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The Journal of neuroscience, ISSN 1529-2401, 03/2012, Volume 32, Issue 12, pp. 4133 - 4144
Potassium (K+) channels are essential to neuronal signaling and survival. Here we show that these proteins are targets of reactive oxygen species in mammalian brain and that their oxidation contributes to neuropathy. Thus, the KCNB1 (Kv2.1... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Fluoresceins - pharmacology | Membrane Potentials - genetics | Electric Stimulation | Age Factors | Cricetulus | Apoptosis - drug effects | Disulfides - toxicity | Embryo, Mammalian | Peptide Fragments - toxicity | 2,2'-Dipyridyl - analogs & derivatives | Humans | Oxidative Stress - physiology | Apoptosis - genetics | Male | 2,2'-Dipyridyl - toxicity | Propanols - pharmacology | Shab Potassium Channels - physiology | Alzheimer Disease - pathology | Cysteine - genetics | Transfection | Alanine - genetics | Neurons - physiology | Oxidation-Reduction - drug effects | Female | Neurons - drug effects | Disease Models, Animal | Shab Potassium Channels - genetics | Alzheimer Disease - physiopathology | Brain - cytology | Cricetinae | Amyloid beta-Peptides - toxicity | Animals, Genetically Modified | Cells, Cultured | Hydrogen Peroxide - pharmacology | Oxidants - toxicity | Oxidative Stress - genetics | Presenilin-1 - genetics | Membrane Potentials - physiology | Caenorhabditis elegans | Patch-Clamp Techniques | Amyloid beta-Protein Precursor - genetics | Animals | Analysis of Variance | Mice | Apoptosis - physiology | Oxidative Stress - drug effects | Alzheimer Disease - genetics | Mass Spectrometry - methods | Index Medicus
Journal Article
Journal Article
Autophagy, ISSN 1554-8627, 09/2011, Volume 7, Issue 9, pp. 1045 - 1051
Multiple stress pathways result in the induction of autophagy and apoptosis. Current methods (e.g., protein gel blot, microscopy) do not offer quantitative... 
Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Autophagy | Multispectral imaging cytometry | Influenza | New methodology | Apoptosis | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Leukocytes, Mononuclear - metabolism | Image Cytometry - methods | Apoptosis - drug effects | Apoptosis - radiation effects | Autophagy - radiation effects | Microtubule-Associated Proteins - metabolism | Humans | Autophagy - drug effects | Lysosomes - radiation effects | Chloroquine - pharmacology | Lysosomes - metabolism | Ultraviolet Rays | Time Factors | Leukocytes, Mononuclear - radiation effects | Signal Transduction - radiation effects | Fibroblasts - metabolism | Lysosomes - drug effects | Leukocytes, Mononuclear - drug effects | Animals | Fibroblasts - radiation effects | Signal Transduction - drug effects | Embryo, Mammalian - cytology | Fibroblasts - drug effects | Leukocytes, Mononuclear - cytology | Fibroblasts - cytology | Mice | Index Medicus | Autophagy/radiation effects | Signal Transduction/radiation effects | Image Cytometry/methods | Autophagy/drug effects | Life Sciences | Lysosomes/drug effects | Immunology | Fibroblasts/metabolism | Leukocytes, Mononuclear/radiation effects | Fibroblasts/drug effects | Leukocytes, Mononuclear/metabolism | Apoptosis/radiation effects | Leukocytes, Mononuclear/drug effects | Fibroblasts/cytology | Apoptosis/drug effects | Fibroblasts/radiation effects | Lysosomes/metabolism | Leukocytes, Mononuclear/cytology | Signal Transduction/drug effects | Chloroquine/pharmacology | Embryo, Mammalian/cytology | Lysosomes/radiation effects | Microtubule-Associated Proteins/metabolism
Journal Article
Autophagy, ISSN 1554-8635, 10/2014, Volume 8, Issue 2, pp. 187 - 199
Neuroepithelial cells undergoing differentiation efficiently remodel their cytoskeleton and shape in an energy-consuming process. The capacity of autophagy to... 
autophagy | differentiation | Ambra1 | neurogenesis | neural progenitors | neuritogenesis | Atg5 | Binding | Proteins | Landes | Calcium | Bioscience | Biology | Cell | Cycle | Cancer | Organogenesis | Neuritogenesis | Differentiation | Autophagy | Neural progenitors | Neurogenesis | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Embryo, Mammalian - drug effects | Embryonic Stem Cells - metabolism | Embryonic Stem Cells - cytology | Pyruvates - pharmacology | Microtubule-Associated Proteins - metabolism | Olfactory Bulb - metabolism | Spheroids, Cellular - cytology | Neural Stem Cells - cytology | Olfactory Bulb - embryology | Autophagy - drug effects | Embryo, Mammalian - metabolism | Mice, Mutant Strains | Haploinsufficiency - drug effects | Neurogenesis - drug effects | Spheroids, Cellular - drug effects | Microtubule-Associated Proteins - deficiency | Neurites - drug effects | Olfactory Bulb - cytology | Adenine - analogs & derivatives | Mice, Inbred C57BL | Spheroids, Cellular - metabolism | Neural Stem Cells - drug effects | Adenine - pharmacology | Neurites - metabolism | Animals | Autophagy-Related Protein 5 | Cell Differentiation - drug effects | Embryo, Mammalian - cytology | Mice | Adaptor Proteins, Signal Transducing - metabolism | Neural Stem Cells - metabolism | Olfactory Bulb - drug effects | Index Medicus
Journal Article
Development (Cambridge), ISSN 1477-9129, 01/2008, Volume 135, Issue 3, pp. 579 - 588
Overexpression of zinc finger E-box binding homeobox transcription factor 1 (Zeb1) in cancer leads to epithelial-to-mesenchymal transition (EMT) and increased... 
Zeb1 | Epithelial-mesenchymal transition | Senescence | Transcription | Life Sciences & Biomedicine | Developmental Biology | Science & Technology | Embryo, Mammalian - drug effects | Brain - embryology | Cadherins - metabolism | Vimentin - metabolism | Homeodomain Proteins - metabolism | Mesoderm - drug effects | Cellular Senescence - drug effects | Cartilage - drug effects | Stem Cells - cytology | Mesoderm - cytology | Epithelium - embryology | Eye - embryology | Kruppel-Like Transcription Factors - metabolism | Mice, Mutant Strains | Vimentin - genetics | Cartilage - cytology | Eye - drug effects | Cadherins - genetics | Palate - cytology | Palate - drug effects | Repressor Proteins - metabolism | Epithelium - drug effects | Brain - cytology | Cartilage - embryology | Mesoderm - embryology | Gene Expression Regulation, Developmental - drug effects | Eye - cytology | Gene Dosage | Mutation - genetics | Homeodomain Proteins - genetics | Palate - embryology | Brain - drug effects | Transforming Growth Factor beta - pharmacology | Animals | Embryo, Mammalian - embryology | Embryo, Mammalian - cytology | Fibroblasts - drug effects | Cyclin-Dependent Kinase Inhibitor p16 - metabolism | Stem Cells - drug effects | Cell Proliferation - drug effects | Fibroblasts - cytology | Mice | Cyclin-Dependent Kinase Inhibitor p15 - metabolism | Kruppel-Like Transcription Factors - genetics | Zinc Finger E-box-Binding Homeobox 1 | Index Medicus
Journal Article
Toxicology letters, ISSN 0378-4274, 04/2018, Volume 286, pp. 10 - 21
•Addition of toxicokinetic data improves the predictivity of developmental toxicity.•Biomarkers of developmental toxicity advance the comparison of in vitro... 
Biomarkers | Embryotoxicity | Azoles | Whole embryo culture | Stem cell test | Placental transfer | Humans | Teratogens/toxicity | Toxicity Tests/methods | Embryo Culture Techniques | Dose-Response Relationship, Drug | Mouse Embryonic Stem Cells/drug effects | Biological Transport | Biological Assay | Female | Alcohol Oxidoreductases/genetics | Cell Line | Cell Differentiation/drug effects | Embryo, Mammalian/drug effects | Reproducibility of Results | Mixed Function Oxygenases/genetics | Retinoic Acid 4-Hydroxylase/genetics | Risk Assessment | Azoles/toxicity | Myocytes, Cardiac/drug effects | Gene Expression Regulation, Developmental/drug effects | Sterol 14-Demethylase/genetics | Placenta/drug effects | Pregnancy | Animals | Mice | Kinetics | Life Sciences & Biomedicine | Toxicology | Science & Technology | Embryo, Mammalian - drug effects | Teratogens - toxicity | Sterol 14-Demethylase - genetics | Mixed Function Oxygenases - metabolism | Retinoic Acid 4-Hydroxylase - metabolism | Retinoic Acid 4-Hydroxylase - genetics | Embryo, Mammalian - metabolism | Mouse Embryonic Stem Cells - drug effects | Mouse Embryonic Stem Cells - metabolism | Alcohol Oxidoreductases - genetics | Toxicity Tests - methods | Azoles - toxicity | Mouse Embryonic Stem Cells - pathology | Embryo, Mammalian - pathology | Gene Expression Regulation, Developmental - drug effects | Alcohol Oxidoreductases - metabolism | Placenta - drug effects | Placenta - metabolism | Myocytes, Cardiac - pathology | Myocytes, Cardiac - drug effects | Cell Differentiation - drug effects | Placenta - pathology | Myocytes, Cardiac - metabolism | Sterol 14-Demethylase - metabolism | Mixed Function Oxygenases - genetics | Index Medicus
Journal Article
Nature genetics, ISSN 1546-1718, 07/2009, Volume 41, Issue 8, pp. 891 - 898
...The accumulation of DNA damage can have important consequences that limit the lifespan of mammalian organisms, such as aging or cancer. Concerning aging, one... 
Life Sciences & Biomedicine | Genetics & Heredity | Science & Technology | Abnormalities, Multiple - pathology | Embryo, Mammalian - drug effects | Protein-Serine-Threonine Kinases - deficiency | Fibroblasts - enzymology | Apoptosis - drug effects | DNA Replication - drug effects | Humans | Aging - drug effects | Brain - enzymology | Embryo, Mammalian - metabolism | DNA-Binding Proteins - metabolism | Aging - genetics | Progeria - embryology | DNA-Activated Protein Kinase - metabolism | Progeria - pathology | Embryo, Mammalian - enzymology | Stress, Physiological - drug effects | Abnormalities, Multiple - genetics | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | DNA Repair - drug effects | Embryo, Mammalian - pathology | Progeria - enzymology | Cell Cycle Proteins - metabolism | Tumor Suppressor Protein p53 - metabolism | Nuclear Proteins - metabolism | Ataxia Telangiectasia Mutated Proteins | Fibroblasts - pathology | Tumor Suppressor Protein p53 - deficiency | Syndrome | Aging - pathology | Phenotype | Animals | Fibroblasts - drug effects | Alleles | Abnormalities, Multiple - enzymology | Brain - pathology | Mice | Protein Kinase Inhibitors - pharmacology | DNA Damage | Embryonic development | Usage | Control | Animal models in research | DNA damage | Causes of | Physiological aspects | Genetic aspects | Research | Proteins | Dwarfism | Studies | Genotype & phenotype | Cell cycle | Kinases | Molecular biology | Cancer | Index Medicus
Journal Article