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Journal Article
JAMA Ophthalmology, ISSN 2168-6165, 06/2013, Volume 131, Issue 6, pp. 731 - 736
Journal Article
Eye (Basingstoke), ISSN 0950-222X, 2014, Volume 28, Issue 2, pp. 231 - 234
Purpose Laser 'toys' can be purchased online and imported with relative ease; the variety of such devices is a potential public safety concern. We describe... 
toy laser | public safety | laser injury | vitelliform maculopathy | laser safety | paediatric | INJURIES | POINTER | OPHTHALMOLOGY | Lasers - adverse effects | Humans | Male | Eye Burns - etiology | Visual Acuity | Retinal Pigment Epithelium - pathology | Adolescent | Female | Eye Burns - diagnosis | Child | Play and Playthings - injuries | Retina - injuries | Fluorescein Angiography | Index Medicus | Case Series
Journal Article
Community Eye Health Journal, ISSN 0953-6833, 2018, Volume 31, Issue 103, pp. 72 - 72
Chemical injury causes severe corneal scarring, but this can be prevented by immediate irrigation of the eye. 
Eye Emergencies | chemical burns | emergency
Journal Article
Cornea, ISSN 0277-3740, 02/2018, Volume 37, Issue 2, pp. 248 - 251
Journal Article
American Journal of Pathology, The, ISSN 0002-9440, 2017, Volume 187, Issue 6, pp. 1327 - 1342
Alkali burns to the eye constitute a leading cause of worldwide blindness. In recent case series, corneal transplantation revealed unexpected damage to the... 
Pathology | EMERGENCY | TUMOR-NECROSIS-FACTOR | IN-VITRO | CYTOKINES | THERAPY | PROTECTION | BOSTON KERATOPROSTHESIS | ENDOTOXIN | EYE BURNS | PATHOLOGY | GLAUCOMA | Neuroprotective Agents - therapeutic use | Tumor Necrosis Factor-alpha - metabolism | Eye Burns - drug therapy | Retina - metabolism | Apoptosis - drug effects | Burns, Chemical - metabolism | Cornea - immunology | Retina - immunology | Infliximab - therapeutic use | Uveitis, Anterior - pathology | Infliximab - pharmacology | Burns, Chemical - etiology | Corneal Injuries - metabolism | Retinal Ganglion Cells - pathology | Receptors, Tumor Necrosis Factor, Type I - deficiency | Eye Burns - pathology | Corneal Injuries - etiology | Neuroprotective Agents - pharmacology | Uvea - metabolism | Disease Models, Animal | Uveitis, Anterior - prevention & control | Alkalies | Drug Evaluation, Preclinical - methods | Rabbits | Corneal Injuries - pathology | Oxidation-Reduction | Mice, Inbred C57BL | Eye Burns - etiology | Receptors, Tumor Necrosis Factor, Type I - genetics | Uveitis, Anterior - chemically induced | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Receptors, Tumor Necrosis Factor, Type II - deficiency | Animals | Uveitis, Anterior - metabolism | Burns, Chemical - drug therapy | Burns, Chemical - pathology | Eye Burns - metabolism | Apoptosis - physiology | Corneal Injuries - drug therapy | Retina - pathology | Sodium Hydroxide | Retina - injuries | Retinal Ganglion Cells - drug effects | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Hydrogen-Ion Concentration | Index Medicus | Abridged Index Medicus | Regular
Journal Article
International Journal of Molecular Medicine, ISSN 1107-3756, 07/2016, Volume 38, Issue 1, pp. 75 - 82
Chemical burns are a major cause of corneal injury. Oxidative stress, inflammatory responses and neovascularization after the chemical burn aggravate corneal... 
inflammation | oxidative stress | NADPH oxidase | neovascularization | corneal injury | alkali burn | Oxidative stress | Alkali burn | Corneal injury | Inflammation | Neovascularization | MEDICINE, RESEARCH & EXPERIMENTAL | RAT | FACTOR-KAPPA-B | NAD(P)H OXIDASE | NOX FAMILY | ENDOTHELIAL-CELLS | SMOOTH-MUSCLE-CELLS | GROWTH-FACTOR | STROMAL CELLS | EXPRESSION | Eye Burns - drug therapy | Inflammation - pathology | Corneal Neovascularization - drug therapy | Humans | NADPH Oxidases - metabolism | Burns, Chemical - complications | Corneal Injuries - complications | Eye Burns - complications | Eye Burns - pathology | Acetophenones - pharmacology | Corneal Neovascularization - enzymology | Corneal Neovascularization - complications | Corneal Injuries - enzymology | Eye Burns - enzymology | Alkalies | Corneal Injuries - pathology | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Onium Compounds - pharmacology | Enzyme Inhibitors - therapeutic use | Animals | Burns, Chemical - drug therapy | Burns, Chemical - pathology | Burns, Chemical - enzymology | Oxidative Stress - drug effects | Corneal Injuries - drug therapy | Corneal Neovascularization - pathology | Oxidases | Complications and side effects | Cornea | Gene expression | Burns, Chemical | Health aspects | Injuries | Enzymes | Angiogenesis | Cytokines | Rodents | Burns | Laboratory animals | Vascular endothelial growth factor | Experiments
Journal Article
Journal Article