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2006, ISBN 9780470012949, xvii, 350 p., [8] p. of plates
Book
Nature (London), ISSN 1476-4687, 2015, Volume 520, Issue 7546, pp. 192 - 197
The metabolism of endothelial cells during vessel sprouting remains poorly studied. Here we report that endothelial loss of CFT1A, a rate-limiting enzyme of... 
OXIDATION | DISTRIBUTIONS | GLYCOLYSIS | REDUCTIVE GLUTAMINE-METABOLISM | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | TUMOR | PROLIFERATION | GROWTH-FACTOR | CANCER | CULTURE | Carnitine O-Palmitoyltransferase - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Neovascularization, Pathologic - pathology | Nucleotides - chemistry | Carnitine O-Palmitoyltransferase - antagonists & inhibitors | Adenosine Triphosphate - metabolism | Carnitine O-Palmitoyltransferase - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Nucleotides - biosynthesis | Nucleotides - pharmacology | Oxidation-Reduction - drug effects | DNA - biosynthesis | Carnitine O-Palmitoyltransferase - deficiency | Fatty Acids - metabolism | Disease Models, Animal | Carbon - metabolism | Fatty Acids - chemistry | Human Umbilical Vein Endothelial Cells - drug effects | Endothelial Cells - metabolism | Gene Silencing | Acetic Acid - pharmacology | Blood Vessels - cytology | Citric Acid Cycle | Animals | Retinopathy of Prematurity - drug therapy | Endothelial Cells - cytology | Neovascularization, Pathologic - drug therapy | Blood Vessels - drug effects | Cell Line, Tumor | Glucose - metabolism | Human Umbilical Vein Endothelial Cells - pathology | Cell Proliferation - drug effects | Mice | Neovascularization, Pathologic - metabolism | Retinopathy of Prematurity - metabolism | Endothelial Cells - enzymology | Endothelial Cells - drug effects | Retinopathy of Prematurity - pathology | Physiological aspects | Deoxyribonucleotides | Fatty acids | Carbon | Endothelium | Proteins | Angiogenesis | Protein synthesis | Glucose | Kinases | Deoxyribonucleic acid--DNA | Cells
Journal Article
2005, ISBN 9780824754242, xviii, 486
This reference serves as the first source to gather current data from endothelial cell biologists in various disciplines to summarize recent progress in the... 
Vascular endothelium | Endothelins | Endothelial Cells | Endothelium | Hematology | Oncology
Book
Nature (London), ISSN 1476-4687, 2018, Volume 561, Issue 7721, pp. 63 - 69
Glutamine synthetase, encoded by the gene GLUL, is an enzyme that converts glutamate and ammonia to glutamine. It is expressed by endothelial cells, but... 
GLYCOLYSIS | MIGRATION | RHO GTPASE | PATHWAY | VASCULATURE | MULTIDISCIPLINARY SCIENCES | ENDOTHELIAL-CELLS | JUNCTIONS | PROTEIN PALMITOYLATION | MODEL | DEFICIENCY | Palmitic Acid - metabolism | Glutamine - biosynthesis | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Actins - metabolism | Neovascularization, Pathologic | Glutamate-Ammonia Ligase - physiology | rho-Associated Kinases - metabolism | Human Umbilical Vein Endothelial Cells - cytology | HEK293 Cells | rho GTP-Binding Proteins - chemistry | Female | Glutamate-Ammonia Ligase - metabolism | Lipoylation | Endothelial Cells - metabolism | Glutamate-Ammonia Ligase - deficiency | Stress Fibers - metabolism | Animals | Human Umbilical Vein Endothelial Cells - enzymology | Glutamate-Ammonia Ligase - genetics | rho GTP-Binding Proteins - metabolism | Mice | Protein Processing, Post-Translational | Endothelial Cells - pathology | Endothelial Cells - enzymology | Cell Movement | Glutamine synthetase | Biosynthesis | Research | Neovascularization | Glutamine | Cell proliferation | Enzymes | Palmitoylation | Activation | Pharmacology | Kinases | Endothelial cells | Rho-associated kinase | Cell adhesion & migration | Fibers | Proteins | Angiogenesis | Ammonia | Clonal deletion | Actin | Skin diseases | Physiology | Inhibition | Localization | Glutamate-ammonia ligase | Umbilical vein | Cell migration | Guanosinetriphosphatase
Journal Article
The EMBO journal, ISSN 1460-2075, 2017, Volume 36, Issue 16, pp. 2334 - 2352
Journal Article
Circulation Research, ISSN 0009-7330, 11/2016, Volume 119, Issue 11, pp. 1183 - 1189
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2014, Volume 111, Issue 22, pp. 7968 - 7973
Journal Article
Circulation research, ISSN 1524-4571, 2019, Volume 125, Issue 1, pp. 43 - 52
Journal Article
Cancer Chemotherapy and Pharmacology, ISSN 0344-5704, 8/2011, Volume 68, Issue 2, pp. 445 - 455
The natural flavonoid fisetin was recently identified as a lead compound that stabilizes endothelial cell microtubules. In this study, we investigated the... 
Fisetin | Lewis lung carcinoma | Angiogenesis | Antitumour activity | Cyclophosphamide | Medicine & Public Health | Cancer Research | Oncology | Cytotoxicity | EA·hy 926 endothelial cells | Pharmacology/Toxicology | Flavonoid | EA•hy 926 endothelial cells | APOPTOSIS | ANTIINFLAMMATORY ACTIVITY | CELL-CYCLE ARREST | PROLIFERATION | FLUOROURACIL | CANCER | IN-VITRO | ONCOLOGY | ENDOTHELIAL-CELLS | PHARMACOLOGY & PHARMACY | EA.hy 926 endothelial cells | INHIBITORS | NIH 3T3 Cells | Cyclophosphamide - administration & dosage | Antineoplastic Combined Chemotherapy Protocols - administration & dosage | Tubulin Modulators - pharmacology | Humans | Antineoplastic Combined Chemotherapy Protocols - adverse effects | Antineoplastic Agents, Alkylating - pharmacology | Flavonoids - adverse effects | Antineoplastic Agents, Alkylating - administration & dosage | Cyclophosphamide - adverse effects | Cyclophosphamide - therapeutic use | Flavonoids - therapeutic use | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Angiogenesis Inhibitors - administration & dosage | Antineoplastic Agents, Phytogenic - administration & dosage | Angiogenesis Inhibitors - therapeutic use | Flavonoids - administration & dosage | Tubulin Modulators - administration & dosage | Female | Flavonoids - pharmacology | Antineoplastic Agents, Phytogenic - therapeutic use | Angiogenesis Inhibitors - adverse effects | Antineoplastic Agents, Phytogenic - adverse effects | Cell Line | Cell Survival - drug effects | Tubulin Modulators - adverse effects | Mice, Inbred C57BL | Angiogenesis Inhibitors - pharmacology | Tubulin Modulators - therapeutic use | Carcinoma, Lewis Lung - drug therapy | Antineoplastic Agents, Alkylating - therapeutic use | Cell Movement - drug effects | Animals | Tumor Burden - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Endothelial Cells - cytology | Neovascularization, Pathologic - drug therapy | Cyclophosphamide - pharmacology | Carcinoma, Lewis Lung - pathology | Cell Proliferation - drug effects | Mice | Antineoplastic Agents, Alkylating - adverse effects | Antineoplastic Agents, Phytogenic - pharmacology | Cell Cycle - drug effects | Endothelial Cells - drug effects | Antimitotic agents | Flavonoids | Flavones | Lung cancer | Bioflavonoids | Accountants | Drug therapy, Combination | Universities and colleges | Antineoplastic agents | Endothelium | Tumors | Index Medicus | cytology | Antineoplastic Agents, Phytogenic | pathology | Cell Proliferation | Endothelial Cells | Tubulin Modulators | Neovascularization, Pathologic | fisetin | administration & dosage | pharmacology | flavonoid | Carcinoma, Lewis Lung | Antineoplastic Agents, Alkylating | cytotoxicity | drug therapy | Cell Survival | angiogenesis | Antineoplastic Combined Chemotherapy Protocols | drug effects | Tumor Burden | Angiogenesis Inhibitors | EA.hy 926 | Cell Cycle | antitumour activity | adverse effects | therapeutic use | Cell Movement
Journal Article
Stem cells (Dayton, Ohio), ISSN 1066-5099, 2007, Volume 25, Issue 9, pp. 2363 - 2370
Recent reports indicated that vascular remodeling and angiogenesis are promoted by conditioned medium from the cells referred to as multipotent stromal cells... 
Angiogenesis | Endothelial cell | Therapeutic potential | Bone marrow stromal cells | Hypoxia | Human aortic endothelial cells Interleukin‐6 | Apoptosis | Human aortic endothelial cells | Interleukin-6 | bone marrow stromal cells | human aortic endothelial cells interleukin-6 | hypoxia | MYOCARDIAL-INFARCTION | endothelial cell | angiogenesis | RATS | apoptosis | ISCHEMIA | MODEL | MESENCHYMAL STEM-CELLS | CELL BIOLOGY | therapeutic potential | HEMATOPOIESIS | ONCOLOGY | HUMAN BONE-MARROW | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GROWTH-FACTOR | NEOVASCULARIZATION | HEMATOLOGY | EXPRESSION | Cell Survival - drug effects | Neovascularization, Physiologic - drug effects | Cell Hypoxia - drug effects | Cell Hypoxia - physiology | Apoptosis - drug effects | Endothelial Cells - metabolism | Humans | Multipotent Stem Cells - metabolism | Stromal Cells - metabolism | Cells, Cultured | Culture Media, Conditioned - chemistry | Culture Media, Conditioned - pharmacology | Phosphatidylinositol 3-Kinases - metabolism | Gene Expression Regulation - drug effects | Phosphatidylinositol 3-Kinases - genetics | Cytokines - isolation & purification | Signal Transduction - drug effects | Endothelial Cells - cytology | Angiogenesis Inducing Agents - metabolism | Angiogenesis Inducing Agents - pharmacology | Oncogene Protein v-akt - metabolism | Stromal Cells - cytology | Culture Media, Conditioned - metabolism | Endothelial Cells - drug effects
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2016, Volume 36, Issue 8, pp. 1517 - 1524
Journal Article
Scientific reports, ISSN 2045-2322, 2015, Volume 5, Issue 1, p. 9409
The porous architectural characteristics of biomaterials play an important role in scaffold revascularization. However, no consensus exists regarding optimal... 
VASCULARIZATION | BIOCERAMICS | INTERNAL STRUCTURE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | ENDOTHELIAL-CELLS | TISSUE | GROWTH | BONE REGENERATION | EXPRESSION | BMP-INDUCED OSTEOGENESIS | Neovascularization, Physiologic - drug effects | Platelet Endothelial Cell Adhesion Molecule-1 - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Biocompatible Materials - chemistry | Humans | Phosphatidylinositol 3-Kinases - metabolism | Structure-Activity Relationship | Vascular Endothelial Growth Factor A - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Vascular Endothelial Growth Factor A - genetics | Proto-Oncogene Proteins c-akt - genetics | Tissue Scaffolds | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Surface Properties | Nitric Oxide Synthase Type III - metabolism | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Calcium Phosphates - pharmacology | Tissue Engineering | Rabbits | Human Umbilical Vein Endothelial Cells - drug effects | Enzyme Inhibitors - pharmacology | Gene Expression Regulation | Morpholines - pharmacology | Calcium Phosphates - chemistry | Biocompatible Materials - pharmacology | Cell Adhesion - drug effects | Nitric Oxide Synthase Type III - genetics | Nitric Oxide Synthase Type III - antagonists & inhibitors | Phosphatidylinositol 3-Kinases - genetics | Cell Movement - drug effects | Animals | Proto-Oncogene Proteins c-akt - agonists | Cell Proliferation - drug effects | Porosity | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Cell proliferation | Femur | Leukocyte migration | AKT protein | Endothelial cells | Cell adhesion molecules | Cell adhesion & migration | Vascularization | 1-Phosphatidylinositol 3-kinase | Tricalcium phosphate | Angiogenesis | Computed tomography | Cell size | Vascular endothelial growth factor | Umbilical vein | Cell migration
Journal Article