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Biochemical Journal, ISSN 0264-6021, 01/2010, Volume 425, Issue 1, pp. 215 - 223
The transcription factor SREBP1c (sterol-regulatory-element-binding protein 1c) is highly expressed in adipose tissue and plays a central role in several... 
Adipocyte | CCAAT/enhancer-binding protein(C/EBP) | Sterol-regulatory-element-binding protein 1c(SREBP1c) | Transcription | Lipid | Adipogenesis | transcription | BIOCHEMISTRY & MOLECULAR BIOLOGY | ELEMENT-BINDING PROTEIN-1C | ALPHA | sterol-regulatory-element-binding protein 1c (SREBP1c) | lipid | BETA | adipogenesis | INSULIN-RESISTANCE | adipocyte | LIPODYSTROPHY | ADIPOCYTE DIFFERENTIATION | PPAR-GAMMA | ADD1/SREBP1 | CCAAT/enhancer-binding protein (C/EBP) | MICE | ADIPOSE-TISSUE | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Humans | Adipocytes - cytology | DNA-Binding Proteins - metabolism | Chromatin Immunoprecipitation | RNA Interference | CCAAT-Enhancer-Binding Protein-delta - metabolism | CCAAT-Enhancer-Binding Proteins - genetics | Sterol Regulatory Element Binding Protein 1 - metabolism | CCAAT-Enhancer-Binding Proteins - metabolism | Proto-Oncogene Proteins - metabolism | Promoter Regions, Genetic | CCAAT-Enhancer-Binding Protein-beta - genetics | Cells, Cultured | Gene Expression Regulation | Proto-Oncogene Proteins - genetics | Transcription Factors - genetics | 3T3-L1 Cells | DNA-Binding Proteins - genetics | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | CCAAT-Enhancer-Binding Protein-beta - metabolism | RUNX1 Translocation Partner 1 Protein | Transcription Factors - metabolism | Animals | Sterol Regulatory Element Binding Protein 1 - genetics | CCAAT-Enhancer-Binding Protein-delta - genetics | Adipocytes - metabolism | CCAAT-Enhancer-Binding Protein-alpha - genetics | Protein Binding | Mice | Index Medicus
Journal Article
Science, ISSN 0036-8075, 12/2012, Volume 338, Issue 6114, pp. 1599 - 1603
The epicardium encapsulates the heart and functions as a source of multipotent progenitor cells and paracrine factors essential for cardiac development and... 
Heart | Transcription factors | HEK293 cells | Transgenic animals | RESEARCH ARTICLES | Neutrophils | Epicardium | Physical trauma | Gene expression | Embryos | Transgenic plants | CELLS | IMMUNITY | RETINOIC ACID | ZEBRAFISH | GRAINY-HEAD | MOUSE HEART | INTEGRITY | REGENERATION | MULTIDISCIPLINARY SCIENCES | BINDING PROTEIN | DROSOPHILA | Myocardial Infarction - genetics | WT1 Proteins - metabolism | Oligonucleotide Array Sequence Analysis | Heart - embryology | Male | Pericardium - metabolism | Neutrophil Infiltration | Ventricular Remodeling | Gene Expression Regulation, Developmental | Uroplakin III - genetics | Pericardium - embryology | Female | CCAAT-Enhancer-Binding Protein-delta - metabolism | CCAAT-Enhancer-Binding Proteins - genetics | Binding Sites | Myocardial Reperfusion Injury - genetics | WT1 Proteins - genetics | Organ Culture Techniques | CCAAT-Enhancer-Binding Proteins - metabolism | Heart - physiopathology | Myocardial Contraction | Signal Transduction | CCAAT-Enhancer-Binding Protein-beta - genetics | Gene Expression Regulation | Mice, Transgenic | Aldehyde Oxidoreductases - genetics | Myocardial Infarction - metabolism | CCAAT-Enhancer-Binding Protein-beta - metabolism | Pericardium - cytology | Aldehyde Oxidoreductases - metabolism | Myocardial Reperfusion Injury - metabolism | Animals | Enhancer Elements, Genetic | CCAAT-Enhancer-Binding Protein-delta - genetics | Mice | Models, Genetic | Uroplakin III - metabolism | Cellular signal transduction | Genetic aspects | Research | Genetic transcription | Regeneration (Biology) | Properties | Observations | Proteins | Signal transduction | Cardiovascular disease | Embryology | Rodents | Index Medicus | Injury prevention | Activation | Mathematical models | Adults | Heart function | Protective | Injuries
Journal Article
PLoS ONE, ISSN 1932-6203, 11/2014, Volume 9, Issue 11, pp. e112073 - e112073
The CCAAT/Enhancer Binding Proteins (C/EBPs) are a family of leucine-zipper transcription factors that regulate physiological processes such as energy... 
BREAST-CANCER | INHIBITORY PROTEIN | PHOTODYNAMIC THERAPY | SKIN TUMORIGENESIS | MULTIDISCIPLINARY SCIENCES | IN-VIVO | GENE-EXPRESSION | DOWN-REGULATION | TUMOR-SUPPRESSOR | BINDING-PROTEIN-BETA | MAMMARY EPITHELIAL-CELLS | CCAAT-Enhancer-Binding Proteins - metabolism | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Phosphorylation | RNA Isoforms | Alternative Splicing | CCAAT-Enhancer-Binding Protein-beta - genetics | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - pathology | Humans | Gene Expression Regulation, Neoplastic | CCAAT-Enhancer-Binding Protein-beta - metabolism | Skin Neoplasms - metabolism | Biopsy | CCAAT-Enhancer-Binding Protein-delta - genetics | Skin Neoplasms - genetics | CCAAT-Enhancer-Binding Protein-alpha - genetics | Cell Line, Tumor | CCAAT-Enhancer-Binding Protein-delta - metabolism | CCAAT-Enhancer-Binding Proteins - genetics | Neoplasm Staging | Squamous cell carcinoma | Ionizing radiation | Skin | DNA binding proteins | Cell differentiation | Protein binding | Cell proliferation | Energy metabolism | Physiological effects | Correlation | Transcription factors | Leukemia | Malignancy | CCAAT/enhancer-binding protein | Leucine | Kinases | Tissues | Western blotting | Skin cancer | Proteins | Rodents | Proliferating cell nuclear antigen | Cell cycle | Post-translation | Tumorigenesis | Deoxyribonucleic acid--DNA | Biomedical engineering | Translation | Dermatology | Melanoma | Breast cancer | Metabolism | Gene expression | Leucine zipper proteins | Digital imaging | Electrophoretic mobility | Isoforms | Differentiation | Tumors | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
AGRONOMY JOURNAL, ISSN 0002-1962, 07/2018, Volume 110, Issue 4, pp. 1628 - 1630
Journal Article
International Journal of Molecular Sciences, ISSN 1661-6596, 07/2018, Volume 19, Issue 7, p. 1918
All-trans-retinoic acid (ATRA) and 1 alpha,25-dihydroxyvitamin D (1,25D) are potent inducers of differentiation of myeloid leukemia cells. During myeloid... 
CCAAT-enhancer-binding proteins | Retinoic acid receptor α | Vitamin D receptor | Differentiation | Nuclear receptors | Acute myeloid leukemia | CEBP genes | TRANSCRIPTION FACTORS | retinoic acid receptor alpha | differentiation | BIOCHEMISTRY & MOLECULAR BIOLOGY | CCAAT/ENHANCER-BINDING PROTEINS | GRANULOCYTE | CHEMISTRY, MULTIDISCIPLINARY | MICE LACKING | BETA | EPSILON | nuclear receptors | LIVER NUCLEAR-PROTEIN | C/EBP-ALPHA | INDUCED DIFFERENTIATION | acute myeloid leukemia | vitamin D receptor | D-2 ANALOGS | Retinoic Acid Receptor alpha - genetics | CCAAT-Enhancer-Binding Protein-beta - genetics | Humans | Leukemia, Myeloid, Acute - metabolism | Retinoic Acid Receptor alpha - metabolism | Gene Silencing | Receptors, Calcitriol - metabolism | Receptors, Calcitriol - genetics | Blotting, Western | CCAAT-Enhancer-Binding Protein-beta - metabolism | Glyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating) - genetics | Glyceraldehyde-3-Phosphate Dehydrogenase (Phosphorylating) - metabolism | Flow Cytometry | CCAAT-Enhancer-Binding Protein-delta - genetics | HL-60 Cells | CCAAT-Enhancer-Binding Protein-delta - metabolism | Leukemia, Myeloid, Acute - genetics | Regulators | Transcription factors | Data points | Myeloid leukemia | Redundancy | Leukemia | Genes | Dihydroxyvitamin D | CCAAT/enhancer-binding protein | Tumor cell lines | Gene expression | Proteins | Vitamin D | Developmental stages | Cell lines | Vitamin D receptors | Retinoic acid | Inducers | trans-Retinoic acid | Index Medicus | retinoic acid receptor α
Journal Article
by Djebali, Sarah and Davis, Carrie A and Merkel, Angelika and Dobin, Alex and Lassmann, Timo and Mortazavi, Ali and Tanzer, Andrea and Lagarde, Julien and Lin, Wei and Schlesinger, Felix and Xue, Chenghai and Marinov, Georgi K and Khatun, Jainab and Williams, Brian A and Zaleski, Chris and Rozowsky, Joel and Röder, Maik and Kokocinski, Felix and Abdelhamid, Rehab F and Alioto, Tyler and Antoshechkin, Igor and Baer, Michael T and Bar, Nadav S and Batut, Philippe and Bell, Kimberly and Bell, Ian and Chakrabortty, Sudipto and Chen, Xian and Chrast, Jacqueline and Curado, Joao and Derrien, Thomas and Drenkow, Jorg and Dumais, Erica and Dumais, Jacqueline and Duttagupta, Radha and Falconnet, Emilie and Fastuca, Meagan and Fejes-Toth, Kata and Ferreira, Pedro and Foissac, Sylvain and Fullwood, Melissa J and Gao, Hui and Gonzalez, David and Gordon, Assaf and Gunawardena, Harsha and Howald, Cedric and Jha, Sonali and Johnson, Rory and Kapranov, Philipp and King, Brandon and Kingswood, Colin and Luo, Oscar J and Park, Eddie and Persaud, Kimberly and Preall, Jonathan B and Ribeca, Paolo and Risk, Brian and Robyr, Daniel and Sammeth, Michael and Schaffer, Lorian and See, Lei-Hoon and Shahab, Atif and Skancke, Jorgen and Suzuki, Ana Maria and Takahashi, Hazuki and Tilgner, Hagen and Trout, Diane and Walters, Nathalie and Wang, Huaien and Wrobel, John and Yu, Yanbao and Ruan, Xiaoan and Hayashizaki, Yoshihide and Harrow, Jennifer and Gerstein, Mark and Hubbard, Tim and Reymond, Alexandre and Antonarakis, Stylianos E and Hannon, Gregory and Giddings, Morgan C and Ruan, Yijun and Wold, Barbara and Carninci, Piero and Guig, Roderic and Gingeras, Thomas R
Nature, ISSN 0028-0836, 09/2012, Volume 489, Issue 7414, pp. 101 - 108
Journal Article
PLoS Genetics, ISSN 1553-7390, 12/2016, Volume 12, Issue 12, pp. e1006474 - e1006474
Increasing energy expenditure through brown adipocyte recruitment is a promising approach to combat obesity. We report here the comprehensive profiling of the... 
ADIPOSE-TISSUE DEVELOPMENT | SUPER-ENHANCERS | MOUSE GENOME | ADAPTIVE THERMOGENESIS | EMBRYONIC STEM-CELLS | GENETICS & HEREDITY | ADIPOCYTE DIFFERENTIATION | PPAR-GAMMA | ADULT HUMANS | FAT DEVELOPMENT | FACTOR HOTSPOTS | CCAAT-Enhancer-Binding Protein-beta - biosynthesis | Homeodomain Proteins - metabolism | Autoantigens - genetics | Obesity - genetics | Cell Differentiation - genetics | Mesenchymal Stromal Cells | Gene Expression Regulation, Developmental | RNA, Long Noncoding - biosynthesis | Basic Helix-Loop-Helix Transcription Factors - biosynthesis | CCAAT-Enhancer-Binding Proteins - biosynthesis | CCAAT-Enhancer-Binding Proteins - genetics | Energy Metabolism - genetics | Adipogenesis - genetics | Cell Lineage - genetics | CCAAT-Enhancer-Binding Proteins - metabolism | Basic Helix-Loop-Helix Transcription Factors - genetics | CCAAT-Enhancer-Binding Protein-beta - genetics | Transcription Factors - biosynthesis | Transcriptome - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | CCAAT-Enhancer-Binding Protein-beta - metabolism | Homeodomain Proteins - genetics | Obesity - metabolism | Obesity - pathology | Animals | Adipose Tissue, Brown - growth & development | Adipose Tissue, Brown - metabolism | Mice | DNA-Binding Proteins - biosynthesis | Obesity | Laboratories | Funding | Biology | Adipocytes | Kinases | Metabolism | Gene expression | Weight control | Stem cells | Expenditures | Life sciences | Standard deviation | Respiration | Bioinformatics | Index Medicus
Journal Article
Molecular Nutrition & Food Research, ISSN 1613-4125, 10/2013, Volume 57, Issue 10, pp. 1729 - 1740
ScopeAdipocytes differentiation is deeply involved in the onset of obesity. 4-Hydroxyderricin (4HD) and xanthoangelol (XAG) are the chalcones that are derived... 
AMP | Peroxisome proliferator‐activated receptor gamma | activated protein kinase | Ashitaba chalcones | Mitogen‐activated protein kinases | CCAAT | enhancer‐binding proteins | Peroxisome proliferator-activated receptor gamma | CCAAT/enhancer-binding proteins | AMP-activated protein kinase | Mitogen-activated protein kinases | METABOLIC SYNDROME | FOOD SCIENCE & TECHNOLOGY | KINASE | ADIPOGENESIS | enhancer-binding proteins | INSULIN | C/EBP-ALPHA | OBESITY | ADIPOSE CONVERSION | PPAR-GAMMA | PRENYLATED CHALCONES | 3T3-L1 CELLS | AMP-Activated Protein Kinases - metabolism | Phosphorylation | Plant Extracts - pharmacology | Adipogenesis - drug effects | Angelica - chemistry | Adipocytes - cytology | JNK Mitogen-Activated Protein Kinases - metabolism | Adipocytes - drug effects | PPAR gamma - metabolism | Chalcone - pharmacology | Chalcone - analogs & derivatives | CCAAT-Enhancer-Binding Proteins - genetics | JNK Mitogen-Activated Protein Kinases - genetics | PPAR gamma - genetics | CCAAT-Enhancer-Binding Proteins - metabolism | Cell Survival - drug effects | Signal Transduction | CCAAT-Enhancer-Binding Protein-beta - genetics | Transcription Factors - genetics | 3T3-L1 Cells | CCAAT-Enhancer-Binding Protein-beta - metabolism | Transcription Factors - metabolism | Animals | Cell Differentiation - drug effects | Adipocytes - metabolism | Lipid Metabolism - drug effects | Mitogen-Activated Protein Kinases - genetics | Cell Proliferation - drug effects | Mice | AMP-Activated Protein Kinases - genetics | Mitogen-Activated Protein Kinases - metabolism | Glycerin | Phosphates | RNA | Glycerol | Binding proteins | Mitogens | Phosphotransferases | Carnitine | Protein binding | Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, pp. e69925 - e69925
This study examined the anti-obesity effect and mechanism of action of blueberry peel extracts (BPE) in 3T3-L1 cells and high-fat diet (HFD)-induced obese... 
C/EBP-ALPHA | ACTIVATED PROTEIN-KINASE | GENE PROMOTER | CANCER CELLS | INSULIN-RESISTANCE | GLUCOSE | MULTIDISCIPLINARY SCIENCES | GROWTH | ADIPOCYTE DIFFERENTIATION | GLYCOGEN-SYNTHASE KINASE | BINDING | CCAAT-Enhancer-Binding Protein-alpha - metabolism | Obesity - drug therapy | Adipogenesis - drug effects | Blueberry Plants - chemistry | Body Weight - drug effects | Male | Adipocytes - drug effects | PPAR gamma - metabolism | Proto-Oncogene Proteins c-akt - genetics | CCAAT-Enhancer-Binding Protein-alpha - antagonists & inhibitors | Diet, High-Fat | Plant Preparations - pharmacology | Cell Differentiation | Proto-Oncogene Proteins c-akt - metabolism | Adipogenesis - genetics | PPAR gamma - genetics | Signal Transduction | Fruit - chemistry | CCAAT-Enhancer-Binding Protein-beta - genetics | Gene Expression Regulation | Rats | Obesity - physiopathology | 3T3-L1 Cells | Adipocytes - pathology | Rats, Sprague-Dawley | CCAAT-Enhancer-Binding Protein-beta - metabolism | Obesity - metabolism | Eating - drug effects | Animals | PPAR gamma - antagonists & inhibitors | Adipocytes - metabolism | CCAAT-Enhancer-Binding Protein-alpha - genetics | Mice | CCAAT-Enhancer-Binding Protein-beta - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Proteins | Obesity | Blood cholesterol | Diet | Body weight | Genetic aspects | Triglycerides | Veterinary colleges | Phosphorylation | Adipose tissue | Genes | AKT protein | Adipocytes | Kinases | Accumulation | High fat diet | Signal transduction | Rodents | Inhibition | Lipoproteins (high density) | Adipogenesis | Oral administration | Preadipocytes | Gene expression | Insulin | Body weight gain | Cholesterol | Substrates | Signaling | Food intake | Weight reduction | Insulin resistance | Fats and oils | Differentiation | Index Medicus
Journal Article