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The Journal of Immunology, ISSN 0022-1767, 06/2006, Volume 176, Issue 11, pp. 6785 - 6793
Fas (CD95)-mediated apoptosis is an essential mechanism for the maintenance of homeostasis, and disruption of this death pathway contributes to many human... 
DRUG-INDUCED APOPTOSIS | PROTEIN-KINASE-B | SMOOTH-MUSCLE-CELLS | ANGIOTENSIN-II | HYDROGEN-PEROXIDE | MEDIATED APOPTOSIS | IMMUNOLOGY | NF-KAPPA-B | TRANSCRIPTION FACTOR | APO-1/FAS RECEPTOR/LIGAND SYSTEM | FLICE-INHIBITORY PROTEIN | Phosphorylation | Membrane Glycoproteins - metabolism | Apoptosis - drug effects | Reactive Oxygen Species - pharmacology | Intracellular Signaling Peptides and Proteins - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | fas Receptor - biosynthesis | fas Receptor - metabolism | Enzyme Activation - immunology | Proto-Oncogene Proteins c-akt - genetics | RNA, Messenger - biosynthesis | Signal Transduction - immunology | Membrane Glycoproteins - physiology | Membrane Glycoproteins - antagonists & inhibitors | RNA, Messenger - antagonists & inhibitors | Epidermis - enzymology | fas Receptor - physiology | Chromones - pharmacology | Proto-Oncogene Proteins c-akt - metabolism | Cell Line | Epidermis - metabolism | Tumor Necrosis Factors - metabolism | Morpholines - pharmacology | Proto-Oncogene Proteins c-akt - physiology | Enzyme Activation - drug effects | Fas Ligand Protein | Tumor Necrosis Factors - physiology | Apoptosis - immunology | Androstadienes - pharmacology | Epidermis - immunology | Signal Transduction - drug effects | Wortmannin | Epidermal Cells | Phosphatidylinositol 3-Kinases - physiology | CASP8 and FADD-Like Apoptosis Regulating Protein | Protein Kinase Inhibitors - pharmacology | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Tumor Necrosis Factors - antagonists & inhibitors
Journal Article
Molecular Carcinogenesis, ISSN 0899-1987, 03/2008, Volume 47, Issue 3, pp. 172 - 183
The cancer preventive action of (−)‐epigallocatechin gallate (EGCG), found in green tea, is strongly supported by epidemiology and laboratory research data.... 
cell transformation | Fyn | epidermal growth factor | EGCG | CANCER-CELLS | ACTIVATED PROTEIN-KINASE | INDUCED PHOSPHORYLATION | TYROSINE KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | ONCOLOGY | HISTONE H3 | EPIDERMAL-GROWTH-FACTOR | BLACK TEA | GENE-EXPRESSION | TUMOR-GROWTH | GREEN TEA COMPONENT | Adaptor Proteins, Signal Transducing - chemistry | Recombinant Fusion Proteins - isolation & purification | Activating Transcription Factor 2 - antagonists & inhibitors | Cell Transformation, Neoplastic - chemically induced | Recombinant Fusion Proteins - metabolism | Transcription Factor AP-1 - metabolism | Epidermal Growth Factor - toxicity | Dose-Response Relationship, Drug | CREB-Binding Protein - metabolism | Time Factors | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Phosphorylation - drug effects | Catechin - metabolism | Catechin - pharmacology | Cell Survival - drug effects | RNA, Small Interfering - pharmacology | Glutathione Transferase - metabolism | Antioxidants - pharmacology | Recombinant Fusion Proteins - chemistry | STAT1 Transcription Factor - antagonists & inhibitors | Animals | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Adaptor Proteins, Signal Transducing - genetics | Protein Binding | Catechin - analogs & derivatives | Mice | Cell Transformation, Neoplastic - drug effects | Kinetics | Adaptor Proteins, Signal Transducing - metabolism | Protein Structure, Tertiary - drug effects
Journal Article
Journal Article
Environmental Health Perspectives, ISSN 0091-6765, 4/2007, Volume 115, Issue 4, pp. 513 - 518
Background: Arsenic contamination has become a major public health concern worldwide. Epidemiologic data show that long-term arsenic exposure results in the... 
Cell growth | Arsenic | Memory interference | Epidermal cells | Gene expression regulation | Skin | Research | Carcinogenesis | Endothelial cells | Arsenites | Apoptosis | COX-2 | NFκB | Arsenite | apoptosis | carcinogenesis | arsenite
Journal Article
Lipids, ISSN 0024-4201, 09/2009, Volume 44, Issue 9, p. 777
Rhizochalin [(2R,3R,26R,27R)-2,27-diamino-3-hydroxy-26-[(2R,3R,4S,6R)-3,4,5-trihydroxy-6-(hydroxymethyl)tetrahydro-2H-pyran-2-yloxy]octacosan-11-one], an... 
Epidermal growth factor | Physiological aspects | Sphingolipids | Research
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 07/2003, Volume 278, Issue 29, pp. 26435 - 26442
Journal Article
Journal Article
Carcinogenesis, ISSN 0143-3334, 08/2006, Volume 27, Issue 8, pp. 1617 - 1626
Dietary freeze-dried black raspberries inhibit tumor induction by N-nitrosomethylbenzylamine in the rat esophagus, but the constituents responsible for this... 
DIOL-EPOXIDE | EPIDERMAL CL41 CELLS | ONCOLOGY | TRANSACTIVATION | RAT ESOPHAGEAL TUMORIGENESIS | ACTIVATOR PROTEIN-1 ACTIVATION | CHEMOPREVENTION | ELLAGIC ACID | C-JUN | CANCER | MOLECULAR-MECHANISMS
Journal Article
by Cai, T and Li, X and Ding, J and Luo, W and Li, J and Huang, C
Current Cancer Drug Targets, ISSN 1568-0096, 2011, Volume 11, Issue 5, pp. 548 - 559
Cyclooxygenase-2 (COX-2) is a critical enzyme implicated in chronic inflammation-associated cancer development. Our studies have shown that the exposure of... 
NF-κB | Nickel | COX-2 | Beas-2B cells | ROS | NFAT | nickel | BRONCHIAL EPITHELIAL-CELLS | CYCLOOXYGENASE-2 INDUCTION | TNF-ALPHA INDUCTION | BREAST-CANCER | LUNG-CANCER | TUMOR-NECROSIS-FACTOR | EPIDERMAL CL41 CELLS | NF-kappa B | ACTIVATED T-CELLS | PLASMINOGEN-ACTIVATOR | ONCOLOGY | NUCLEAR-FACTOR
Journal Article
Cancer Research, ISSN 0008-5472, 08/2005, Volume 65, Issue 15, pp. 6601 - 6611
Transcription factor p53 and phosphatase PTEN are two tumor suppressors that play essential roles in suppression of carcinogenesis. However, the mechanisms by... 
DIOL-EPOXIDE | EPIDERMAL CL41 CELLS | AP-1 TRANSACTIVATION | INDUCED PHOSPHORYLATION | ONCOLOGY | INDUCED APOPTOSIS | GROWTH ARREST | DNA-DAMAGE | P38 KINASE | HUMAN CANCER | PIFITHRIN-ALPHA
Journal Article
Journal Article
Journal Article
Journal Article
Neuroendocrinology, ISSN 0028-3835, 06/2010, Volume 91, Issue 3, pp. 268 - 278
Gastroenteropancreatic (GEP) endocrine tumors are hypervascular tumors able to synthesize and secrete high amounts of VEGF. We aimed to study the regulation of... 
Neuroendocrine Tumors | Angiogenesis | Neuroendocrine cell lines | Octreotide | VEGF-mTOR inhibitors | HYPOXIA-INDUCIBLE FACTOR | SOMATOSTATIN ANALOGS | PHOSPHATIDYLINOSITOL 3-KINASE | MICROVESSEL DENSITY | NEUROSCIENCES | RAD001 EVEROLIMUS | EPIDERMAL CL41 CELLS | IN-VITRO | SIGNALING PATHWAY | ENDOCRINOLOGY & METABOLISM | UP-REGULATION | ENDOTHELIAL GROWTH-FACTOR | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Hypoxia-Inducible Factor 1, alpha Subunit - antagonists & inhibitors | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Phosphatidylinositol 3-Kinases - antagonists & inhibitors | Neuroendocrine Tumors - enzymology | Somatostatin - drug effects | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Flavonoids - pharmacology | Phosphorylation - drug effects | Chromones - pharmacology | Insulin Secretion | Somatostatin - metabolism | Cell Survival - drug effects | MAP Kinase Kinase 1 - antagonists & inhibitors | Neuroendocrine Tumors - metabolism | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | Morpholines - pharmacology | Rats | Imidazoles - pharmacology | Octreotide - pharmacology | Sirolimus - pharmacology | Drug Synergism | Insulin - metabolism | Animals | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cell Line, Tumor | Mice | Pyridines - pharmacology | TOR Serine-Threonine Kinases | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Life Sciences | Cancer
Journal Article