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Cell, ISSN 0092-8674, 02/2018, Volume 172, Issue 4, pp. 811 - 824.e14
Type I interferon (IFN) is produced when host sensors detect foreign nucleic acids, but how sensors differentiate self from nonself nucleic acids, such... 
type I interferon | neuronal progenitor cells | RNA editing | innate immunity | PKR | Aicardi-Goutieres syndrome | Alu elements | translation | AGS | ADAR1 | MDA5 | INTERFERON | COMPLEX | INHIBITION | PROTEIN-KINASE PKR | LANDSCAPE | BIOCHEMISTRY & MOLECULAR BIOLOGY | BINDING | CELL BIOLOGY | Inflammation - pathology | RNA-Binding Proteins - genetics | Interferon-Induced Helicase, IFIH1 - genetics | Protein Biosynthesis | Nervous System Malformations - immunology | Adenosine Deaminase - metabolism | eIF-2 Kinase - metabolism | Humans | Cell Death - immunology | eIF-2 Kinase - immunology | RNA Polymerase II - metabolism | Neural Stem Cells - cytology | RNA Polymerase II - immunology | Inflammation - metabolism | Neural Stem Cells - immunology | Cell Death - genetics | Nervous System Malformations - metabolism | Autoimmune Diseases of the Nervous System - genetics | HEK293 Cells | RNA, Double-Stranded - metabolism | Autoimmune Diseases of the Nervous System - metabolism | Nervous System Malformations - genetics | eIF-2 Kinase - genetics | Adenosine Deaminase - genetics | RNA-Binding Proteins - immunology | Interferon-Induced Helicase, IFIH1 - metabolism | Inflammation - immunology | Gene Knockout Techniques | Interferon-Induced Helicase, IFIH1 - immunology | Neural Stem Cells - pathology | Autoimmune Diseases of the Nervous System - immunology | RNA, Double-Stranded - genetics | RNA, Double-Stranded - immunology | Inflammation - genetics | RNA Polymerase II - genetics | Alu Elements | Neural Stem Cells - metabolism | Adenosine Deaminase - immunology | RNA-Binding Proteins - metabolism | Prevention | Enzymes | Medical colleges | Messenger RNA | Microbiology | Neurons | Cell death | Communicable diseases | Interferon | Sensors | Biological response modifiers | Index Medicus
Journal Article
SCIENCE, ISSN 0036-8075, 09/2015, Volume 349, Issue 6252, pp. 1115 - 1120
Adenosine-to-inosine (A-to-I) editing is a highly prevalent posttranscriptional modification of RNA, mediated by ADAR (adenosine deaminase acting on RNA)... 
APOPTOSIS | INTERFERON | ADENOSINE-DEAMINASE | GENE | MULTIDISCIPLINARY SCIENCES | MOUSE | SITES | MUTATIONS | IDENTIFICATION | ENZYME ADAR1 | GENOME
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2013, Volume 110, Issue 3, pp. 1041 - 1046
The molecular etiology of human progenitor reprogramming into self-renewing leukemia stem cells (LSC) has remained elusive. Although DNA sequencing has... 
Protein isoforms | Up regulation | RNA editing | Cord blood | Genes | Leukemia | Stem cells | Chronic myeloid leukemia | Progenitor cells | Cells | CHRONIC MYELOGENOUS LEUKEMIA | ADENOSINE-DEAMINASE | CML | MESSENGER-RNA | GENE | MULTIDISCIPLINARY SCIENCES | HEMATOPOIETIC STEM-CELLS | IMATINIB | HUMAN TRANSCRIPTOME | ACCURATE IDENTIFICATION | EDITING SITES | Alternative Splicing | Adenosine Deaminase - metabolism | RNA Editing | Humans | Leukemia, Myeloid, Chronic-Phase - pathology | Transcriptome | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Glycogen Synthase Kinase 3 beta | Transplantation, Heterologous | RNA-Binding Proteins | Gene Knockdown Techniques | Neoplastic Stem Cells - metabolism | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Inflammation Mediators - metabolism | Neoplastic Stem Cells - pathology | Leukemia, Myeloid, Chronic-Phase - metabolism | Tumor Stem Cell Assay | Blast Crisis - pathology | Adenosine Deaminase - genetics | Blast Crisis - etiology | Glycogen Synthase Kinase 3 - metabolism | Disease Progression | Fusion Proteins, bcr-abl - genetics | Animals | Glycogen Synthase Kinase 3 - genetics | Cell Transformation, Neoplastic | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Blast Crisis - genetics | Mice | Blast Crisis - metabolism | Fusion Proteins, bcr-abl - metabolism | Leukemia, Myeloid, Chronic-Phase - genetics | Physiological aspects | Nuclear reprogramming | Research | Adenosine deaminase | Health aspects | Enzymes | Mutation | Gene expression | Ribonucleic acid--RNA | Index Medicus | Biological Sciences
Journal Article
Molecular Cell, ISSN 1097-2765, 03/2019, Volume 73, Issue 5, pp. 866 - 868
Three recent studies by , and show that deleting RNA editing enzyme ADAR1 could induce higher cell lethality and render tumor cells more vulnerable to... 
RNA | BIOCHEMISTRY & MOLECULAR BIOLOGY | CELL BIOLOGY | Enzymes | Immunotherapy | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 01/2019, Volume 565, Issue 7737, pp. 43 - 48
Most patients with cancer either do not respond to immune checkpoint blockade or develop resistance to it, often because of acquired mutations that impair... 
RECRUITMENT | RESPONSES | CELLS | THERAPY | PD-1 BLOCKADE | MULTIDISCIPLINARY SCIENCES | EVASION | RADIATION | CANCER | I INTERFERON | RNA-Binding Proteins - genetics | Receptors, G-Protein-Coupled - metabolism | Adenosine Deaminase - metabolism | RNA Editing | Melanoma, Experimental - drug therapy | Melanoma, Experimental - radiotherapy | Programmed Cell Death 1 Receptor - antagonists & inhibitors | Interferons - immunology | Melanoma, Experimental - immunology | Immunotherapy | Female | Adenosine Deaminase - deficiency | Adenosine Deaminase - genetics | Mice, Inbred C57BL | Histocompatibility Antigens Class I - immunology | Interferon-Induced Helicase, IFIH1 - metabolism | Inflammation - immunology | CRISPR-Cas Systems - genetics | Drug Resistance, Neoplasm - genetics | Phenotype | Animals | Melanoma, Experimental - genetics | Cell Cycle Checkpoints - drug effects | RNA, Double-Stranded - genetics | Cell Line, Tumor | Inflammation - genetics | Mice | RNA-Binding Proteins - metabolism | Drug Resistance, Neoplasm - drug effects | Enzymes | Oncology, Experimental | Physiological aspects | Genetic aspects | Research | Tumors | Cancer | PD-1 protein | CD8 antigen | Editing | Cytotoxicity | Lymphocytes T | Genomes | Inactivation | Immunity | Anticancer properties | Cell recognition | Lymphocytes | Dimensional analysis | DNA methylation | Bioinformatics | Antigen presentation | CRISPR | RNA editing | Deactivation | Double-stranded RNA | Inflammation | Gene expression | Ribonucleic acid--RNA | Immune checkpoint | Ligands | Interferon | Mutation | Chemokines | Index Medicus
Journal Article