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1. Full Text Increased dendritic spine densities on cortical projection neurons in autism spectrum disorders
by Hutsler, Jeffrey J and Zhang, Hong
Brain Research, ISSN 0006-8993, 2009, Volume 1309, pp. 83 - 94
Abstract Multiple types of indirect evidence have been used to support theories of altered cortical connectivity in autism spectrum disorders (ASD). In other... 
Neurology | Dendritic spine | Cerebral cortex | Pyramidal cell | Autistic disorder | Neuroanatomy | QUANTITATIVE GOLGI ANALYSIS | MENTAL-RETARDATION | WHITE-MATTER | HUMAN PREFRONTAL CORTEX | NEUROSCIENCES | HIGH-FUNCTIONING AUTISM | PYRAMIDAL NEURONS | HUMAN CEREBRAL-CORTEX | FRAGILE-X-SYNDROME | HEAD CIRCUMFERENCE | DEVELOPMENTAL DISORDERS | Humans | Middle Aged | Child Development Disorders, Pervasive - physiopathology | Male | Synapses - pathology | Cerebral Cortex - physiopathology | Young Adult | Adult | Female | Neural Pathways - physiopathology | Cerebral Cortex - abnormalities | Child | Mental Processes - physiology | Cognition Disorders - physiopathology | Child Development Disorders, Pervasive - pathology | Silver Staining | Cognition Disorders - pathology | Intellectual Disability - pathology | Organ Size - physiology | Aging - pathology | Efferent Pathways - physiopathology | Intellectual Disability - physiopathology | Neural Pathways - abnormalities | Dendritic Spines - pathology | Adolescent | Pyramidal Cells - pathology | Cell Shape - physiology | Efferent Pathways - abnormalities
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2. Full Text The Cerebral Signature for Pain Perception and Its Modulation
by Tracey, Irene and Mantyh, Patrick W
Neuron, ISSN 0896-6273, 08/2007, Volume 55, Issue 3, pp. 377 - 391
Clinical pain is a serious public health issue. Treatment of pain-related suffering requires knowledge of how pain signals are initially interpreted and... 
Pain - psychology | Animals | Pain - physiopathology | Brain - physiopathology | Humans | Nociceptors | Brain Stem - physiopathology | Cognition | Central Nervous System - physiopathology | Affect | Emotions | Efferent Pathways - physiopathology | Studies | Influence | Medical imaging | Behavior | Pain management | Animal cognition
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3. Full Text Treatment of motor and non-motor features of Parkinson's disease with deep brain stimulation
by Fasano, Alfonso, MD and Daniele, Antonio, MD and Albanese, Alberto, Prof
Lancet Neurology, The, ISSN 1474-4422, 2012, Volume 11, Issue 5, pp. 429 - 442
Summary Deep brain stimulation (DBS) is an established procedure for the symptomatic treatment of Parkinson's disease. Several deep brain nuclei have been... 
Neurology | SUBTHALAMIC NUCLEUS STIMULATION | IMPULSE CONTROL DISORDERS | LA-TOURETTE-SYNDROME | INTERMEDIATE THALAMIC NUCLEUS | HUMAN PEDUNCULOPONTINE NUCLEUS | INTERNAL PALLIDAL STIMULATION | HIGH-FREQUENCY STIMULATION | QUALITY-OF-LIFE | RANDOMIZED CONTROLLED-TRIAL | CAUDAL ZONA INCERTA | CLINICAL NEUROLOGY | Parkinson Disease - therapy | Motor Activity - physiology | Mental Disorders - therapy | Follow-Up Studies | Speech Disorders - therapy | Humans | Deep Brain Stimulation - adverse effects | Intralaminar Thalamic Nuclei - physiopathology | Globus Pallidus - physiopathology | Disability Evaluation | Motor Skills - physiology | Pedunculopontine Tegmental Nucleus - physiopathology | Subthalamic Nucleus - physiopathology | Basal Ganglia - physiopathology | Dyskinesias - physiopathology | Parkinson Disease - physiopathology | Disease Progression | Speech Disorders - physiopathology | Efferent Pathways - physiopathology | Mental Disorders - physiopathology | Ventral Thalamic Nuclei - physiopathology | Magnetic Resonance Imaging | Dyskinesias - therapy | Neurologic Examination | Brain Mapping | Quality of Life | Care and treatment | Parkinson's disease | Drugs | Brain | tremor | Neurodegenerative diseases | Deep brain stimulation | Globus pallidus | Data processing | Pedunculopontine tegmental nucleus | Electrical stimuli | subthalamic nucleus | Thalamus | Movement disorders
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4. Full Text Proportional recovery after stroke depends on corticomotor integrity
by Byblow, Winston D and Stinear, Cathy M and Barber, P. Alan and Petoe, Matthew A and Ackerley, Suzanne J
Annals of Neurology, ISSN 0364-5134, 12/2015, Volume 78, Issue 6, pp. 848 - 859
Objective For most patients, resolution of upper limb impairment during the first 6 months poststroke is 70% of the maximum possible. We sought to identify... 
CORTICOSPINAL TRACT INTEGRITY | TRANSCRANIAL MAGNETIC STIMULATION | BDNF | MOTOR CORTEX EXCITABILITY | CORTICAL REORGANIZATION | INTERHEMISPHERIC DIFFERENCES | ISCHEMIC-STROKE | NEUROSCIENCES | BRAIN | CLINICAL NEUROLOGY | POSTSTROKE | IMPAIRMENT | Outcome Assessment (Health Care) | Humans | Middle Aged | Stroke Rehabilitation | Male | Brain Ischemia - physiopathology | Stroke - physiopathology | Cerebral Cortex - physiopathology | Efferent Pathways - physiopathology | Transcranial Magnetic Stimulation | Upper Extremity - physiopathology | Evoked Potentials, Motor | Brain Ischemia - rehabilitation | Muscle, Skeletal - physiopathology | Diffusion Magnetic Resonance Imaging | Aged, 80 and over | Recovery of Function - physiology | Adult | Female | Aged | Electromyography | Cohort Studies
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5. Full Text Synaptopathy in the noise-exposed and aging cochlea: Primary neural degeneration in acquired sensorineural hearing loss
by Kujawa, Sharon G and Liberman, M. Charles
Hearing Research, ISSN 0378-5955, 12/2015, Volume 330, Issue Pt B, pp. 191 - 199
The classic view of sensorineural hearing loss (SNHL) is that the “primary” targets are hair cells, and that cochlear-nerve loss is “secondary” to hair cell... 
INNER-EAR | HAIR CELL LOSS | GLUTAMATE SYNAPSE | OLIVOCOCHLEAR EFFERENTS | AUDIOLOGY & SPEECH-LANGUAGE PATHOLOGY | BRAIN-STEM | GUINEA-PIG | NEUROSCIENCES | SPIRAL GANGLION NEURONS | AFFERENT SYNAPSES | OTORHINOLARYNGOLOGY | MAMMALIAN COCHLEA | AUDITORY-NERVE FIBERS | Cochlear Nerve - physiopathology | Hair Cells, Auditory - pathology | Hearing Loss, Noise-Induced - physiopathology | Guinea Pigs | Acoustic Stimulation | Humans | Hearing Loss, Sensorineural - pathology | Cochlea - innervation | Auditory Threshold | Hearing Loss, Sensorineural - psychology | Synaptic Transmission | Auditory Pathways - pathology | Hearing | Animals | Evoked Potentials, Auditory, Brain Stem | Cochlear Nerve - pathology | Hearing Loss, Noise-Induced - pathology | Auditory Pathways - physiopathology | Mice | Noise - adverse effects | Auditory Perception | Nerve Degeneration | Disease Models, Animal | Hearing Loss, Sensorineural - physiopathology | Physiological aspects | Hearing loss
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6. Full Text Central circuitries for body temperature regulation and fever
by Nakamura, Kazuhiro
American Journal of Physiology - Regulatory Integrative and Comparative Physiology, ISSN 0363-6119, 11/2011, Volume 301, Issue 5, pp. R1207 - R1228
Nakamura K. Central circuitries for body temperature regulation and fever. Am J Physiol Regul Integr Comp Physiol 301: R1207-R1228, 2011. First published... 
Brown adipose tissue thermogenesis | Shivering | Sympathetic nervous system | Skin vasoconstriction | brown adipose tissue thermogenesis | skin vasoconstriction | PHYSIOLOGY | THERMOREGULATORY VASOMOTOR CONTROL | sympathetic nervous system | INTERMEDIOLATERAL CELL COLUMN | BROWN ADIPOSE-TISSUE | EXPRESSING PREOPTIC NEURONS | ROSTRAL MEDULLARY RAPHE | INDUCED MEMBRANE DEPOLARIZATION | shivering | SYMPATHETIC PREMOTOR NEURONS | CENTRAL EFFERENT PATHWAYS | PROSTAGLANDIN EP3 RECEPTOR | STRESS-INDUCED HYPERTHERMIA | Thermoreceptors - physiopathology | Central Nervous System - metabolism | Signal Transduction | Fever - metabolism | Humans | Body Temperature Regulation | Awards and Prizes | Preoptic Area - physiopathology | Fever - physiopathology | Thermosensing | Fever - psychology | Skin - blood supply | Feedback, Physiological | Animals | Skin - innervation | Central Nervous System - physiopathology | Fever - immunology | Neural Pathways - physiopathology | Skin Temperature | Body temperature | Physiological aspects | Homeostasis | Development and progression | Research | Health aspects | Hyperthermia | Fever
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7. Full Text Altered cortico-basal ganglia motor pathways reflect reduced volitional motor activity in schizophrenia
by Bracht, Tobias and Schnell, Susanne and Federspiel, Andrea and Razavi, Nadja and Horn, Helge and Strik, Werner and Wiest, Roland and Dierks, Thomas and Müller, Thomas J and Walther, Sebastian
Schizophrenia Research, ISSN 0920-9964, 2012, Volume 143, Issue 2, pp. 269 - 276
Abstract Little is known about the neurobiology of hypokinesia in schizophrenia. Therefore, the aim of this study was to investigate alterations of white... 
Psychiatry | Schizophrenia | Actigraphy | White matter | Tractography | Movement disorders | Diffusion tensor imaging | ACTIVATION | MRI | PSYCHIATRY | SYMPTOMS | 1ST-EPISODE SCHIZOPHRENIA | CONNECTIVITY | CORTEX | DIFFUSION-TENSOR | CEREBRAL-BLOOD-FLOW | PARKINSONS-DISEASE | Motor Activity - physiology | Basal Ganglia - physiopathology | Humans | Male | Motor Cortex - physiopathology | Actigraphy - instrumentation | Volition - physiology | Efferent Pathways - physiopathology | Monitoring, Ambulatory | Diffusion Tensor Imaging - methods | Schizophrenia - physiopathology | Actigraphy - methods | Hypokinesia - physiopathology | Adult | Diffusion Tensor Imaging - instrumentation | Female
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8. Full Text Efferent pathways modulate hyperactivity in inferior colliculus
by Mulders, Wilhelmina Henrica A. M and Seluakumaran, Kumar and Robertson, Donald
Journal of Neuroscience, ISSN 0270-6474, 07/2010, Volume 30, Issue 28, pp. 9578 - 9587
Animal models have demonstrated that mild hearing loss caused by acoustic trauma results in spontaneous hyperactivity in the central auditory pathways. This... 
OUTER HAIR-CELLS | COCHLEAR NUCLEUS NEURONS | CROSSED OLIVOCOCHLEAR BUNDLE | PROJECTION PATTERNS | ELECTRICAL-STIMULATION | GUINEA-PIG COCHLEA | GENE-EXPRESSION | NEURAL ACTIVITY | BRAIN-STEM | NEUROSCIENCES | AUDITORY-NERVE FIBERS | Hearing Loss, Noise-Induced - physiopathology | Inferior Colliculi - physiopathology | Guinea Pigs | Acoustic Stimulation | Cochlea - physiopathology | Electrophysiology | Male | Hyperkinesis - physiopathology | Efferent Pathways - physiopathology | Animals | Neurons - physiology | Auditory Pathways - physiopathology | Female
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9. Full Text Animal models of neurological deficits: how relevant is the rat?
by Schallert, Timothy and Cenci, M. Angela and Whishaw, Ian Q and Neuronano Research Center (NRC) and Basala gangliernas patofysiologi and Lund University and Basal Ganglia Pathophysiology and Lunds universitet
Nature Reviews Neuroscience, ISSN 1471-003X, 07/2002, Volume 3, Issue 7, pp. 574 - 579
Animal models of neurological deficits are essential for the assessment of new therapeutic options. It has been suggested that rats are not as appropriate as... 
OPIOID RECEPTOR-BINDING | DOPA-INDUCED DYSKINESIA | LEVODOPA-INDUCED DYSKINESIAS | DESCENDING PATHWAYS | BASAL GANGLIA | CORTICOSPINAL TRACT | SPINAL-CORD | 6-HYDROXYDOPAMINE LESION | BEHAVIORAL-ASSESSMENT | NEUROSCIENCES | PARKINSONS-DISEASE | Reproducibility of Results | Species Specificity | Humans | Dyskinesias - physiopathology | Rats | Brain Ischemia - physiopathology | Brain Injuries - physiopathology | Parkinson Disease - physiopathology | Efferent Pathways - physiopathology | Animals | Primates | Nervous System Diseases - physiopathology | Disease Models, Animal | Human | Parkinson Disease : physiopathology | Disease Models | Basic Medicine | Neurosciences | Medical and Health Sciences | Medicin och hälsovetenskap | Nervous System Diseases : physiopathology | Animal | Medicinska och farmaceutiska grundvetenskaper | Efferent Pathways : physiopathology | Dyskinesias : physiopathology | Brain Ischemia : physiopathology | Brain Injuries : physiopathology | Neurovetenskaper
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