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Scientific Reports, ISSN 2045-2322, 2013, Volume 3, Issue 1, pp. 2767 - 2767
Myocardial ischemia, while causing cardiomyocyte injury, can activate innate protective processes, enhancing myocardial tolerance to ischemia. Such processes... 
BETA-KLOTHO | SURVIVAL | ENDOTHELIAL PROGENITOR CELLS | ARTERIAL ELASTIC LAMINAE | PPAR-ALPHA | ACTIVATION | MULTIDISCIPLINARY SCIENCES | REPERFUSION INJURY | BONE-MARROW | C-KIT | AKT | Up-Regulation | Liver - pathology | Phosphorylation | Cardiotonic Agents - metabolism | Adipose Tissue - physiopathology | Caspase 3 - metabolism | Receptor, Fibroblast Growth Factor, Type 1 - metabolism | Glucuronidase | Liver - physiopathology | Phosphatidylinositol 3-Kinases | Myocardial Reperfusion Injury - enzymology | Myocytes, Cardiac - enzymology | Adipose Tissue - metabolism | Fibroblast Growth Factors - metabolism | Myocardial Reperfusion Injury - pathology | Endocrine System - metabolism | Myocardial Ischemia - physiopathology | bcl-Associated Death Protein - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Heart Function Tests | Heart Ventricles - pathology | Signal Transduction | Liver - metabolism | Adipose Tissue - pathology | Gene Silencing | Myocardial Ischemia - prevention & control | Myocardial Ischemia - enzymology | Myocardial Reperfusion Injury - physiopathology | Endocrine System - pathology | Myocardial Ischemia - pathology | Myocytes, Cardiac - pathology | Animals | Heart Ventricles - physiopathology | Protein Binding | Heart Ventricles - metabolism | Mice | Hemodynamics | RNA, Small Interfering - metabolism | Myocardial infarction | AKT1 protein | Adipose tissue | Liver | Caspase | Myocardial ischemia | Cardiomyocytes | Adipocytes | Caspase-3 | 1-Phosphatidylinositol 3-kinase | Membrane proteins | Reperfusion | Ischemia | Cell death | Klotho protein | Rodents | Myocardium | Fibroblast growth factor receptor 1 | Index Medicus
Journal Article
1980, Frontiers of matrix biology, ISBN 9783805530781, Volume 8, viii, 230
Book
Science, ISSN 0036-8075, 4/2006, Volume 312, Issue 5770, pp. 117 - 121
Aortic aneurysm and dissection are manifestations of Marfan syndrome (MFS), a disorder caused by mutations in the gene that encodes fibrillin-1. Selected... 
Airspace | Receptors | Architecture | Dilatation | Root growth | Placebos | Reports | Mice | Aortic aneurysm | Elastic tissue | Marfan syndrome | PATHOGENESIS | GROWTH-FACTOR-BETA | ACTIVATION | FIBRILLIN | MULTIDISCIPLINARY SCIENCES | RATS | RECEPTOR | SMOOTH-MUSCLE-CELLS | BLOCKADE | CONTRIBUTES | DILATATION | Angiotensin II Type 1 Receptor Blockers - therapeutic use | Propranolol - therapeutic use | Neutralization Tests | Aortic Aneurysm - etiology | Lung Diseases - drug therapy | Marfan Syndrome - drug therapy | Propranolol - administration & dosage | Antibodies - immunology | Transforming Growth Factor beta - antagonists & inhibitors | Female | Marfan Syndrome - complications | Microfilament Proteins - genetics | Adrenergic beta-Antagonists - administration & dosage | Adrenergic beta-Antagonists - therapeutic use | Angiotensin II Type 1 Receptor Blockers - administration & dosage | Aortic Aneurysm - prevention & control | Disease Models, Animal | Transforming Growth Factor beta - immunology | Fibrillin-1 | Lung - pathology | Pulmonary Alveoli - pathology | Signal Transduction | Fibrillins | Pregnancy Complications - drug therapy | Aorta - pathology | Pregnancy | Elastic Tissue - pathology | Animals | Receptor, Angiotensin, Type 1 - metabolism | Losartan - therapeutic use | Marfan Syndrome - metabolism | Marfan Syndrome - pathology | Losartan - administration & dosage | Lung Diseases - pathology | Mutation | Transforming Growth Factor beta - metabolism | Complications and side effects | Aortic aneurysms | Genetic aspects | Research | Risk factors | Signal transduction | Pharmacology | Models | Genetic disorders | Antagonist drugs | Rodents | Index Medicus
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2015, Volume 290, Issue 35, pp. 21443 - 21459
Background: Mutations in fibulin-4 cause autosomal recessive cutis laxa 1B, characterized by loose skin with vascular, lung, and skeletal abnormalities.... 
aneurysm | AORTIC-ANEURYSM | BIOCHEMISTRY & MOLECULAR BIOLOGY | collagen | skin | MATRIX PROTEINS | lysyl oxidase | LATENT TGF-BETA | elastin | TISSUE LOCALIZATION | ELASTOGENESIS | tendon | connective tissue | ARTERIAL TORTUOSITY | aorta | OSTEOGENESIS IMPERFECTA | forelimb | OF-THE-LITERATURE | GENDER-DIFFERENCES | extracellular matrix | Bone and Bones - pathology | Fibroblasts - enzymology | Protein Biosynthesis | Blood Vessels - abnormalities | Elastic Tissue - ultrastructure | Blood Vessels - pathology | Humans | Protein Multimerization | Molecular Sequence Data | Forelimb - diagnostic imaging | Forelimb - pathology | Forelimb - abnormalities | Base Sequence | HEK293 Cells | Extracellular Matrix Proteins - metabolism | Skin - pathology | Disease Models, Animal | Cutis Laxa - metabolism | Tendons - ultrastructure | Amino Acid Sequence | Extracellular Matrix Proteins - chemistry | Collagen Type I - metabolism | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Elastic Tissue - abnormalities | Fibroblasts - pathology | Collagen Type I - ultrastructure | Gene Knock-In Techniques | Cross-Linking Reagents - metabolism | Radiography | Elastic Tissue - pathology | Animals | Models, Biological | Tendons - pathology | Protein-Lysine 6-Oxidase - metabolism | Mutation | Bone and Bones - abnormalities | Cutis Laxa - pathology | Amino Acid Substitution | Tendons - abnormalities | Index Medicus | Glycobiology and Extracellular Matrices
Journal Article
Sleep, ISSN 0161-8105, 11/2014, Volume 37, Issue 11, pp. 1817 - 1824
Journal Article
The American Journal of dermatopathology, ISSN 0193-1091, 02/2018, Volume 40, Issue 2, pp. e18 - E18
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2018, Volume 13, Issue 8, pp. e0200872 - e0200872
Ligamentum flavum (LF) hypertrophy in lumbar spinal canal stenosis (LSCS) is characterized by a loss of elastic fibers and fibrosis. Chronic inflammation is... 
SPINAL-CANAL STENOSIS | PATHOGENESIS | FIBROSIS | HYPERTROPHY | ANGIOGENESIS | PATHWAY | MULTIDISCIPLINARY SCIENCES | PATHOMECHANISM | DERMAL FIBROBLASTS | INTERLEUKIN-6 | EXPRESSION | Constriction, Pathologic - congenital | Constriction, Pathologic - pathology | Fibroblasts - enzymology | Humans | Middle Aged | Male | RNA, Messenger - metabolism | Young Adult | Ligamentum Flavum - surgery | Lumbar Vertebrae - surgery | Matrix Metalloproteinase 9 - metabolism | Aged, 80 and over | Lumbar Vertebrae - abnormalities | Adult | Female | Interleukin-6 - administration & dosage | Interleukin-6 - metabolism | STAT3 Transcription Factor - metabolism | Signal Transduction | Matrix Metalloproteinase 2 - metabolism | Cells, Cultured | Gene Expression Regulation | Fibroblasts - pathology | Ligamentum Flavum - enzymology | Elastic Tissue - pathology | Constriction, Pathologic - surgery | Fibroblasts - drug effects | Aged | Lumbar Vertebrae - pathology | Constriction, Pathologic - enzymology | STAT3 Transcription Factor - antagonists & inhibitors | Elastic Tissue - enzymology | Ligamentum Flavum - pathology | Lumbar Vertebrae - enzymology | Care and treatment | Inflammation | Research | Interleukins | Correlation | Nuclear magnetic resonance--NMR | Transcription | Disease | Interleukin | Stenosis | Matrix metalloproteinase | Kinases | Males | Bone surgery | Fibers | Stains & staining | Degradation | Interleukin 6 | Proteins | Angiogenesis | Signal transduction | Surgery | Fibroblasts | Life sciences | Degeneration | Metalloproteinase | Growth factors | Cytokines | Secretion | Elastin | Stat3 protein | Gene expression | Patients | Gelatinase B | Gelatinase A | Signaling | Orthopedics | Collagen | Fibrosis | Hypertrophy | Index Medicus | Nuclear magnetic resonance | NMR
Journal Article