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Oncogene, ISSN 1476-5594, 2013, Volume 33, Issue 18, pp. 2307 - 2316
Signals from the tumor microenvironment trigger cancer cells to adopt an invasive phenotype through epithelial-mesenchymal transition (EMT). Relatively little... 
Vimentin | Epithelial-mesenchymal transition | Signal transduction | Breast cancer | Calcium | STAT3 | MIGRATION | ACTIVATION | calcium | METASTASIS | signal transduction | BIOCHEMISTRY & MOLECULAR BIOLOGY | PHENOTYPE | breast cancer | vimentin | CELL BIOLOGY | ONCOLOGY | GENETICS & HEREDITY | GENE-EXPRESSION | epithelial-mesenchymal transition | TRPM7 | CHANNELS | UP-REGULATION | CONTRIBUTES | PROGRESSION | RNA, Small Interfering - genetics | Phosphorylation | Vimentin - biosynthesis | Calcium - metabolism | Epithelial-Mesenchymal Transition - physiology | Humans | Protein-Serine-Threonine Kinases | Epidermal Growth Factor - metabolism | Epithelial-Mesenchymal Transition - drug effects | Epithelial-Mesenchymal Transition - genetics | Breast Neoplasms - metabolism | Cell Hypoxia | TRPM Cation Channels - antagonists & inhibitors | Mitogen-Activated Protein Kinase 3 - metabolism | Breast Neoplasms - pathology | Cell Line, Tumor | TRPM Cation Channels - genetics | Female | TRPM Cation Channels - metabolism | Epidermal Growth Factor - pharmacology | Calcium Signaling | STAT3 Transcription Factor - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Care and treatment | Calcium channels | Physiological aspects | Genetic aspects | Cellular signal transduction | Research | Risk factors | Cellular biology | Epithelial-mesenchymal transition (EMT)
Journal Article
Cancer Letters, ISSN 0304-3835, 2015, Volume 367, Issue 1, pp. 1 - 11
Highlights • Sorafenib resistance of advanced HCC has raised global concern and understanding the underlying mechanism is in urgent need. • Several growth... 
Hematology, Oncology and Palliative Medicine | Advanced hepatocellular carcinoma (HCC) | Cancer stem cells (CSCs) | Acquired resistance | Microenvironment | Epithelial–mesenchymal transitions (EMT) and mesenchymal–epithelial transitions (MET) | Epithelial-mesenchymal transitions (EMT) and mesenchymal-epithelial transitions (MET) | PLUS SORAFENIB | PHASE-III | ALPHA-B-CRYSTALLIN | PROMOTES METASTASIS | Epithelial-mesenchymal transitions (EMT) and mesenchymal epithelial transitions (MET) | EPITHELIAL-MESENCHYMAL TRANSITION | BREAST-CANCER | LIVER-CANCER CELLS | ONCOLOGY | DEPENDENT MECHANISM | UP-REGULATION | HEDGEHOG SIGNALING PATHWAY | Niacinamide - analogs & derivatives | Tumor Microenvironment - drug effects | Neoplastic Stem Cells - drug effects | Humans | Liver Neoplasms - drug therapy | Niacinamide - therapeutic use | Phenylurea Compounds - therapeutic use | Drug Resistance, Neoplasm | Epithelial-Mesenchymal Transition - drug effects | Antineoplastic Agents - therapeutic use | Carcinoma, Hepatocellular - enzymology | Molecular Targeted Therapy | Animals | Carcinoma, Hepatocellular - drug therapy | Signal Transduction - drug effects | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Protein Kinase Inhibitors - therapeutic use | Carcinoma, Hepatocellular - pathology | Neoplastic Stem Cells - pathology | Liver Neoplasms - pathology | Liver Neoplasms - enzymology | Neoplastic Stem Cells - enzymology | Antimitotic agents | Antineoplastic agents | Cancer
Journal Article
Cellular and molecular life sciences : CMLS, ISSN 1420-9071, 2011, Volume 68, Issue 18, pp. 3033 - 3046
Journal Article
International journal of cancer, ISSN 0020-7136, 2015, Volume 137, Issue 11, pp. 2566 - 2577
Journal Article