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Journal of Clinical Investigation, ISSN 0021-9738, 12/2007, Volume 117, Issue 12, pp. 4003 - 4008
Phosphate homeostasis is maintained by a counterbalance between efflux from the kidney and influx from intestine and bone. FGF23 is a bone-derived phosphaturic... 
PHOSPHATE | MEDICINE, RESEARCH & EXPERIMENTAL | HORMONE GENE-EXPRESSION | IN-VIVO | CALCIUM | GROWTH | KLOTHO PROTEIN | SECONDARY HYPERPARATHYROIDISM | VITAMIN-D-RECEPTOR | DOMINANT HYPOPHOSPHATEMIC RICKETS | MICE | MAP Kinase Signaling System - physiology | Humans | Mitogen-Activated Protein Kinase 3 - antagonists & inhibitors | Fibroblast Growth Factors - genetics | Intestines - metabolism | Parathyroid Hormone - genetics | Fibroblast Growth Factors - metabolism | Parathyroid Glands - cytology | RNA, Messenger - biosynthesis | Kidney - metabolism | Parathyroid Hormone - biosynthesis | Bone and Bones - metabolism | Mitogen-Activated Protein Kinase 1 - genetics | Early Growth Response Protein 1 - genetics | Receptors, Fibroblast Growth Factor - genetics | Homeostasis - drug effects | Mitogen-Activated Protein Kinase 3 - genetics | Mitogen-Activated Protein Kinase 1 - antagonists & inhibitors | RNA, Messenger - genetics | Cells, Cultured | Gene Expression Regulation - physiology | Rats | Fibroblast Growth Factors - pharmacology | Gene Expression Regulation - drug effects | Phosphates - metabolism | Animals | Glucuronidase - biosynthesis | MAP Kinase Signaling System - drug effects | Early Growth Response Protein 1 - biosynthesis | Glucuronidase - genetics | Mitogen-Activated Protein Kinase 3 - metabolism | Homeostasis - physiology | Receptors, Fibroblast Growth Factor - metabolism | Vitamin D - metabolism | Protein Kinase Inhibitors - pharmacology | Mitogen-Activated Protein Kinase 1 - metabolism | Parathyroid Glands - metabolism | Homeostasis | Parathyroid glands | Fibroblast growth factors | Dosage and administration | Genetic aspects | Research | Health aspects | Index Medicus | Abridged Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 04/2016, Volume 7, Issue 1, pp. 11022 - 11022
Journal Article
Journal Article
Journal of Experimental Medicine, ISSN 0022-1007, 2013, Volume 210, Issue 10, pp. 2071 - 2086
Toxoplasma gondii, the causative agent of toxoplasmosis, is an obligate intracellular protozoan parasite that resides inside a parasitophorous vacuole. During... 
MEDICINE, RESEARCH & EXPERIMENTAL | GONDII INFECTION | DENDRITIC CELLS | DOCKING INTERACTIONS | IFN-GAMMA | GENE-EXPRESSION | RESISTANCE | P38-ALPHA | IMMUNOLOGY | KINASE ACTIVATION | BINDING | AUTOPHOSPHORYLATION | Toxoplasma - immunology | Phosphorylation | Toxoplasmosis - immunology | Humans | Protein Multimerization | Molecular Sequence Data | Gene Expression Profiling | Protozoan Proteins - genetics | Cell Nucleus - metabolism | Protozoan Proteins - metabolism | Toxoplasmosis - metabolism | Gene Deletion | Early Growth Response Protein 1 - genetics | Female | Protozoan Proteins - chemistry | Protein Interaction Domains and Motifs | p38 Mitogen-Activated Protein Kinases - metabolism | Gene Order | Macrophages - immunology | Amino Acid Sequence | Cell Line | Chemokines - biosynthesis | Proto-Oncogene Proteins c-fos - metabolism | Models, Molecular | Inflammation - immunology | Amino Acid Motifs | Protein Transport | Macrophages - metabolism | Sequence Alignment | Animals | Toxoplasma - genetics | Inflammation - genetics | Protein Binding | Protein Conformation | Proto-Oncogene Proteins c-fos - genetics | Mice | Enzyme Activation | p38 Mitogen-Activated Protein Kinases - chemistry | Early Growth Response Protein 1 - metabolism | Protozoan Proteins - immunology | Cluster Analysis | Index Medicus | Life Sciences | Biochemistry, Molecular Biology | Genomics
Journal Article
PLoS ONE, ISSN 1932-6203, 2011, Volume 6, Issue 5, pp. e19481 - e19481
Background: The heart responds to myriad stresses by well-described transcriptional responses that involve long-term changes in gene expression as well as more... 
SKELETAL-MUSCLE | HYPERTROPHY | STAGE HEART-FAILURE | C-FOS | HEAVY-CHAIN GENE | PORCINE MICRORNAS | BIOLOGY | MICE | DILATED CARDIOMYOPATHY | GROWTH-FACTOR | MICRORNA EXPRESSION | Early Growth Response Protein 2 - genetics | Humans | SOXD Transcription Factors - metabolism | Myosin Heavy Chains - genetics | Mice, Transgenic | MicroRNAs - metabolism | Muscle, Skeletal - metabolism | Blotting, Western | Actins - genetics | Blotting, Northern | Animals | Myocardium - metabolism | Early Growth Response Protein 1 - genetics | Polymerase Chain Reaction | Proto-Oncogene Proteins c-fos - genetics | Mice | MicroRNAs - genetics | SOXD Transcription Factors - genetics | Heart | MicroRNA | Heart enlargement | Analysis | Genes | Myosin | Muscles | Genetic engineering | Genetic transcription | Muscle proteins | Adaptations | Pediatrics | Transcription | Cardiomyopathy | Heart surgery | Gene regulation | Genomics | Chains | Cardiovascular disease | Epidermal growth factor | Fos protein | Actin | Transgenic animals | Rodents | Tomography | Egr-2 protein | Stress response | Vascular endothelial growth factor | Bioinformatics | Heart diseases | Heart failure | Stresses | Adenosine | Cardiac muscle | EGR-1 protein | Transgenic mice | MiRNA | Cardiomyocytes | Gene expression | Skeletal muscle | Studies | Musculoskeletal system | Ribonucleic acids | Microscopy | MicroRNAs | Ventricle | Hypertrophy | Index Medicus
Journal Article
Blood, ISSN 0006-4971, 01/2017, Volume 129, Issue 4, pp. 473 - 483
Follicular lymphoma(FL) is the most common form of indolent non-Hodgkin lymphoma, yet it remains only partially characterized at the genomic level. To improve... 
POINT MUTATIONS | CHRONIC LYMPHOCYTIC-LEUKEMIA | NON-HODGKIN-LYMPHOMA | MARGINAL ZONE LYMPHOMA | GATED PROTON CHANNEL | SEQUENCING DATA | HEMATOLOGY | CODING VARIANTS | MALT LYMPHOMA | INACTIVATING MUTATIONS | HISTONE GENES | Vacuolar Proton-Translocating ATPases - genetics | Protein-Tyrosine Kinases - metabolism | Receptors, Notch - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Lymphoma, Follicular - diagnosis | Ion Channels - genetics | Male | Receptors, Notch - genetics | Gene Expression Profiling | Receptors, Antigen, B-Cell - metabolism | Vacuolar Proton-Translocating ATPases - metabolism | CREB-Binding Protein - genetics | CREB-Binding Protein - metabolism | Protein-Tyrosine Kinases - genetics | Lymphoma, Follicular - genetics | Early Growth Response Protein 1 - genetics | Trans-Activators - genetics | Aged, 80 and over | Adult | Female | Receptors, CXCR4 - genetics | Repressor Proteins - metabolism | Lymphoma, Follicular - pathology | Lymphoma, Follicular - mortality | Repressor Proteins - genetics | Signal Transduction - genetics | Receptors, CXCR4 - metabolism | Disease-Free Survival | Histones - genetics | Ion Channels - metabolism | Receptors, Antigen, B-Cell - genetics | Trans-Activators - metabolism | Aged | Histones - metabolism | Mutation | Early Growth Response Protein 1 - metabolism | Index Medicus | Abridged Index Medicus | Lymphoid Neoplasia
Journal Article
Cell Stem Cell, ISSN 1934-5909, 01/2014, Volume 14, Issue 1, pp. 40 - 52
Journal Article