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BBA - Molecular and Cell Biology of Lipids, ISSN 1388-1981, 06/2016, Volume 1861, Issue 6, pp. 491 - 500
Journal Article
Science, ISSN 0036-8075, 11/2011, Volume 334, Issue 6057, pp. 809 - 813
Phospholipase A₂ (PLA₂) enzymes are considered the primary source of arachidonic acid for cyclooxygenase (COX)-mediated biosynthesis of prostaglandins. Here,... 
Datasets | Brain | Enzymes | Prostaglandins | Cytokines | Neurodegenerative diseases | REPORTS | Eicosanoids | Lipids | Endocannabinoids | Vehicles | SYSTEM | INHIBITION | CYCLOOXYGENASE-2 | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | INJURY | LIPOPOLYSACCHARIDE | PHOSPHOLIPASE A | MICE | PARKINSONS-DISEASE | Cannabinoid Receptor Modulators - metabolism | Inflammation - pathology | Metabolomics | Arachidonic Acid - metabolism | Monoacylglycerol Lipases - antagonists & inhibitors | Monoacylglycerol Lipases - genetics | Brain - metabolism | Parkinsonian Disorders - metabolism | Monoacylglycerol Lipases - metabolism | Arachidonic Acids - metabolism | Inflammation - metabolism | Neuroprotective Agents - pharmacology | Piperidines - pharmacology | Inflammation Mediators - pharmacology | Lung - metabolism | Cytokines - metabolism | Signal Transduction | Liver - metabolism | Mice, Inbred C57BL | Enzyme Inhibitors - pharmacology | Phospholipases A2 - metabolism | Prostaglandins - biosynthesis | Glycerides - metabolism | Brain - drug effects | Hydrolysis | Animals | Parkinsonian Disorders - pathology | Eicosanoids - metabolism | Lipopolysaccharides - pharmacology | Brain - pathology | Mice | Cyclooxygenase 1 - metabolism | Benzodioxoles - pharmacology | Phospholipases A2 - genetics | Prostaglandins - metabolism | Physiological aspects | Inflammation | Research | Risk factors | Hydrology | Neurology | Inflammatory diseases | Animal models | Index Medicus
Journal Article
Gut, ISSN 0017-5749, 08/2014, Volume 63, Issue 8, pp. 1300 - 1312
Objective Enteric glia activation has been reported to amplify intestinal inflammation via the enteroglial-specific S100B protein. This neurotrophin promotes... 
NITRIC-OXIDE PRODUCTION | INHIBITION | CROHNS-DISEASE | MACROPHAGES | ULCERATIVE-COLITIS | FATTY-ACID AMIDE | S100B PROTEIN | N-PALMITOYL-ETHANOLAMINE | GLIAL-CELLS | GASTROENTEROLOGY & HEPATOLOGY | BOWEL-DISEASE | Tumor Necrosis Factor-alpha - metabolism | Dextran Sulfate | Ethanolamines - pharmacology | Endocannabinoids - pharmacology | Rectum - pathology | Humans | Middle Aged | Palmitic Acids - therapeutic use | Male | NF-kappa B - metabolism | Glial Fibrillary Acidic Protein - metabolism | Nitric Oxide - analysis | Colon, Sigmoid - chemistry | Anti-Inflammatory Agents, Non-Steroidal - pharmacology | Neuroglia - drug effects | Colitis - chemically induced | Female | Indoles - pharmacology | Colitis - drug therapy | S100 Calcium Binding Protein beta Subunit - metabolism | Colitis, Ulcerative - drug therapy | Palmitic Acids - pharmacology | Colon, Sigmoid - pathology | Endocannabinoids - therapeutic use | Neutrophil Infiltration - drug effects | Severity of Illness Index | PPAR alpha - antagonists & inhibitors | Signal Transduction | Colitis, Ulcerative - metabolism | Cells, Cultured | Colitis, Ulcerative - pathology | Toll-Like Receptor 4 - metabolism | Dinoprostone - metabolism | Nerve Tissue Proteins - metabolism | Animals | PPAR gamma - antagonists & inhibitors | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Anilides - pharmacology | Colitis - metabolism | Cyclooxygenase 2 - metabolism | Rectum - chemistry | Ethanolamines - therapeutic use | Neuroglia - metabolism | Mice | PPAR alpha - metabolism | Nitric Oxide - metabolism | Nitric Oxide Synthase Type II - metabolism | Influence | Amides | Inflammation | Neuroglia | Analysis | Risk factors | Index Medicus | Abridged Index Medicus
Journal Article
American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 08/2007, Volume 293, Issue 2, pp. 909 - 918
Recent studies have uncovered important cross talk between inflammation, generation of reactive oxygen and nitrogen species, and lipid metabolism in the... 
Pressure-volume relationship | Cardiac function | Endocannabinoids | Anandamide | CARDIAC & CARDIOVASCULAR SYSTEMS | PHYSIOLOGY | ANANDAMIDE HYDROLYSIS | HEART-FAILURE | ENDOGENOUS CANNABINOID SYSTEM | ENDOCANNABINOID SYSTEM | RECEPTOR | KNOCKOUT MICE | CORONARY-ARTERIES | HEMODYNAMIC PROFILE | pressure-volume relationship | NITRIC-OXIDE | PERIPHERAL VASCULAR DISEASE | cardiac function | endocannabinoids | VASCULAR ENDOTHELIAL-CELLS | anandamide | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Humans | Monocytes - metabolism | NF-kappa B - metabolism | Coronary Vessels - metabolism | Receptors, Cannabinoid - metabolism | Arachidonic Acids - metabolism | Inflammation - metabolism | Aging - genetics | Ventricular Dysfunction, Left - genetics | Myocardium - metabolism | Ventricular Dysfunction, Left - pathology | Ventricular Dysfunction, Left - enzymology | Amidohydrolases - deficiency | Amidohydrolases - metabolism | Endothelial Cells - metabolism | Reactive Nitrogen Species - metabolism | Amidohydrolases - genetics | Cells, Cultured | Gene Expression Regulation | Polyunsaturated Alkamides - metabolism | Cell Adhesion | Aging - pathology | Mice, Knockout | Ventricular Dysfunction, Left - metabolism | Intercellular Adhesion Molecule-1 - metabolism | Myocardium - enzymology | Animals | Coronary Vessels - cytology | Inflammation - genetics | Mice | Vascular Cell Adhesion Molecule-1 - metabolism | Inflammation - enzymology | Aging - metabolism | Apoptosis | Studies | Cardiovascular system | Rodents | Aging | Gene expression | Fatty acids | Index Medicus
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 11/2007, Volume 152, Issue 5, pp. 734 - 743
Background and purpose: Although CB1 receptor activation evokes neuroprotection in response to cannabinoids, some cannabinoids have been reported to be... 
Neuroprotection | Peroxisome proliferator-activated receptors (PPARs) | Cannabinoids | N-oleoylethanolamine | GAMMA | CELLS | FOCAL CEREBRAL-ISCHEMIA | ANANDAMIDE | ENDOCANNABINOID SYSTEM | NUCLEAR RECEPTOR | peroxisome proliferator-activated receptors (PPARs) | neuroprotection | cannabinoids | THERAPEUTIC TARGETS | PHARMACOLOGY & PHARMACY | NF-KAPPA-B | TRANSCRIPTION FACTOR | BRAIN | Luciferases - metabolism | Humans | Benzoxazines - metabolism | Male | Recombinant Fusion Proteins - metabolism | Cerebral Cortex - metabolism | Neuroprotective Agents - metabolism | Luciferases - genetics | Oleic Acids - metabolism | Arachidonic Acids - metabolism | Fenofibrate - pharmacology | Neuroprotective Agents - pharmacology | Transfection | I-kappa B Kinase - metabolism | Cerebral Cortex - drug effects | Oleic Acids - pharmacology | Dronabinol - pharmacology | Naphthalenes - metabolism | Cannabinoids - metabolism | Endocannabinoids | Fenofibrate - metabolism | Mice, Inbred C57BL | Morpholines - pharmacology | Fatty Acids, Unsaturated - pharmacology | Morpholines - metabolism | PPAR alpha - genetics | Polyunsaturated Alkamides - metabolism | Arachidonic Acids - pharmacology | Cannabinoids - pharmacology | Mice, Knockout | Naphthalenes - pharmacology | Animals | Benzoxazines - pharmacology | Cyclooxygenase 2 - metabolism | Polyunsaturated Alkamides - pharmacology | Protein Binding | Recombinant Fusion Proteins - genetics | Ligands | PPAR alpha - agonists | Mice | HeLa Cells | PPAR alpha - metabolism | Dronabinol - metabolism | Index Medicus | Research Papers
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 09/2017, Volume 102, Issue 12, pp. 4486 - 4495
Journal Article
Obesity, ISSN 1930-7381, 03/2008, Volume 16, Issue 3, pp. 553 - 565
Journal Article
Endocrinology, ISSN 0013-7227, 10/2017, Volume 158, Issue 10, pp. 3462 - 3476
Phthalates, used as plasticizers, have become a ubiquitous contaminant and have been reported for their potential to induce toxicity in living organisms. Among... 
DANIO-RERIO | HUMAN EXPOSURE | LIPID-METABOLISM | FOOD-INTAKE | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | ADIPOCYTE DIFFERENTIATION | ACTIVATED RECEPTOR-ALPHA | DIISONONYL PHTHALATE | ACYL-COENZYME | NEUROPEPTIDE-Y | Endocannabinoids - metabolism | Plasticizers - pharmacology | Gene Expression - drug effects | Lipoprotein Lipase - drug effects | Receptor, Cannabinoid, CB2 - genetics | Brain - metabolism | Dose-Response Relationship, Drug | Arachidonic Acids - metabolism | Phospholipase D - drug effects | Endocrine Disruptors - pharmacology | Liver - drug effects | Female | Phospholipase D - genetics | Phthalic Acids - pharmacology | Lipoprotein Lipase - metabolism | Fatty Liver - metabolism | Lipoprotein Lipase - genetics | Receptor, Cannabinoid, CB2 - drug effects | Zebrafish | Glycerides - metabolism | Polyunsaturated Alkamides - metabolism | Phospholipase D - metabolism | Brain - drug effects | Receptor, Cannabinoid, CB1 - metabolism | Receptor, Cannabinoid, CB2 - metabolism | Animals | Receptor, Cannabinoid, CB1 - genetics | Lipid Metabolism - drug effects | Receptor, Cannabinoid, CB1 - drug effects | Endocrine disruptors | Neuroimaging | Brain | Infrared spectroscopy | Deregulation | Transcription | Image processing | Toxicity | Liver | Lipids | Infrared analysis | Fatty liver | Infrared imaging | Lipid metabolism | Appetite | Plasticizers | Histology | Metabolism | Infrared imagery | Phthalates | Studies | Image analysis | Contaminants | Aquatic organisms | Index Medicus | Abridged Index Medicus
Journal Article
Nature, ISSN 0028-0836, 11/2016, Volume 539, Issue 7630, pp. 555 - 559
Cellular activity in the brain depends on the high energetic support provided by mitochondria, the cell organelles which use energy sources to generate... 
OXIDATIVE-PHOSPHORYLATION | CATALYTIC SUBUNIT | SOLUBLE ADENYLYL-CYCLASE | METABOLISM | MULTIDISCIPLINARY SCIENCES | ENDOCANNABINOID SYSTEM | SUBCELLULAR-LOCALIZATION | NEURONS | PROTEINS | RECEPTORS | BRAIN | Memory - drug effects | Electron Transport - drug effects | Memory Disorders - metabolism | Male | Hippocampus - drug effects | Cannabinoids - adverse effects | Mitochondrial Membranes - drug effects | Oxidative Phosphorylation - drug effects | Cell Respiration - drug effects | Female | Synaptic Transmission - drug effects | Mitochondrial Membranes - enzymology | Cannabinoids - metabolism | Memory Disorders - pathology | Cyclic AMP-Dependent Protein Kinases - metabolism | Memory Disorders - enzymology | Hippocampus - pathology | Mitochondria - metabolism | Adenylyl Cyclases - metabolism | Mitochondria - drug effects | NADH Dehydrogenase - metabolism | Mitochondrial Membranes - metabolism | Hippocampus - metabolism | Receptor, Cannabinoid, CB1 - metabolism | Animals | Receptor, Cannabinoid, CB1 - genetics | Signal Transduction - drug effects | Mice | Receptor, Cannabinoid, CB1 - deficiency | GTP-Binding Protein alpha Subunits, Gi-Go - metabolism | Energy Metabolism - drug effects | Memory - physiology | Memory Disorders - chemically induced | Mitochondria | Cannabinoids | Health aspects | Memory | Proteins | Phosphorylation | Rodents | Respiration | Chemical compounds | Index Medicus
Journal Article