO | LOWER-LIMB ISCHEMIA | NITRIC-OXIDE | EXPRESSION | ISCHEMIA/REPERFUSION | Cell Proliferation - genetics | Reperfusion Injury - pathology | Endothelial Cells - metabolism | Gene Expression Regulation - genetics | Humans | Rats | Apoptosis - genetics | Male | Signal Transduction - genetics | von Willebrand Factor - genetics | Nitric Oxide Synthase Type III - genetics | Phosphorylation - genetics | Animals | Nitric Oxide - genetics | Phosphofructokinase-2 - genetics | Superoxide Dismutase-1 - genetics | MicroRNAs - genetics | Thrombomodulin - genetics | Endothelial Cells - pathology | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | AMP-Activated Protein Kinases - genetics | Disease Models, Animal | Endothelin | MicroRNA | Analysis | Nitric oxide | Von Willebrand factor | Genetic engineering | Superoxide | Fructose | Reperfusion injury | Endothelium | Cell proliferation | Phosphorylation | Animal models | Superoxide dismutase | Kinases | Tissues | Proteins | Reperfusion | Ischemia | Restoration | miRNA | Thrombomodulin | Injuries | Adenosine monophosphate | AMP | Ribonucleic acid--RNA | Malondialdehyde | Nitric-oxide synthase | Endothelial cells | Polymerase chain reaction | Gene silencing | Cell injury | Protein kinase | Adenosine kinase | Vascular tissue
Journal Article
Arthritis & Rheumatology, ISSN 2326-5191, 11/2018, Volume 70, Issue 11, pp. 1879 - 1889
Objective Immune complex (IC) deposition activates polymorphonuclear neutrophils (PMNs), increases vascular permeability, and leads to organ damage in systemic... 
Human Umbilical Vein Endothelial Cells | Receptors, Lysosphingolipid - antagonists & inhibitors | Cadherins - metabolism | Skin - metabolism | Humans | Capillary Permeability - drug effects | Adherens Junctions - metabolism | Capillary Permeability - genetics | Antigens, CD - metabolism | Oxadiazoles - pharmacology | Apolipoproteins M - pharmacology | Lysophospholipids - pharmacology | Receptors, Lysosphingolipid - genetics | Lung - metabolism | Antigen-Antibody Complex - metabolism | Arthus Reaction | Receptors, Lysosphingolipid - metabolism | Cardiac Myosins - metabolism | Cardiac Myosins - drug effects | Endothelial Cells - metabolism | Thiophenes - pharmacology | Myosin Light Chains - drug effects | Antigens, CD - drug effects | Cadherins - drug effects | Mice, Knockout | Skin - blood supply | Sphingosine - pharmacology | Organophosphonates - pharmacology | Sphingosine - analogs & derivatives | Animals | Lung - drug effects | Adherens Junctions - drug effects | Anilides - pharmacology | Receptors, Lysosphingolipid - agonists | Mice | Myosin Light Chains - metabolism | Lung - blood supply | Skin - drug effects | Endothelial Cells - drug effects | Indans - pharmacology | Phosphates | Phosphorylation | Target recognition | Lipids | Antagonists | Arthritis | Contraction | Systemic lupus erythematosus | Myosin | Leukocytes (polymorphonuclear) | Attenuation | Capillaries | Deposition | Arthus reaction | Leukocytes (neutrophilic) | Adherens junctions | Inflammation | Permeability | Cadherin | Substrates | Endothelial cells | White blood cells | Signaling | Chronic conditions | Injury prevention | Lungs | Rheumatoid arthritis | Skin | Disruption | Autoimmune diseases | Immunofluorescence | In vitro methods and tests | Umbilical vein | Destabilization | Electrical junctions
Journal Article
Kidney International, ISSN 0085-2538, 08/2012, Volume 82, Issue 4, pp. 412 - 427
Endothelial progenitor cells are known to reverse acute kidney injury by paracrine mechanisms. We previously found that microvesicles released from these... 
exosome | ischemia–reperfusion | acute kidney injury | ischemia-reperfusion | HORIZONTAL TRANSFER | STEM-CELLS | CONTRIBUTE | REGENERATION | FAILURE | EXPERIMENTAL GLOMERULONEPHRITIS | REPAIR | EPITHELIAL-CELLS | MESSENGER-RNA | UROLOGY & NEPHROLOGY | PLATELET-ACTIVATING-FACTOR | Chemotaxis, Leukocyte | Cell Proliferation | Epithelial Cells - metabolism | Kidney - blood supply | Capillaries - pathology | Kidney - pathology | Rats, Wistar | Endothelial Cells - transplantation | Cell-Derived Microparticles - transplantation | Male | MicroRNAs - metabolism | Acute Kidney Injury - genetics | Stem Cells - metabolism | Cell Hypoxia | Stem Cell Transplantation | Kidney - metabolism | Transfection | RNA Interference | Time Factors | Cell-Derived Microparticles - pathology | Cell-Derived Microparticles - metabolism | Kidney Tubules - pathology | Kidney Tubules - metabolism | Capillaries - metabolism | Reperfusion Injury - genetics | Reperfusion Injury - metabolism | Disease Models, Animal | Ribonuclease III - genetics | Ribonuclease III - metabolism | Reperfusion Injury - pathology | Acute Kidney Injury - pathology | Endothelial Cells - metabolism | Cells, Cultured | Gene Expression Regulation | Rats | Epithelial Cells - pathology | Acute Kidney Injury - prevention & control | Regeneration | Oligonucleotides - metabolism | Animals | Reperfusion Injury - prevention & control | Fibrosis | Stem Cells - pathology | Acute Kidney Injury - metabolism | Endothelial Cells - pathology | Apoptosis | Cell proliferation | Intravenous administration | Paracrine signalling | mRNA | Leukocytes | Ribonuclease | Endothelial cells | Kidney | Angiogenesis | Ischemia | Stem cells | Hypoxia | miRNA | Capillaries | Injuries
Journal Article
Journal of the American Society of Nephrology, ISSN 1046-6673, 03/2017, Volume 28, Issue 3, pp. 776 - 784
Journal Article