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Advances in Chronic Kidney Disease, ISSN 1548-5595, 05/2018, Volume 25, Issue 3, pp. 251 - 260
Magnesium is the most abundant intracellular divalent cation and essential for maintaining normal cellular physiology and metabolism, acting as a cofactor of... 
Heart failure | Magnesium | Arrhythmia | Cardiovascular | Coronary artery disease | TORSADES-DE-POINTES | DIETARY MAGNESIUM | ATRIAL-FIBRILLATION | SERUM MAGNESIUM | ATHEROSCLEROSIS-RISK | EXTRACELLULAR MAGNESIUM | CALCIUM-CHANNELS | ACUTE MYOCARDIAL-INFARCTION | UROLOGY & NEPHROLOGY | CORONARY-HEART-DISEASE | INTRAVENOUS-MAGNESIUM
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 2, p. e89525
Magnesium reduces vascular smooth muscle cell (VSMC) calcification in vitro but the mechanism has not been revealed so far. This work used only slightly... 
SEVELAMER | HOMEOSTASIS | CORONARY-ARTERY CALCIFICATION | BINDER | MULTIDISCIPLINARY SCIENCES | CALCIUM | HEMODIALYSIS-PATIENTS | PARATHYROID-HORMONE | PHOSPHATE-INDUCED CALCIFICATION | CHRONIC KIDNEY-DISEASE | MODULATION | Muscle, Smooth, Vascular - metabolism | Humans | Protein-Serine-Threonine Kinases | Wnt Proteins - metabolism | Vascular Calcification - metabolism | Wnt Proteins - genetics | Osteoprotegerin - genetics | Real-Time Polymerase Chain Reaction | Extracellular Matrix Proteins - metabolism | Muscle, Smooth, Vascular - drug effects | Calcium-Binding Proteins - metabolism | Boron Compounds - pharmacology | Extracellular Matrix Proteins - genetics | Osteogenesis - drug effects | RNA, Messenger - genetics | Cells, Cultured | Vascular Calcification - drug therapy | Magnesium - pharmacology | Muscle, Smooth, Vascular - cytology | Reverse Transcriptase Polymerase Chain Reaction | beta Catenin - metabolism | beta Catenin - genetics | TRPM Cation Channels - antagonists & inhibitors | Signal Transduction - drug effects | beta Catenin - antagonists & inhibitors | TRPM Cation Channels - genetics | TRPM Cation Channels - metabolism | Osteoprotegerin - metabolism | Wnt Proteins - antagonists & inhibitors | Calcium-Binding Proteins - genetics | Calcification (ectopic) | Phosphates | Transformation | Transcription factors | Wnt protein | Calcium | Frizzled protein | Homeostasis | Smooth muscle | Activation | Kinases | Osteoprotegerin | Proteins | Transient receptor potential proteins | β-catenin | Signal transduction | Biomedical materials | Biocompatibility | Magnesium | Inhibition | Translocation | Cbfa-1 protein | Muscles | siRNA | Gene expression | Studies | Signaling | Lymphocytes B | Calcification | Transport | Dkk1 protein | Apoptosis
Journal Article
Molecular Neurobiology, ISSN 0893-7648, 9/2018, Volume 55, Issue 9, pp. 7118 - 7131
Poor Mg status is a risk factor for Alzheimer’s disease (AD), and the underlying mechanisms remain elusive. Here, we provided the first evidence that elevated... 
Neurology | Neurosciences | Biomedicine | Alzheimer’s disease | Caveolae | Neurobiology | TRPM7 | MagT1 | LRP | RAGE | Cell Biology | PICALM | ALZHEIMERS-DISEASE | MEDIATED DOWN-REGULATION | PLASMA-MEMBRANE | NEUROSCIENCES | P-GLYCOPROTEIN | IN-VITRO MODEL | ENDOTHELIAL-CELLS | SMOOTH-MUSCLE-CELLS | ION CHANNELS | Alzheimer's disease | RECEPTOR-RELATED PROTEIN-1 | CAVEOLIN-1 | Endothelial Cells - metabolism | Membrane Microdomains - metabolism | Humans | Receptor for Advanced Glycation End Products - metabolism | Low Density Lipoprotein Receptor-Related Protein-1 - metabolism | Permeability | Magnesium - pharmacology | Blood-Brain Barrier - drug effects | Blood-Brain Barrier - metabolism | Caveolin 1 - metabolism | Extracellular Space - metabolism | Models, Biological | Monomeric Clathrin Assembly Proteins - metabolism | Amyloid beta-Peptides - metabolism | Protein Binding | TRPM Cation Channels - metabolism | Transcytosis - drug effects | Protein-Serine-Threonine Kinases - metabolism | Endothelial Cells - drug effects | Caveolin-1 | Clathrin | Phosphorylation | Advanced glycosylation end products | Immunoprecipitation | Neurodegenerative diseases | Caveolin | Membrane permeability | Health risks | Glycosylation | Risk factors | Endothelial cells | Phosphatidylinositol | Transient receptor potential proteins | Blood-brain barrier | β-Amyloid | Receptor density | Magnesium | Vascular endothelial growth factor
Journal Article
Molecular Microbiology, ISSN 0950-382X, 05/2019, Volume 111, Issue 5, pp. 1283 - 1301
Summary A proper response to elevated extracellular calcium levels helps to most organisms to keep this secondary messenger under strict control, thereby... 
YEAST | TARGET | PROTEIN | PATHWAY | CA2 | PHOSPHORYLATION | CALCIUM HOMEOSTASIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MICROBIOLOGY | HOMOLOG | IDENTIFICATION | STRESS | Analysis | Genetic aspects | Genetic transcription | Calcium signalling | Fungi | Phosphorylation | Phenotypes | Calcium | Calcineurin | Magnesium | Gene expression | Zinc | Calcium (extracellular) | Calcium influx
Journal Article
Stem Cell Research and Therapy, ISSN 1757-6512, 02/2017, Volume 8, Issue 1, p. 39
Background: Magnesium is essential for numerous physiological functions. Magnesium exists mostly in bone and the amount is dynamically regulated by skeletal... 
Mesenchymal stromal cells | Magnesium transporter | SLC41A1 | Mineralization | Osteogenic differentiation | MEDICINE, RESEARCH & EXPERIMENTAL | STEM-CELLS | MARROW | MATRIX GLA PROTEIN | PROLIFERATION | CELL BIOLOGY | IN-VITRO | EXTRACELLULAR MAGNESIUM | MOLECULAR-IDENTIFICATION | CALCIUM | CALCIFICATION | DIFFERENTIATION | Osteopontin - genetics | RNA, Small Interfering - genetics | Cation Transport Proteins - antagonists & inhibitors | Humans | Alkaline Phosphatase - metabolism | Osteocalcin - genetics | Male | Osteopontin - metabolism | Intercellular Signaling Peptides and Proteins - metabolism | Mesenchymal Stromal Cells - cytology | Collagen Type I - genetics | Protein Isoforms - metabolism | Cation Transport Proteins - metabolism | Bone Morphogenetic Protein 2 - metabolism | Bone Marrow Cells - drug effects | Cation Transport Proteins - genetics | Cell Differentiation | Ion Transport | Extracellular Matrix Proteins - metabolism | Osteogenesis - genetics | Wnt Signaling Pathway | Calcification, Physiologic - genetics | Calcium-Binding Proteins - metabolism | Bone Morphogenetic Protein 2 - genetics | Mesenchymal Stromal Cells - drug effects | Alkaline Phosphatase - genetics | Collagen Type I - metabolism | Calcification, Physiologic - drug effects | Bone Marrow Cells - cytology | Osteocalcin - metabolism | Extracellular Matrix Proteins - genetics | Osteogenesis - drug effects | Gene Expression Regulation | Intercellular Signaling Peptides and Proteins - genetics | Mesenchymal Stromal Cells - metabolism | Magnesium - metabolism | Transcription Factors - genetics | Magnesium - pharmacology | Transcription Factors - metabolism | Animals | Sp7 Transcription Factor | Mice | Mice, Inbred BALB C | Bone Marrow Cells - metabolism | Protein Isoforms - antagonists & inhibitors | Calcium-Binding Proteins - genetics | Protein Isoforms - genetics | RNA, Small Interfering - metabolism | Growth | Analysis | Physiological aspects | Bone morphogenetic proteins | Bones | Genetic aspects | Research | Gene expression | Magnesium compounds | Density
Journal Article
Effect of magnesium chloride and guanidinium chloride on the extraction of components of extracellular matrix from chicken cartilage, 12/1994
In order to evaluate the effect of chaotropic agents on proteoglycan and non-collagenous proteins, chicken xiphoid cartilage was treated with guanidine-HCl and... 
proteoglycans - non-collagenous proteins - cartilage - extracellular matrix
Journal
Nephrology Dialysis Transplantation, ISSN 0931-0509, 02/2012, Volume 27, Issue 2, pp. 514 - 521
Journal Article