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2015, 2015, Advances in experimental medicine and biology, ISBN 9783319188812, Volume 868, x, 228
"Novel topics compiled in this one-of-a-kind volume provide tantalizing hints for clinical application through future translational research. Only recently... 
Spermatozoa | Motility | Semen | abnormalities | Disorders | Aneuploidy | Transplantation | Embryo Implantation | Immunological aspects | genetics | Infertility, Male | Human reproduction | immunology | Human embryo | Human genetics | Life Sciences | Cytogenetics | Embryology | Reproductive Medicine | Pregnancy | Generative organs, Male | Obstetrical endocrinology
Book
Scientific Reports, ISSN 2045-2322, 12/2018, Volume 8, Issue 1, pp. 5169 - 13
Adult Cebpb KO mice incisors present amelogenin-positive epithelium pearls, enamel and dentin allopathic hyperplasia, fewer Sox2-positive cells in labial... 
EPITHELIAL-MESENCHYMAL TRANSITION | DECORIN | TARGETED DISRUPTION | MULTIDISCIPLINARY SCIENCES | BIGLYCAN | GENE-EXPRESSION | CBFA1 | MICE | DIFFERENTIATION | CELL FATE | RUNX2 | Dentin | Cbfa-1 protein | Decorin | Mesenchyme | Enamel | Hyperplasia | Teeth | Stem cell transplantation | Epithelium | Pearls | Dental enamel | Ameloblasts | Rodents | Stem cells | Incisors | Polarity | Supernumerary | Cementum | Dental pulp
Journal Article
Nature Genetics, ISSN 1061-4036, 06/2011, Volume 43, Issue 6, pp. 595 - 600
Journal Article
Nature Structural and Molecular Biology, ISSN 1545-9993, 2010, Volume 17, Issue 6, pp. 688 - 695
Germ-line mutations in breast cancer 1, early onset (BRCA1) result in predisposition to breast and ovarian cancer. BRCA1-mutated tumors show genomic... 
STEM-CELLS | GENE BRCA1 | DNA-DAMAGE RESPONSE | BIOCHEMISTRY & MOLECULAR BIOLOGY | CONDITIONAL MOUSE MODEL | DOUBLE-STRAND BREAKS | OVARIAN-CANCER | CELL BIOLOGY | GENOMIC INSTABILITY | MAMMARY-TUMORS | BIOPHYSICS | TUMOR-SUPPRESSOR | CELL-CYCLE | Embryonic Stem Cells - metabolism | Cell Proliferation | Embryonic Stem Cells - cytology | Humans | Breast Neoplasms - metabolism | Intracellular Signaling Peptides and Proteins - deficiency | Gene Deletion | Genes, BRCA2 | Female | Genes, BRCA1 | Tumor Cells, Cultured | BRCA1 Protein - deficiency | Intracellular Signaling Peptides and Proteins - genetics | DNA-Binding Proteins | Intracellular Signaling Peptides and Proteins - antagonists & inhibitors | Chromosomal Proteins, Non-Histone | Breast Neoplasms - drug therapy | Mice, Knockout | BRCA1 Protein - genetics | Drug Resistance, Neoplasm - genetics | Animals | Apoptosis Regulatory Proteins | Breast Neoplasms - genetics | Cell Cycle | Breast Neoplasms - pathology | DNA Repair | BRCA2 Protein - deficiency | Mice | Mutagenesis, Insertional | DNA Damage | Mutation | Tumor Suppressor p53-Binding Protein 1 | Drug Resistance, Neoplasm - physiology | BRCA2 Protein - genetics | BRCA mutations | DNA damage | Physiological aspects | Breast cancer | Genetic aspects | Research | Health aspects | DNA repair | Risk factors | Molecular biology | Ovarian cancer
Journal Article
Science, ISSN 0036-8075, 9/2012, Volume 337, Issue 6101, pp. 1541 - 1546
De-ubiquitinating enzyme BAP1 is mutated in a hereditary cancer syndrome with increased risk of mesothelioma and uveal melanoma. Somatic BAP1 mutations occur... 
Spleen | Myeloid cells | Genes | DNA | REPORTS | Stem cells | Neutrophils | Bone marrow | Bone marrow cells | Genetic mutation | Tumors | ASXL1 | MULTIDISCIPLINARY SCIENCES | MOUSE | GENE-EXPRESSION | MUTATIONS | LEUKEMIA | UBIQUITIN HYDROLASE | PREDISPOSE | MYELODYSPLASTIC SYNDROMES | Myeloid Cells - cytology | Humans | Leukemia, Myelomonocytic, Chronic - genetics | Myeloid Cells - physiology | Host Cell Factor C1 - metabolism | Leukemia, Myelomonocytic, Chronic - metabolism | Chromatin Immunoprecipitation | Hematopoiesis | Gene Deletion | Tumor Suppressor Proteins - chemistry | Tumor Suppressor Proteins - genetics | Bone Marrow Transplantation | Myeloid Progenitor Cells - cytology | Ubiquitin Thiolesterase - metabolism | Leukemia, Myelomonocytic, Chronic - pathology | Genes, Tumor Suppressor | Repressor Proteins - metabolism | Promoter Regions, Genetic | Myelodysplastic Syndromes - metabolism | Tumor Suppressor Proteins - metabolism | Gene Expression Regulation | Embryonic Development | Ubiquitin Thiolesterase - genetics | N-Acetylglucosaminyltransferases - metabolism | Gene Knock-In Techniques | Mice, Knockout | Animals | Myeloid Progenitor Cells - physiology | Cell Transformation, Neoplastic | Mice | Myelodysplastic Syndromes - genetics | Myelodysplastic Syndromes - pathology | Ubiquitin Thiolesterase - chemistry | Gene silencing | Tumor suppressor genes | Genetic engineering | Research | Properties | Observations | Enzymes | Mutation | Gene expression | Molecular biology | Leukemia
Journal Article
Journal of Immunology, ISSN 0022-1767, 04/2013, Volume 190, Issue 7, pp. 3639 - 3647
Journal Article