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1. Full Text The genomic landscape and evolution of endometrial carcinoma progression and abdominopelvic metastasis
by Gibson, William J and Hoivik, Erling A and Halle, Mari K and Taylor-Weiner, Amaro and Cherniack, Andrew D and Berg, Anna and Holst, Frederik and Zack, Travis I and Werner, Henrica M.J and Staby, Kjersti M and Rosenberg, Mara and Stefansson, Ingunn M and Kusonmano, Kanthida and Chevalier, Aaron and Mauland, Karen K and Trovik, Jone and Krakstad, Camilla and Giannakis, Marios and Hodis, Eran and Woie, Kathrine and Bjorge, Line and Vintermyr, Olav K and Wala, Jeremiah A and Lawrence, Michael S and Getz, Gad and Carter, Scott L and Beroukhim, Rameen and Salvesen, Helga B
Nature Genetics, ISSN 1061-4036, 08/2016, Volume 48, Issue 8, pp. 848 - 855
Recent studies have detailed the genomic landscape of primary endometrial cancers, but the evolution of these cancers into metastases has not been... 
BREAST-CANCER | THERAPY | INSTABILITY | ADENOCARCINOMA | PANCREATIC-CANCER | GENETICS & HEREDITY | MUTATIONS | HYPERPLASIA | ARID1A | ESTROGEN-RECEPTOR | REVEALS | Abdominal Neoplasms - secondary | Genomics | Humans | Pelvic Neoplasms - genetics | Endometrial Hyperplasia - genetics | Endometrial Hyperplasia - pathology | Phylogeny | Mutation - genetics | Disease Progression | Pelvic Neoplasms - secondary | Exome - genetics | Endometrial Neoplasms - genetics | Abdominal Neoplasms - genetics | Endometrial Neoplasms - pathology | Female | Biomarkers, Tumor - genetics | High-Throughput Nucleotide Sequencing | Evolution, Molecular | Complications and side effects | Genome-wide association studies | Endometrial cancer | Gene mutations | Genetic aspects | Metastasis | Health aspects | Risk factors | Methods | Studies | Biomedical research | Biopsy | Software | Mutation | Patients | Tumors | Precursor | Cancer genomics | Cancer evolution | Cancer
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2. Full Text β-catenin mediates glandular formation and dysregulation of β-catenin induces hyperplasia formation in the murine uterus
by Jeong, J.-W and Lee, H.S and Franco, H.L and Broaddus, R.R and Taketo, M.M and Tsai, S.Y and Lydon, J.P and DeMayo, F.J
Oncogene, ISSN 0950-9232, 01/2009, Volume 28, Issue 1, pp. 31 - 40
Endometrioid adenocarcinoma is the most frequent form of endometrial cancer, usually developing in pre- and perimenopausal women. beta-catenin abnormalities... 
β-catenin | Uterus | Hyperplasia | Estrogen | beta-catenin | estrogen | ENDOMETRIAL CARCINOMAS | CYCLIN D1 | MICROSATELLITE INSTABILITY | BIOCHEMISTRY & MOLECULAR BIOLOGY | LEUKEMIA INHIBITORY FACTOR | E-CADHERIN | CELL BIOLOGY | NUCLEAR-LOCALIZATION | uterus | ONCOLOGY | GENETICS & HEREDITY | hyperplasia | MOUSE UTERUS | TRANSCRIPTIONAL TARGET | FEMALE REPRODUCTIVE-TRACT | COLON-CARCINOMA | Cell Proliferation | Endometrium - growth & development | Endometrial Hyperplasia - genetics | Endometrial Hyperplasia - pathology | Infertility, Female - genetics | beta Catenin - genetics | Cell Differentiation - genetics | Animals | Cell Transformation, Neoplastic - genetics | Mice, Mutant Strains | Female | Mice | beta Catenin - physiology | Cell Transformation, Neoplastic - pathology | Endometrium - pathology | Disease Models, Animal
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3. Full Text Clonal evolution in paired endometrial intraepithelial neoplasia/atypical hyperplasia and endometrioid adenocarcinoma
by Russo, Mariano and Broach, James and Sheldon, Kathryn and Houser, Kenneth R and Liu, Dajiang J and Kesterson, Joshua and Phaeton, Rebecca and Hossler, Carrie and Hempel, Nadine and Baker, Maria and Newell, Jordan M and Zaino, Richard and Warrick, Joshua I
Human Pathology, ISSN 0046-8177, 09/2017, Volume 67, pp. 69 - 77
Endometrial intraepithelial neoplasia (EIN) and atypical endometrial hyperplasia (AH) are histomorphologically defined precursors to endometrioid... 
Clonal evolution | Endometrial hyperplasia | Endometrial intraepithelial neoplasia | Endometrioid adenocarcinoma | Next generation sequencing | PTEN | EIN | PATHOLOGY | CANCER | GENES | MUTATIONS | CARCINOMA | EXPRESSION | Immunohistochemistry | Microsatellite Instability | Cell Proliferation | Endometrial Hyperplasia - enzymology | Humans | Middle Aged | Endometrial Hyperplasia - pathology | DNA Copy Number Variations | Carcinoma in Situ - genetics | Carcinoma, Endometrioid - surgery | Carcinoma in Situ - enzymology | Carcinoma in Situ - surgery | Endometrial Neoplasms - genetics | DNA Mismatch Repair | Endometrial Hyperplasia - surgery | Aged, 80 and over | Female | Carcinoma, Endometrioid - genetics | Genetic Predisposition to Disease | Endometrial Neoplasms - enzymology | Carcinoma, Endometrioid - enzymology | Biomarkers, Tumor - analysis | Carcinoma in Situ - pathology | Endometrial Hyperplasia - genetics | Gene Dosage | Hysterectomy | DNA Repair Enzymes - analysis | Disease Progression | Phenotype | Biopsy | Endometrial Neoplasms - surgery | Endometrial Neoplasms - pathology | Aged | Biomarkers, Tumor - genetics | High-Throughput Nucleotide Sequencing | Mutation | Carcinoma, Endometrioid - pathology | Clonal Evolution | Adenocarcinoma | Gene mutations | Analysis | Hyperplasia | Genomics | Genetic research | Medical colleges | Proteins | Genes | Gynecology | Genomes | Deoxyribonucleic acid--DNA | Cancer | Esophagus
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4. Full Text Molecular profiles of benign and (pre)malignant endometrial lesions
by Putten, L.J.M. van der and Hoof, R. van and Tops, B.B.J and Snijders, M and Berg-van Erp, S.H. van den and Wurff, A.A. van der and Bulten, J and Pijnenborg, J.M.A and Massuger, L.F.A.G
Carcinogenesis, ISSN 0143-3334, 2017, Volume 38, Issue 3, pp. 329 - 335
Endometrial carcinomas are histologically classified as endometrioid, assumed to originate from hyperplastic endometrium, or non-endometrioid carcinomas,... 
AMPLIFICATION | PIK3CA | ONCOLOGY | BRAF MUTATIONS | CLASSIFICATION | ATYPICAL HYPERPLASIA | KRAS | CARCINOMA | CANCER | INTRAEPITHELIAL-NEOPLASIA | PTEN EXPRESSION | Receptor, Epidermal Growth Factor - genetics | Endometrial Hyperplasia - enzymology | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Uterine Diseases - pathology | Endometrial Hyperplasia - pathology | Proto-Oncogene Proteins c-akt - genetics | Endometrial Neoplasms - genetics | Female | Precancerous Conditions - pathology | Carcinoma, Endometrioid - genetics | Precancerous Conditions - enzymology | Endometrial Neoplasms - enzymology | Carcinoma, Endometrioid - enzymology | Carcinogenesis - genetics | Endometrial Hyperplasia - genetics | Precancerous Conditions - genetics | Carcinogenesis - pathology | Phosphatidylinositol 3-Kinases - genetics | Class I Phosphatidylinositol 3-Kinases | Uterine Diseases - enzymology | Uterine Diseases - genetics | Endometrial Neoplasms - pathology | DNA, Neoplasm - genetics | Mutation | Carcinoma, Endometrioid - pathology
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5. Full Text DNA methylation as a biomarker for the detection of hidden carcinoma in endometrial atypical hyperplasia
by Lai, Hung-Cheng and Wang, Yu-Chi and Yu, Mu-Hsien and Huang, Rui-Lan and Yuan, Chiou-Chung and Chen, Kuan-Ju and Wu, Chia-Chun and Chiang, Kai-Jo and Chao, Tai-Kuang
Gynecologic Oncology, ISSN 0090-8258, 2014, Volume 135, Issue 3, pp. 552 - 559
Abstract Objective Women with atypical hyperplasia (AH) are often found to have endometrial carcinoma (EC) at hysterectomy. The purpose of this study was to... 
Hematology, Oncology and Palliative Medicine | Obstetrics and Gynecology | Epigenetics | Methylation | Endometrial carcinoma | Endometrium | Atypical hyperplasia | BLADDER-CANCER | DIAGNOSIS | CELLS | PROMOTER METHYLATION | ADENOCARCINOMA | MARKERS | CERVICAL-CANCER | IDENTIFICATION | OBSTETRICS & GYNECOLOGY | ONCOLOGY | GENES | EXPRESSION | DNA Methylation | Epigenomics | Endometrial Neoplasms - genetics | Humans | Endometrial Hyperplasia - genetics | Endometrial Neoplasms - pathology | Female | Male | Biomarkers, Tumor - genetics | Endometrial Hyperplasia - pathology | Carcinoma | Hyperplasia | DNA | Genetic research | Genetic aspects | Cancer
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6. Full Text ARID1A loss is prevalent in endometrial hyperplasia with atypia and low-grade endometrioid carcinomas
by Werner, Henrica M. J and Berg, Anna and Wik, Elisabeth and Birkeland, Even and Krakstad, Camilla and Kusonmano, Kanthida and Petersen, Kjell and Kalland, Karl H and Oyan, Anne M and Akslen, Lars A and Trovik, Jone and Salvesen, Helga B
Modern Pathology, ISSN 0893-3952, 03/2013, Volume 26, Issue 3, pp. 428 - 434
ARID1A (AT-rich interactive domain 1A) has recently been identified as a tumor suppressor gene in various, predominantly gynecological cancers. We wanted to... 
ARID1A | carcinoma | endometrial | REMODELING GENE ARID1A | PROTEIN EXPRESSION | TISSUE MICROARRAYS | TUMOR-SUPPRESSOR | MUTATIONS | PATHOLOGY | CLEAR-CELL CARCINOMA | CANCER | PROGRESSION | Immunohistochemistry | Nuclear Proteins - analysis | Prognosis | Prospective Studies | Oligonucleotide Array Sequence Analysis | Endometrial Neoplasms - mortality | Tissue Array Analysis | Endometrial Hyperplasia - mortality | Humans | Middle Aged | RNA, Messenger - analysis | Endometrial Hyperplasia - pathology | Carcinoma, Endometrioid - therapy | Carcinoma, Endometrioid - chemistry | Endometrial Hyperplasia - metabolism | Neoplasm Grading | Endometrial Neoplasms - genetics | Time Factors | Female | Retrospective Studies | Endometrial Hyperplasia - therapy | Nuclear Proteins - genetics | Carcinoma, Endometrioid - genetics | Carcinoma, Endometrioid - secondary | Endometrial Neoplasms - chemistry | Biomarkers, Tumor - analysis | Neoplasm Invasiveness | Down-Regulation | Kaplan-Meier Estimate | Endometrial Hyperplasia - genetics | Transcription Factors - genetics | Chi-Square Distribution | Carcinoma, Endometrioid - mortality | Endometrial Neoplasms - therapy | Endometrial Neoplasms - pathology | Aged | Biomarkers, Tumor - genetics | Transcription Factors - analysis | Index Medicus
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7. Full Text Expression of miRNAs and PTEN in endometrial specimens ranging from histologically normal to hyperplasia and endometrial adenocarcinoma
by Lee, Heejeong and Choi, Hyun Joo and Kang, Chang Suk and Lee, Hee Jin and Lee, Weon Sun and Park, Chul Soo
Modern Pathology, ISSN 0893-3952, 11/2012, Volume 25, Issue 11, pp. 1508 - 1515
We investigated the relationship between frequently deregulated microRNAs (miRNAs) and enodometrial pathology in an attempt to find the most dependable miRNA... 
PTEN | endometrial hyperplasia and carcinoma | microRNA | OVARIAN-CANCER | PATHOLOGY | SIGNATURE | PROFILE | MICRORNA EXPRESSION | CARCINOMA | BIOPSIES | PROGRESSION | CLINICOPATHOLOGICAL FEATURES | EPIGENETIC ALTERATIONS | IMMUNOHISTOCHEMISTRY | Immunohistochemistry | Predictive Value of Tests | Adenocarcinoma - pathology | Endometrium - enzymology | Endometrial Hyperplasia - enzymology | Humans | Middle Aged | PTEN Phosphohydrolase - analysis | Endometrial Hyperplasia - pathology | Formaldehyde | Paraffin Embedding | Endometrial Neoplasms - genetics | Fixatives | Cell Transformation, Neoplastic - genetics | MicroRNAs - analysis | Sensitivity and Specificity | Adult | Female | Precancerous Conditions - pathology | Adenocarcinoma - genetics | Tissue Fixation | Real-Time Polymerase Chain Reaction | Precancerous Conditions - enzymology | Endometrial Neoplasms - enzymology | Biomarkers, Tumor - analysis | Adenocarcinoma - enzymology | Endometrial Hyperplasia - genetics | Gene Expression Profiling - methods | Chi-Square Distribution | Reverse Transcriptase Polymerase Chain Reaction | Cell Transformation, Neoplastic - metabolism | Precancerous Conditions - genetics | Disease Progression | Endometrial Neoplasms - pathology | Aged | Cell Transformation, Neoplastic - pathology | Endometrium - pathology
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8. Full Text Molecular pathology of endometrial hyperplasia and carcinoma
by Matias-guiu, Xavier and Catasus, Lluis and Bussaglia, Elena and Lagarda, Helena and Garcia, Arnald and Pons, Cristina and Muñoz, Josefina and Argüelles, Rosmary and Machin, Pilar and Prat, Jaime
Human Pathology, ISSN 0046-8177, 06/2001, Volume 32, Issue 6, pp. 569 - 577
Four different genetic abnormalities may occur in endometrioid adenocarcinomas of the endometrium (mircosatellite instability and mutations in the , and... 
microsatellite instability | β-catenin | MLH-1 methylation | PTEN, k-RAS | Endometrial carcinoma | molecular pathology | PTEN | Molecular pathology | k-RAS | Microsatellite instability | beta-catenin | BETA-CATENIN GENE | PROMOTER HYPERMETHYLATION | endometrial carcinoma | GENETICALLY UNSTABLE CANCERS | PATHOLOGY | ISLAND METHYLATOR PHENOTYPE | COLORECTAL-CANCER | SEROUS CARCINOMA | SOMATIC FRAMESHIFT MUTATIONS | REPLICATION ERRORS | KI-RAS ONCOGENE | Phosphoric Monoester Hydrolases - genetics | beta Catenin | Cytoskeletal Proteins - genetics | Humans | Trans-Activators | Endometrial Hyperplasia - genetics | Genes, p53 - genetics | Microsatellite Repeats - genetics | DNA Methylation | Endometrial Neoplasms - genetics | PTEN Phosphohydrolase | Female | Mutation | Tumor Suppressor Proteins | Genes, ras - genetics | Carcinoma | Colon cancer | Hyperplasia | Development and progression | Bone morphogenetic proteins | Tumor proteins | Transforming growth factors | Methylation | Chromosomes | Cancer
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9. Full Text Sox9 overexpression in uterine epithelia induces endometrial gland hyperplasia
by Gonzalez, Gabriel and Mehra, Shyamin and Wang, Ying and Akiyama, Haruhiko and Behringer, Richard R
Differentiation, ISSN 0301-4681, 10/2016, Volume 92, Issue 4, pp. 204 - 215
SOX9 is a high mobility group transcription factor that is required in many biological processes, including cartilage differentiation, endoderm progenitor... 
Transgenic mouse | Sox9 | Uterus | Endometrium | Cancer | CELLS | DEVELOPMENTAL BIOLOGY | TUMORIGENICITY | POLYPS | CELL BIOLOGY | IN-VIVO | MUTATIONS | DIFFERENTIATION | UP-REGULATION | CARCINOMA | Uterine Neoplasms - genetics | Uterus - pathology | Epithelium - pathology | Epithelium - growth & development | Uterine Neoplasms - pathology | Humans | Uterus - metabolism | Endometrium - growth & development | Gene Expression Regulation, Neoplastic | Endometrial Hyperplasia - genetics | Endometrial Hyperplasia - pathology | Receptors, Progesterone - genetics | Animals | Female | Mice | Endometrium - pathology | SOX9 Transcription Factor - genetics | endometrium | cancer | uterus | transgenic mouse
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10. Full Text Abnormal mismatch repair and other clinicopathologic predictors of poor response to progestin treatment in young women with endometrial complex atypical hyperplasia and well‐differentiated endometrial adenocarcinoma: a consecutive case series
by Zakhour, M and Cohen, JG and Gibson, A and Walts, AE and Karimian, B and Baltayan, A and Aoyama, C and Garcia, L and Dhaliwal, SK and Elashoff, D and Amneus, M and Walsh, C
BJOG: An International Journal of Obstetrics & Gynaecology, ISSN 1470-0328, 09/2017, Volume 124, Issue 10, pp. 1576 - 1583
Objective To report the response to progestin therapy in young women with endometrial complex atypical hyperplasia (CAH) or FIGO grade 1 endometrial... 
endometrial cancer | mismatch repair | Complex atypical hyperplasia | Lynch syndrome | PROGNOSTIC-FACTORS | PREMENOPAUSAL WOMEN | FAMILY-HISTORY | CANCER | OBSTETRICS & GYNECOLOGY | REPRODUCTIVE OUTCOMES | THERAPY | FERTILITY-SPARING MANAGEMENT | ORAL PROGESTIN | CARCINOMA | Immunohistochemistry | Adenocarcinoma - pathology | Progestins - therapeutic use | Humans | Middle Aged | Endometrial Hyperplasia - genetics | Endometrial Hyperplasia - pathology | Adenocarcinoma - drug therapy | Endometrial Hyperplasia - drug therapy | Endometrial Neoplasms - genetics | DNA Mismatch Repair | Adult | Endometrial Neoplasms - drug therapy | Endometrial Neoplasms - pathology | Female | Adenocarcinoma - genetics | Adenocarcinoma | Proteins | Medical research | Endometrial cancer | Hyperplasia | Young women | Medicine, Experimental | Invasiveness | Gynecology | Malignancy | DNA repair | Body mass index | MMR protein | Body mass | Progestin | Medical prognosis | Mismatch repair | Biomarkers | Protein expression | Hormone replacement therapy | Health risk assessment | Age | Endometrium
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11. Full Text Uterine epithelial cell proliferation and endometrial hyperplasia: Evidence from a mouse model
by Gao, Yang and Li, Shu and Li, Qinglei
Molecular Human Reproduction, ISSN 1360-9947, 2014, Volume 20, Issue 8, pp. 776 - 786
In the uterus, epithelial cell proliferation changes during the estrous cycle and pregnancy. Uncontrolled epithelial cell proliferation results in implantation... 
Endometrial hyperplasia | Proliferation | Uterus | epithelial cell | Transforming growth factor β | TGF-BETA | proliferation | PROGESTERONE | transforming growth factor beta | DEVELOPMENTAL BIOLOGY | PREGNANCY | OBSTETRICS & GYNECOLOGY | GROWTH-FACTOR-BETA | REPRODUCTIVE BIOLOGY | uterus | endometrial hyperplasia | SUPERFAMILY | PROTEINS | EXPRESSION | FOXA2 | FGF10 | Cell Proliferation - genetics | Receptors, Transforming Growth Factor beta - genetics | Epithelial Cells - metabolism | Transforming Growth Factor beta1 - metabolism | Uterus - metabolism | Cell Proliferation - physiology | Cells, Cultured | Endometrial Hyperplasia - genetics | Endometrial Hyperplasia - pathology | Transforming Growth Factor beta1 - genetics | Receptors, Peptide - genetics | Mice, Knockout | Animals | Endometrial Hyperplasia - metabolism | Receptors, Transforming Growth Factor beta - metabolism | Uterus - cytology | Female | Mice | Epithelial Cells - cytology | Receptors, Peptide - metabolism | transforming growth factor β
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