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Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2013, Volume 110, Issue 5, pp. 1714 - 1719
Expression of the Notch ligand Jagged 1 (JAG1) and Notch activation promote poor-prognosis in breast cancer. We used high throughput screens to identify... 
Tumor cell line | Cell lines | Small interfering RNA | Breast cancer | Cells | Regulator genes | Genetic screening | Tumors | Mesenchymal stem cells | Cancer | EPITHELIAL-MESENCHYMAL TRANSITION | STEM-CELLS | IN-VITRO | ACTIVATED PROTEIN-KINASE | SIGNALING PATHWAY | TRIBBLES HOMOLOG | MULTIDISCIPLINARY SCIENCES | HYPOXIA | EXPRESSION | ESTROGEN-RECEPTOR | MAMMARY-GLAND | Humans | Breast Neoplasms - metabolism | MAP Kinase Signaling System | Intercellular Signaling Peptides and Proteins - metabolism | RNA Interference | Serrate-Jagged Proteins | Protein-Serine-Threonine Kinases - antagonists & inhibitors | Cell Cycle Proteins - genetics | Female | Membrane Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Repressor Proteins - metabolism | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Cell Line | Membrane Proteins - genetics | Cell Cycle Proteins - metabolism | Intercellular Signaling Peptides and Proteins - genetics | Protein-Serine-Threonine Kinases - genetics | Repressor Proteins - genetics | Protein Kinase Inhibitors - isolation & purification | Receptor, Notch1 - metabolism | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Interleukin Receptor Common gamma Subunit - genetics | Blotting, Western | Hep G2 Cells | Mice, Knockout | Interleukin Receptor Common gamma Subunit - deficiency | Xenograft Model Antitumor Assays | Animals | Breast Neoplasms - genetics | Transforming Growth Factor beta - genetics | Signal Transduction - drug effects | Breast Neoplasms - pathology | Cell Line, Tumor | Mice, Inbred NOD | Mice | Protein Kinase Inhibitors - pharmacology | Receptor, Notch1 - genetics | Transforming Growth Factor beta - metabolism | Calcium-Binding Proteins - genetics | Index Medicus | Biological Sciences
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2014, Volume 289, Issue 32, pp. 22117 - 22127
GPR30, or G protein-coupled estrogen receptor, is a G protein-coupled receptor reported to bind 17 beta-estradiol (E-2), couple to the G proteins G(s) and... 
SIGNAL-TRANSDUCTION | GROWTH-FACTOR RECEPTOR | BREAST-CANCER CELLS | AMPA RECEPTORS | PDZ PROTEINS | ADENYLYL-CYCLASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRAFFICKING | PSD-95 | ENDOCYTOSIS | ESTROGEN-RECEPTOR | Guanylate Kinases - genetics | Receptors, Estrogen - metabolism | Cricetulus | Receptors, G-Protein-Coupled - metabolism | Humans | Multiprotein Complexes - genetics | A Kinase Anchor Proteins - chemistry | Guanylate Kinases - chemistry | Gene Knockdown Techniques | A Kinase Anchor Proteins - genetics | Multiprotein Complexes - metabolism | HEK293 Cells | Cell Membrane - metabolism | Membrane Proteins - metabolism | A Kinase Anchor Proteins - metabolism | CHO Cells | Recombinant Proteins - metabolism | Cell Line | Cricetinae | Receptors, Estrogen - genetics | Signal Transduction | Membrane Proteins - genetics | Recombinant Proteins - chemistry | Recombinant Proteins - genetics | Multiprotein Complexes - chemistry | Animals | Receptors, Estrogen - chemistry | Cyclic AMP - biosynthesis | Membrane Proteins - chemistry | Models, Biological | Dogs | Guanylate Kinases - metabolism | PDZ Domains | Receptors, G-Protein-Coupled - genetics | Receptors, G-Protein-Coupled - chemistry | Index Medicus | Membrane Trafficking | Protein Kinase A (PKA) | PDZ Domain | GPR30 | Membrane-associated Guanylate Kinase | Cyclic AMP (cAMP) | G Protein-coupled Receptor (GPCR) | A Kinase-anchoring Protein (AKAP) | Adenylate Cyclase (Adenylyl Cyclase) | G Protein-coupled Estrogen Receptor
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2015, Volume 10, Issue 8, pp. e0135851 - e0135851
Overexpression of Twist, a highly conserved basic helix-loop-helix transcription factor, is associated with epithelial-mesenchymal transition (EMT) and... 
TRANSCRIPTION FACTORS | METASTASIS | PROTEIN | LOCAL RECURRENCE | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | GLYCOGEN-SYNTHASE KINASE-3 | RESISTANCE | BETA-CATENIN | CONSERVING THERAPY | CARCINOMA | Carcinoma, Ductal, Breast - genetics | Prognosis | Cadherins - metabolism | Receptor, ErbB-2 - genetics | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Receptor, ErbB-2 - metabolism | Ki-67 Antigen - metabolism | Receptors, Progesterone - genetics | Epithelial-Mesenchymal Transition - genetics | Carcinoma, Ductal, Breast - mortality | Proto-Oncogene Proteins c-akt - genetics | Tumor Suppressor Protein p53 - genetics | Receptors, Progesterone - metabolism | Neoplasm Grading | Biomarkers, Tumor - metabolism | Adult | Carcinoma, Ductal, Breast - pathology | Estrogen Receptor alpha - metabolism | Female | Cadherins - genetics | Nuclear Proteins - genetics | Proto-Oncogene Proteins c-akt - metabolism | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | Lymphatic Metastasis | Carcinoma, Ductal, Breast - diagnosis | Vascular Endothelial Growth Factor C - genetics | Breast Neoplasms - genetics | Estrogen Receptor alpha - genetics | Ki-67 Antigen - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Breast Neoplasms - pathology | Vascular Endothelial Growth Factor C - metabolism | Survival Analysis | Twist-Related Protein 1 - genetics | Cell Line, Tumor | Breast Neoplasms - mortality | Biomarkers, Tumor - genetics | Breast Neoplasms - diagnosis | Neoplasm Staging | Twist-Related Protein 1 - metabolism | Transcription factors | Physiological aspects | Breast cancer | Genetic aspects | Research | Gene expression | Immunohistochemistry | Mesenchyme | Laboratories | Estrogens | Estrogen receptors | AKT protein | Activation | Metastasis | Kinases | Cancer therapies | E-cadherin | Proteins | Signal transduction | Tumorigenesis | Bioindicators | Helix-loop-helix proteins | Vascular endothelial growth factor | Medical research | Immunoglobulins | Invasiveness | Extracellular signal-regulated kinase | Mammography | Patients | ErbB-2 protein | Hospitals | Protein kinase | Biomarkers | Breast | Progesterone | Prostate | Cell migration | Cancer | Index Medicus
Journal Article
The EMBO Journal, ISSN 0261-4189, 03/2006, Volume 25, Issue 6, pp. 1231 - 1241
The expression of metastasis‐associated protein 1 (MTA1) correlates well with tumor metastases; however, the associated molecular mechanism is not fully... 
hypoxia | HIF‐1α | MTA1 | HDAC1 | metastasis | Hypoxia | HIF-1α | Metastasis | HUMAN MTA1 GENE | TRANSCRIPTIONAL ACTIVITY | HIF-1 alpha | BIOCHEMISTRY & MOLECULAR BIOLOGY | VIRUS-X PROTEIN | ALPHA | ESTROGEN-RECEPTOR | CELL BIOLOGY | BREAST-CANCER | HYPOXIA-INDUCIBLE FACTOR-1-ALPHA | TRANSACTIVATION FUNCTIONS | EXPRESSION | CANCER CELL INVASIVENESS
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2016, Volume 11, Issue 8, pp. e0160312 - e0160312
Heat shock proteins (Hsps) are known to associate with estrogen receptors (ER) and regulate ER-mediated cell proliferation. Historically, the studies in this... 
GLUCOCORTICOID-RECEPTOR | MITOCHONDRIAL LOCALIZATION | SPECTRAL ABUNDANCE FACTORS | NUCLEAR RECEPTORS | MULTIDISCIPLINARY SCIENCES | BREAST-CANCER-CELLS | PROGESTERONE-RECEPTOR | PLASMA-MEMBRANE | BINDING | EXPRESSION | STEROID-RECEPTORS | Chromatin - metabolism | Humans | Cytoplasm - metabolism | Tacrolimus Binding Proteins - metabolism | Heat-Shock Proteins - genetics | Cell Nucleus - metabolism | MCF-7 Cells | Prostaglandin-E Synthases - genetics | HEK293 Cells | Estrogen Receptor alpha - metabolism | Proteomics - methods | Estradiol - pharmacology | Chromatin - chemistry | Gene Expression | Prostaglandin-E Synthases - metabolism | Heat-Shock Proteins - metabolism | Ubiquitin-Protein Ligases - metabolism | HSP70 Heat-Shock Proteins - genetics | HSP70 Heat-Shock Proteins - metabolism | Protein Interaction Mapping | Tacrolimus Binding Proteins - genetics | Estrogen Receptor alpha - genetics | Protein Binding | Cell Nucleus - drug effects | Ubiquitin-Protein Ligases - genetics | Cytoplasm - drug effects | Cell proliferation | Ubiquitin | Cell culture | Hsp90 protein | Chromatin | Transcription | Estrogens | Estrogen | Estrogen receptors | Nuclei | Estradiol | Proteins | Receptors | Historical account | Rodents | Penicillin | Trends | Prostaglandin E | Localization | Ubiquitin-protein ligase | Peptidylprolyl isomerase | Starvation | Heat shock proteins | Breast cancer | Hsp70 protein | Sex hormones | Isoforms | Phenols | Ligands | Hsc70 protein | Nuclei (cytology) | Endoplasmic reticulum | Endocrinology | Cytoplasm | Cancer | Heat shock | Index Medicus
Journal Article
Journal Article
by Chan, QKY and Lam, HM and Ng, CF and Lee, AYY and Chan, ESY and Ng, HK and Ho, SM and Lau, KM
Cell Death and Differentiation, ISSN 1350-9047, 09/2010, Volume 17, Issue 9, pp. 1511 - 1523
G-protein-coupled receptor-30 (GPR30) shows estrogen-binding affinity and mediates non-genomic signaling of estrogen to regulate cell growth. We here showed... 
COUPLED RECEPTOR GPR30 | ER-BETA | signal transduction | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | G-protein-coupled receptor-30 | G2/M | PROLIFERATION | G-PROTEIN | non-genomic estrogen signaling | ESTROGEN-RECEPTOR | 17-BETA-ESTRADIOL | CELL BIOLOGY | NUCLEAR ACCUMULATION | Y TRANSCRIPTION FACTOR | EXPRESSION | 1-[4-(6-bromobenzo[1,3]dioxol-5yl)-3a,4,5,9b-tetrahydro-3H-cyclopenta[c]quinolin-8-yl]-ethanone | Prostatic Neoplasms - metabolism | RNA, Small Interfering - genetics | Gene Expression - drug effects | Gene Expression - genetics | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Extracellular Signal-Regulated MAP Kinases - antagonists & inhibitors | Male | Extracellular Signal-Regulated MAP Kinases - metabolism | Receptors, G-Protein-Coupled - agonists | G2 Phase - drug effects | Quinolines - pharmacology | Cell Nucleus - metabolism | Cyclin-Dependent Kinase Inhibitor p21 - genetics | Cyclin-Dependent Kinase Inhibitor p21 - metabolism | Antineoplastic Agents - pharmacology | Flavonoids - pharmacology | Phosphorylation - drug effects | Prostatic Neoplasms - drug therapy | Proto-Oncogene Proteins - metabolism | Prostatic Neoplasms - pathology | Proto-Oncogene Proteins c-jun - genetics | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | Proto-Oncogene Proteins c-fos - metabolism | Receptors, Estrogen - antagonists & inhibitors | Proto-Oncogene Proteins - genetics | Cyclopentanes - pharmacology | Xenograft Model Antitumor Assays | Animals | Mitogen-Activated Protein Kinase 3 - metabolism | Signal Transduction - drug effects | Mice, Nude | Proto-Oncogene Proteins c-jun - metabolism | Cell Line, Tumor | Proto-Oncogene Proteins c-fos - genetics | Signal Transduction - physiology | Cell Proliferation - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Receptors, G-Protein-Coupled - genetics | Cell Cycle - drug effects | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus
Journal Article