X
Search Filters
Format Format
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
index medicus (1101) 1101
animals (811) 811
endocardium - metabolism (524) 524
humans (455) 455
male (419) 419
myocardium - metabolism (371) 371
female (338) 338
cardiac & cardiovascular systems (299) 299
endocardium - pathology (293) 293
heart (251) 251
endocardium (213) 213
middle aged (182) 182
dogs (179) 179
myocardium - pathology (174) 174
endocardium - cytology (169) 169
mice (169) 169
abridged index medicus (159) 159
adult (157) 157
developmental biology (149) 149
endocardium - embryology (145) 145
rats (143) 143
cell biology (141) 141
peripheral vascular disease (120) 120
aged (112) 112
pericardium - metabolism (112) 112
biopsy (111) 111
time factors (105) 105
heart - embryology (100) 100
immunohistochemistry (98) 98
physiology (98) 98
expression (94) 94
cells (91) 91
heart ventricles - metabolism (91) 91
cardiovascular system (89) 89
endocardium - physiology (89) 89
endocardium - drug effects (87) 87
endothelium (85) 85
gene expression regulation, developmental (85) 85
hematology (84) 84
endocardium - physiopathology (82) 82
myocardium (82) 82
myocardium - cytology (79) 79
gene expression (78) 78
in vitro techniques (77) 77
morphogenesis (73) 73
calcium - metabolism (72) 72
cells, cultured (71) 71
coronary circulation (70) 70
article (68) 68
electrocardiography (66) 66
myocytes, cardiac - metabolism (66) 66
endothelium, vascular - metabolism (65) 65
signal transduction (65) 65
disease models, animal (62) 62
rabbits (60) 60
cardiology (59) 59
research (59) 59
heart - physiology (58) 58
endocardium - ultrastructure (57) 57
chick embryo (56) 56
epicardium (56) 56
physiological aspects (56) 56
analysis (55) 55
gene-expression (53) 53
microscopy, electron (49) 49
action potentials (48) 48
anatomy & morphology (48) 48
endocardium - enzymology (48) 48
swine (48) 48
endothelial cells - metabolism (47) 47
myocardial contraction (47) 47
heart development (46) 46
transplantation (46) 46
heart failure (45) 45
heart valves - embryology (45) 45
pathology (45) 45
rna, messenger - metabolism (45) 45
zebrafish (45) 45
hemodynamics (44) 44
biochemistry & molecular biology (43) 43
multidisciplinary sciences (43) 43
adolescent (42) 42
cardiovascular (42) 42
electrophysiology (42) 42
in situ hybridization (42) 42
ischemia (42) 42
potassium - metabolism (42) 42
transcription factor (42) 42
cardiac development (41) 41
mouse (41) 41
cardiomyopathy (40) 40
cell proliferation (40) 40
endothelial-cells (40) 40
fibrosis (40) 40
heart - drug effects (40) 40
heart transplantation (40) 40
receptor (40) 40
mice, knockout (39) 39
patch-clamp techniques (39) 39
pharmacology & pharmacy (39) 39
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (1121) 1121
German (9) 9
Japanese (8) 8
Russian (8) 8
French (5) 5
Chinese (3) 3
Italian (2) 2
Serbian (1) 1
Spanish (1) 1
Ukrainian (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Developmental Cell, ISSN 1534-5807, 07/2013, Volume 26, Issue 1, pp. 45 - 58
Vegf signaling specifies arterial fate during early vascular development by inducing the transcription of ( ), the earliest Notch ligand gene expressed in... 
VASCULAR DEVELOPMENT | ARTERIOVENOUS-MALFORMATIONS | CELLS | ENDOTHELIAL-GROWTH-FACTOR | NOTCH PATHWAY | MICE | DEVELOPMENTAL BIOLOGY | DIFFERENTIATION | TUBE FORMATION | EXPRESSION | FORKHEAD TRANSCRIPTION FACTORS | CELL BIOLOGY | Transcriptional Regulator ERG | Human Umbilical Vein Endothelial Cells - metabolism | Receptors, Notch - metabolism | Humans | Receptors, Notch - genetics | Green Fluorescent Proteins - genetics | Intracellular Signaling Peptides and Proteins - metabolism | Vascular Endothelial Growth Factor A - metabolism | Aorta - metabolism | Vascular Endothelial Growth Factor A - genetics | Zebrafish - embryology | Endocardium - metabolism | MAP Kinase Signaling System | Gene Expression Regulation, Developmental | Trans-Activators - genetics | Transcription, Genetic | Membrane Proteins - metabolism | Binding Sites | Intracellular Signaling Peptides and Proteins - genetics | Proto-Oncogene Proteins - metabolism | Green Fluorescent Proteins - metabolism | Endocardium - embryology | Membrane Proteins - genetics | Proto-Oncogene Proteins - genetics | Organ Specificity | Animals, Genetically Modified - metabolism | Zebrafish - genetics | Animals | Animals, Genetically Modified - embryology | Enhancer Elements, Genetic | Zebrafish - metabolism | Trans-Activators - metabolism | Mice | Aorta - physiology | Receptor, Notch4 | Index Medicus
Journal Article
Circulation Research, ISSN 0009-7330, 04/2008, Volume 102, Issue 7, pp. 752 - 760
Journal Article
NATURE COMMUNICATIONS, ISSN 2041-1723, 04/2019, Volume 10, Issue 1, pp. 1929 - 14
Genetically modified mice have advanced our understanding of valve development and disease. Yet, human pathophysiological valvulogenesis remains poorly... 
EPITHELIAL-MESENCHYMAL TRANSITION | PROGENITOR CELLS | POPULATION | ARTERIAL | HEART-DISEASE | FIELD | MULTIDISCIPLINARY SCIENCES | MITRAL-VALVE-PROLAPSE | MUTATIONS | EXPRESSION | REVEALS | Bone Morphogenetic Protein 2 - pharmacology | Heart Atria - pathology | Cadherins - metabolism | Embryo, Mammalian | Humans | Endothelial Cells - transplantation | Hedgehog Proteins - metabolism | Epithelial-Mesenchymal Transition - drug effects | GATA5 Transcription Factor - metabolism | Gene Expression Profiling | Endocardium - pathology | Antigens, CD - genetics | Mitral Valve Prolapse - genetics | Mitral Valve - metabolism | Antigens, CD - metabolism | Endocardium - metabolism | Pluripotent Stem Cells - pathology | Wnt3A Protein - pharmacology | GATA5 Transcription Factor - genetics | Hedgehog Proteins - genetics | Cadherins - genetics | Biomarkers - metabolism | Endothelial Cells - metabolism | Gene Expression Regulation | Mitral Valve Prolapse - metabolism | Mitral Valve Prolapse - pathology | Mitral Valve Prolapse - therapy | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Heart Atria - metabolism | Pluripotent Stem Cells - metabolism | Animals | Cell Differentiation - drug effects | Models, Biological | Pluripotent Stem Cells - drug effects | Mice | Mitral Valve - pathology | Primary Cell Culture | Endothelial Cells - pathology | Endothelial Cells - drug effects | Mitral valve | Cushions | Bone morphogenetic protein 2 | Mesenchyme | Secretion | Therapeutic applications | Heart transplantation | Stem cell transplantation | Interstitial cells | Embryos | Heart valves | Hedgehog protein | Stem cells | Extracellular matrix | Ventricle | Pluripotency | Genetic modification | Index Medicus
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 07/2014, Volume 124, Issue 7, pp. 2921 - 2934
Journal Article
Journal Article
Developmental Biology, ISSN 0012-1606, 01/2012, Volume 361, Issue 2, pp. 377 - 391
Endocardial cells form the inner endothelial layer of the heart tube, surrounded by the myocardium. Signaling pathways that regulate endocardial cell... 
Heart | Hedgehog | Smoothened | Cyclopamine | Endocardium | Zebrafish | NFATc1 | Notochord | Fibronectin | Endoderm | HEART FIELD | TRANSGENIC ZEBRAFISH | CARDIOVASCULAR PROGENITORS | DEVELOPMENTAL BIOLOGY | ENDODERM FORMATION | NEURONAL PRECURSORS | VERTEBRATE HEART | EMBRYONIC VASCULAR DEVELOPMENT | ENDOTHELIAL DIFFERENTIATION | TRANSCRIPTION FACTOR | SONIC-HEDGEHOG | Notochord - cytology | Embryo, Nonmammalian - cytology | Receptors, Notch - metabolism | Embryo, Nonmammalian - metabolism | Hedgehog Proteins - metabolism | Endoderm - embryology | Stem Cells - cytology | Vascular Endothelial Growth Factor A - metabolism | Stem Cells - metabolism | Zebrafish - embryology | Embryo, Nonmammalian - drug effects | Time-Lapse Imaging | Endocardium - metabolism | Cell Differentiation - genetics | In Situ Hybridization | Notochord - metabolism | Hedgehog Proteins - genetics | Endoderm - cytology | Myocardium - metabolism | Veratrum Alkaloids - pharmacology | Endocardium - cytology | Biomarkers - metabolism | Zebrafish Proteins - metabolism | NFATC Transcription Factors - metabolism | Gene Expression Regulation, Developmental - drug effects | Signal Transduction - genetics | Notochord - embryology | Fibronectins - metabolism | Zebrafish - genetics | Endoderm - metabolism | Myocardium - cytology | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Endoderm - drug effects | Zebrafish - metabolism | Morphogenesis - drug effects | Stem Cells - drug effects | Cell Proliferation - drug effects | Zebrafish Proteins - genetics | Endocardium - drug effects | NFATC Transcription Factors - genetics | Notochord - drug effects | Fibronectins | Analysis | Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 368 - 16
Journal Article
Developmental Cell, ISSN 1534-5807, 02/2008, Volume 14, Issue 2, pp. 298 - 311
Journal Article
Advances in Experimental Medicine and Biology, ISSN 0065-2598, 11/2016, Volume 913, pp. 229 - 239
Journal Article
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1079-5642, 2017, Volume 37, Issue 9, pp. E116 - +
Objective-Fstl1 (Follistatin-like 1) is a secreted protein that is expressed in the atrioventricular valves throughout embryonic development, postnatal... 
heart failure | mitral valve | animals | endocardium | echocardiography | PROGENITOR CELLS | TGF-BETA | MOUSE HEART | ACUTE CORONARY SYNDROME | CARDIAC DEVELOPMENT | EPITHELIAL-MESENCHYMAL TRANSITION | PERIPHERAL VASCULAR DISEASE | EXTRACELLULAR-MATRIX | MORPHOGENETIC PROTEIN BMP | DEVELOPING HEART-VALVES | HEMATOLOGY | CHICKEN HEART | Cell Proliferation | Ventricular Function, Left | Heart Failure - physiopathology | Endocardium - pathology | Mitral Valve Prolapse - genetics | Mitral Valve - metabolism | Mitral Valve - physiopathology | Endocardium - metabolism | Mitral Valve Insufficiency - genetics | Bone Morphogenetic Proteins - metabolism | Ventricular Remodeling | Time Factors | Ventricular Dysfunction, Left - genetics | Epithelial-Mesenchymal Transition | Ventricular Dysfunction, Left - pathology | Disease Models, Animal | Follistatin-Related Proteins - genetics | Mitral Valve Prolapse - physiopathology | Genetic Predisposition to Disease | Heart Conduction System - metabolism | Signal Transduction | Endothelial Cells - metabolism | Mitral Valve Prolapse - metabolism | Mitral Valve Prolapse - pathology | Heart Failure - genetics | Heart Failure - metabolism | Heart Failure - pathology | Disease Progression | Ventricular Dysfunction, Left - physiopathology | Follistatin-Related Proteins - deficiency | Mice, Knockout | Receptor, TIE-2 - genetics | Transcription Factors - metabolism | Ventricular Dysfunction, Left - metabolism | Cell Lineage | Mitral Valve Insufficiency - pathology | Phenotype | Animals | Heart Conduction System - physiopathology | Mitral Valve - pathology | Endothelial Cells - pathology | Integrases - genetics | Mitral Valve Insufficiency - physiopathology | Transforming Growth Factor beta - metabolism | Mitral Valve Insufficiency - metabolism | Index Medicus
Journal Article
Clinical and Experimental Pharmacology and Physiology, ISSN 0305-1870, 11/2007, Volume 34, Issue 11, pp. 1165 - 1172
SUMMARY • The role of haemodynamic changes in left ventricular remodelling has been poorly investigated, especially in the context of volume overload cardiac... 
aortocaval fistula | coronary perfusion pressure | metalloproteinases | remodelling | cytokines | fibrosis | subendocardium | Remodelling | Aortocaval fistula | Cytokines | Coronary perfusion pressure | Fibrosis | Subendocardium | Metalloproteinases | MATRIX | PHYSIOLOGY | MYOCARDIAL-INFARCTION | VOLUME OVERLOAD | HEART-FAILURE | RATS | BLOOD-FLOW | CARDIAC-HYPERTROPHY | PHARMACOLOGY & PHARMACY | HYPERTENSION | EXPRESSION | Blood Pressure | Tumor Necrosis Factor-alpha - metabolism | Vena Cava, Inferior - surgery | Rats, Wistar | Arteriovenous Fistula - metabolism | Male | Endocardium - pathology | Aortic Diseases - metabolism | Endocardium - metabolism | Ventricular Remodeling | Time Factors | Interleukin-1beta - metabolism | Ventricular Dysfunction, Left - pathology | Interleukin-10 - metabolism | Interleukin-6 - metabolism | Aortic Diseases - pathology | Disease Models, Animal | Arteriovenous Fistula - complications | Heart Rate | Echocardiography | Matrix Metalloproteinase 2 - metabolism | Rats | Ventricular Dysfunction, Left - etiology | Thiobarbituric Acid Reactive Substances - metabolism | Ventricular Dysfunction, Left - physiopathology | Endocardium - physiopathology | Aortic Diseases - physiopathology | Animals | Aortic Diseases - complications | Arteriovenous Fistula - pathology | Arteriovenous Fistula - physiopathology | Ventricular Pressure | Endocardium - enzymology | Peroxidase - metabolism | Vena Cava, Inferior | Arteriovenous Fistula, physiopathology | Arteriovenous Fistula, metabolism | Thiobarbituric Acid Reactive Substances, metabolism | Endocardium, pathology | Ventricular Dysfunction, Left, physiopathology | Tumor Necrosis Factor-alpha, metabolism | Ventricular Dysfunction, Left, etiology | Interleukin-10, metabolism | Interleukin-6, metabolism | Arteriovenous Fistula, complications | Endocardium, metabolism | Aortic Diseases, complications | Arteriovenous Fistula, pathology | Endocardium, physiopathology | Peroxidase, metabolism | Aortic Diseases, pathology | Vena Cava, Inferior, surgery | Aortic Diseases, metabolism | Aortic Diseases, physiopathology | Endocardium, enzymology | Matrix Metalloproteinase 2, metabolism | Ventricular Dysfunction, Left, pathology | Interleukin-1beta, metabolism | Fistula | Index Medicus
Journal Article