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Journal Article
Journal Article
The Journal of Immunology, ISSN 0022-1767, 03/2007, Volume 178, Issue 6, pp. 3447 - 3456
Journal Article
The Journal of immunology (1950), ISSN 1550-6606, 2015, Volume 195, Issue 5, pp. 1974 - 1983
IL-17-producing CD4(+) T (Th17) cells, along with IFN-gamma-expressing Th1 cells, represent two major pathogenic T cell subsets in experimental autoimmune encephalomyelitis (EAE... 
ROR-GAMMA-T | MULTIPLE-SCLEROSIS | IFN-GAMMA | IN-VIVO | HELPER-CELLS | IMMUNOLOGY | IL-23 | EXPRESSION | CYTOKINE GM-CSF | CUTTING EDGE | TH17 CELLS | T-Lymphocyte Subsets - immunology | Encephalomyelitis, Autoimmune, Experimental - metabolism | Humans | Encephalomyelitis, Autoimmune, Experimental - immunology | Interferon-gamma - metabolism | Th1 Cells - immunology | CD4-Positive T-Lymphocytes - immunology | Receptors, Interleukin - metabolism | Th1 Cells - metabolism | Cell Differentiation - genetics | Interleukin-7 - genetics | Signal Transduction - immunology | Receptors, Interleukin-7 - genetics | Flow Cytometry | Interleukin-23 - metabolism | Th17 Cells - metabolism | Encephalomyelitis, Autoimmune, Experimental - genetics | Interferon-gamma - genetics | Receptors, Interleukin-7 - immunology | Interleukin-7 - metabolism | Disease Models, Animal | Multiple Sclerosis - metabolism | Receptors, Interleukin-7 - metabolism | Receptors, Interleukin - immunology | Mice, Inbred C57BL | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Mice, Transgenic | Multiple Sclerosis - genetics | Signal Transduction - genetics | Receptors, Interleukin - genetics | Interleukin-23 - immunology | Mice, Knockout | Cell Differentiation - immunology | Animals | Interleukin-7 - immunology | T-Lymphocyte Subsets - metabolism | Interferon-gamma - immunology | Multiple Sclerosis - immunology | Th17 Cells - immunology
Journal Article
PloS one, ISSN 1932-6203, 2010, Volume 5, Issue 11, p. e15531
Background: There is consensus that experimental autoimmune encephalomyelitis (EAE) can be mediated by myelin specific T cells of Th1 as well as of Th17... 
MYELIN OLIGODENDROCYTE GLYCOPROTEIN | IFN-GAMMA | MULTIDISCIPLINARY SCIENCES | CENTRAL-NERVOUS-SYSTEM | MICE | CYTOKINE | VASCULAR ENDOTHELIAL-CELLS | INDUCTION | EXPRESSION | T-CELLS | CUTTING EDGE | Cell Proliferation | Spleen - immunology | Encephalomyelitis, Autoimmune, Experimental - metabolism | Coculture Techniques | Interleukin-17 - immunology | Encephalomyelitis, Autoimmune, Experimental - immunology | Th17 Cells - transplantation | Adoptive Transfer | Interferon-gamma - metabolism | Th1 Cells - immunology | Th1 Cells - transplantation | Brain - metabolism | Th1 Cells - metabolism | Astrocytes - immunology | Th17 Cells - metabolism | Encephalomyelitis, Autoimmune, Experimental - genetics | Spleen - pathology | Mice, Inbred C57BL | Cells, Cultured | Myelin-Oligodendrocyte Glycoprotein | Mice, Transgenic | Mice, Knockout | Cell Differentiation - immunology | Interleukin-17 - metabolism | Myelin-Associated Glycoprotein - immunology | Animals | Spleen - metabolism | Interferon-gamma - immunology | Brain - pathology | Myelin Proteins | Th17 Cells - immunology | Mice | Cytotoxicity, Immunologic - immunology | Brain - immunology | Astrocytes - metabolism | Autoimmunity | Encephalomyelitis | Cytokines | Genetic engineering | B cells | T cells | Myelin proteins | Biotechnology | Neurosciences | Multiple sclerosis | Oligodendrocyte-myelin glycoprotein | Pathogenesis | Neurobiology | Helper cells | Central nervous system | Cytotoxicity | Lymphocytes T | Biology | Adoptive transfer | T-cell receptor | Lymphocytes | Rodents | Fibroblasts | Lesions | Localization | Antigens | Myelin | Astrocytes | Neutrophils | Transgenic mice | Glycoproteins | Gene expression | Experimental allergic encephalomyelitis | Interleukin 17 | CD4 antigen | Studies | RAG2 protein | Lymphopenia | Biopsy | Production methods | γ-Interferon | Cell lines | Interferon
Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2014, Volume 9, Issue 1, p. e85007
... from subcutaneous inguinal fat pads of mice with established experimental autoimmune encephalomyelitis (EAE... 
IMMUNE-RESPONSE | MCP-1 | MULTIPLE-SCLEROSIS | MULTIDISCIPLINARY SCIENCES | BONE-MARROW | TISSUE | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | CCL2 | STROMAL CELLS | METALLOPROTEINASES | EXPRESSION | Mesenchymal Stem Cells - immunology | Humans | Adipose Tissue - cytology | Encephalomyelitis, Autoimmune, Experimental - immunology | Adipocytes - cytology | Transplantation, Autologous | Adipose Tissue - immunology | Mesenchymal Stem Cells - cytology | Treatment Failure | Female | Cell Differentiation | Encephalomyelitis, Autoimmune, Experimental - genetics | Cytokines - genetics | Adipocytes - immunology | Cytokines - immunology | Disease Models, Animal | Biomarkers - metabolism | Gene Expression | Encephalomyelitis, Autoimmune, Experimental - pathology | Mice, Inbred C57BL | Multiple Sclerosis - genetics | Multiple Sclerosis - therapy | Disease Progression | Encephalomyelitis, Autoimmune, Experimental - therapy | Animals | Multiple Sclerosis - immunology | Multiple Sclerosis - pathology | Mice | Mesenchymal Stem Cell Transplantation | Medical research | Nervous system diseases | Multiple sclerosis | Encephalomyelitis | Analysis | Stem cells | Medicine, Experimental | Models | Inflammation | Transplantation | Animal models | Transplants & implants | Disease | Laboratories | Syngeneic grafts | Central nervous system | Cytology | Stem cell transplantation | Nervous system | Cell surface | Cell morphology | Proteins | Infusion | Keratin | Biomedical materials | Allografts | Immunology | Demyelination | Rodents | Bone marrow | Biocompatibility | Immune system | Autografts | Cytokines | Medical treatment | Experimental allergic encephalomyelitis | Whooping cough | Medicine | Studies | Brain research | Monocyte chemoattractant protein | Infiltration | Molecular biology | Differentiation | Chemokines | Monocyte chemoattractant protein 1
Journal Article
Nature immunology, ISSN 1529-2916, 2007, Volume 8, Issue 3, pp. 247 - 256
.... This reduction was associated with much less inflammatory disease in vivo in both autoimmune encephalomyelitis and dextran sodium sulfate-induced colitis. Our data show that Act1 is essential in IL-17-dependent signaling in autoimmune and inflammatory disease. 
PATHWAYS | ENCEPHALOMYELITIS | EPITHELIAL-CELLS | MULTIPLE-SCLEROSIS | FAMILY-MEMBERS | RECEPTOR COMPLEX | DEFICIENT MICE | IMMUNOLOGY | EXPRESSION | T-CELLS | CUTTING EDGE | Autoimmunity | Encephalomyelitis, Autoimmune, Experimental - metabolism | Humans | Receptors, Interleukin-17 - immunology | Interleukin-17 - immunology | Encephalomyelitis, Autoimmune, Experimental - immunology | Colitis - pathology | Adoptive Transfer | Signal Transduction - immunology | CD40 Antigens - metabolism | Transfection | Adaptor Proteins, Signal Transducing - immunology | Female | Receptors, Interleukin-17 - metabolism | Colitis - immunology | B-Cell Activation Factor Receptor - immunology | Tumor Necrosis Factor Receptor-Associated Peptides and Proteins | Gene Expression | Encephalomyelitis, Autoimmune, Experimental - pathology | Enzyme-Linked Immunosorbent Assay | Gene Expression Regulation - immunology | Inflammation - immunology | Reverse Transcriptase Polymerase Chain Reaction | Interleukin-17 - metabolism | B-Cell Activation Factor Receptor - metabolism | Animals | Autoimmune Diseases | Colitis - metabolism | Fluorescent Antibody Technique | T-Lymphocytes - immunology | Mice | HeLa Cells | Adaptor Proteins, Signal Transducing - metabolism | CD40 Antigens - immunology | Immune response | Interleukins | Physiological aspects | Cellular signal transduction | Inflammation | Genetic aspects | Research | Binding proteins | Risk factors
Journal Article