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PLoS Pathogens, ISSN 1553-7366, 04/2012, Volume 8, Issue 4, p. e1002655
CLEC5A/MDL-1, a member of the myeloid C-type lectin family expressed on macrophages and neutrophils, is critical for dengue virus (DV)-induced hemorrhagic... 
ACTIVATION | MICROBIOLOGY | MYELOID CELLS | DAP12-ASSOCIATING LECTIN (MDL)-1 | DENGUE | MDL-1 CLEC5A | VIROLOGY | MICROGLIA | NEURONAL DEATH | CENTRAL-NERVOUS-SYSTEM | MICE | PARASITOLOGY | WEST-NILE | Lectins, C-Type - immunology | Encephalitis, Japanese - genetics | Lectins, C-Type - genetics | Lectins, C-Type - metabolism | STAT1 Transcription Factor - metabolism | Macrophages - virology | Astrocytes - immunology | Encephalitis Virus, Japanese - metabolism | Adaptor Proteins, Signal Transducing - immunology | Blood-Brain Barrier - virology | Neurons - metabolism | Encephalitis, Japanese - metabolism | Cytokines - genetics | Astrocytes - virology | Macrophages - immunology | Cytokines - immunology | Neurons - virology | Blood-Brain Barrier - immunology | Immunity, Cellular - genetics | Cytokines - metabolism | Immunity, Humoral - genetics | Neurons - immunology | Receptors, Cell Surface - metabolism | Inflammation | STAT1 Transcription Factor - genetics | Receptors, Cell Surface - immunology | STAT1 Transcription Factor - immunology | Blood-Brain Barrier - metabolism | Encephalitis Virus, Japanese - genetics | Mice, Knockout | Macrophages - metabolism | Animals | Encephalitis, Japanese - immunology | Adaptor Proteins, Signal Transducing - genetics | Mice | Adaptor Proteins, Signal Transducing - metabolism | Antibodies, Monoclonal, Murine-Derived - pharmacology | Encephalitis Virus, Japanese - immunology | Astrocytes - metabolism | Receptors, Cell Surface - genetics | Encephalitis | Physiological aspects | Development and progression | Virulence (Microbiology) | Research | Macrophages | Health aspects | Flow cytometry | Phosphorylation | Cytokines | Pathogenesis | Dengue fever | Viruses | Infections | Experiments | Proteins | Studies | Brain research | Rodents | Labeling
Journal Article
Acta Crystallographica Section F, ISSN 2053-230X, 03/2017, Volume 73, Issue 3, pp. 116 - 122
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2012, Volume 7, Issue 2, p. e32270
Background: Japanese Encephalitis virus (JEV) is a common cause of acute and epidemic viral encephalitis. JEV infection is associated with microglial... 
OXIDATIVE STRESS | MICROGLIAL ACTIVATION | CASPASE-1 | INTERLEUKIN-1-BETA | MULTIDISCIPLINARY SCIENCES | WEST NILE VIRUS | IL-1-BETA | INFLUENZA-VIRUS | HOST RESPONSE | EXPRESSION | NALP3 INFLAMMASOME | Carrier Proteins - physiology | Protein Structure, Tertiary | Cell Line | Microglia - metabolism | Reactive Oxygen Species | Cytokines - metabolism | Encephalitis, Japanese - virology | Signal Transduction | NLR Family, Pyrin Domain-Containing 3 Protein | Encephalitis, Japanese - genetics | Caspase 1 - metabolism | Inflammation | Carrier Proteins - genetics | Animals | Encephalitis Virus, Japanese - metabolism | Interleukin-1beta - metabolism | Mice | Mice, Inbred BALB C | Interleukin-18 - metabolism | Virus diseases | Medical research | Encephalitis | Enzymes | Interleukins | Analysis | Medicine, Experimental | Health aspects | Epidemics | Oxidative stress | Reactive oxygen species | Animal models | Transcription factors | Interleukin | Viruses | Infections | Activation | Neuroblastoma | Caspase-1 | Proteins | Signal transduction | Receptors | E coli | Rodents | Interleukin 1 | Tumor necrosis factor-TNF | Vector-borne diseases | Efflux | Cytokines | Maturation | Caspase | Hazards | Pattern recognition | Gene expression | Microglia | Interleukin 18 | Pathology | Signaling | Brain research | Influenza | Sepsis | Potassium | Viral infections | Apoptosis
Journal Article
Journal of General Virology, ISSN 0022-1317, 04/2015, Volume 96, Issue 4, pp. 793 - 803
Japanese encephalitis virus (JEV), one of encephalitic flaviviruses, is naturally transmitted by mosquitoes. During infection, JEV generally enters host cells... 
Cell Line | Cricetinae | Encephalitis, Japanese - virology | Molecular Sequence Data | Clathrin - metabolism | Virus Internalization | Endocytosis - physiology | HSP70 Heat-Shock Proteins - metabolism | Culicidae - virology | Animals | Encephalitis Virus, Japanese - metabolism | Protein Isoforms | Encephalitis, Japanese - metabolism
Journal Article
Journal Article
European Journal of Immunology, ISSN 0014-2980, 05/2014, Volume 44, Issue 5, pp. 1363 - 1374
The mechanisms underlying Japanese encephalitis virus (JEV) pathogenesis need to be thoroughly explored to delineate therapeutic approaches. It is believed... 
Regulatory T cells | Host–pathogen interactions | Infectious diseases | Immune evasion | PD‐L1 | PD-L1 | Regulatory T cells | Host-pathogen interactions | ACTIVATION | CD40 | PROTECTION | C-REL | INDUCTION | IMMUNOLOGY | CEREBROSPINAL-FLUID | DC-SIGN | Regulatory Tcells | INFECTION | MICE | DIFFERENTIATION | T-Lymphocytes, Regulatory - metabolism | Cell Proliferation | Dendritic Cells - immunology | Humans | Dendritic Cells - pathology | Encephalitis, Japanese - genetics | Male | Monocytes - metabolism | Monocytes - immunology | T-Lymphocytes, Regulatory - pathology | T-Lymphocytes, Regulatory - immunology | Encephalitis Virus, Japanese - metabolism | Immune Evasion - genetics | Monocytes - pathology | Female | Encephalitis, Japanese - metabolism | Dendritic Cells - virology | Dendritic Cells - metabolism | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | B7-H1 Antigen - genetics | B7-H1 Antigen - immunology | Encephalitis Virus, Japanese - genetics | Monocytes - virology | Antigens, Differentiation - biosynthesis | Encephalitis, Japanese - pathology | B7-H1 Antigen - biosynthesis | Encephalitis, Japanese - immunology | Antigens, Differentiation - genetics | Immune Evasion - immunology | Encephalitis Virus, Japanese - immunology | Antigens, Differentiation - immunology | Dendritic cells | T cells | Health aspects | Analysis | Japanese encephalitis | Encephalitis | Vaccines | Immune system
Journal Article
Future Microbiology, ISSN 1746-0913, 10/2016, Volume 11, Issue 10, pp. 1227 - 1248
Aim: To investigate the detailed mechanism of Japanese encephalitis virus (JEV) cell entry. Materials & methods: Utilize a siRNA library targeting cellular... 
Caveolin-1 | Japanese encephalitis virus | endocytosis | neuronal cells | actin regulation | EGFR | TYROSINE PHOSPHORYLATION | MICROBIOLOGY | MAMMALIAN-CELLS | FAMILY | SIGNAL-TRANSDUCTION | GROWTH-FACTOR RECEPTOR | PATHWAY | MEDIATED ENDOCYTOSIS | DYNAMIN | CAVEOLAE | INFECTIOUS ENTRY | RNA, Small Interfering - genetics | Phosphorylation | Encephalitis, Japanese - virology | Protein-Tyrosine Kinases - metabolism | Humans | rhoA GTP-Binding Protein - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Virus Internalization - drug effects | Actin Depolymerizing Factors - metabolism | Cell Membrane - chemistry | Protein-Tyrosine Kinases - pharmacology | Receptor, Epidermal Growth Factor - metabolism | Transfection | Actin Cytoskeleton - drug effects | Encephalitis Virus, Japanese - metabolism | Membrane Transport Proteins - genetics | Caveolin 1 - drug effects | HEK293 Cells | Membrane Transport Proteins - metabolism | Cell Membrane - metabolism | Dynamin II - metabolism | Life Cycle Stages - physiology | Cell Line | Neurons - virology | Actin Depolymerizing Factors - pharmacology | Cricetinae | Actin Cytoskeleton - metabolism | Signal Transduction | Caveolin 1 - genetics | Dynamin II - genetics | Encephalitis Virus, Japanese - physiology | rhoA GTP-Binding Protein - pharmacology | Cholesterol - metabolism | Encephalitis Virus, Japanese - genetics | Endocytosis - physiology | Caveolin 1 - metabolism | Actin Cytoskeleton - virology | Animals | Virus Attachment | Cell Membrane - virology | rac1 GTP-Binding Protein - pharmacology | Encephalitis Virus, Japanese - pathogenicity | rac1 GTP-Binding Protein - metabolism | Host-Pathogen Interactions - physiology
Journal Article