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index medicus (446) 446
animals (414) 414
humans (298) 298
glucagon-like peptide-1 (267) 267
enteroendocrine cells - metabolism (253) 253
mice (218) 218
enteroendocrine cells (215) 215
endocrinology & metabolism (203) 203
male (182) 182
glucagon-like peptide 1 - metabolism (140) 140
glucose (138) 138
secretion (135) 135
glp-1 (126) 126
glucagon (122) 122
peptides (99) 99
glucagon-like peptide 1 - secretion (97) 97
cell line (92) 92
digestive, oral, and skin physiology (92) 92
enteroendocrine cells - drug effects (92) 92
diabetes (91) 91
obesity (89) 89
rats (89) 89
expression (88) 88
physiology (88) 88
physiological aspects (85) 85
insulin (83) 83
cell biology (82) 82
receptors, g-protein-coupled - metabolism (81) 81
mice, inbred c57bl (80) 80
enteroendocrine cells - cytology (79) 79
female (78) 78
intestine (76) 76
endocrine system (75) 75
hormones (75) 75
gastrointestinal-tract (73) 73
food-intake (69) 69
rodents (67) 67
hormones, hormone substitutes, and hormone antagonists (65) 65
type 2 diabetes (65) 65
abridged index medicus (64) 64
enteroendocrine cells - secretion (64) 64
enteroendocrine cells - physiology (63) 63
dextrose (62) 62
gene expression (62) 62
research (62) 62
glucagon-like peptide 1 (61) 61
immunohistochemistry (59) 59
signal transduction (59) 59
article (57) 57
fatty acids (56) 56
homeostasis (56) 56
analysis (55) 55
small intestine (53) 53
gastroenterology & hepatology (52) 52
pharmacology & pharmacy (52) 52
cholecystokinin (50) 50
receptors (50) 50
glucose metabolism (49) 49
intestinal mucosa - metabolism (49) 49
biochemistry & molecular biology (48) 48
cells, cultured (48) 48
insulin - metabolism (48) 48
release (48) 48
taste (48) 48
endocrine-cells (47) 47
gastric-inhibitory polypeptide (47) 47
gut (47) 47
insulin-secretion (47) 47
receptor (47) 47
glucose - metabolism (45) 45
mice, transgenic (45) 45
glucagon-like peptide 1 - genetics (44) 44
receptors, g-protein-coupled - genetics (44) 44
nutrition & dietetics (43) 43
digestive system (42) 42
protein-coupled receptor (42) 42
mice, knockout (41) 41
taste receptors (41) 41
adult (40) 40
gastrointestinal tract - metabolism (40) 40
insulin secretion (40) 40
internal medicine (40) 40
middle aged (40) 40
pancreas (40) 40
proteins (40) 40
gastrointestinal system (39) 39
glucagon-like peptide 1 - blood (39) 39
colon (38) 38
l cells (38) 38
metabolism (38) 38
peptide yy - metabolism (37) 37
small-intestine (37) 37
blood glucose - metabolism (36) 36
cells (36) 36
surgery (36) 36
appetite (35) 35
multidisciplinary sciences (35) 35
bariatric surgery (34) 34
diabetes mellitus, type 2 - metabolism (34) 34
health aspects (34) 34
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1. Full Text Glucose Sensing in L Cells: A Primary Cell Study
by Reimann, Frank and Habib, Abdella M and Tolhurst, Gwen and Parker, Helen E and Rogers, Gareth J and Gribble, Fiona M
Cell Metabolism, ISSN 1550-4131, 12/2008, Volume 8, Issue 6, pp. 532 - 539
Glucagon-like peptide-1 (GLP-1) is an enteric hormone that stimulates insulin secretion and improves glycaemia in type 2 diabetes. Although GLP-1-based... 
HUMDISEASE | SYSTEM | GLYCEMIC CONTROL | PROTEIN | GLUCAGON-LIKE PEPTIDE-1 | LINE | ENDOCRINOLOGY & METABOLISM | SMALL-INTESTINE | RECEPTOR | TASTE | SECRETION | RELEASE | CELL BIOLOGY | KATP Channels - physiology | Glucagon-Like Peptide 1 - metabolism | Calcium - metabolism | Electrophysiology | Mice, Transgenic | Glucose - pharmacology | Glucokinase - metabolism | Calcium - chemistry | Glucagon-Like Peptide 1 - secretion | Animals | Biological Transport | Glucose - metabolism | Mice | Enteroendocrine Cells - metabolism | Type 2 diabetes | Fluorescence | Glucose | Peptides | Dextrose | Tolbutamide | Short
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2. Full Text Interleukin-6 enhances insulin secretion by increasing glucagon-like peptide-1 secretion from L cells and alpha cells
by Ellingsgaard, Helga and Hauselmann, Irina and Schuler, Beat and Habib, Abdella M and Baggio, Laurie L and Meier, Daniel T and Eppler, Elisabeth and Bouzakri, Karim and Wueest, Stephan and Muller, Yannick D and Hansen, Ann Maria Kruse and Reinecke, Manfred and Konrad, Daniel and Gassmann, Max and Reimann, Frank and Halban, Philippe A and Gromada, Jesper and Drucker, Daniel J and Gribble, Fiona M and Ehses, Jan A and Donath, Marc Y
Nature Medicine, ISSN 1078-8956, 11/2011, Volume 17, Issue 11, pp. 1481 - 1489
Exercise, obesity and type 2 diabetes are associated with elevated plasma concentrations of interleukin-6 (IL-6). Glucagon-like peptide-1 (GLP-1) is a hormone... 
MEDICINE, RESEARCH & EXPERIMENTAL | GLP-1 | BENEFICIAL ROLE | BIOCHEMISTRY & MOLECULAR BIOLOGY | EXERCISE | CELL BIOLOGY | SKELETAL-MUSCLE | IN-VITRO | GLUCOSE-HOMEOSTASIS | HORMONE | BETA-CELL | MICE | EXPRESSION | Interleukin-6 - antagonists & inhibitors | Humans | Male | Diabetes Mellitus, Type 2 - metabolism | Glucagon-Secreting Cells - drug effects | Diet, High-Fat | Glucagon-Secreting Cells - metabolism | Female | Enteroendocrine Cells - metabolism | Insulin Secretion | Interleukin-6 - metabolism | Physical Conditioning, Animal | Disease Models, Animal | Glucose Tolerance Test | Glucagon-Like Peptide 1 - metabolism | Interleukin-6 - genetics | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Insulin - metabolism | Animals | Diabetes Mellitus, Type 2 - physiopathology | Interleukin-6 - pharmacology | Signal Transduction - drug effects | Signal Transduction - physiology | Mice | Blood Glucose - metabolism | Enteroendocrine Cells - drug effects | Type 2 diabetes | Care and treatment | Physiological aspects | Genetic aspects | Research | Insulin | Interleukin-6 | Peptides | Cellular biology | Pharmacology | Diabetes | Pancreas | Endocrinology
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3. Full Text Bacterial Metabolite Indole Modulates Incretin Secretion from Intestinal Enteroendocrine L Cells
by Chimerel, Catalin and Emery, Edward and Summers, David K and Keyser, Ulrich and Gribble, Fiona M and Reimann, Frank
Cell Reports, ISSN 2211-1247, 11/2014, Volume 9, Issue 4, pp. 1202 - 1208
It has long been speculated that metabolites, produced by gut microbiota, influence host metabolism in health and diseases. Here, we reveal that indole,... 
TRYPTOPHAN | COLONIC BACTERIA | LIRAGLUTIDE | CHAIN FATTY-ACIDS | MOUSE | LINE | GUT MICROBIOTA | RECEPTOR AGONIST | PEPTIDE-1 SECRETION | SIGNAL INDOLE | CELL BIOLOGY | Colon - cytology | Calcium - metabolism | Cells, Cultured | Ions | Adenosine Triphosphate - biosynthesis | Enteroendocrine Cells - secretion | Glucagon-Like Peptide 1 - secretion | Animals | Potassium Channels, Voltage-Gated - metabolism | Adenosine Triphosphate - metabolism | Potassium Chloride - pharmacology | Incretins - secretion | Indoles - pharmacology | Mice | Adenosine Diphosphate - metabolism | NADP - metabolism | Enteroendocrine Cells - drug effects | Action Potentials - drug effects
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4. Full Text Intestinal mTOR regulates GLP-1 production in mouse L cells
by Xu, Geyang and Li, Ziru and Ding, Li and Tang, Hong and Guo, Song and Liang, Hongbin and Wang, Huadong and Zhang, Weizhen
Diabetologia, ISSN 0012-186X, 8/2015, Volume 58, Issue 8, pp. 1887 - 1897
Glucagon-like peptide (GLP-1), an intestinal incretin produced in L cells through proglucagon processing, is released in response to meal intake. The... 
Blood glucose | GLP-1 | Medicine & Public Health | Human Physiology | Intestine | Metabolic Diseases | mTOR | Internal Medicine | Enteroendocrine L cells | Neurogenin 3 | MAMMALIAN TARGET | RAPAMYCIN | PROGLUCAGON GENE-EXPRESSION | RAT MODEL | GLUCAGON-LIKE PEPTIDE-1 | GLUCOSE | IN-VIVO | ENDOCRINOLOGY & METABOLISM | PEPTIDASE-4 INHIBITOR SITAGLIPTIN | ENDOCRINE-CELLS | MOLECULAR-MECHANISMS | Intestines - drug effects | Leucine - pharmacology | Glucagon-Like Peptide 1 - metabolism | TOR Serine-Threonine Kinases - metabolism | Down-Regulation | Mice, Inbred C57BL | Male | Intestines - metabolism | Sirolimus - pharmacology | Mice, Knockout | Animals | Diet, High-Fat | Mice | Enteroendocrine Cells - metabolism | Fasting - metabolism | Diabetes Mellitus, Experimental - metabolism | Enteroendocrine Cells - drug effects | Proglucagon - metabolism | Physiological aspects | Enzymes | Sensors | Blood sugar | Cells
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5. Intestinal enteroendocrine L cells in amino acid sensing and diseases
by Ding, Xueqin and Hu, Chien-An Andy and Huang, Pengfei and Li, Yali and He, Shanping and Yang, Huansheng and Li, Jianzhong and Xie, Huaping and Yin, Yulong
Frontiers in Bioscience - Landmark, ISSN 1093-9946, 03/2018, Volume 23, Issue 9, pp. 1740 - 1753
Enteroendocrine L cells are open-type enteroendocrine cells that play an important role in amino acid sensing. They detect amino acids by a number of membrane... 
Disease | Electrical activity | Amino acid sensing | Intestinal enteroendocrine cells | L cells | Review | Hormone secretion | SIGNALING PATHWAYS | FOOD-INTAKE | BIOCHEMISTRY & MOLECULAR BIOLOGY | MICROELECTRODE ARRAYS | CELL BIOLOGY | TYPE-2 DIABETIC-PATIENTS | GLUCAGON-LIKE PEPTIDE-1 | LINE STC-1 | PROTEIN-COUPLED RECEPTOR | COLORECTAL-CANCER | GASTROINTESTINAL HORMONES | GENE-TRANSCRIPTION | Humans | Receptors, Cell Surface - metabolism | Diabetes Mellitus, Type 2 - metabolism | Obesity - metabolism | Amino Acids - metabolism | Animals | Signal Transduction - drug effects | Gastrointestinal Hormones - metabolism | Enteroendocrine Cells - physiology | Signal Transduction - physiology | Enteroendocrine Cells - metabolism | Glutamine - pharmacology | Enteroendocrine Cells - drug effects
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6. Full Text Ghrelin regulates GLP-1 production through mTOR signaling in L cells
by Xu, Geyang and Hong, Xiaosi and Tang, Hong and Jiang, Sushi and Liu, Fenting and Shen, Zhemin and Li, Ziru and Zhang, Weizhen
Molecular and Cellular Endocrinology, ISSN 0303-7207, 11/2015, Volume 416, Issue C, pp. 9 - 18
Glucagon-like peptide (GLP-1), an intestinal incretin produced in L-cells and released in response to meal intake, functions to promote insulin secretion and... 
Incretin | GLP-1 | Enteroendocrine L cells | Ghrelin | GHSR1a | mTORC1 | MTORC1 | GLYCEMIC CONTROL | HUMANS | INSULIN-RELEASE | CELL BIOLOGY | GLUCAGON-LIKE PEPTIDE-1 | GENE | GLUCOSE-METABOLISM | BIOLOGY | ENDOCRINOLOGY & METABOLISM | DEGRADATION | IMPROVES | SECRETION | Glucagon-Like Peptide 1 - metabolism | TOR Serine-Threonine Kinases - metabolism | Mice, Inbred C57BL | Ileum - metabolism | Incretins - metabolism | Ghrelin - pharmacology | Male | Oligopeptides - metabolism | Receptors, Ghrelin - genetics | Mice, Knockout | Ghrelin - metabolism | Insulin - metabolism | Animals | Cell Line, Tumor | Mice | Enteroendocrine Cells - metabolism | Blood Glucose - metabolism | Enteroendocrine Cells - drug effects | Insulin Secretion | Glucagon | Dextrose | Glucose
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7. Full Text Enteroendocrine K and L cells in healthy and type 2 diabetic individuals
by Jorsal, Tina and Jorsal, Tina and Rhee, Nicolai A and Rhee, Nicolai A and Pedersen, Jens and Pedersen, Jens and Wahlgren, Camilla D and Wahlgren, Camilla D and Mortensen, Brynjulf and Mortensen, Brynjulf and Jepsen, Sara L and Jepsen, Sara L and Jelsing, Jacob and Jelsing, Jacob and Dalbøge, Louise S and Dalbøge, Louise S and Vilmann, Peter and Vilmann, Peter and Hassan, Hazem and Hassan, Hazem and Hendel, Jakob W and Hendel, Jakob W and Poulsen, Steen S and Poulsen, Steen S and Holst, Jens J and Holst, Jens J and Vilsbøll, Tina and Vilsbøll, Tina and Knop, Filip K and Knop, Filip K
Diabetologia, ISSN 0012-186X, 2/2018, Volume 61, Issue 2, pp. 284 - 294
Enteroendocrine K and L cells are pivotal in regulating appetite and glucose homeostasis. Knowledge of their distribution in humans is sparse and it is unknown... 
Immunohistochemistry | Type 2 diabetes | Chromogranin A | Glucose-dependent insulinotropic polypeptide | Human Physiology | Metabolic Diseases | Double-balloon enteroscopy | Internal Medicine | Peptide YY | mRNA expression | Prohormone convertase | Glucagon-like peptide-1 | Medicine & Public Health | Enteroendocrine cells | ChromograninA | FOOD-INTAKE | CHROMOGRANIN | SMALL-INTESTINE | GLUCOSE | ENDOCRINOLOGY & METABOLISM | DEPENDENT INSULINOTROPIC POLYPEPTIDE | SECRETION | GASTRIC-INHIBITORY POLYPEPTIDE | HUMAN GASTROINTESTINAL-TRACT | ENDOCRINE-CELLS | Appetite | Glucagon | Diabetes mellitus | Mucosa | Homeostasis | L cells | GIP protein | Gene expression | Small intestine | Biopsy | Large intestine | Prohormone convertase 2 | Prohormone convertase 1 | Diabetes | Colon | Glucagon-like peptide 1
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8. Full Text High fat diet impairs the function of glucagon-like peptide-1 producing L-cells
by Richards, Paul and Pais, Ramona and Habib, Abdella M and Brighton, Cheryl A and Yeo, Giles S.H and Reimann, Frank and Gribble, Fiona M
Peptides, ISSN 0196-9781, 03/2016, Volume 77, pp. 21 - 27
Glucagon-like peptide-1 (GLP-1) acts as a satiety signal and enhances insulin release. This study examined how GLP-1 production from intestinal L-cells is... 
Enteroendocrine | GLP-1 | L-cell | High fat diet | NUMBER | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | INTESTINE | GLUCOSE | HELIX TRANSCRIPTION FACTORS | ENTEROENDOCRINE CELLS | PHARMACOLOGY & PHARMACY | SECRETION | DIFFERENTIATION | EXPRESSION | Gene Expression | Animals | Glucagon-Like Peptide 1 - metabolism | Mice, Inbred C57BL | Cells, Cultured | Diet, High-Fat - adverse effects | Enteroendocrine Cells - physiology | Male | Primary Cell Culture | Glucagon-Like Peptide 1 - genetics | Glucagon-Like Peptide 1 - secretion | Glucagon | Diets | Reduction | Nutrients | Mathematical models | Mice | Gene expression | Secretions | Insulin
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9. Full Text Hypertrophy Dependent Doubling of L-Cells in Roux-en-Y Gastric Bypass Operated Rats
by Hansen, Carl Frederik and Bueter, Marco and Theis, Nadine and Lutz, Thomas and Paulsen, Sarah and Dalbøge, Louise S and Vrang, Niels and Jelsing, Jacob
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e65696
Background and Aims: Roux-en-Y gastric bypass (RYGB) leads to a rapid remission of type 2 diabetes mellitus (T2DM), but the underlying mode of action remains... 
DIABETES-MELLITUS | GUT HORMONES | JEJUNOILEAL BYPASS | GLUCAGON-LIKE PEPTIDE-1 | GLUCOSE-METABOLISM | BILIOPANCREATIC DIVERSION | MULTIDISCIPLINARY SCIENCES | WEIGHT-LOSS | BARIATRIC SURGERY | MORBID-OBESITY | INTESTINAL EPITHELIAL PROLIFERATION | Hypertrophy - etiology | Glucagon-Like Peptide 1 - metabolism | Peptide YY - metabolism | Rats, Wistar | Gastric Bypass - adverse effects | Rats | Male | Enteroendocrine Cells - cytology | Glucagon-Like Peptide 1 - genetics | Hypertrophy - physiopathology | Mucous Membrane - metabolism | Animals | Proglucagon - genetics | Enteroendocrine Cells - metabolism | Peptide YY - genetics | Proglucagon - metabolism | Type 2 diabetes | Glucagon | RNA | Body weight | Gastric bypass | Gene expression | Hypertrophy | Transcription | Mucosa | Adaptability | Hormones | Small intestine | Cell density | Dietary restrictions | Food availability | Surgery | Rodents | Remission | Digestive tract | Glucagon-like peptide 1 | Food | Obesity | Digestive system | Diabetes mellitus | Mode of action | Cell number | Gastrointestinal surgery
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10. Full Text Farnesoid X receptor inhibits glucagon-like peptide-1 production by enteroendocrine L cells
by Trabelsi, Mohamed-Sami and Daoudi, Mehdi and Prawitt, Janne and Ducastel, Sarah and Touche, Véronique and Sayin, Sama I and Perino, Alessia and Brighton, Cheryl A and Sebti, Yasmine and Kluza, Jérôme and Briand, Olivier and Dehondt, Hélène and Vallez, Emmanuelle and Dorchies, Emilie and Baud, Grégory and Spinelli, Valeria and Hennuyer, Nathalie and Caron, Sandrine and Bantubungi, Kadiombo and Caiazzo, Robert and Reimann, Frank and Marchetti, Philippe and Lefebvre, Philippe and Bäckhed, Fredrik and Gribble, Fiona M and Schoonjans, Kristina and Pattou, François and Tailleux, Anne and Staels, Bart and Lestavel, Sophie and Institute of Medicine, Department of Molecular and Clinical Medicine and Wallenberg Laboratory and Göteborgs universitet and Gothenburg University and Sahlgrenska Academy and Sahlgrenska akademin and Institutionen för medicin, avdelningen för molekylär och klinisk medicin and Wallenberglaboratoriet
Nature Communications, ISSN 2041-1723, 2015, Volume 6, Issue 1, p. 7629
Bile acids are signalling molecules, which activate the transmembrane receptor TGR5 and the nuclear receptor FXR. BA sequestrants (BAS) complex bile acids in... 
GLP-1 SECRETION | FXR | BILE-ACID RECEPTORS | GLUCOSE-HOMEOSTASIS | OBESITY | MULTIDISCIPLINARY SCIENCES | RAT SMALL-INTESTINE | MICE | TYPE-2 DIABETES-MELLITUS | METABOLIC-RATE | EXPRESSION | Colon - cytology | Intestinal Mucosa - metabolism | Sequestering Agents - pharmacology | Humans | Ileum - metabolism | RNA, Messenger - metabolism | Colesevelam Hydrochloride - pharmacology | Obesity - genetics | Glucagon-Like Peptide 1 - genetics | Jejunum - metabolism | Insulin-Secreting Cells - metabolism | Proglucagon - drug effects | Diet, High-Fat | Jejunum - cytology | Enteroendocrine Cells - metabolism | Ileum - cytology | Proglucagon - metabolism | Insulin Secretion | Glucagon-Like Peptide 1 - metabolism | Signal Transduction | Bile Acids and Salts - metabolism | Nuclear Proteins - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Colon - metabolism | Mice, Knockout | Obesity - metabolism | Transcription Factors - metabolism | Insulin - metabolism | Animals | Glycolysis | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - metabolism | Mice, Obese | Mice | Proglucagon - genetics | Receptors, G-Protein-Coupled - genetics | Blood Glucose - metabolism | Anticholesteremic Agents - pharmacology | Intestines - cytology | Carbohydrates | Glucose | Gene expression | Insulin | Cell and Molecular Biology | Endokrinologi och diabetes | Multidisciplinary Sciences | Cell- och molekylärbiologi | Endocrinology and Diabetes
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11. Full Text Lipid derivatives activate GPR119 and trigger GLP-1 secretion in primary murine L-cells
by Moss, Catherine E and Glass, Leslie L and Diakogiannaki, Eleftheria and Pais, Ramona and Lenaghan, Carol and Smith, David M and Wedin, Marianne and Bohlooly-Y, Mohammad and Gribble, Fiona M and Reimann, Frank
Peptides, ISSN 0196-9781, 03/2016, Volume 77, pp. 16 - 20
Glucagon-like peptide-1 (GLP-1) is an incretin hormone derived from proglucagon, which is released from intestinal L-cells and increases insulin secretion in a... 
Incretin | GPR119 | GLP-1 | PATHWAYS | GLYCEMIC CONTROL | BIOCHEMISTRY & MOLECULAR BIOLOGY | OLEOYLETHANOLAMIDE | RELEASE | AGONISTS | DISCOVERY | G-PROTEIN-COUPLED-RECEPTOR-119 | GLUCAGON-LIKE PEPTIDE-1 | DEPENDENT INSULIN-SECRETION | PROTEIN-COUPLED RECEPTOR | PHARMACOLOGY & PHARMACY | Receptors, G-Protein-Coupled - metabolism | Cells, Cultured | Male | Mice, Transgenic | Olive Oil - administration & dosage | Corn Oil - pharmacology | Enteroendocrine Cells - secretion | Glucagon-Like Peptide 1 - secretion | Olive Oil - pharmacology | Animals | Female | Primary Cell Culture | Enteroendocrine Cells - drug effects | Cyclic AMP - metabolism | Second Messenger Systems | Cyclic adenylic acid | Sensors | Intestines | Fretting | Lipids | Ligands | Mice | Derivatives | Secretions | Culture | Fsk, forskolin | WT, wildtype | KO, knockout | GLP-1, glucagon-like peptide-1 | IBMX, 3-isobutyl-1-methylxanthine | 2-OG, 2-oleoylglycerol | OEA, oleoylethanolamide | PPAR, peroxisome-proliferator-activated receptor
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12. Full Text Signalling pathways involved in the detection of peptones by murine small intestinal enteroendocrine L-cells
by Pais, Ramona and Gribble, Fiona M and Reimann, Frank
Peptides, ISSN 0196-9781, 03/2016, Volume 77, pp. 9 - 15
Glucagon like peptide-1 is an insulinotropic hormone released from intestinal L-cells in response to food ingestion. Here, we investigated mechanisms... 
Enteroendocrine | GLP-1 | L-cell | Peptones | PROTEIN | INCRETIN HORMONE | STIMULATE | BIOCHEMISTRY & MOLECULAR BIOLOGY | GLUTAG CELLS | HYDROLYSATE | GLUCAGON-LIKE PEPTIDE-1 | CALCIUM-SENSING RECEPTOR | AMINO-ACIDS | ENDOCRINOLOGY & METABOLISM | PHARMACOLOGY & PHARMACY | SECRETION | CA2+-SENSING RECEPTOR | Animals | Peptones - physiology | Tissue Culture Techniques | Enteroendocrine Cells - physiology | Mice, Transgenic | Peptones - pharmacology | Calcium Channels, L-Type - metabolism | Transient Receptor Potential Channels - metabolism | Enteroendocrine Cells - drug effects | Calcium Signaling | Glucagon-Like Peptide 1 - secretion | Type 2 diabetes | Enzymes | Protein hydrolysates | Receptors | Calcium | Pathways | Inhibition | Detection | Channels | Meat
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13. Full Text Enteroendocrine L Cells Sense LPS after Gut Barrier Injury to Enhance GLP-1 Secretion
by Lebrun, Lorène J and Lenaerts, Kaatje and Kiers, Dorien and Pais de Barros, Jean-Paul and Le Guern, Naig and Plesnik, Jiri and Thomas, Charles and Bourgeois, Thibaut and Dejong, Cornelis H.C and Kox, Matthijs and Hundscheid, Inca H.R and Khan, Naim Akhtar and Mandard, Stéphane and Deckert, Valérie and Pickkers, Peter and Drucker, Daniel J and Lagrost, Laurent and Grober, Jacques
Cell Reports, ISSN 2211-1247, 10/2017, Volume 21, Issue 5, pp. 1160 - 1168
Glucagon-like peptide 1 (GLP-1) is a hormone released from enteroendocrine L cells. Although first described as a glucoregulatory incretin hormone, GLP-1 also... 
enteroendocrine cells | lipopolysaccharides | intestinal ischemia | inflammation | glucagon-like peptide 1 | TLR4 | gut injury | MORTALITY | PHYSIOLOGY | INHIBITION | GLUCAGON-LIKE PEPTIDE-1 | MECHANISM | RECEPTOR AGONISTS | INFLAMMATION | INSULIN-SECRETION | LIPOPOLYSACCHARIDE | MICE | CELL BIOLOGY | Life Sciences | Food and Nutrition
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