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Toxicon, ISSN 0041-0101, 02/2016, Volume 110, pp. 90 - 109
Ureases are metalloenzymes that hydrolyze urea into ammonia and carbon dioxide. They were the first enzymes to be crystallized and, with them, the notion that... 
Neurotoxicity | Insecticide | Antifungal | Canavalia ensiformis | Helicobacter pylori | Virulence factor | Eicosanoids | Proteolytic activation | Glycine max | Pro-inflammatory activity | URINARY-TRACT-INFECTION | BACILLUS-PASTEURII | VECTOR RHODNIUS-PROLIXUS | NITRIC-OXIDE SYNTHASE | JACK-BEAN UREASE | CANAVALIA-ENSIFORMIS UREASE | HELICOBACTER-PYLORI UREASE | NEUTROPHIL-ACTIVATING PROTEIN | PHARMACOLOGY & PHARMACY | GASTRIC EPITHELIAL-CELLS | CENTRAL-NERVOUS-SYSTEM | TOXICOLOGY | Metalloproteins - toxicity | Humans | Fungicides, Industrial - toxicity | Neurotoxins - genetics | Fungicides, Industrial - pharmacology | Plant Proteins - toxicity | Urease - pharmacology | Metalloproteins - metabolism | Bacterial Proteins - toxicity | Isoenzymes - toxicity | Isoenzymes - metabolism | Apoenzymes - metabolism | Metalloproteins - genetics | Plant Proteins - metabolism | Insecticides - pharmacology | Recombinant Proteins - metabolism | Plant Proteins - pharmacology | Isoenzymes - genetics | Bacterial Proteins - genetics | Neurotoxins - toxicity | Apoenzymes - pharmacology | Recombinant Proteins - pharmacology | Apoenzymes - toxicity | Neurotoxins - pharmacology | Plant Proteins - genetics | Bacterial Proteins - pharmacology | Animals | Insecticides - metabolism | Urease - toxicity | Apoenzymes - genetics | Isoenzymes - pharmacology | Recombinant Proteins - toxicity | Bacterial Proteins - metabolism | Metalloproteins - pharmacology | Neurotoxins - metabolism | Urease - metabolism | Urease - genetics | Insecticides - toxicity | Enzymes | Urea | Bacterial infections | Enzyme inhibitors | Peptides | Proteolysis | Analysis | Physiological aspects | Hydrolases | Virulence (Microbiology) | Metalloenzymes | Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 2015, Volume 308, Issue 9, pp. H1020 - H1029
This study addressed the hypothesis that inhibiting the soluble epoxide hydrolase (sEH)-mediated degradation of epoxy-fatty acids, notably epoxyeicosatrienoic... 
Insulin resistance | Cardiac function | Soluble epoxide hydrolase | Endothelium | endothelium | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | HEART-FAILURE | RATS | MECHANISMS | soluble epoxide hydrolase | DIABETES-MELLITUS | CARDIOVASCULAR-DISEASE | PERIPHERAL VASCULAR DISEASE | cardiac function | DYSFUNCTION | HYPERTENSION | ARTERY | insulin resistance | EXPRESSION | Obesity - drug therapy | Coronary Vessels - physiopathology | Endothelium, Vascular - drug effects | Heart Diseases - prevention & control | Male | Endothelium, Vascular - enzymology | Obesity - blood | Dose-Response Relationship, Drug | Glyburide - pharmacology | Time Factors | Lipids - blood | Urea - analogs & derivatives | Inflammation Mediators - metabolism | Disease Models, Animal | Coronary Vessels - drug effects | Coronary Vessels - enzymology | Vasodilator Agents - pharmacology | Heart Diseases - physiopathology | Obesity - complications | Ventricular Function, Left - drug effects | Endothelium, Vascular - physiopathology | Enzyme Inhibitors - pharmacology | Insulin Resistance | Obesity - physiopathology | Heart Diseases - enzymology | Hypoglycemic Agents - pharmacology | Blood Glucose - drug effects | Heart Diseases - etiology | Animals | Eicosanoids - metabolism | Benzoates - pharmacology | Mice | Obesity - enzymology | Epoxide Hydrolases - metabolism | Vasodilation - drug effects | Blood Glucose - metabolism | Nitric Oxide - metabolism | Epoxide Hydrolases - antagonists & inhibitors | Ventricular Remodeling - drug effects | Urea - pharmacology | Prevention | Obesity | Enzymes | Physiological aspects | Hydrolases | Research | Heart diseases | Homeostasis | Physiology | Nitric oxide | Life Sciences | Call for Papers
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 10/2013, Volume 288, Issue 40, pp. 28641 - 28655
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2000, Volume 97, Issue 11, pp. 6155 - 6160
Journal Article
Science, ISSN 0036-8075, 11/2011, Volume 334, Issue 6057, pp. 809 - 813
Phospholipase A₂ (PLA₂) enzymes are considered the primary source of arachidonic acid for cyclooxygenase (COX)-mediated biosynthesis of prostaglandins. Here,... 
Datasets | Brain | Enzymes | Prostaglandins | Cytokines | Neurodegenerative diseases | REPORTS | Eicosanoids | Lipids | Endocannabinoids | Vehicles | SYSTEM | INHIBITION | CYCLOOXYGENASE-2 | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | INJURY | LIPOPOLYSACCHARIDE | PHOSPHOLIPASE A | MICE | PARKINSONS-DISEASE | Cannabinoid Receptor Modulators - metabolism | Inflammation - pathology | Metabolomics | Arachidonic Acid - metabolism | Monoacylglycerol Lipases - antagonists & inhibitors | Monoacylglycerol Lipases - genetics | Brain - metabolism | Parkinsonian Disorders - metabolism | Monoacylglycerol Lipases - metabolism | Arachidonic Acids - metabolism | Inflammation - metabolism | Neuroprotective Agents - pharmacology | Piperidines - pharmacology | Inflammation Mediators - pharmacology | Lung - metabolism | Cytokines - metabolism | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Phospholipases A2 - metabolism | Prostaglandins - biosynthesis | Glycerides - metabolism | Brain - drug effects | Hydrolysis | Animals | Parkinsonian Disorders - pathology | Eicosanoids - metabolism | Lipopolysaccharides - pharmacology | Brain - pathology | Mice | Cyclooxygenase 1 - metabolism | Benzodioxoles - pharmacology | Phospholipases A2 - genetics | Prostaglandins - metabolism | Physiological aspects | Inflammation | Research | Risk factors | Hydrology | Neurology | Inflammatory diseases
Journal Article
Cancer Research, ISSN 0008-5472, 08/2000, Volume 60, Issue 16, pp. 4629 - 4637