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1. Full Text Site-specific binding of a PPR protein defines and stabilizes 5′ and 3′ mRNA termini in chloroplasts
by Prikryl, Jana and Barkan, Alice and Bayraktar, Omer Ali and Pfalz, Jeannette
The EMBO Journal, ISSN 0261-4189, 07/2009, Volume 28, Issue 14, pp. 2042 - 2052
Chloroplast mRNA populations are characterized by overlapping transcripts derived by processing from polycistronic precursors. The mechanisms and functional... 
RNA stability | pentatricopeptide repeat | plastid | RNA‐binding protein | mitochondria | Mitochondria | RNA-binding protein | Pentatricopeptide repeat | Plastid | BIOCHEMISTRY & MOLECULAR BIOLOGY | ESCHERICHIA-COLI | RIBOSOMAL-RNA | TRANSLATION | CELL BIOLOGY | MAIZE CHLOROPLASTS | CHLAMYDOMONAS-REINHARDTII | IN-VIVO | GENE-EXPRESSION | PENTATRICOPEPTIDE REPEAT PROTEIN | ARABIDOPSIS | NUCLEUS-ENCODED FACTOR | RNA Cap-Binding Proteins - metabolism | Zea mays - genetics | RNA Processing, Post-Transcriptional | RNA, Messenger - genetics | Gene Expression Regulation, Plant | Molecular Sequence Data | Plant Proteins - metabolism | Zea mays - metabolism | RNA Stability | RNA, Messenger - metabolism | Chloroplasts - genetics | Chloroplasts - metabolism | Plant biology | Plant mitochondria | RNA-protein interactions | Cellular biology | Chloroplasts | Molecular biology
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2. Full Text SATB1 Defines a Subtype of Cutaneous CD30+ Lymphoproliferative Disorders Associated with a T-Helper 17 Cytokine Profile
by Sun, Jingru and Yi, Shengguo and Qiu, Lei and Fu, Wenjing and Wang, Lin and Wang, Lei and Wang, Tingting and Wang, Yang and Wang, Anqi and Liu, Fengjie and Chen, Hao and Kadin, Marshall E and Tu, Ping
Journal of Investigative Dermatology, ISSN 0022-202X, 08/2018, Volume 138, Issue 8, pp. 1795 - 1804
Cutaneous CD30 lymphoproliferative disorders (LPDs), including lymphomatoid papulosis (LyP) and primary cutaneous anaplastic large-cell lymphoma, comprise the... 
PATHOGENESIS | LARGE-CELL LYMPHOMAS | TRANSLOCATIONS | DISEASE | IL-17 | GENE-EXPRESSION | ORGANIZER SATB1 | CLASSIFICATION | PROLIFERATION | TUMOR-GROWTH | DERMATOLOGY | Lymphoma, Large-Cell, Anaplastic - immunology | Up-Regulation | DNA Methylation - immunology | Skin Neoplasms - drug therapy | Follow-Up Studies | Humans | Middle Aged | Male | Th1 Cells - immunology | Promoter Regions, Genetic - genetics | Th1 Cells - metabolism | Lymphomatoid Papulosis - pathology | Th17 Cells - metabolism | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Female | Retrospective Studies | Child | Lymphomatoid Papulosis - drug therapy | Cytokines - immunology | Lymphoma, Large-Cell, Anaplastic - genetics | Skin Neoplasms - pathology | Cytokines - metabolism | Skin Neoplasms - immunology | Matrix Attachment Region Binding Proteins - metabolism | Lymphomatoid Papulosis - genetics | Gene Expression Regulation, Neoplastic - immunology | Treatment Outcome | Ki-1 Antigen - metabolism | Matrix Attachment Region Binding Proteins - immunology | Antineoplastic Combined Chemotherapy Protocols - therapeutic use | Lymphoma, Large-Cell, Anaplastic - drug therapy | Skin Neoplasms - genetics | Lymphomatoid Papulosis - immunology | Cell Line, Tumor | Th17 Cells - immunology | Aged | Lymphoma, Large-Cell, Anaplastic - pathology | Biomarkers, Tumor - immunology
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3. Full Text Stromal gene expression defines poor-prognosis subtypes in colorectal cancer
by Calon, Alexandre and Lonardo, Enza and Berenguer-Llergo, Antonio and Espinet, Elisa and Hernando-Momblona, Xavier and Iglesias, Mar and Sevillano, Marta and Palomo-Ponce, Sergio and Tauriello, Daniele V.F and Byrom, Daniel and Cortina, Carme and Morral, Clara and Barceló, Carles and Tosi, Sebastien and Riera, Antoni and Attolini, Camille Stephan-Otto and Rossell, David and Sancho, Elena and Batlle, Eduard
Nature Genetics, ISSN 1061-4036, 04/2015, Volume 47, Issue 4, pp. 320 - 329
Recent molecular classifications of colorectal cancer (CRC) based on global gene expression profiles have defined subtypes displaying resistance to therapy and... 
MOLECULAR SUBTYPES | GROWTH-FACTOR-BETA | TGF-BETA | METASTASIS | HUMAN-COLON | GENETICS & HEREDITY | STEM-CELL | RECEPTOR | TUMOR-GROWTH | TUMORIGENESIS | GENOME | Prognosis | Neoplasm Invasiveness | Stromal Cells - pathology | Colorectal Neoplasms - genetics | Humans | Stromal Cells - metabolism | Gene Expression Regulation, Neoplastic | Transcriptome | Colorectal Neoplasms - classification | Fibroblasts - pathology | Colorectal Neoplasms - diagnosis | HT29 Cells | Neoplasm Metastasis | Animals | Neoplastic Stem Cells - metabolism | Mice, Nude | Microarray Analysis | Neoplastic Stem Cells - pathology | Mice | Colorectal Neoplasms - pathology | Cluster Analysis | Fibroblasts - metabolism | Oncology, Experimental | Colorectal cancer | Genetic research | Development and progression | Genetic aspects | Research | Gene expression | Cancer | Datasets | Medical prognosis | Metastasis | Experiments | Cancer therapies | Patients | Tumors
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4. Full Text Impaired intracellular trafficking defines early Parkinson's disease
by Hunn, Benjamin H.M and Cragg, Stephanie J and Bolam, J. Paul and Spillantini, Maria-Grazia and Wade-Martins, Richard
Trends in Neurosciences, ISSN 0166-2236, 2016, Volume 38, Issue 3, pp. 178 - 188
Highlights • The dopamine neurons that die in Parkinson's disease are anatomically complex. • Cell trafficking is impaired by the key pathogenic protein in... 
Neurology | cell trafficking | Tau | Parkinson's disease | α-synuclein | Cell trafficking | INCREASED EXPRESSION | TRANSGENIC MOUSE | alpha-synuclein | NEUROSCIENCES | BODY PATHOLOGY | ALPHA-SYNUCLEIN OLIGOMERS | DOPAMINE NEURONS | RECYCLING-POOL | IN-VIVO | ECONOMIC-IMPACT | MOTOR PROTEINS | LRRK2 CONTROLS | Neurons - pathology | Parkinson Disease - pathology | Animals | Biological Transport | Humans | Neurons - metabolism | Parkinson Disease - metabolism | Neurosciences | Review
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5. Full Text BAP1 loss defines a new class of renal cell carcinoma
by Peña-Llopis, Samuel and Vega-Rubín-De-Celis, Silvia and Liao, Arnold and Leng, Nan and Pavía-Jiménez, Andrea and Wang, Shanshan and Yamasaki, Toshinari and Zhrebker, Leah and Sivanand, Sharanya and Spence, Patrick and Kinch, Lisa and Hambuch, Tina and Jain, Suneer and Lotan, Yair and Margulis, Vitaly and Sagalowsky, Arthur I and Summerour, Pia Banerji and Kabbani, Wareef and Wong, S W Wendy and Grishin, Nick and Laurent, Marc and Xie, Xian-Jin and Haudenschild, Christian D and Ross, Mark T and Bentley, David R and Kapur, Payal and Brugarolas, James
Nature Genetics, ISSN 1061-4036, 07/2012, Volume 44, Issue 7, pp. 751 - 759
The molecular pathogenesis of renal cell carcinoma (RCC) is poorly understood. Whole-genome and exome sequencing followed by innovative tumorgraft analyses (to... 
COMPLEX | OCULOCUTANEOUS ALBINISM | GENETICS & HEREDITY | GENE-EXPRESSION | FREQUENT MUTATION | TUMOR-SUPPRESSOR | FACTOR-I | UBIQUITIN HYDROLASE | BRCA1-ASSOCIATED PROTEIN-1 | P-GENE | SOMATIC MUTATIONS | Kidney Neoplasms - genetics | Gene Expression - genetics | Humans | Middle Aged | Carcinoma, Renal Cell - genetics | Cell Growth Processes - physiology | Male | Kidney Neoplasms - metabolism | Host Cell Factor C1 - metabolism | Exome | Tumor Suppressor Proteins - deficiency | Tumor Suppressor Proteins - genetics | Ubiquitin Thiolesterase - deficiency | Ubiquitin Thiolesterase - metabolism | Female | Protein Interaction Domains and Motifs | Nuclear Proteins - genetics | Tumor Suppressor Proteins - metabolism | Carcinoma, Renal Cell - pathology | Cells, Cultured | Nuclear Proteins - metabolism | Transcription Factors - genetics | Ubiquitin Thiolesterase - genetics | Host Cell Factor C1 - genetics | Transcription Factors - metabolism | Carcinoma, Renal Cell - metabolism | Kidney Neoplasms - pathology | Aged | Mutation | Gene mutations | Genetic aspects | Research | Carcinoma, Renal cell | Gene expression | Health aspects | Risk factors | Studies | Proteins | Cancer | Tumors
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6. Full Text CCR7 Is Mainly Expressed in Teleost Gills, Where It Defines an IgD +IgM- B Lymphocyte Subset
by Castro, Rosario and Bromage, Erin and Abós, Beatriz and Pignatelli, Jaime and Granja, Aitor González and Luque, Alfonso and Tafalla, Carolina
Journal of Immunology, ISSN 0022-1767, 02/2014, Volume 192, Issue 3, pp. 1257 - 1266
Chemokine receptor CCR7, the receptor for both CCL19 and CCL21 chemokines, regulates the recruitment and clustering of circulating leukocytes to secondary... 
SALMO-GAIRDNERI | RAINBOW-TROUT | FISH | IG HEAVY-CHAIN | MONOCLONAL-ANTIBODY | MUCOSAL | IMMUNOLOGY | CHEMOKINE RECEPTORS | IDENTIFICATION | IMMUNOGLOBULIN-D | T-CELLS | Antibody Specificity | Receptors, CCR7 - immunology | Novirhabdovirus - physiology | Gills - cytology | Immunoglobulin M - analysis | Lymphoid Tissue - growth & development | Receptors, CCR7 - biosynthesis | Gene Expression Regulation, Developmental | Female | Head Kidney - metabolism | Oncorhynchus mykiss - immunology | Oncorhynchus mykiss - metabolism | Immunoglobulin D - analysis | Receptors, CCR7 - genetics | Lymphoid Tissue - metabolism | Gills - metabolism | Oncorhynchus mykiss - genetics | Organ Specificity | Head Kidney - growth & development | Oncorhynchus mykiss - growth & development | Lymphoid Tissue - cytology | Animals | Gills - growth & development | B-Lymphocyte Subsets - metabolism | Head Kidney - cytology | Hemorrhagic Septicemia, Viral - immunology
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7. Full Text CD161 defines the subset of FoxP3+ T cells capable of producing proinflammatory cytokines
by Pesenacker, Anne M and Pesenacker, Anne M and Bending, David and Bending, David and Ursu, Simona and Ursu, Simona and Wu, Qiong and Wu, Qiong and Nistala, Kiran and Nistala, Kiran and Wedderburn, Lucy R and Wedderburn, Lucy R
Blood, ISSN 0006-4971, 2013, Volume 121, Issue 14, pp. 2647 - 2658
Regulatory FoxP3+CD4+ T cells (Treg) are vital for maintaining the balance between tolerance, adequate immune response, and autoimmunity. Despite this... 
INTERLEUKIN-2 | GENE | INFLAMMATION | T(H)17 | REGULATORY T-CELLS | INDUCTION | JOINTS | ARTHRITIS | HEMATOLOGY | EXPRESSION | FEATURES | Forkhead Transcription Factors - immunology | T-Lymphocyte Subsets - immunology | T-Lymphocytes, Regulatory - metabolism | T-Lymphocyte Subsets - cytology | Humans | Interleukin-17 - immunology | Child, Preschool | Infant | Male | Receptors, Antigen, T-Cell, alpha-beta - immunology | T-Lymphocytes, Regulatory - immunology | Flow Cytometry | Interleukin-2 - immunology | Th17 Cells - metabolism | Forkhead Transcription Factors - metabolism | T-Lymphocytes, Regulatory - cytology | Female | Th17 Cells - cytology | Child | Interleukin-2 - metabolism | CD4 Antigens - immunology | NK Cell Lectin-Like Receptor Subfamily B - metabolism | Immunophenotyping | NK Cell Lectin-Like Receptor Subfamily B - immunology | Interleukin-17 - metabolism | T-Lymphocyte Subsets - metabolism | Adolescent | Th17 Cells - immunology | Receptors, Antigen, T-Cell, alpha-beta - metabolism | CD4 Antigens - metabolism | Cell Lineage - immunology | Immunobiology
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8. Full Text Comprehensive genomic characterization defines human glioblastoma genes and core pathways
by Chin, L and Meyerson, M and Aldape, K and Bigner, D and Mikkelsen, T and VandenBerg, S and Kahn, A and Penny, R and Gerhard, D. S and Getz, G and Brennan, C and Taylor, B. S and Winckler, W and Park, P and Ladanyi, M and Hoadley, K. A and Verhaak, R. G. W and Hayes, D. N and Spellman, Paul T and Absher, D and Weir, B. A and Ding, L and Wheeler, D and Lawrence, M. S and Cibulskis, K and Mardis, E and Zhang, Jinghui and Wilson, R. K and Donehower, L and Wheeler, D. A and Purdom, E and Wallis, J and Laird, P. W and Herman, J. G and Schuebel, K. E and Weisenberger, D. J and Baylin, S. B and Schultz, N and Yao, Jun and Wiedemeyer, R and Weinstein, J and Sander, C and Gibbs, R. A and Gray, J and Kucherlapati, R and Lander, E. S and Myers, R. M and Perou, C. M and McLendon, Roger and Friedman, Allan and Tissue Source Sites and Canc Genome Atlas Res Network and Canc Genome Characterization Ctr and Genome Sequencing Ctr and Project Teams and Cancer Genome Atlas Research Network and The Cancer Genome Atlas Research Network
Nature, ISSN 0028-0836, 10/2008, Volume 455, Issue 7216, pp. 1061 - 1068
Human cancer cells typically harbour multiple chromosomal aberrations, nucleotide substitutions and epigenetic modifications that drive malignant... 
PIK3CA GENE | ALKYLATING-AGENTS | HIGH-FREQUENCY | MALIGNANT GLIOMAS | MISMATCH REPAIR | CELL-LINES | NF1 GENE | TUMORS | NEUROFIBROMATOSIS TYPE-1 | SOMATIC MUTATIONS | INACTIVATION | MGMT | MULTIDISCIPLINARY SCIENCES | CANCER | TEMOZOLOMIDE | Genomics | Humans | Middle Aged | DNA Repair Enzymes - genetics | Gene Expression Regulation, Neoplastic | Male | DNA Repair - genetics | DNA Methylation | Glioblastoma - genetics | Tumor Suppressor Proteins - genetics | Aged, 80 and over | Adult | Female | Retrospective Studies | Genes, Tumor Suppressor | Protein Structure, Tertiary | Genes, erbB-1 - genetics | Brain Neoplasms - genetics | Models, Molecular | Gene Dosage | Signal Transduction - genetics | Mutation - genetics | Genome, Human - genetics | Phosphatidylinositol 3-Kinases - genetics | DNA Modification Methylases - genetics | Adolescent | Aged | Neurofibromin 1 - genetics | Genetic aspects | Research | Gene expression | Glioblastoma multiforme | Cancer cells | Genotype & phenotype | Public access | Genetics | Patients | Deoxyribonucleic acid--DNA | Methods | Cancer
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