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1. Full Text Endocrine protection of ischemic myocardium by FGF21 from the liver and adipose tissue
by Liu, Shu Q and Roberts, Derek and Kharitonenkov, Alexei and Zhang, Brian and Hanson, Samuel M and Li, Yan Chun and Zhang, Li-Qun and Wu, Yu H
Scientific Reports, ISSN 2045-2322, 2013, Volume 3, Issue 1, pp. 2767 - 2767
Myocardial ischemia, while causing cardiomyocyte injury, can activate innate protective processes, enhancing myocardial tolerance to ischemia. Such processes... 
BETA-KLOTHO | SURVIVAL | ENDOTHELIAL PROGENITOR CELLS | ARTERIAL ELASTIC LAMINAE | PPAR-ALPHA | ACTIVATION | MULTIDISCIPLINARY SCIENCES | REPERFUSION INJURY | BONE-MARROW | C-KIT | AKT | Up-Regulation | Liver - pathology | Phosphorylation | Cardiotonic Agents - metabolism | Adipose Tissue - physiopathology | Caspase 3 - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Glucuronidase | Liver - physiopathology | Phosphatidylinositol 3-Kinases | Myocardial Reperfusion Injury - enzymology | Myocytes, Cardiac - enzymology | Adipose Tissue - metabolism | Fibroblast Growth Factors - metabolism | Myocardial Reperfusion Injury - pathology | Endocrine System - metabolism | Myocardial Ischemia - physiopathology | bcl-Associated Death Protein - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Heart Function Tests | Heart Ventricles - pathology | Signal Transduction | Liver - metabolism | Adipose Tissue - pathology | Gene Silencing | Myocardial Ischemia - prevention & control | Myocardial Ischemia - enzymology | Myocardial Reperfusion Injury - physiopathology | Endocrine System - pathology | Myocardial Ischemia - pathology | Myocytes, Cardiac - pathology | Animals | Heart Ventricles - physiopathology | Protein Binding | Heart Ventricles - metabolism | Mice | Hemodynamics | RNA, Small Interfering - metabolism | Myocardial infarction | AKT1 protein | Adipose tissue | Liver | Caspase | Myocardial ischemia | Cardiomyocytes | Adipocytes | Caspase-3 | 1-Phosphatidylinositol 3-kinase | Membrane proteins | Reperfusion | Ischemia | Cell death | Klotho protein | Rodents | Myocardium | Fibroblast growth factor receptor 1 | Index Medicus
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2. Biology and pathology of elastic tissues
by Robert, A. M and Robert, Ladislas
1980, Frontiers of matrix biology, ISBN 9783805530781, Volume 8, viii, 230
physiology | Elastic Tissue | pathology | Elastin
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3. Full Text Losartan, an AT1 Antagonist Prevents Aortic Aneurysm in a Mouse Model of Marfan Syndrome
by Jennifer P. Habashi and Daniel P. Judge and Tammy M. Holm and Ronald D. Cohn and Bart L. Loeys and Timothy K. Cooper and Loretha Myers and Erin C. Klein and Guosheng Liu and Carla Calvi and Megan Podowski and Enid R. Neptune and Marc K. Halushka and Djahida Bedja and Kathleen Gabrielson and Daniel B. Rifkin and Luca Carta and Francesco Ramirez and David L. Huso and Harry C. Dietz
Science, ISSN 0036-8075, 4/2006, Volume 312, Issue 5770, pp. 117 - 121
Aortic aneurysm and dissection are manifestations of Marfan syndrome (MFS), a disorder caused by mutations in the gene that encodes fibrillin-1. Selected... 
Airspace | Receptors | Architecture | Dilatation | Root growth | Placebos | Reports | Mice | Aortic aneurysm | Elastic tissue | Marfan syndrome | PATHOGENESIS | GROWTH-FACTOR-BETA | ACTIVATION | FIBRILLIN | MULTIDISCIPLINARY SCIENCES | RATS | RECEPTOR | SMOOTH-MUSCLE-CELLS | BLOCKADE | CONTRIBUTES | DILATATION | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Propranolol - therapeutic use | Neutralization Tests | Aortic Aneurysm - etiology | Lung Diseases - drug therapy | Marfan Syndrome - drug therapy | Propranolol - administration & dosage | Antibodies - immunology | Transforming Growth Factor beta - antagonists & inhibitors | Female | Marfan Syndrome - complications | Microfilament Proteins - genetics | Adrenergic beta-Antagonists - administration & dosage | Adrenergic beta-Antagonists - therapeutic use | Angiotensin II Type 1 Receptor Blockers - administration & dosage | Aortic Aneurysm - prevention & control | Disease Models, Animal | Transforming Growth Factor beta - immunology | Fibrillin-1 | Lung - pathology | Pulmonary Alveoli - pathology | Signal Transduction | Fibrillins | Pregnancy Complications - drug therapy | Aorta - pathology | Pregnancy | Elastic Tissue - pathology | Animals | Receptor, Angiotensin, Type 1 - metabolism | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Losartan - administration & dosage | Lung Diseases - pathology | Mutation | Transforming Growth Factor beta - metabolism | Complications and side effects | Aortic aneurysms | Genetic aspects | Research | Risk factors | Signal transduction | Pharmacology | Models | Genetic disorders | Antagonist drugs | Rodents | Index Medicus
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4. Full Text Novel Histologic Finding: Adipose Tissue Is Prevalent Within Penile Tunica Albuginea and Corpora Cavernosa: An Anatomic Study of 63 Specimens and Considerations for Cancer Invasion
by Rodriguez, Ingrid M and Cuevas, Martin and Silvero, Arturo and Cañete-Portillo, Sofía and Sanchez, Diego F and Barreto, Jose and Cubilla, Antonio L
The American Journal of Surgical Pathology, ISSN 0147-5185, 09/2017, Volume 41, Issue 11, pp. 1542 - 1546
Adipose tissue, along with arteries, veins, and peripheral nerves, is a normal constituent of mesenchymal tissues encasing the corpora cavernosa at the level... 
SURGERY | corpora cavernosa | carcinoma | penile anatomy | adipose tissue | penis | PATHOLOGY | Adipose Tissue - surgery | Neoplasm Invasiveness | Carcinoma, Squamous Cell - pathology | Humans | Adipose Tissue - pathology | Carcinoma, Squamous Cell - surgery | Male | Elastic Tissue - pathology | Penile Neoplasms - surgery | Elastic Tissue - surgery | Biopsy | Penile Neoplasms - pathology | Fascia - pathology | Index Medicus
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5. Full Text Restrictive allograft syndrome post lung transplantation is characterized by pleuroparenchymal fibroelastosis
by Ofek, Efrat and Sato, Masaaki and Saito, Tomohito and Wagnetz, Ute and Roberts, Heidi C and Chaparro, Cecilia and Waddell, Thomas K and Singer, Lianne G and Hutcheon, Michael A and Keshavjee, Shaf and Hwang, David M
Modern Pathology, ISSN 0893-3952, 03/2013, Volume 26, Issue 3, pp. 350 - 356
We previously described restrictive allograft syndrome as a form of chronic lung allograft dysfunction, demonstrating restrictive pulmonary function decline.... 
lung allograft rejection | lung transplantation | interstitial lung disease | pleuroparenchymal fibroelastosis | pulmonary fibrosis | BRONCHIOLITIS | FIBROSIS | BONE-MARROW-TRANSPLANTATION | PATHOLOGY | ENTITY | REJECTION | DISEASE | DYSFUNCTION | SPECTRUM | Lung Diseases, Interstitial - etiology | Pleural Diseases - etiology | Lung Diseases, Interstitial - metabolism | Lung Diseases, Interstitial - pathology | Humans | Middle Aged | Lung - chemistry | Male | Young Adult | Pleura - chemistry | Autopsy | Adult | Female | Retrospective Studies | Bronchiolitis Obliterans - pathology | Pulmonary Fibrosis - etiology | Lung - pathology | Bronchiolitis Obliterans - etiology | Pulmonary Fibrosis - pathology | Lung Transplantation - adverse effects | Syndrome | Elastic Tissue - pathology | Pleural Diseases - metabolism | Biopsy | Adolescent | Pleura - pathology | Collagen - analysis | Pleural Diseases - pathology | Index Medicus
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6. Full Text Fibulin-4 E57K knock-in mice recapitulate cutaneous, vascular and skeletal defects of recessive cutis laxa 1B with both elastic fiber and collagen fibril abnormalities
by Igoucheva, Olga and Alexeev, Vitali and Halabi, Carmen M and Adams, Sheila M and Stoilov, Ivan and Sasaki, Takako and Arita, Machiko and Donahue, Adele and Mecham, Robert P and Birk, David E and Chu, Mon-Li
Journal of Biological Chemistry, ISSN 0021-9258, 08/2015, Volume 290, Issue 35, pp. 21443 - 21459
Background: Mutations in fibulin-4 cause autosomal recessive cutis laxa 1B, characterized by loose skin with vascular, lung, and skeletal abnormalities.... 
aneurysm | AORTIC-ANEURYSM | BIOCHEMISTRY & MOLECULAR BIOLOGY | collagen | skin | MATRIX PROTEINS | lysyl oxidase | LATENT TGF-BETA | elastin | TISSUE LOCALIZATION | ELASTOGENESIS | tendon | connective tissue | ARTERIAL TORTUOSITY | aorta | OSTEOGENESIS IMPERFECTA | forelimb | OF-THE-LITERATURE | GENDER-DIFFERENCES | extracellular matrix | Bone and Bones - pathology | Fibroblasts - enzymology | Protein Biosynthesis | Blood Vessels - abnormalities | Elastic Tissue - ultrastructure | Blood Vessels - pathology | Humans | Protein Multimerization | Molecular Sequence Data | Forelimb - diagnostic imaging | Forelimb - pathology | Forelimb - abnormalities | Base Sequence | HEK293 Cells | Extracellular Matrix Proteins - metabolism | Skin - pathology | Disease Models, Animal | Cutis Laxa - metabolism | Tendons - ultrastructure | Amino Acid Sequence | Extracellular Matrix Proteins - chemistry | Collagen Type I - metabolism | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Elastic Tissue - abnormalities | Fibroblasts - pathology | Collagen Type I - ultrastructure | Gene Knock-In Techniques | Cross-Linking Reagents - metabolism | Radiography | Elastic Tissue - pathology | Animals | Models, Biological | Tendons - pathology | Protein-Lysine 6-Oxidase - metabolism | Mutation | Bone and Bones - abnormalities | Cutis Laxa - pathology | Amino Acid Substitution | Tendons - abnormalities | Index Medicus | Glycobiology and Extracellular Matrices
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7. Full Text Myofibroblasts are increased in the lung parenchyma in asthma
by Boser, Stacey R and Mauad, Thais and De Araújo-Paulino, Bianca Bergamo and Mitchell, Ian and Shrestha, Grishma and Chiu, Andrea and Butt, John and Kelly, Margaret M and Caldini, Elia and James, Alan and Green, Francis H. Y
PLoS ONE, ISSN 1932-6203, 08/2017, Volume 12, Issue 8, pp. e0182378 - e0182378
Background Increased airway smooth muscle is observed in large and small airways in asthma. Semi-quantitative estimates suggest that cells containing alpha... 
MECHANICS | STIMULATION | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | ELASTIC RECOIL | TISSUE | DEATH | AIRWAY SMOOTH-MUSCLE | MAST-CELL | Myofibroblasts - pathology | Lung - pathology | Cell Count | Humans | Middle Aged | Actins - metabolism | Myocytes, Smooth Muscle - pathology | Male | Parenchymal Tissue - pathology | Young Adult | Adolescent | Adult | Female | Asthma - pathology | Myocytes, Smooth Muscle - metabolism | Physiological aspects | Fibroblasts | Development and progression | Genetic aspects | Research | Muscle proteins | Asthma | Lung | Acetylcysteine | Parenchyma | Smooth muscle | Inflammation | Forensic pathology | Medicine | Respiratory tract | Airway management | Actin | Alveoli | Chronic illnesses | Index Medicus
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8. Full Text Disseminated atrophic sarcoidosis with elastophagocytosis and elastic tissue loss
by Fancher, W and Braniecki, M and Guzman, G and Groth, J and Krunic, A
British Journal of Dermatology, ISSN 0007-0963, 04/2015, Volume 172, Issue 4, pp. 1154 - 1156
CUTANEOUS SARCOIDOSIS | HUMAN METALLOELASTASE MMP-12 | EXPRESSION | DERMATOLOGY | Macrophages - physiology | Elastic Tissue - pathology | Connective Tissue Diseases - pathology | Humans | Atrophy - pathology | Phagocytosis - physiology | Adult | Male | Matrix Metalloproteinase 12 - metabolism | Skin - pathology | Sarcoidosis - pathology
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9. Full Text Chronic sleep fragmentation induces endothelial dysfunction and structural vascular changes in mice
by Carreras, Alba and Zhang, Shelley X and Peris, Eduard and Qiao, Zhuanhong and Gileles-Hillel, Alex and Li, Richard C and Wang, Yang and Gozal, David
Sleep, ISSN 0161-8105, 11/2014, Volume 37, Issue 11, pp. 1817 - 1824
Study Objectives: Sleep fragmentation (SF) is a common occurrence and constitutes a major characteristic of obstructive sleep apnea (OSA). SF has been... 
Cell senescence | Sleep apnea | Endothelial function | Atherosclerosis | endothelial function | atherosclerosis | NONOBESE CHILDREN | OXIDATIVE STRESS | DISRUPTED SLEEP | ACCELERATED ATHEROSCLEROSIS | ELASTIC LAMINA DEFECTS | NEUROSCIENCES | CLINICAL NEUROLOGY | SYSTEMIC INFLAMMATION | INTERMITTENT HYPOXIA | INSULIN-RESISTANCE | CARDIOVASCULAR-DISEASE | sleep apnea | cell senescence | ADIPOSE-TISSUE | Blood Pressure | Sleep Apnea, Obstructive - physiopathology | Sleep Deprivation - pathology | Insulin-Like Growth Factor Binding Protein 3 - blood | Sleep Apnea, Obstructive - blood | Male | Sleep Apnea, Obstructive - complications | Sleep Apnea, Obstructive - pathology | Cellular Senescence | Interleukin-6 - blood | Plaque, Atherosclerotic - pathology | Eating | Heart Rate | Mice, Inbred C57BL | Endothelium, Vascular - physiopathology | Aorta - immunology | Chemokine CXCL1 - blood | Biomarkers - blood | Sleep Deprivation - complications | Aorta - pathology | Elastic Tissue - pathology | Sleep Deprivation - physiopathology | Animals | Endothelium, Vascular - pathology | Mice | Sleep Deprivation - blood | Index Medicus
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10. Full Text Increment of subcutaneous adipose tissue is associated with decrease of elastic fibres in the dermal layer
by Ezure, Tomonobu and Amano, Satoshi
Experimental Dermatology, ISSN 0906-6705, 12/2015, Volume 24, Issue 12, pp. 924 - 929
Obesity is a significant risk factor for various skin disorders, including pressure ulcer and delayed wound healing. We previously showed that increment of... 
subcutaneous adipose layer | dermal layer | elastin | MMP9 | adipocyte | Adipocyte | Subcutaneous adipose layer | Elastin | Dermal layer | Body Mass Index | Humans | Middle Aged | Elasticity | Obesity - physiopathology | Cell Enlargement | Elastic Tissue - physiopathology | 3T3-L1 Cells | Adipocytes - pathology | Subcutaneous Fat - pathology | Transcription Factor AP-1 - metabolism | MAP Kinase Signaling System | Obesity - pathology | Elastic Tissue - pathology | Matrix Metalloproteinase 9 - metabolism | Animals | Adipocytes - metabolism | Subcutaneous Fat - physiopathology | Adult | Female | Mice | Skin - physiopathology | Skin - pathology | Adipose tissues | Index Medicus
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11. Full Text Changes in Dermal Fibroblasts from Abcc6−/− Mice Are Present before and after the Onset of Ectopic Tissue Mineralization
by Boraldi, Federica and Bartolomeo, Angelica and Li, Qiaoli and Uitto, Jouni and Quaglino, Daniela
Journal of Investigative Dermatology, ISSN 0022-202X, 07/2014, Volume 134, Issue 7, pp. 1855 - 1861
Pseudoxanthoma elasticum (PXE), a rare genetic disease caused by mutations in the gene, is characterized by progressive calcification of elastic fibers in the... 
PATHOGENESIS | OXIDATIVE STRESS | OSTEOPONTIN | ALKALINE-PHOSPHATASE | VASCULAR CALCIFICATION | MOUSE MODEL | TRANSCRIPTION | PSEUDOXANTHOMA ELASTICUM PATIENTS | MUTATIONS | MATRIX-GLA-PROTEIN | DERMATOLOGY | Calcinosis - genetics | Pseudoxanthoma Elasticum - metabolism | Cell Proliferation | Reactive Oxygen Species - metabolism | Cells, Cultured | Extracellular Matrix - metabolism | Pseudoxanthoma Elasticum - pathology | Disease Progression | Aging - pathology | Mice, Knockout | Elastic Tissue - metabolism | Elastic Tissue - pathology | Animals | ATP-Binding Cassette Transporters - genetics | Pseudoxanthoma Elasticum - genetics | Fibroblasts - cytology | Mice | Calcinosis - metabolism | Calcinosis - pathology | Dermis - cytology | Extracellular Matrix - pathology | Aging - metabolism | Disease Models, Animal | Index Medicus
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12. Full Text Bilateral and Symmetrical Linear Plaques on the Hands: Challenge
by Wu, Zhouwei and Xu, Mingyuan and Chen, Zhuo and Shi, Weimin and Liu, Yeqiang
The American Journal of dermatopathology, ISSN 0193-1091, 02/2018, Volume 40, Issue 2, pp. e18 - E18
DERMATOLOGY | Elastic Tissue - pathology | Collagen | Humans | Middle Aged | Female | Keratosis - pathology | Hand - pathology | Index Medicus
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13. Full Text Matrix metalloproteinase promotes elastic fiber degradation in ligamentum flavum degeneration
by Sugimoto, Kazuki and Nakamura, Takayuki and Tokunaga, Takuya and Uehara, Yusuke and Okada, Tatsuya and Taniwaki, Takuya and Fujimoto, Toru and Mizuta, Hiroshi
PLoS ONE, ISSN 1932-6203, 08/2018, Volume 13, Issue 8, pp. e0200872 - e0200872
Ligamentum flavum (LF) hypertrophy in lumbar spinal canal stenosis (LSCS) is characterized by a loss of elastic fibers and fibrosis. Chronic inflammation is... 
SPINAL-CANAL STENOSIS | PATHOGENESIS | FIBROSIS | HYPERTROPHY | ANGIOGENESIS | PATHWAY | MULTIDISCIPLINARY SCIENCES | PATHOMECHANISM | DERMAL FIBROBLASTS | INTERLEUKIN-6 | EXPRESSION | Constriction, Pathologic - congenital | Constriction, Pathologic - pathology | Fibroblasts - enzymology | Humans | Middle Aged | Male | RNA, Messenger - metabolism | Young Adult | Ligamentum Flavum - surgery | Lumbar Vertebrae - surgery | Matrix Metalloproteinase 9 - metabolism | Aged, 80 and over | Lumbar Vertebrae - abnormalities | Adult | Female | Interleukin-6 - administration & dosage | Interleukin-6 - metabolism | STAT3 Transcription Factor - metabolism | Signal Transduction | Matrix Metalloproteinase 2 - metabolism | Cells, Cultured | Gene Expression Regulation | Fibroblasts - pathology | Ligamentum Flavum - enzymology | Elastic Tissue - pathology | Constriction, Pathologic - surgery | Fibroblasts - drug effects | Aged | Lumbar Vertebrae - pathology | Constriction, Pathologic - enzymology | STAT3 Transcription Factor - antagonists & inhibitors | Elastic Tissue - enzymology | Ligamentum Flavum - pathology | Lumbar Vertebrae - enzymology | Care and treatment | Inflammation | Research | Interleukins | Correlation | Nuclear magnetic resonance--NMR | Transcription | Disease | Interleukin | Stenosis | Matrix metalloproteinase | Kinases | Males | Bone surgery | Fibers | Stains & staining | Degradation | Interleukin 6 | Proteins | Angiogenesis | Signal transduction | Surgery | Fibroblasts | Life sciences | Degeneration | Metalloproteinase | Growth factors | Cytokines | Secretion | Elastin | Stat3 protein | Gene expression | Patients | Gelatinase B | Gelatinase A | Signaling | Orthopedics | Collagen | Fibrosis | Hypertrophy | Index Medicus | Nuclear magnetic resonance | NMR
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14. Full Text Histopathologic findings in ascending aortas from individuals with Loeys-Dietz Syndrome (LDS)
by Maleszewski, Joseph J and Miller, Dylan V and Lu, Jie and Dietz, Harry C and Halushka, Marc K
American Journal of Surgical Pathology, ISSN 0147-5185, 02/2009, Volume 33, Issue 2, pp. 194 - 201
Loeys-Dietz syndrome (LDS) is an autosomal dominant connective tissue disorder resulting from genetic Mutations in the transforming growth factor beta... 
Ascending aorta | Medial degeneration | Pathology | Loeys-Dietz Syndrome | Transforming growth factor-b | SURGERY | pathology | medial degeneration | RECEPTOR | transforming growth factor-beta | PATHOGENESIS | Loeys-Dietz syndrome | MARFAN-SYNDROME | MOUSE MODEL | MUTATIONS | ANEURYSM | TGF-BETA ACTIVATION | ascending aorta | Tunica Media - pathology | Receptors, Transforming Growth Factor beta - genetics | Humans | Middle Aged | Child, Preschool | Infant | Male | Aorta - metabolism | Fibrosis - metabolism | DNA Mutational Analysis | Adult | Female | Child | Protein-Serine-Threonine Kinases - genetics | Connective Tissue Diseases - metabolism | Syndrome | Aorta - pathology | Collagen - metabolism | Elastic Tissue - pathology | Connective Tissue Diseases - pathology | Adolescent | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Connective Tissue Diseases - genetics | Fibrosis - pathology | Index Medicus
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15. Full Text Conditional overexpression of connective tissue growth factor disrupts postnatal lung development
by Wu, Shu and Platteau, Astrid and Chen, Shaoyi and McNamara, George and Whitsett, Jeffrey and Bancalari, Eduardo
American Journal of Respiratory Cell and Molecular Biology, ISSN 1044-1549, 05/2010, Volume 42, Issue 5, pp. 552 - 563
Connective tissue growth factor (CTGF) is a member of an emerging family of immediate-early gene products that coordinates complex biological processes during... 
Development | Transgenic | Connective tissue growth factor | Postnatal lung | development | INTEGRIN-LINKED KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | MOUSE | MESENCHYMAL TRANSITION | NEWBORN MICE | CELL BIOLOGY | connective tissue growth factor | BRONCHOPULMONARY DYSPLASIA | MORPHOGENESIS | RESPIRATORY SYSTEM | TRANSFORMING GROWTH-FACTOR-BETA-1 | ALVEOLARIZATION | transgenic | EXPRESSION | postnatal lung | PULMONARY-FIBROSIS | Cell Proliferation | Lung - enzymology | Elastic Tissue - metabolism | Pulmonary Alveoli - metabolism | Cell Differentiation