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Biochemical journal, ISSN 0264-6021, 01/2010, Volume 425, Issue 1, pp. 215 - 223
The transcription factor SREBP1c (sterol-regulatory-element-binding protein 1c) is highly expressed in adipose tissue and plays a central role in several... 
Adipocyte | CCAAT/enhancer-binding protein(C/EBP) | Sterol-regulatory-element-binding protein 1c(SREBP1c) | Transcription | Lipid | Adipogenesis | transcription | BIOCHEMISTRY & MOLECULAR BIOLOGY | ELEMENT-BINDING PROTEIN-1C | ALPHA | sterol-regulatory-element-binding protein 1c (SREBP1c) | lipid | BETA | adipogenesis | INSULIN-RESISTANCE | adipocyte | LIPODYSTROPHY | ADIPOCYTE DIFFERENTIATION | PPAR-GAMMA | ADD1/SREBP1 | CCAAT/enhancer-binding protein (C/EBP) | MICE | ADIPOSE-TISSUE | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Humans | Adipocytes - cytology | DNA-Binding Proteins - metabolism | Chromatin Immunoprecipitation | RNA Interference | CCAAT-Enhancer-Binding Protein-delta - metabolism | CCAAT-Enhancer-Binding Proteins - genetics | Sterol Regulatory Element Binding Protein 1 - metabolism | CCAAT-Enhancer-Binding Proteins - metabolism | Proto-Oncogene Proteins - metabolism | Promoter Regions, Genetic | CCAAT-Enhancer-Binding Protein-beta - genetics | Cells, Cultured | Gene Expression Regulation | Proto-Oncogene Proteins - genetics | Transcription Factors - genetics | 3T3-L1 Cells | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | CCAAT-Enhancer-Binding Protein-beta - metabolism | RUNX1 Translocation Partner 1 Protein | Transcription Factors - metabolism | Animals | Sterol Regulatory Element Binding Protein 1 - genetics | CCAAT-Enhancer-Binding Protein-delta - genetics | Adipocytes - metabolism | CCAAT-Enhancer-Binding Protein-alpha - genetics | Protein Binding | Mice
Journal Article
Journal Article
Science (American Association for the Advancement of Science), ISSN 1095-9203, 2012, Volume 338, Issue 6114, pp. 1599 - 1603
The epicardium encapsulates the heart and functions as a source of multipotent progenitor cells and paracrine factors essential for cardiac development and... 
Heart | Transcription factors | HEK293 cells | Transgenic animals | RESEARCH ARTICLES | Neutrophils | Epicardium | Physical trauma | Gene expression | Embryos | Transgenic plants | CELLS | IMMUNITY | RETINOIC ACID | ZEBRAFISH | GRAINY-HEAD | MOUSE HEART | INTEGRITY | REGENERATION | MULTIDISCIPLINARY SCIENCES | BINDING PROTEIN | DROSOPHILA | Myocardial Infarction - genetics | WT1 Proteins - metabolism | Oligonucleotide Array Sequence Analysis | Heart - embryology | Male | Pericardium - metabolism | Neutrophil Infiltration | Ventricular Remodeling | Gene Expression Regulation, Developmental | Uroplakin III - genetics | Pericardium - embryology | Female | CCAAT-Enhancer-Binding Protein-delta - metabolism | CCAAT-Enhancer-Binding Proteins - genetics | Binding Sites | Myocardial Reperfusion Injury - genetics | WT1 Proteins - genetics | Organ Culture Techniques | CCAAT-Enhancer-Binding Proteins - metabolism | Heart - physiopathology | Myocardial Contraction | Signal Transduction | CCAAT-Enhancer-Binding Protein-beta - genetics | Gene Expression Regulation | Mice, Transgenic | Aldehyde Oxidoreductases - genetics | Myocardial Infarction - metabolism | CCAAT-Enhancer-Binding Protein-beta - metabolism | Pericardium - cytology | Aldehyde Oxidoreductases - metabolism | Myocardial Reperfusion Injury - metabolism | Animals | Enhancer Elements, Genetic | CCAAT-Enhancer-Binding Protein-delta - genetics | Mice | Models, Genetic | Uroplakin III - metabolism | Cellular signal transduction | Genetic aspects | Research | Genetic transcription | Regeneration (Biology) | Properties | Observations | Proteins | Signal transduction | Cardiovascular disease | Embryology | Rodents | Injury prevention | Activation | Mathematical models | Adults | Heart function | Protective | Injuries
Journal Article
Genes and Development, ISSN 0890-9369, 03/2019, Volume 33, Issue 5-6, pp. 255 - 257
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 11, p. e112073
The CCAAT/Enhancer Binding Proteins (C/EBPs) are a family of leucine-zipper transcription factors that regulate physiological processes such as energy... 
BREAST-CANCER | INHIBITORY PROTEIN | PHOTODYNAMIC THERAPY | SKIN TUMORIGENESIS | MULTIDISCIPLINARY SCIENCES | IN-VIVO | GENE-EXPRESSION | DOWN-REGULATION | TUMOR-SUPPRESSOR | BINDING-PROTEIN-BETA | MAMMARY EPITHELIAL-CELLS | CCAAT-Enhancer-Binding Proteins - metabolism | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Phosphorylation | RNA Isoforms | Alternative Splicing | CCAAT-Enhancer-Binding Protein-beta - genetics | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | Gene Expression Regulation, Neoplastic | CCAAT-Enhancer-Binding Protein-beta - metabolism | Skin Neoplasms - metabolism | Biopsy | CCAAT-Enhancer-Binding Protein-delta - genetics | Skin Neoplasms - genetics | CCAAT-Enhancer-Binding Protein-alpha - genetics | Cell Line, Tumor | CCAAT-Enhancer-Binding Protein-delta - metabolism | CCAAT-Enhancer-Binding Proteins - genetics | Neoplasm Staging | Squamous cell carcinoma | Ionizing radiation | Skin | DNA binding proteins | Cell differentiation | Protein binding | Cell proliferation | Energy metabolism | Physiological effects | Correlation | Transcription factors | Leukemia | Malignancy | CCAAT/enhancer-binding protein | Leucine | Kinases | Tissues | Western blotting | Skin cancer | Proteins | Rodents | Proliferating cell nuclear antigen | Cell cycle | Post-translation | Tumorigenesis | Deoxyribonucleic acid--DNA | Biomedical engineering | Translation | Dermatology | Melanoma | Breast cancer | Metabolism | Gene expression | Leucine zipper proteins | Digital imaging | Electrophoretic mobility | Isoforms | Differentiation | Tumors | Deoxyribonucleic acid | DNA
Journal Article
PLoS genetics, ISSN 1553-7404, 2016, Volume 12, Issue 12, p. e1006474
Increasing energy expenditure through brown adipocyte recruitment is a promising approach to combat obesity. We report here the comprehensive profiling of the... 
ADIPOSE-TISSUE DEVELOPMENT | SUPER-ENHANCERS | MOUSE GENOME | ADAPTIVE THERMOGENESIS | EMBRYONIC STEM-CELLS | GENETICS & HEREDITY | ADIPOCYTE DIFFERENTIATION | PPAR-GAMMA | ADULT HUMANS | FAT DEVELOPMENT | FACTOR HOTSPOTS | CCAAT-Enhancer-Binding Protein-beta - biosynthesis | Homeodomain Proteins - metabolism | Autoantigens - genetics | Obesity - genetics | Cell Differentiation - genetics | Mesenchymal Stromal Cells | Gene Expression Regulation, Developmental | RNA, Long Noncoding - biosynthesis | Basic Helix-Loop-Helix Transcription Factors - biosynthesis | CCAAT-Enhancer-Binding Proteins - biosynthesis | CCAAT-Enhancer-Binding Proteins - genetics | Energy Metabolism - genetics | Adipogenesis - genetics | Cell Lineage - genetics | CCAAT-Enhancer-Binding Proteins - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | CCAAT-Enhancer-Binding Protein-beta - genetics | Transcription Factors - biosynthesis | Transcriptome - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | CCAAT-Enhancer-Binding Protein-beta - metabolism | Homeodomain Proteins - genetics | Obesity - metabolism | Obesity - pathology | Animals | Adipose Tissue, Brown - growth & development | Adipose Tissue, Brown - metabolism | Mice | DNA-Binding Proteins - biosynthesis | Obesity | Laboratories | Funding | Biology | Adipocytes | Kinases | Metabolism | Gene expression | Weight control | Stem cells | Expenditures | Life sciences | Standard deviation | Respiration | Bioinformatics
Journal Article
International journal of molecular sciences, ISSN 1422-0067, 2018, Volume 19, Issue 7, p. 1918
All-trans-retinoic acid (ATRA) and 1 alpha,25-dihydroxyvitamin D (1,25D) are potent inducers of differentiation of myeloid leukemia cells. During myeloid... 
CCAAT-enhancer-binding proteins | Retinoic acid receptor α | Vitamin D receptor | Differentiation | Nuclear receptors | Acute myeloid leukemia | CEBP genes | TRANSCRIPTION FACTORS | retinoic acid receptor alpha | differentiation | BIOCHEMISTRY & MOLECULAR BIOLOGY | CCAAT/ENHANCER-BINDING PROTEINS | GRANULOCYTE | CHEMISTRY, MULTIDISCIPLINARY | MICE LACKING | BETA | EPSILON | nuclear receptors | LIVER NUCLEAR-PROTEIN | C/EBP-ALPHA | INDUCED DIFFERENTIATION | acute myeloid leukemia | vitamin D receptor | D-2 ANALOGS | Retinoic Acid Receptor alpha - genetics | CCAAT-Enhancer-Binding Protein-beta - genetics | Humans | Leukemia, Myeloid, Acute - metabolism | Retinoic Acid Receptor alpha - metabolism | Gene Silencing | Receptors, Calcitriol - metabolism | Receptors, Calcitriol - genetics | Blotting, Western | CCAAT-Enhancer-Binding Protein-beta - metabolism | Glyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating) - genetics | Glyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating) - metabolism | Flow Cytometry | CCAAT-Enhancer-Binding Protein-delta - genetics | HL-60 Cells | CCAAT-Enhancer-Binding Protein-delta - metabolism | Leukemia, Myeloid, Acute - genetics | Regulators | Transcription factors | Data points | Myeloid leukemia | Redundancy | Leukemia | Genes | Dihydroxyvitamin D | CCAAT/enhancer-binding protein | Tumor cell lines | Gene expression | Proteins | Vitamin D | Developmental stages | Vitamin D receptors | Retinoic acid | Inducers | trans-Retinoic acid | Tretinoin | retinoic acid receptor α
Journal Article
by Djebali, Sarah and Davis, Carrie A and Merkel, Angelika and Dobin, Alex and Lassmann, Timo and Mortazavi, Ali and Tanzer, Andrea and Lagarde, Julien and Lin, Wei and Schlesinger, Felix and Xue, Chenghai and Marinov, Georgi K and Khatun, Jainab and Williams, Brian A and Zaleski, Chris and Rozowsky, Joel and Röder, Maik and Kokocinski, Felix and Abdelhamid, Rehab F and Alioto, Tyler and Antoshechkin, Igor and Baer, Michael T and Bar, Nadav S and Batut, Philippe and Bell, Kimberly and Bell, Ian and Chakrabortty, Sudipto and Chen, Xian and Chrast, Jacqueline and Curado, Joao and Derrien, Thomas and Drenkow, Jorg and Dumais, Erica and Dumais, Jacqueline and Duttagupta, Radha and Falconnet, Emilie and Fastuca, Meagan and Fejes-Toth, Kata and Ferreira, Pedro and Foissac, Sylvain and Fullwood, Melissa J and Gao, Hui and Gonzalez, David and Gordon, Assaf and Gunawardena, Harsha and Howald, Cedric and Jha, Sonali and Johnson, Rory and Kapranov, Philipp and King, Brandon and Kingswood, Colin and Luo, Oscar J and Park, Eddie and Persaud, Kimberly and Preall, Jonathan B and Ribeca, Paolo and Risk, Brian and Robyr, Daniel and Sammeth, Michael and Schaffer, Lorian and See, Lei-Hoon and Shahab, Atif and Skancke, Jorgen and Suzuki, Ana Maria and Takahashi, Hazuki and Tilgner, Hagen and Trout, Diane and Walters, Nathalie and Wang, Huaien and Wrobel, John and Yu, Yanbao and Ruan, Xiaoan and Hayashizaki, Yoshihide and Harrow, Jennifer and Gerstein, Mark and Hubbard, Tim and Reymond, Alexandre and Antonarakis, Stylianos E and Hannon, Gregory and Giddings, Morgan C and Ruan, Yijun and Wold, Barbara and Carninci, Piero and Guigó, Roderic and Gingeras, Thomas R
Nature (London), ISSN 1476-4687, 2012, Volume 489, Issue 7414, pp. 101 - 108
Journal Article
by Chen, Han and Li, Chunyan and Peng, Xinxin and Weinstein, John N and Ferguson, Martin L and Hutter, Carolyn M and Sofia, Heidi J and Tarnuzzer, Roy and Wang, Zhining and Zhang, Jiashan (Julia) and Chudamani, Sudha and Liu, Jia and Lolla, Laxmi and Cho, Juok and DeFreitas, Timothy and Gehlenborg, Nils and Heiman, David I and Kim, Jaegil and Lawrence, Michael S and Lin, Pei and Meier, Sam and Saksena, Gordon and Bernard, Brady and Dhankani, Varsha and Knijnenburg, Theo and Leinonen, Kalle and Liu, Yuexin and Reynolds, Sheila and Shmulevich, Ilya and Akbani, Rehan and Broom, Bradley M and Hegde, Apurva M and Kanchi, Rupa S and Li, Jun and Lu, Yiling and Ng, Kwok-Shing and Rao, Arvind and Ryan, Michael and Wang, Jing and Zhang, Jiexin and Abeshouse, Adam and Armenia, Joshua and Chakravarty, Debyani and Chatila, Walid K and de Bruijn, Ino and Gao, Jianjiong and Gross, Benjamin E and La, Konnor and Ladanyi, Marc and Nissan, Moriah G and Ochoa, Angelica and Reznik, Ed and Sanchez-Vega, Francisco and Schultz, Nikolaus and Sheridan, Robert and Sun, Yichao and Taylor, Barry S and Wang, Jioajiao and Zhang, Hongxin and Anur, Pavana and Benz, Christopher and Hayes, D. Neil and Parker, Joel S and Wilkerson, Matthew D and Balasundaram, Miruna and Chuah, Eric and Dhalla, Noreen and Lee, Darlene and Marra, Marco A and Moore, Richard A and Mungall, Karen and Robertson, A. Gordon and Sadeghi, Sara and Schein, Jacqueline E and Tam, Angela and Thiessen, Nina and Wong, Tina and Beroukhim, Rameen and Cherniack, Andrew D and Cibulskis, Carrie and Gabriel, Stacey B and Gao, Galen F and Ha, Gavin and Meyerson, Matthew and Schumacher, Steven E and Shih, Juliann and Kucherlapati, Raju S and Baylin, Stephen and Cope, Leslie and Bootwalla, Moiz S and Lai, Phillip H and Van Den Berg, David J and Weisenberger, Daniel J and Auman, J. Todd and Balu, Saianand and Bodenheimer, Tom and Fan, Cheng and Hoyle, Alan P and Jefferys, Stuart R and Jones, Corbin D and ... and The Cancer Genome Atlas Research Network and Canc Genome Atlas Res Network and Cancer Genome Atlas Research Network
Cell (Cambridge), ISSN 0092-8674, 04/2018, Volume 173, Issue 2, pp. 386 - 399.e12
Journal Article