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Journal Article
Journal Article
PloS one, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, p. e76495
.... In current study, astragaloside IV (ASI), a natural saponin molecule isolated from Astragalus membranceus, given at 20 mg/kg daily attenuated the severity of experimental autoimmune encephalomyelitis (EAE... 
IN-VITRO | INHIBITION | ACID | MULTIDISCIPLINARY SCIENCES | SCLEROSIS | TAU | BAX | CENTRAL-NERVOUS-SYSTEM | EMERGING THERAPIES | CELL-DEATH | P53 | Reactive Oxygen Species - metabolism | Triterpenes - pharmacology | Encephalomyelitis, Autoimmune, Experimental - metabolism | Humans | Demyelinating Diseases - metabolism | Glial Fibrillary Acidic Protein | Th1 Cells - metabolism | CD11b Antigen - genetics | Inflammation - metabolism | Th17 Cells - drug effects | Th17 Cells - metabolism | Inflammation - drug therapy | Apoptosis Regulatory Proteins - genetics | Female | Encephalomyelitis, Autoimmune, Experimental - genetics | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Demyelinating Diseases - drug therapy | Disease Models, Animal | Multiple Sclerosis - metabolism | Cell Line | Th1 Cells - drug effects | Drugs, Chinese Herbal | Cytokines - metabolism | Encephalomyelitis, Autoimmune, Experimental - drug therapy | RNA, Messenger - genetics | Multiple Sclerosis - genetics | Antioxidants - pharmacology | Forkhead Transcription Factors - genetics | Nerve Tissue Proteins - genetics | T-Box Domain Proteins - genetics | Disease Progression | Apoptosis Regulatory Proteins - metabolism | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Animals | Nitric Oxide Synthase Type II - genetics | Mice | CD11b Antigen - metabolism | Oxidative Stress - drug effects | Multiple Sclerosis - drug therapy | Nitric Oxide Synthase Type II - metabolism | Saponins - pharmacology | Antioxidants | Oxidative stress | Nervous system diseases | Multiple sclerosis | Cytokines | Encephalomyelitis | T cells | Tumor proteins | Neurophysiology | Cell culture | Phosphorylation | Hydrogen peroxide | Bax protein | Bcl-2 protein | Laboratories | Pathogenesis | Hydrogen | p53 Protein | Differentiation (biology) | Central nervous system | Lymphocytes T | Chinese medicine | Blood-brain barrier | Neurodegeneration | Rodents | Modulation | Cell cycle | Inhibition | Free radicals | Young adults | Inflammation | Gene expression | Cell differentiation | Experimental allergic encephalomyelitis | Nitric-oxide synthase | Molecular chains | Microglia | Tau protein | Nitric oxide | γ-Interferon | Adults | Infiltration | Apoptosis
Journal Article
PloS one, ISSN 1932-6203, 2011, Volume 6, Issue 2, p. e17103
...). The impact of Rituximab on T cell responses remains largely unexplored. In the experimental autoimmune encephalomyelitis (EAE... 
B-CELLS | REMITTING MULTIPLE-SCLEROSIS | PROTEIN | TOLERANCE | BIOLOGY | EAE | DEPLETION | CTLA-4 | MICE | INDUCTION | IL-10 | Organ Specificity - drug effects | Encephalomyelitis, Autoimmune, Experimental - immunology | Antibodies, Monoclonal - therapeutic use | Organ Specificity - immunology | Immunity, Cellular - drug effects | Autoimmunity - immunology | T-Lymphocytes - drug effects | Drug Evaluation, Preclinical | Multiple Sclerosis, Relapsing-Remitting - complications | Multiple Sclerosis, Relapsing-Remitting - pathology | Encephalomyelitis, Autoimmune, Experimental - pathology | Multiple Sclerosis, Relapsing-Remitting - immunology | Multiple Sclerosis, Relapsing-Remitting - drug therapy | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Mice, Inbred C57BL | Rituximab | Mice, Transgenic | Down-Regulation - drug effects | Animals | Down-Regulation - immunology | Encephalomyelitis, Autoimmune, Experimental - complications | T-Lymphocytes - immunology | Mice | Autoimmunity - drug effects | Antibodies, Monoclonal, Murine-Derived - therapeutic use | Immunologic Factors - therapeutic use | Development and progression | Multiple sclerosis | B cells | Encephalomyelitis | T cells | Immunosuppressive agents | Cell proliferation | Therapy | Hypersensitivity (delayed) | Clinical trials | Nervous system | Lymphocytes T | Cell growth | Immunology | Lymphocytes | Animal tissues | Immune system | Medical research | Immune response | Cytokines | Dendritic cells | Hypersensitivity | Glycoproteins | T cell receptors | Experimental allergic encephalomyelitis | Interleukin 17 | Neurology | Depletion | Lymphocytes B | CD20 antigen
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 0027-8424, 2/2013, Volume 110, Issue 9, pp. 3543 - 3548
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 2012, Volume 189, Issue 11, pp. 5277 - 5283
IL-9-producing Th9 cells have been associated with autoimmune diseases, such as experimental autoimmune encephalitis... 
ENCEPHALOMYELITIS | ALLERGIC INFLAMMATION | INTERLEUKIN-27 | CD4(+) T-CELLS | CENTRAL-NERVOUS-SYSTEM | MICE | PROLIFERATION | DIFFERENTIATION | INDUCTION | IMMUNOLOGY | CUTTING EDGE | Inflammation - chemically induced | Myelin-Oligodendrocyte Glycoprotein - pharmacology | Gene Expression - drug effects | Dendritic Cells - immunology | Interleukin-17 - immunology | Encephalomyelitis, Autoimmune, Experimental - immunology | Peptide Fragments - pharmacology | T-Box Domain Proteins - immunology | Interleukin-9 - biosynthesis | T-Lymphocytes, Helper-Inducer - drug effects | Inflammation - complications | Encephalomyelitis, Autoimmune, Experimental - chemically induced | Inflammation - drug therapy | T-Lymphocytes, Helper-Inducer - immunology | Interleukins - immunology | T-Lymphocytes, Helper-Inducer - cytology | Dendritic Cells - drug effects | Interleukins - biosynthesis | Interleukins - pharmacology | Encephalomyelitis, Autoimmune, Experimental - drug therapy | Mice, Inbred C57BL | Inflammation - immunology | Interleukin-17 - biosynthesis | STAT1 Transcription Factor - genetics | T-Box Domain Proteins - genetics | STAT1 Transcription Factor - immunology | Mice, Knockout | Cell Differentiation - immunology | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Encephalomyelitis, Autoimmune, Experimental - complications | Interferon-gamma - immunology | Dendritic Cells - cytology | Mice | Interleukin-9 - immunology | Interferon-gamma - pharmacology | IL-27 | EAE | Th9 cells | IFN-γ | Inflammation
Journal Article
PloS one, ISSN 1932-6203, 2013, Volume 8, Issue 2, p. e54841
We observed the therapeutic effect of Fasudil and explored its mechanisms in experimental autoimmune encephalomyelitis (EAE... 
MIGRATION | INHIBITION | MULTIPLE-SCLEROSIS | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | RHO-KINASE | MONOCYTES | CROSSTALK | POLARIZATION | T-CELLS | ALTERNATIVE ACTIVATION | Tumor Necrosis Factor-alpha - metabolism | Spleen - immunology | Encephalomyelitis, Autoimmune, Experimental - metabolism | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - analogs & derivatives | Encephalomyelitis, Autoimmune, Experimental - immunology | Spleen - drug effects | rho-Associated Kinases - antagonists & inhibitors | T-Lymphocytes - metabolism | rho-Associated Kinases - metabolism | T-Lymphocytes - drug effects | Interleukin-10 - metabolism | Female | Macrophages - immunology | Severity of Illness Index | Encephalomyelitis, Autoimmune, Experimental - drug therapy | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - therapeutic use | Macrophages - metabolism | Animals | Spleen - metabolism | Protein Kinase Inhibitors - therapeutic use | 1-(5-Isoquinolinesulfonyl)-2-Methylpiperazine - pharmacology | Macrophages - drug effects | Mice | Protein Kinase Inhibitors - pharmacology | Nitric Oxide Synthase Type II - metabolism | Animal experimentation | Immunohistochemistry | Multiple sclerosis | Encephalomyelitis | T cells | Macrophages | Health aspects | Polarization | Neurosciences | CD40 antigen | Pathogenesis | Science | Lymphocytes T | CD14 antigen | Medical schools | Cell adhesion & migration | Cords | Chinese medicine | Demyelination | Lymphocytes | Rodents | Toll-like receptors | Inhibition | Spleen | Antigens | Immunoregulation | Interleukin 12 | Inflammation | T cell receptors | TLR4 protein | Experimental allergic encephalomyelitis | Interleukin 17 | Nitric-oxide synthase | Rho-associated kinase | CD4 antigen | Neurology | CD16 antigen | Tumor necrosis factor | Interleukin 10 | Laboratory animals | Monocyte chemoattractant protein 1
Journal Article
Immunological reviews, ISSN 1600-065X, 2009, Volume 229, Issue 1, pp. 337 - 355
...‐approved therapies for autoimmune disease primarily focus on the global inhibition of immune inflammatory activity... 
experimental autoimmune encephalitis/multiple sclerosis | T cells | antigens/peptides/epitopes | Th1/Th2/Th17cells | cell surface molecules | cell activation | Cell surface molecules | Antigens/peptides/epitopes | Cell activation | Experimental autoimmune encephalitis/multiple sclerosis | DEPENDENT DIABETES-MELLITUS | MULTIPLE-SCLEROSIS LESIONS | NOD MICE | ANTI-CD3 MONOCLONAL-ANTIBODY | DENDRITIC CELLS | multiple sclerosis | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | peptides | Th17cells | IMMUNOLOGY | antigens | ANTIGEN-SPECIFIC TOLERANCE | experimental autoimmune encephalitis | epitopes | Th1 | IMMUNOLOGICAL SYNAPSE | MYELIN PROTEOLIPID PROTEIN | Th2 | E3 UBIQUITIN LIGASE | Protein Kinases - metabolism | Antigens, CD - immunology | Humans | Immunosuppressive Agents - therapeutic use | Antibodies, Monoclonal - therapeutic use | Antigens, CD - metabolism | CD28 Antigens - metabolism | CD4-Positive T-Lymphocytes - immunology | Signal Transduction - immunology | Lymphocyte Activation - immunology | NFATC Transcription Factors - immunology | Receptors, Antigen, T-Cell - immunology | Immunosuppressive Agents - pharmacology | Antibodies, Monoclonal - immunology | Autoimmune Diseases - drug therapy | B7-1 Antigen - immunology | NFATC Transcription Factors - metabolism | Autoimmune Diseases - immunology | B7-1 Antigen - metabolism | CD28 Antigens - immunology | Clinical Trials as Topic | CTLA-4 Antigen | Animals | Protein Kinases - immunology | Signal Transduction - drug effects | Lymphocyte Activation - drug effects | CD4-Positive T-Lymphocytes - drug effects | Autoimmunity | Multiple sclerosis | Peptides | Type 1 diabetes | Biological response modifiers | Antigenic determinants | Interferon gamma
Journal Article