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Science (American Association for the Advancement of Science), ISSN 1095-9203, 2011, Volume 334, Issue 6057, pp. 809 - 813
Phospholipase A₂ (PLA₂) enzymes are considered the primary source of arachidonic acid for cyclooxygenase (COX)-mediated biosynthesis of prostaglandins. Here,... 
Datasets | Brain | Enzymes | Prostaglandins | Cytokines | Neurodegenerative diseases | REPORTS | Eicosanoids | Lipids | Endocannabinoids | Vehicles | SYSTEM | INHIBITION | CYCLOOXYGENASE-2 | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | INJURY | LIPOPOLYSACCHARIDE | PHOSPHOLIPASE A | MICE | PARKINSONS-DISEASE | Cannabinoid Receptor Modulators - metabolism | Inflammation - pathology | Metabolomics | Arachidonic Acid - metabolism | Monoacylglycerol Lipases - antagonists & inhibitors | Monoacylglycerol Lipases - genetics | Brain - metabolism | Parkinsonian Disorders - metabolism | Monoacylglycerol Lipases - metabolism | Arachidonic Acids - metabolism | Inflammation - metabolism | Neuroprotective Agents - pharmacology | Piperidines - pharmacology | Inflammation Mediators - pharmacology | Lung - metabolism | Cytokines - metabolism | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Phospholipases A2 - metabolism | Prostaglandins - biosynthesis | Glycerides - metabolism | Brain - drug effects | Hydrolysis | Animals | Parkinsonian Disorders - pathology | Eicosanoids - metabolism | Lipopolysaccharides - pharmacology | Brain - pathology | Mice | Cyclooxygenase 1 - metabolism | Benzodioxoles - pharmacology | Phospholipases A2 - genetics | Prostaglandins - metabolism | Physiological aspects | Inflammation | Research | Risk factors | Hydrology | Neurology | Inflammatory diseases
Journal Article
Nature (London), ISSN 1476-4687, 2012, Volume 490, Issue 7418, pp. 107 - 111
Detection of microbial products by host inflammasomes is an important mechanism of innate immune surveillance. Inflammasomes activate the caspase-1 (CASP1)... 
CYTOSOLIC PHOSPHOLIPASE A | FLAGELLIN | III SECRETION APPARATUS | MECHANISM | MICE DEFICIENT | MULTIDISCIPLINARY SCIENCES | NLRC4 INFLAMMASOME | INFECTION | PYROPTOSIS | CASPASE-1 ACTIVATION | IMMUNE RECOGNITION | Inflammation - pathology | Inflammasomes - metabolism | Hematocrit | Salmonella Infections - immunology | Eicosanoids - biosynthesis | Caspase 1 - metabolism | Male | Inflammation - metabolism | Time Factors | Flagellin - metabolism | Bacterial Toxins - genetics | Antigens, Bacterial - chemistry | Neuronal Apoptosis-Inhibitory Protein - deficiency | Macrophages, Peritoneal - immunology | Calcium-Binding Proteins - deficiency | Interleukin-1beta | Bacterial Toxins - chemistry | Apoptosis Regulatory Proteins - metabolism | Cyclooxygenase 1 - deficiency | Body Fluids - metabolism | Caspase 1 - deficiency | Recombinant Fusion Proteins - genetics | Cytosol - metabolism | Death | Mice | Antigens, Bacterial - metabolism | Capillary Permeability | Peritoneal Cavity | Legionella pneumophila | Intestines - metabolism | Antigens, Bacterial - genetics | Recombinant Fusion Proteins - metabolism | Peritoneal Lavage | Apoptosis Regulatory Proteins - deficiency | Flagellin - genetics | Female | Interleukin-18 | Calcium Signaling | Calcium-Binding Proteins - metabolism | Flagellin - immunology | Salmonella typhimurium - immunology | Mice, Inbred C57BL | Inflammation - immunology | Body Temperature | Neuronal Apoptosis-Inhibitory Protein - metabolism | Immunity, Innate - immunology | Bacterial Toxins - metabolism | Animals | Eicosanoids - metabolism | Fluid Shifts | Cellular signal transduction | Research | Properties | Bacterial proteins | Flagella (Microbiology) | Immune system | Proteins | Bone marrow | Pathology | Infections | Biosynthesis | Rodents
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 5/2012, Volume 109, Issue 22, pp. 8517 - 8522
... inhibit cyclooxygenase pathways. This inhibition was able to shunt metabolism of AA to lipoxygenase pathways, augmenting leukotriene and other lipoxygenase mediator synthesis... 
Prostaglandins | Receptors | Cellular metabolism | Cyclooxygenase inhibitors | Eicosanoids | Lipids | Phospholipids | Cell membranes | Lipid metabolism | Fatty acids | P388D MACROPHAGES | CYTOSOLIC PHOSPHOLIPASE A | INHIBITION | RAW264.7 CELLS | PROSTAGLANDIN-E SYNTHASE-1 | MULTIDISCIPLINARY SCIENCES | PURIFICATION | N-3 FATTY-ACIDS | CELL-PROLIFERATION | EXPRESSION | ARACHIDONATE | Arachidonic Acid - metabolism | Gas Chromatography-Mass Spectrometry | Male | Macrophages, Peritoneal - cytology | Adenosine Triphosphate - pharmacology | Lipoxygenase - metabolism | Fatty Acids, Omega-3 - pharmacology | Toll-Like Receptor 4 - agonists | Cell Membrane - metabolism | Macrophages, Peritoneal - drug effects | Cell Membrane - drug effects | Cell Line | Fatty Acids, Omega-3 - metabolism | Cells, Cultured | Fatty Acids, Unsaturated - pharmacology | Phospholipases A2 - metabolism | Fatty Acids, Unsaturated - metabolism | Macrophages - cytology | Toll-Like Receptor 4 - metabolism | Blotting, Western | Docosahexaenoic Acids - pharmacology | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Prostaglandin-Endoperoxide Synthases - metabolism | Eicosanoids - metabolism | Arachidonic Acid - pharmacology | Lipopolysaccharides - pharmacology | Membrane Lipids - metabolism | Receptors, Purinergic P2X7 - metabolism | Macrophages - drug effects | Mice | Macrophages, Peritoneal - metabolism | Docosahexaenoic Acids - metabolism | Physiological aspects | Eicosanoic acid | Research | Health aspects | Omega-3 fatty acids | Proteomics | Biological Sciences
Journal Article
Journal of lipid research, ISSN 1539-7262, 2014, Volume 55, Issue 12, pp. 2471 - 2478
Journal Article
Cellular and molecular life sciences : CMLS, ISSN 1420-9071, 2015, Volume 73, Issue 1, pp. 57 - 77
As the primary protective barrier for neurons in the brain, the blood–brain barrier (BBB) exists between the blood microcirculation system and the brain... 
Life Sciences | Biochemistry, general | Life Sciences, general | Agrin | MicroRNAs | Inflammatory mediators | Annexins | Anesthetics | Biomedicine general | Cell Biology | Integrins | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MICROVESSEL ENDOTHELIAL-CELLS | FOCAL CEREBRAL-ISCHEMIA | ACID PHENETHYL ESTER | BASEMENT-MEMBRANE PROTEINS | NECROSIS-FACTOR-ALPHA | CELL BIOLOGY | TIGHT JUNCTIONS | MULTIPLE-SCLEROSIS | MICROVASCULAR BASAL LAMINA | CENTRAL-NERVOUS-SYSTEM | Brain Diseases - metabolism | Cytokines - analysis | Humans | Capillary Permeability - drug effects | Anesthetics - pharmacology | MicroRNAs - metabolism | Agrin - metabolism | Integrins - metabolism | Brain Diseases - drug therapy | MicroRNAs - analysis | Platelet Endothelial Cell Adhesion Molecule-1 - metabolism | Tight Junction Proteins - metabolism | Receptor-Like Protein Tyrosine Phosphatases, Class 3 - analysis | Tight Junction Proteins - analysis | Annexins - metabolism | Cytokines - metabolism | Platelet Endothelial Cell Adhesion Molecule-1 - analysis | Blood-Brain Barrier - drug effects | Integrins - analysis | Iron - metabolism | Blood-Brain Barrier - metabolism | Annexins - analysis | Blood-Brain Barrier - pathology | Animals | Brain Diseases - pathology | Eicosanoids - analysis | Signal Transduction - drug effects | Receptor-Like Protein Tyrosine Phosphatases, Class 3 - metabolism | Eicosanoids - metabolism | Agrin - analysis | Medical colleges | MicroRNA | Endothelial growth factors | Neurons | Pharmacy | Physiological aspects | Permeability | Neurophysiology | Endothelium | Brain | Anesthesia | Blood
Journal Article
Journal of lipid research, ISSN 0022-2275, 11/2013, Volume 54, Issue 11, pp. 3085 - 3097
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2019, Volume 116, Issue 13, pp. 6292 - 6297
Inflammation in the tumor microenvironment is a strong promoter of tumor growth. Substantial epidemiologic evidence suggests that aspirin, which suppresses... 
Eicosanoids | Metabolomics | Inflammation | Metastasis | Resolvins | CELLS | MULTIDISCIPLINARY SCIENCES | PEPTIDE RECEPTOR 2 | LIPID MEDIATORS | eicosanoids | RESPONSES | RESOLVIN D1 | LOW-DOSE ASPIRIN | metabolomics | inflammation | metastasis | NONSTEROIDAL ANTIINFLAMMATORY DRUGS | IN-VIVO | DISEASE | resolvins | Microtubule-Associated Proteins - metabolism | Neoplasms - prevention & control | Aspirin - administration & dosage | Neoplasm Metastasis - drug therapy | Inflammation - drug therapy | Neoplasm Metastasis - prevention & control | Female | Antineoplastic Agents - pharmacology | Chemokine CCL2 - metabolism | Aspirin - pharmacology | Disease Models, Animal | Phagocytosis - drug effects | Cytokines - metabolism | Mice, Inbred C57BL | Fatty Acids, Unsaturated - metabolism | Plasminogen Inactivators - metabolism | Neoplasms - drug therapy | Nerve Tissue Proteins - metabolism | Macrophages - metabolism | Animals | Eicosanoids - metabolism | Macrophages - drug effects | Chemokines - metabolism | Mice | Mice, Inbred BALB C | Lipoxins - metabolism | Docosahexaenoic Acids - metabolism | Prostaglandins - metabolism | Carbon-carbon composites | Aspirin | Lipoxin A4 | Leukocyte migration | Cytokines | Toxicity | Health risks | Pharmacology | Biosynthesis | Macrophages | Epidemiology | Anticancer properties | Proteins | Monocytes | Prostaglandins | Migration inhibitory factor | Plasminogen activator inhibitors | Antitumor activity | Monocyte chemoattractant protein 1 | Cell migration | Phagocytosis | Cancer | Tumors | Biological Sciences
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 09/2013, Volume 33, Issue 9, pp. 2193 - 2201
...) 2 affect eicosanoids metabolism in atherosclerosis. APPROACH AND RESULTS—Analysis of human carotid plaques revealed that NOD2 was abundantly expressed at both mRNA and protein levels by endothelial cells and macrophages... 
atherosclerosis | pattern recognition receptors | eicosanoids | peptidoglycan | immunity, innate | inflammation | ACTIVATED PROTEIN-KINASE | CROHNS-DISEASE | HUMAN SYNOVIAL-CELLS | HUMAN ENDOTHELIAL-CELLS | LEUKOTRIENE B-4 | MURAMYL DIPEPTIDE | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | E-DEFICIENT MICE | STIMULATES COLLAGENASE | PROSTAGLANDIN E-2 | Carotid Arteries - metabolism | Tumor Necrosis Factor-alpha - metabolism | Up-Regulation | Arachidonate 12-Lipoxygenase - metabolism | Humans | Transcriptional Activation | Nod2 Signaling Adaptor Protein - genetics | Case-Control Studies | Time Factors | Carotid Artery Diseases - pathology | Interleukin-1beta - metabolism | Transcription, Genetic | p38 Mitogen-Activated Protein Kinases - metabolism | Carotid Artery Diseases - metabolism | Fibroblasts - metabolism | Carotid Artery Diseases - immunology | Arachidonate 5-Lipoxygenase - metabolism | Signal Transduction | Endothelial Cells - metabolism | Tissue Culture Techniques | Cells, Cultured | Acetylmuramyl-Alanyl-Isoglutamine - metabolism | Arachidonate 15-Lipoxygenase - metabolism | Immunity, Innate | Plaque, Atherosclerotic | Dinoprostone - metabolism | Endothelial Cells - immunology | Nod2 Signaling Adaptor Protein - metabolism | Eicosanoids - metabolism | Carotid Arteries - immunology | Carotid Artery Diseases - genetics | Cyclooxygenase 2 - metabolism | Fibroblasts - immunology | Ligands | Carotid Arteries - pathology | Kardiologi | Klinisk medicin | Medicin och hälsovetenskap | Cell- och molekylärbiologi | Medicinska och farmaceutiska grundvetenskaper
Journal Article
PloS one, ISSN 1932-6203, 11/2013, Volume 8, Issue 11, p. e80316
...), affects milk production, metabolism, and health during this period. Thirty-three Holstein cows... 
INSULIN | TRANSITION | ETIOLOGY | TUMOR-NECROSIS-FACTOR | ORGANIC NUTRIENT METABOLISM | PHOSPHORYLATION | MULTIDISCIPLINARY SCIENCES | FATTY LIVER | PREVENTION | MASTITIS | STRESS | Tumor Necrosis Factor-alpha - blood | Lactation - drug effects | Haptoglobins - metabolism | Insulin - blood | Inflammation - blood | Inflammation - metabolism | Liver - drug effects | Cattle | Fatty Acids, Nonesterified - blood | Female | Eating | 3-Hydroxybutyric Acid - blood | Liver - metabolism | Recombinant Proteins - pharmacology | Methylhistidines - blood | Triglycerides - metabolism | Blood Glucose - drug effects | Recombinant Proteins - blood | Tumor Necrosis Factor-alpha - pharmacology | Animals | Milk - metabolism | Lactation - metabolism | Lipid Metabolism - drug effects | Triglycerides - blood | Glucose - metabolism | Blood Glucose - metabolism | Energy Metabolism - drug effects | Parturition | Phosphorylation | Lactation | Liver | Body weight | Disorders | Eicosanoids | Dry matter | Lipids | Glucose | Kinases | Dosage | Turnover rate | Animal lactation | Proteins | Glucose metabolism | E coli | Haptoglobin | Rodents | Tumor necrosis factor-TNF | Oxidation | Water intakes | Lipid metabolism | Milk | Cytokines | Health | Animal sciences | Glycerol | Inflammation | Tumor necrosis factor-α | Gene expression | Metabolism | Insulin | Fatty acids | Esterification | Milk production | Water intake | Feeds | Metabolic disorders
Journal Article