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Genes and Development, ISSN 0890-9369, 06/2006, Volume 20, Issue 12, pp. 1569 - 1574
The tumor suppressor PTEN is frequently inactivated in human cancers. A major downstream effector of PTEN is Akt, which is hyperactivated via PTEN... 
Intestinal polyps | PIN | Endometrium carcinoma | Thyroid and adrenal tumors | Cancer therapy | AKT/PKB | GENETICALLY-ENGINEERED MICE | PTEN | DEVELOPMENTAL BIOLOGY | PHOSPHOINOSITIDE 3-KINASE | CANCER | CELL BIOLOGY | cancer therapy | PATHWAY | thyroid and adrenal tumors | NEOPLASIA | RELEVANCE | intestinal polyps | MUTATION | GENETICS & HEREDITY | endometrium carcinoma | Prostatic Intraepithelial Neoplasia - pathology | Thyroid Neoplasms - enzymology | Proto-Oncogene Proteins c-akt - deficiency | Male | Intestinal Polyps - enzymology | Proto-Oncogene Proteins c-akt - genetics | Adrenal Gland Neoplasms - enzymology | Prostatic Neoplasms - genetics | Endometrial Neoplasms - genetics | Neoplasms - genetics | Female | Pseudolymphoma - genetics | Pseudolymphoma - enzymology | Adrenal Gland Neoplasms - pathology | PTEN Phosphohydrolase - genetics | Prostatic Neoplasms - pathology | Endometrial Neoplasms - enzymology | Neoplasms - enzymology | PTEN Phosphohydrolase - metabolism | Pseudolymphoma - pathology | Thyroid Neoplasms - genetics | Animals | Prostatic Intraepithelial Neoplasia - enzymology | Intestinal Polyps - pathology | Prostatic Neoplasms - enzymology | Endometrial Neoplasms - pathology | Heterozygote | Mice | Prostatic Intraepithelial Neoplasia - genetics | Neoplasms - pathology | Adrenal Gland Neoplasms - genetics | Thyroid Neoplasms - pathology | Index Medicus | Research Communication
Journal Article
American Journal of Surgical Pathology, ISSN 0147-5185, 12/2009, Volume 33, Issue 12, pp. 1869 - 1877
Endometrial carcinomas (ECs) in Young women (<= 40 y) are Usually managed conservatively in selected patients. Whether oophorectomy with total hysterectomy is... 
HNPCC/Lynch syndrome | Endometrial carcinoma | Young patients | SURGERY | FERTILITY-PRESERVING TREATMENT | LYNCH-SYNDROME | PROGNOSTIC-FACTORS | NONPOLYPOSIS COLORECTAL-CANCER | SYNCHRONOUS PRIMARY CANCERS | MICROSATELLITE INSTABILITY | endometrial carcinoma | PATHOLOGY | MLH1 PROMOTER METHYLATION | ESTROGEN-RECEPTOR-ALPHA | OVARIAN CARCINOMAS | PROGESTERONE RECEPTORS | Immunohistochemistry | MutL Protein Homolog 1 | Nuclear Proteins - analysis | Age Factors | Endometrial Neoplasms - mortality | Adenocarcinoma, Clear Cell - mortality | Humans | Ovarian Neoplasms - pathology | DNA-Binding Proteins - analysis | Neoplasms, Multiple Primary | Ovarian Neoplasms - mortality | Carcinoma, Endometrioid - therapy | Patient Selection | Ovarian Neoplasms - genetics | Young Adult | Endometrial Neoplasms - genetics | Time Factors | Adenosine Triphosphatases - analysis | DNA Mismatch Repair | Adult | Female | Mismatch Repair Endonuclease PMS2 | Carcinoma, Endometrioid - genetics | Endometrial Neoplasms - enzymology | Carcinoma, Endometrioid - enzymology | Risk Assessment | Down-Regulation | Risk Factors | Treatment Outcome | DNA Repair Enzymes - analysis | Ovarian Neoplasms - enzymology | Adaptor Proteins, Signal Transducing - analysis | Biopsy | Carcinoma, Endometrioid - mortality | Adenocarcinoma, Clear Cell - therapy | Endometrial Neoplasms - therapy | Ovarian Neoplasms - therapy | Endometrial Neoplasms - pathology | Adenocarcinoma, Clear Cell - pathology | MutS Homolog 2 Protein - analysis | Neoplasm Staging | Adenocarcinoma, Clear Cell - enzymology | Adenocarcinoma, Clear Cell - genetics | Carcinoma, Endometrioid - pathology | Index Medicus
Journal Article
AMERICAN JOURNAL OF SURGICAL PATHOLOGY, ISSN 0147-5185, 02/2016, Volume 40, Issue 2, pp. 155 - 165
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2012, Volume 109, Issue 21, pp. 8155 - 8160
Metastasis from primary tumors remains a major problem for tumor therapy. In the search for markers of metastasis and more effective therapies, the tumor... 
Enzymes | Cell lines | Small interfering RNA | Lipids | Breast cancer | Lipid metabolism | Metastasis | Endometrial neoplasms | Tumors | Cancer | Glycerophosphodiester phosphodiesterase domain containing 6 | Glycerophosphocholine phosphodiesterase GDE1 homolog (Saccharomyces cerevisiae) | Lysophosphatidic acid | Glycerophosphodiesterase 5 | Phosphatidic acid | PHOSPHOLIPID-METABOLISM | MULTIDISCIPLINARY SCIENCES | PKC-ALPHA | KINASE | glycerophosphodiesterase 5 | glycerophosphodiester phosphodiesterase domain containing 6 | KAPPA-B | glycerophosphocholine phosphodiesterase GDE1 homolog (Saccharomyces cerevisiae) | BREAST-CANCER | LUNG-CANCER | OVEREXPRESSION | lysophosphatidic acid | phosphatidic acid | PHOSPHODIESTERASE | EXPRESSION | Choline - metabolism | Breast Neoplasms - secondary | Endometrial Neoplasms - enzymology | Phosphoric Diester Hydrolases - metabolism | Protein Kinase C-alpha - metabolism | Humans | Mice, Inbred C57BL | Phosphoric Diester Hydrolases - genetics | Cell Movement - physiology | Ovarian Neoplasms - enzymology | Phospholipases - genetics | Breast Neoplasms - enzymology | Ovarian Neoplasms - secondary | Animals | Endometrial Neoplasms - secondary | Phospholipases - metabolism | Cell Line, Tumor | Female | Signal Transduction - physiology | Mice | Choline | Physiological aspects | Development and progression | Research | Phosphodiesterases | Health aspects | Cell migration | Proteins | Genotype & phenotype | Metabolism | Cells | Index Medicus | Biological Sciences
Journal Article
Gynecologic Oncology, ISSN 0090-8258, 2009, Volume 116, Issue 1, pp. 15 - 20
Journal Article
Cancer Cell, ISSN 1535-6108, 12/2015, Volume 28, Issue 6, pp. 785 - 799
Polymorphonuclear neutrophils (PMNs) are largely considered to foster cancer development despite wielding an arsenal of cytotoxic agents. Using a mouse model... 
LUNG | ONCOLOGY | INFLAMMATION | IN-VIVO | CXCR2 | SENESCENCE | MICE | ANTITUMOR IMMUNITY | SUPPRESSION | TUMORIGENESIS | ENDOMETRIAL CANCER | CELL BIOLOGY | Cell Proliferation | Humans | Ovarian Neoplasms - pathology | Tumor Microenvironment | Gene Expression Profiling | Uterus - immunology | Neutrophil Infiltration | Receptors, Colony-Stimulating Factor - deficiency | Ovarian Neoplasms - genetics | Receptors, Colony-Stimulating Factor - genetics | Time Factors | Bone Marrow Transplantation | Neutrophils - metabolism | Gene Transfer Techniques | PTEN Phosphohydrolase - genetics | PTEN Phosphohydrolase - deficiency | Neutrophil Activation | Ovarian Neoplasms - prevention & control | Ovariectomy | Computational Biology | Myeloid Differentiation Factor 88 - genetics | Carcinoma, Endometrioid - immunology | Tumor Burden | Cell Adhesion | Ovarian Neoplasms - enzymology | Mice, Knockout | Carcinoma, Endometrioid - prevention & control | Myeloid Differentiation Factor 88 - deficiency | Carcinoma, Endometrioid - mortality | Receptors, Interleukin-8B - genetics | Survival Analysis | Cell Line, Tumor | Carcinoma, Endometrioid - pathology | Phagocytosis | Uterus - pathology | Databases, Genetic | Ovarian Neoplasms - mortality | Cell Hypoxia | Inflammation Mediators - metabolism | Female | Carcinoma, Endometrioid - genetics | Carcinoma, Endometrioid - enzymology | Mice, Inbred C57BL | Neutrophils - immunology | Chemotaxis | Animals | Receptors, Interleukin-8B - metabolism | Uterus - enzymology | Neoplasm Staging | Ovarian Neoplasms - immunology | Development and progression | Carcinogenesis | Analysis | Tumors | Medical colleges | Lymphocytes | Inflammation | Genetic engineering | Cancer | Index Medicus
Journal Article
International Journal of Cancer, ISSN 0020-7136, 10/2016, Volume 139, Issue 7, pp. 1557 - 1563
Journal Article
Journal Article
Modern Pathology, ISSN 0893-3952, 11/2013, Volume 26, Issue 11, pp. 1525 - 1535
The switch/sucrose non-fermentable (SWI/SNF) subunit ARID1A (AT-rich interactive domain 1A gene) has been recently postulated as a novel tumor suppressor of... 
endometrial cancer | microsatellite instability | ARID1A | Lynch syndrome | METHYLATION | COMPLEX | MSI | REMODELING GENE ARID1A | PATHOLOGY | HNPCC | TUMOR-SUPPRESSOR | GRADE | COMPONENT | MUTATIONS | Immunohistochemistry | MutL Protein Homolog 1 | ras Proteins - genetics | Microsatellite Instability | Proto-Oncogene Proteins p21(ras) | Nuclear Proteins - analysis | Prognosis | Humans | Middle Aged | PTEN Phosphohydrolase - analysis | Gene Expression Regulation, Neoplastic | DNA-Binding Proteins - analysis | SMARCB1 Protein | Tumor Suppressor Protein p53 - analysis | Endometrial Neoplasms - genetics | DNA Mutational Analysis | Aged, 80 and over | Adult | Colorectal Neoplasms, Hereditary Nonpolyposis - enzymology | Female | Nuclear Proteins - genetics | Promoter Regions, Genetic | Genetic Predisposition to Disease | Endometrial Neoplasms - enzymology | Signal Transduction | Colorectal Neoplasms, Hereditary Nonpolyposis - genetics | Down-Regulation | Gene Silencing | Proto-Oncogene Proteins - genetics | Phosphatidylinositol 3-Kinases - genetics | Phenotype | Class I Phosphatidylinositol 3-Kinases | Chromosomal Proteins, Non-Histone - analysis | Adaptor Proteins, Signal Transducing - genetics | Proto-Oncogene Proteins c-akt - analysis | Endometrial Neoplasms - pathology | Aged | Transcription Factors - analysis | Mutation | Index Medicus
Journal Article
The American Journal of Surgical Pathology, ISSN 0147-5185, 05/2016, Volume 40, Issue 5, pp. 656 - 663
Several morphologic features have been reported to be predictive of abnormal expression of mismatch repair (MMR) proteins in endometrial and colon carcinomas.... 
DNA mismatch repair proteins | Ovarian clear cell carcinoma | Microsatellite instability | Lynch syndrome | SURGERY | microsatellite instability | LYNCH-SYNDROME | DEFECTS | HMSH2 | NONPOLYPOSIS COLORECTAL-CANCER | ovarian clear cell carcinoma | HMLH1 | PATHOLOGY | ENDOMETRIAL CANCER | MICROSATELLITE INSTABILITY STATUS | FREQUENCY | MUTATIONS | AGE | Immunohistochemistry | MutL Protein Homolog 1 | Nuclear Proteins - analysis | Stromal Cells - pathology | Colorectal Neoplasms, Hereditary Nonpolyposis - pathology | Humans | Middle Aged | Ovarian Neoplasms - pathology | Carcinoma - mortality | DNA-Binding Proteins - analysis | Ovarian Neoplasms - mortality | Incidence | Ovarian Neoplasms - genetics | Colorectal Neoplasms, Hereditary Nonpolyposis - mortality | Time Factors | Adenosine Triphosphatases - analysis | Carcinoma - enzymology | DNA Mismatch Repair | Aged, 80 and over | Boston - epidemiology | Adult | Colorectal Neoplasms, Hereditary Nonpolyposis - enzymology | Female | Mismatch Repair Endonuclease PMS2 | Carcinoma - pathology | Biomarkers, Tumor - analysis | California - epidemiology | Colorectal Neoplasms, Hereditary Nonpolyposis - genetics | Kaplan-Meier Estimate | DNA Repair Enzymes - analysis | Ovarian Neoplasms - enzymology | Adaptor Proteins, Signal Transducing - analysis | Lymphocytes, Tumor-Infiltrating - pathology | Biopsy | Carcinoma - genetics | Aged | MutS Homolog 2 Protein - analysis | Neoplasm Staging | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 3/2009, Volume 106, Issue 12, pp. 4834 - 4839
Although 75% of endometrial cancers are treated at an early stage, 15% to 20% of these recur. We performed an integrated analysis of genome-wide expression and... 
Phenotypes | Genomics | Genes | Histology | Genomes | Signatures | Endometrial neoplasms | Genetic mutation | Tumors | Cancer | Amplification | Prognosis | Comparative genomic hybridization | Endometrial cancer | Stathmin expression | POPULATION | CANCERS | MULTIDISCIPLINARY SCIENCES | ADENOCARCINOMAS | stathmin expression | prognosis | comparative genomic hybridization | SIGNATURE | amplification | P110-BETA | PIK3CA | endometrial cancer | MUTATIONS | ABERRATIONS | INHIBITOR | EXPRESSION | Proto-Oncogene Proteins - metabolism | Proto-Oncogene Proteins p21(ras) | Endometrial Neoplasms - enzymology | Humans | Gene Expression Regulation, Neoplastic | ras Proteins - metabolism | Gene Dosage | Gene Expression Profiling | Phosphatidylinositol 3-Kinases - metabolism | Endometrial Neoplasms - diagnosis | Genome, Human - genetics | Loss of Heterozygosity - genetics | Class I Phosphatidylinositol 3-Kinases | Endometrial Neoplasms - genetics | Survival Analysis | Biomarkers, Tumor - metabolism | Endometrial Neoplasms - pathology | Female | Enzyme Activation | Stathmin - metabolism | Cluster Analysis | Genetic aspects | Research | Phosphotransferases | Health aspects | Mutation | Gene expression | Kinases | Carcinoma | Transcription | Data processing | Survival | 1-Phosphatidylinositol 3-kinase | Loss of heterozygosity | genomics | Endometrium | Oncogenes | PTEN protein | Index Medicus | Biological Sciences
Journal Article
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